FAI - exam 2 Flashcards

1
Q

what is femoral acetabular impingement (FAI)?

A

abnormal hip joint morphology or bony shape and arrangement

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

If FAI is symptomatic, what two structures are contacting?

A

proximal femur and acetabulum

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

who is more likely to get FAI?

A

males
higher with vigorous or end range activities (dance)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

what are risk factors of FAI?

A

genetics and gender
- higher risk for sibling

susceptible populations and activities such as:
- vigorous loading in athletics
- use of excessive motion
- peds hip conditions

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

what are abnormal hip/pelvis kinematics that could be risk factors for FAI?

A

ant pelvic tilt
limited post tilt, may limit coupled hip ER
excessive hip adduction
limited hip IR (more likely due to bony abutment than capsular tightness)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

although the cause of FAI is largely unknown, what are more often thought to be causes of FAI?

A

abnormal hip mechanics
vigorous athletic loading
combo of both above

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

what are less often causes of FAI?

A

slipped capital femoral epiphysis*
legg-calve-perthes’ disease*
femoral neck fx or malunion

  • peds hip conditions
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

3 congenital types of FAI
1. Cam:
- _____ spherical femoral head
- head contacts _________ __________ (___ o’clock position)
- more common in _____

A
  • less
  • anterosuperior acetabulum or 12 o’clock
  • males
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

is a cam FAI more present in general population without pain or athletes without pain? why?

A

athletes without P!
if you don’t go into end range positions a lot you may not get symptoms

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

overtime, what can a cam type FAI cause?

A

labrum tear
damage articular cartilage on femur head

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

3 congenital types of FAI
2. Pincer:
- deeper _______ or _______ ________
- neck primarily contacts _______ but may also contact ______. explain
- most common in _____

A
  • acetabulum or anterior osteophyte
  • anterior; posterior. impact on one side can cause rebound on the back side.
  • middle aged athletic females
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

what is the 3rd and most common type of FAI?

A

mixed

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

what structures are involved with or without age related joint changes/labral tears?

A

articular cartilage damage (83%)
labral damage aka fibrocartilage (93%)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

what is the labrum primarily made up of? can FAI cause gradual damage to the labrum?

A

type I collagen (resists tension)
yes, up to 75%

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

an active patient comes in complaining of mechanical groin pain with activity and without an alternative radiological dx. what should you consider has happened?

A

labral tears

20% of athletes w groin P!
55% prevalence in those with hip and groin P!

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

what is happening to cause FAI?
what other condition does this sound like?**

A

mechanical impingement leading to degenerative cascade of events
Shoulder impingement

17
Q

Symptoms of FAI:
–worsened with:
–_________P! (100% specificity)

A

gradual onset of hip P! into ant. hip/groin (deep pinch)

–repetitive and/or prolonged hip flexion (squatting, stairs, and sitting) (bony CPP)

–groin P! (lateral hip P! possible)

18
Q

A pt. with FAI will likely report clicking and/or locking ant. hip? T or F

A

False; minimal to no support

19
Q

Signs of FAI:
-Observation
-Functional test

A

impaired LE control

impaired balance and LE control
Quad dominant squatting pattern

20
Q

Signs of FAI:
-A/PROM

A

Primarily P! and loss of motion w/flex to 90º, IR <20º @ 90º of hip flexion and/or H add.

Possible limitation w/abd & dysplasia if >20º difference of hip flx and/or H Add.

Hip maltracking may be present - hip deviates into and while moving into flexion at 90º-100º flexion

<85º total rot. @ 90º flx is LARGEST predictor of groin P!

21
Q

Signs of FAI:
resisted test/MMT:
CM:
Stress Tests:

A

decreased activation of G.Med and Max and ERs
—weak ER and Abd in chronic conditions

possibly consistent block

possibly (+) w/ compression; (-) with distx

22
Q

Signs of FAI:
AM:
Special test:
Palpation:

A

possible hypomobile
FIR, FADDIR, FABER likey (+); possible (+) femoral torsion
TTP over ant. hip jt.@ 12oclock region

23
Q

PT Rx: FAI

A

POLICED
-load management including cross training
-foot orthotics to limit hip add/IR

Pt. Education:
–limit hip flexion >90º
–verbal cues for LE control

JM for cartilage integrity & possibly mobility

MET:
–Primarily for cartilage integrity, mm. function, and possibly mobility (local mm./anti-gravity)
–empathize LE control

24
Q

FAI prognosis:

A

66% return to play thru PT in athletes w/laberal tears (no sx)

ARJC poor prognosis (older pt.)

25
Q

MD Rx:

A

Ultrasound/Fluoroscopic guided injections
Sx: open or arthroscopy for FAI
-iliopsoas release and/or labral address
—NO evidence to suggest sx is better than PT

26
Q

Age-Related Jt. Change, what is happening?

A

lose GAGs; decreased hydration
articular cartilage is thinning. joint space narrow
doesn’t absorb shock well

27
Q

Prevalence:
Most common cause of _________ P!
Up to 25% of ______

A

hip P!
adults

28
Q

What are the structures involved with AJRC?

A

synovial membrane, joint capsule, articular cartilage, joint space, synovial fluid

29
Q

Symptoms of ARJC:

A

like FAI plus BUT more of labrum, articular cartilage not primary structure
AM stiffness < 30 min
Less tolerant to WBing activities and sitting w/possibly limping (articular cartilage more shock absorbent)
C-sign of P! groin, lateral hip, and buttock; may even refer to knee
*maybe nociplastic

30
Q

Trendelenburg gait the hip drops to ____________affected hip and pt. leans to ____________ side of the affected hip

A

contralateral; ipsilateral

31
Q

Signs of AJRC:
planes?
capsular pattern?
CM -
Resisted/MMT-
Compression; Distx-
AM-

A

like FAI plus
≥3 planes of motion restricted (high specificity)
Inconclusive capsular pattern of restriction at the hip
CM - consistent block
Resisted/MMT: P! and possible weakness w/ABD (high specificity)
Compression- possibly (+); Distx possibly reliving (-)
AM- hypomobility

32
Q

Specials Tests of AJRC?

A

OA CPR ≥ 3 present
-P! w/ squatting
-P! w/ hip flx
-P! w/ hip ext
-P! IR < 25º
-(+) Scour and FABER

OA Combined Results
-Hip P!
-IR < 15º
-IR P!
-AM stiffness < 60 min
->50 yrs. or age

Impaired functional performance testing
-6 min walk test
-Timed up and Go Test
Impaired balance test like Berg Balance Scale

33
Q

PT Rx: ARJC
_________P!/inflammation: (3)

___________mod support: (4)

A

POLICED
Modalities for P!/Inflammation
–no more than 2 wks.
–only short-term P! influence
–assistive device to minimize/avoid limping

PT. education -mod support
–limit hip flexion >90º
–avoid low seats and sit on a wedge
–no knee to chest stretches
–Wt. management (10-20 lbs)

34
Q

Left hip P! which UE would you use a cane? Why?

A

Right; increase base of support—> weak hip abductors on left so cane go in R.

35
Q

PT Rx: ARJC
JM for:
MET primarily for:

A

cartilage integrity and mobility
–better than usual care out to 1 yr.

MET: mobility, cartilage integrity, and mm. function
**include trunk and hip anti-gravity mm. groups
**balance training as WBing is tolerated
**1-5x/wk for 6-12wks.
–effective for improving P! and function
–better than usual care out of 1 yr.
–mod support
–aerobic component beneficial