Otorhinolaryngology (ENT) Flashcards

Ear: - Sensorineural & Conductive hearing loss - Tumours of ear Nose: -Rhinitis

1
Q

NasoOropharyngeal squamous cell carcinomas (OPSCC)

A
  • In the head and neck, OPSCC are associated with HPV
    -Ebstein Barr virus (suspect 2nd)
  • Arise in the soft palate, tonsils, base of
    tongue, pharyngeal wall, and vallecula, the fold located between the base of tongue
    and the epiglottis. Often present with neck masses
  • Classic symptoms of odynophagia and otalgia (usually no other clinical complaints)
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2
Q

Squamous cell carcinomas risk factors

A
  • smoking & drinking (strongest)
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3
Q

Oropharyngeal squamous cell carcinomas (OPSCC) differentials

A
  • Branchial cleft cyst carcinomas (exceptionally rare and its diagnosis should be one of exclusion rather than presumption)
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4
Q

Oropharyngeal squamous cell carcinomas (OPSCC) diagnosis

A

-Test for HPV
- CT head & neck (assessing the degree of local infiltration, involvement of regional
lymph nodes, and presence of distant metastases or second primary tumours)

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5
Q

1st line treatment for moderate to severe rhinitis?

A

Intranasal corticosteroid glucocorticoid (fluticasone)

-include antihistamines, leukotriene-receptor blockers, and topical glucocorticoids

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6
Q

Types of Rhinitis

A
  • Allergic
  • vasomotor (perennial and
    is not associated with itching)
  • infectious (nasal turbinates red and inflamed)
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7
Q

Allergic rhinitis

A
  • Caused by allergens that trigger a local hypersensitivity reaction (Specific IgE antibodies attach to circulating mast cells or basophils)
  • Rhinorrhoea and pruritus
  • nasal turbinates appear pale and
    boggy
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8
Q

If all conservative approaches to rhinitis have been unsuccessful, what the next step?

A

Immunotherapy:
- requires identification of specific antigen by dermal or serum testing
- 3-to-5-year course (treatment duration of less than a year is ineffective)

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9
Q

Pleomorphic adenoma diagnosis

A
  • CT to assess for deep lo be involvement
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10
Q

Pleomorphic adenoma management

A
  • If there’s no deep lobe involvement: superficial parotidectomy (treatment of choice for most cases)
  • If there’s deep lobe involvement: Total parotidectomy
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11
Q

clinical features of otitis media

A
  • one-week history of unwellness with URTI
  • unilateral earache
  • tympanic membrane red and bulging
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12
Q

Acute Otitis media risk factors

A
  • Age (6-18 months)
  • Lack of breastfeeding
  • Day care attendance
  • Smoke exposure
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13
Q

Pathogens that give arise to acute otitis media

A
  • Streptococcus pneumoniae
  • Nontypeable (?) Haemophilus influenzae
  • Moraxella catarrhali
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14
Q
A
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15
Q

Chronic Suppurative Otitis Media treatment

A

Ear toileting + ciprofloxacin ear drops

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16
Q

Complications of Otitis media

A

Acute mastoiditis

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17
Q

Acute mastoiditis initial treatment

A

sample from the ear discharge should be taken for culture DONE BY ENT

flucloxacillin + a third-generation cephalosporin

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18
Q

Acute otitis media treatment

A
  • Amoxicillin 1st choice (used for 1 week)
  • 2nd line: amoxicillin-clavulanate
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19
Q

Acute otitis media px that’s allergic to penicillin

A
  • clindamycin
  • azithromycin
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20
Q

Aboriginal px with otitis media management

A

Antibiotics straight away

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21
Q

Chronic suppurative otitis media treatment

A

Assess if tympanic membrane intact or perforated:
If intact: ciprofloxacin ear drops to treat on going infection
If perforated: initial treatment ear toilet with povidone-iodine solution, followed by
dry mopping with rolled toilet papers2 to 3 times a day using 20ml syringe with plastic tubing. In addition, ciprofloxacin ear drops

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22
Q

Untreated a tympanic membrane perforation secondary to chronic otitis media

A
  • Marginal perforation with discharge
  • Perforation that is surrounded by granulation tissue
  • Continuously discharging central perforation
  • Perforation associated with a cholesteatoma
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23
Q

