Pain Flashcards

1
Q

Nociceptive Pain

A

results from injury to tissues
called somatic or visceral pain
Responds to opioids, NSAIDs, acetaminophen depending on tissue type

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2
Q

Neuropathic pain

A

results from injury to peripheral nerves

responds poorly to opioids

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3
Q

Non pharamlogical ways for pain management

A
hot/cold
repositioning
message
distraction
acupuncture
spiritual practices
TENS
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4
Q

Opioid recepters

A

Mu
Kappa
Delta

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5
Q

Mu

A

2 types

  • brain, spinal chord, peripheral nervous system
  • produces analgesia and sedation
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6
Q

Kappa

A

brain, spinal chord, PNS

produces analgesia and sedation

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7
Q

Delta

A

located in the brain and PNS

Analgesia, antidepressant effects

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8
Q

Morphine

A

binds to Mu and Kappa receptors to produce analgesia

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9
Q

Caffeine

A

used to enhance the distribution and absorption of the analgesic

relief of head aches (vasoconstrict and decrease intracranial pressure)

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10
Q

Tolerance

A

increasing doses to obtain same response

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11
Q

Addiction

A

the continued used of a substance despite its negative health and social consequences

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12
Q

Physical dependence

A

people with this experience highly uncomfortable symptoms that make them want to continue to use them

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13
Q

Meds used to “get off”

A

methadone, buprenophine suboxone

uses different receptors

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14
Q

Prostaglandins and Cox 1 and 2

A

promote inflammation for healing, but cause pain and fever

cox 1: supports blood clotting and protects the lining of the stomach

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15
Q

NSAIDs and COX

A

NSAIDs block the COX enzymes and reduce prostaglandins throughout the body

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16
Q

What to assess with acetominophine

A

liver function

17
Q

What to assess with NSAIDs

A

GI bleeding

18
Q

lab values to check prior to admin of NSAIDs are…

A

eGFR, Creatinine

19
Q

When to go to PRN meds

A

when pain is greater than 4/10

20
Q

ASA side effects

A

gastric ulcers
bleeding
renal failure

21
Q

Side effect occurring in children taking ASA

A

Reyes syndrome

22
Q

Narcan

A

opioid antagonist

23
Q

Migraine tx

A

begins with acetaminophen or NSAIDs

then triptans

then alkaloids

24
Q

Triptans

A

quiets down overactive brain nerves

25
Q

Sumatriptan

A

people are more at risk of myocardial infarction

26
Q

Local anesthetics

A

produce a rapid loss of sensation to a limited part of the body by blocking the entry of sodium ions into neurons by blocking sodium ion channels