knee tendinopathies - exam 3 Flashcards

1
Q

ITB syndrome:
-prevalence

A

5-14% of runner
2nd leading cause of Knee P! in runners
Males compromise 50-81% of cases

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2
Q

Tendinopathy of the distal IT band:

A

Iliotibial Band Syndrome

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3
Q

RF of ITB Syndrome:

A

running
training errors
weak ERs and aBd
Excessive pronation
Increased hip aDd and IR
Trunk lean in unilateral stance
Associated with GTPS and PFPS

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4
Q

Etiology of ITB Syndrome:
-consider

A

Little is known that leads to abnormal mechanical loading
consider lumbar hypermobility/instability w/impaired LE control and excessive recruitment of TFL

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5
Q

Structures involved in ITB Syndrome:

A

TFL/Band
Lateral femoral epicondyle, Gerdys tubercle (lateral tibia) insertion and asscoiated burase and fat pad

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6
Q

Symptoms of ITB syndrome:

A

gradual onset of lateral knee P!
worse with activities involving repetitive knee motion, grades, and dynamic U stance i.e. , running

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7
Q

ITB Syndrome: Sign
–observations
–ROM
–Resisted/MMT
–special test

A

impaired LE control
P! likley with hip add
possible hip ER weakness
possible hip aBd weakness w/P! paricualry in a lengthened position
possibly (+) Obers tests
TTP over lat. femoral condyle and Gerdy’s tubercle

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8
Q

PT Rx: ITB Syndrome
–MET parameters
—primary purpose

A

3x15 heavy loads to fatigue
tendon proliferation and stabilization (hip and lumabr)

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9
Q

Tendinosis prescription: ITB Syndrome

A

isometric loading without compression leneghthing
isotonic loading without compression from lengthening
Isotonic loading with compression from lengthening
isometric loading in weight-bearing -CC hip, aBd, ER, and EXT
plyometric loading

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10
Q

Patellar Tendinopathy aka:

A

jumpers knee or ant. knee P!

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11
Q

Incidence & RF of tendinopathy
up to_________ % of athletes
gender?
more common in____________

A

up to 50% of athletes
males>females
more common in jumping sports

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12
Q

Structures involved: Patellar Tendinopathy

A

patellar tendon
infrapatellar bursae and fat pad

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13
Q

Bursae: Patellar Tendinopathy
–superficial infrapatellar between ______ and ____________
–deep infrapatellar between _______ and __________

A

skin and patellar tendon
patellar tendon and tibia

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14
Q

Etiology of Patellar Tendinopathy
Pathomechanics: ________origin

A

abnormal mechanical loading
tendinopathy

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15
Q

S&S of Patellar Tendinopathy?
- onset
- increased with:

A

overuse of gradual onset of P!
increased w/activity, jumping, lunging, and squatting

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16
Q

S&S of Patellar Tendinopathy:
–observation
–ROM

A

possible thickened tendon
impaired LE control, particularly knee ant. to toes

ROM: possible P! and limitation with end range flexion, especially of hip extended

17
Q

S&S of Patellar Tendinopathy:
Resisted/MMT
AM:
Special Test:
Palpation:

A

possible P! w/ knee ext, espcially in lengthened position; may be weak
possible impaired patellar motion i.e., limited inf glide
(+) Thomas Test for shortened rectus femoris
TTP: localized ; patella alta adding to tendon tension/compression

18
Q

PT Rx: Patellar Tendinopathy

A

lack of high-quality evidence
Pt. edu.
–soreness rule
–load management
–movement cues
POLICED
Modalities- no additional benefit when added to MET
Orthotic-tendon strap may help but might cause more P!
JM

19
Q

PT Rx Patellar Tendinopathy Specifics :
–Tendinosis Rx + :
- how?

A

increased trunk flexion with landing limits tendon stresses

  • leaning forward draws the knees back to a better position (think squatting - back straight vs leaning forward)
20
Q

Prognosis: Patellar Tendinopathy

A

50-70% improvement 3-6 mths

21
Q

prevalence of Tibial Tubercle apophysitis AKA Osgood Schlatter’s disease:

A

most common causes of anterior knee pain in children
peaks at 12-15 years

22
Q

structures involved with osgood schlatters:

A

tibial tuberosity apophysis or epiphyseal plate
patellar tendon

23
Q

risk factors of osgood shlatters:

A

growth spurt
high activity
shortened quads and hamstrings
weak quads
high BMI
reduced core stability

24
Q

cause of osgood shlatters:

A

overuse

25
Q

how does osgood schlatters happen?
- bone growth exceeds:
- increased:
- weak spot:
- complications:

A

bone growth exceeds quadriceps lengthening
increased tendon tension
growth plate is the weak spot so also where inflammation begins
complications: avulsion and/or premature closure

26
Q

symptoms of osgood schlatters:

A

gradual onset of ant knee p! with overuse
a “pop” may indicate an avulsion (dont disregard!)
possible loss of vertical jump

27
Q

osgood schlatters: Signs:
- observation
- ROM
- resisted

A
  • impaired LE control
    possibly enlarged tibial tuberosity
  • possible P! with end range flexion esp if hip is extended
  • likely P! with ext, esp in lengthened position
28
Q

osgood schlatters: Signs:
accessory motion:
special tests:
palpation:

A
  • patellar hypomobility i.e., limited inf glide
  • maybe (+) thomas test for rectus femoris shortening
  • TTP over tibial tuberosity, possibly tendon.
    possible patella alta positioning
29
Q

what 3 things should you tell your patient with patient education?

A

soreness rule
load management (cross training good)
movement cues for LE mechanics

30
Q

Rx for osgood schlatters:

A

POLICED
JM - PF glides
Stretching - careful stretching quads if P! occurs at growth plate

31
Q

PT Rx for osgood schlatters:
orthotic:

A

sleeve but may cause compression P!
strap on tendon but may cause traction P!
foot to control LE

32
Q

PT Rx for osgood schlatters:
MET:

A

possibly for trunk and hip stabilization (quads aren’t the problem here)
caution with muscle/tendon attached to growth plate to avoid greater overuse

33
Q

prognosis for osgood schlatters

A

PT 90% successful
can become a recurrent/persistent problem