Benign growth of the squamous epithelium squamous epithelium and
accumulation of keratin debris within the middle ear

A

Cholesteatoma

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24
Q

Cholesteatoma risk factors

A
  • history of recurrent acute otitis media
  • chronic middle ear effusion
  • tympanostomy tube placement
  • history of cleft palate
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25
Cholesteatoma features
- pearly white mass in the anterosuperior quadrant of the TM - chronic otorrhea (runny ear) - conductive hearing loss
26
Cholesteatoma treatment
surgical excision
27
Chronic sinusitis
- Symptoms that persist beyond 3 months (**symptoms that improve with therapy then reoccur**)
28
Chronic sinusitis treatment
- 2-3 weeks amoxicillin/clavulanate or fluoroquinolones - if above are unsuccessful, high-dose amoxicillin or a cephalosporin for 14 to 21 days
29
Chronic sialadenitis features
- recurrent jaw pain - swelling of the affected salivary gland - minimal saliva expression
30
Salivary duct calculi are composed predominantly of
calcium salts
31
Chronic sialadenitis investigation
- **Intraoral plain X-ray** (80% stones are radio-opaque) - CT - US (best used for parotid glands)
32
Parotid gland tumour features
- painless firm and mobile mass (same side) - facial nerve dysfunction (same side)
33
Parotid gland tumour investigation
CT head & neck
34
Midline neck swelling which moves with tongue protrusion
Thyroglossal cyst
35
Painless neck lumps in adults
- Malignant unless proven otherwise - FNAC and CT investigations of choice first -Excisional biopsy after CT
36
Lateral neck swelling which doesn't move with tongue protrusion
Branchial cysts
37
38
Sleep apnoea features
– Excessive daytime sleepiness. – Fatigue. – Snoring at night. – Choking or gasping while asleep. – Morning headaches. – Moodiness, irritability or depression - Impaired vigilance - Depression - Narcolepsy
39
Difference between obstructive sleep apnoea and central sleep apnoea
- sleep maintenance insomnia (experiencing a lot of prolonged awakenings during the night or one long wakeful period associated with daytime tiredness)
40
Sleep apnoea treatment
Continuous positive airway pressure **(CPAP)** - sleep studies & respiratory specialist referral for diagnosis confirmation of the
41
Sleep apnoea most important risk factor
**Congestive cardiac failure** (most important) - obesity (50%) - myotonic dystrophy - Ehlers-Danlos syndrome - smoking
42
Sleep apnoea stroke mechanisms
- Large swings in blood pressure - Increased coagulopathy - Development of atrial fibrillation - Local vibrational damage to the carotid artery bifurcation - Paradoxical emboli through the asymptomatic patent foramen ovale opening during transient sleep-related hypoxia with pulmonary hypertension.
43
Anterior triangle of the neck mass
**BCC** - Branchial cyst - Carotid body tumour - Carotid aneurysm
44
Branchial Cyst
- 20 – 40 y/o - Location: inferior to the EAM and anterior to the sternomastoid muscle - diagnosis: Mainly clinical - management: Excision
45
Carotid body tumour
- 40 – 60 y/o - Location: opposite thyroid cartilage - Pulsatile mass that moves laterally - Management: referral for excision
46
Posterior triangle of the neck mass
**CPP** - Cystic hygroma - Cervical rib - Pancoast tumour - (Naso/oropharyngeal squamous cell carcinomas)
47
Cystic hygroma
Paediatric condition (developmental remnant) * Transluminal mass Treatment: * refer Surgery * Sclerosis
48
Pancoast tumour
-Tumour of lung APEX - non-small cell cancer. - Unilateral or bilateral
49
Pancoast tumour Complications
Mainly compression of: 1. Subclavian artery: subclavian syndrome 2. Brachial plexus: painful paraesthesia 3. **Sympathetic ganglion: Horner’s syndrome** 4. brachiocephalic vein compression 5. phrenic nerve: cough 6. recurrent laryngeal nerve: hoarseness
50
Pancoast tumour treatment
- Surgery - Radiotherapy if life-threatening (subclavian syndrome) - Chemotherapy if surgery too difficult
51
Horner's Syndrome
- Anhidrosis – pseudo enophthalmos - Ptosis - Miosis
52
Midline of the neck mass
**TTD ** - Thyroid nodule - Thyroglossal cyst - Dermoid cyst
53
Thyroglossal cyst
**Paeds** remnant - most common cause of midline neck masses - **will move upwards with protrusion of the tongue**
54
Dermoid cyst
- **teratoma** that contains an array of developmentally mature, solid tissues - Management: imaging (CT) and surgical resection
55
Epistaxis management
1. Sitting forward to prevent blood dripping down the throat. 2. Compress the cartilaginous portion of nose for 5 -10 minutes without interruption
56
Most common cause of epistaxis
Local incidental trauma
57
Recurrent epistaxis in a sleep apnoeic patient
CPAP use - recommend warming & humidification of CPAP - nasal saline irrigation
58
Painless swelling in front of the ear and slight drooping of mouth
Parotid gland carcinoma Parotid gland carcinoma
59
Ramsay Hunt syndrome
- Also termed **herpes zoster oticus** (reactivation of varicella-zoster virus) - acute peripheral facial neuropathy Triad of symptoms: 1. ipsilateral facial paralysis (lower motor neuron cranial nerve 7) palsy 2. ear pain 3. **erythematous vesicular rash** of the skin of the ear canal, auricle, and mucous membrane of the oropharynx. - may be a loss of taste at anterior two-thirds of the tongue and hyperacusis
60
Ramsay Hunt syndrome treatment
- antiviral therapy and corticosteroid within 72 hours of presentation (rash onset). - Oral Acyclovir (800 mg five times per day), **valaciclovir** (1 g three times per day for 7–10 days) and **famciclovir** (250 mg three times per day) - valaciclovir and famciclovir have replaced acyclovir as drug of choice - oral corticosteroids **combined with antivirals** (eg, 40 mg prednisolone daily for 7 days, tapering to 5 mg daily over the next 2 weeks – total of 14 days)
61
Cancer of the oral cavity
- >50 years - Heavy smoker -Heavy drinker If no ulcerations or lesions present, **follow up in 12 months**
62
Rinne test
63
Weber test
64
Diseases of the middle ear
Otitis media Otosclerosis
65
Types of sensorineural hearing loss
- Meniere's - Labyrinthitis - Vestibular neuritis
66
Sensorineural deafness diseases are all found in
inner ear
67
Types of conductive hearing loss
- Otitis media - Otosclerosis - Cholesteatoma - TM perforation - Head trauma
68
Under normal circumstances, bone conduction is ________ than air conduction
worse
69
Under the Weber test, sensorineural loss localises to
the good ear, therefore the bad ear won't be able to hear it as well
70
Ménière's syndrome diagnosis
vertigo + vomiting + tinnitus + aural fullness + sensorineural deafness Abrupt onset. Attacks last 30 minutes to several hours.
71
Ménière's syndrome. Acute severe attack TREATMENT
Diazepam ± Prochlorperazine, or if episodic, a thiazide diuretic IF **Anticipation** of attack (Aural fullness, tinnitus) : Prochlorperazine
72
Ménière's syndrome: Prophylaxis
Hydrochlorothiazide or Triamterene + amiloride (K+ sparing diuretic) combination
73
Labyrinthitis diagnosis
acute onset vertigo provoked with changes in head position + tinnitus + hearing loss + URTI
74
URTI + vertigo + loss of balance + nystagmus + change in head position + reduced caloric test
Vestibular neuritis
75
Vestibular neuritis cause
Herpes Zoster URTI
76
Investigation of choice in a patient suspected with hearing loss + tinnitus + vertigo
Pure tone audiometry (audiogram)
77
BPPV investigation
Hallpike manoeuvre
78
BPPV treatment
Epley manoeuvre
79
Red flags for dizziness/vertigo
– Neurological signs – Ataxia out of proportion to vertigo – Nystagmus out of proportion to vertigo – Central nystagmus – Central eye movement abnormalities
80
Vestibular Neuronitis VS Labyrinthitis
Vestibular Neuronitis: Acute vertigo Nausea Vomiting Labyrinthitis: PLUS Hearing loss ± Tinnitus
81
Vestibular Neuronitis and Labyrinthitis MANAGEMENT
Bed rest Prochlorperazine or Ondansetron or Promethazine or Diazepam for the first 2 days
82