Near Drowning Flashcards

1
Q

New Drowning Outcome According to WHO

A

Death

With Morbidity

With no morbidity

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2
Q

Near Drowning Definition

A

When a victim survives a liquid submersion, at least temporarily

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3
Q

Dry Drowning Definition

A

As soon as fluid is inhaled the bronchi constricts in response to a parasympathetic mediated reflex

Due to involuntary laryngospasm liquid does not enter the lungs

10-20% of victims maintain the laryngospasm until cardiac arrest

Have lidocaine ready for the laryngospasm

Lungs of near-dry drowning victims are normal

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4
Q

Wet Drowning Definition

A

As oxygen levels fall the glottis relaxes and the liquid enters the lungs

When there is fluid in the lungs it will interfere with surfactant ability and look similar to ARDS

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5
Q

Aquatic Distress

A

The “classic” picture of drowning

Often, but not always, precedes drowning

Visible, active/violent/abrupt movements

This will only last 20-60 seconds

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6
Q

Instictive Drowning Response

A
  • The more typical picture of drowning
  • Behaviorsinstinctively taken by someone drowning (or close to drowning)
  • Noiseless and subtle behaviorsthat are instinctive
    • Will remain in the same position and extend arms out to the sides
    • Mouth is sinking below and reappearing above the water
    • Most drowning victims cannot yell for help because they are having trouble breathing
    • Young children will struggle less than adults and can look like they are doing the dog paddle in the water
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7
Q

Sequence of Drowning

A

Panic and violent struggle to return to the surface

Period of calmness and apnea

Swallowing of large amounts of fluid, followed by vomiting

Gasping inspirations and aspirations

Convulsion

Coma

Death

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8
Q

SALT WATER VS FRESH WATER

A
  • Pathologic changes in the lungs essentially the same
    • Reduction in pulmonary surfactant, alveolar injury, atelectasis, and pulmonary edema
  • Salt water near-drowning victims often have more electrolyte disturbances
    • Also if you swallow enough fresh water you will have electrolyte imbalance
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9
Q

Cold Vs. Warm Water

A

> 20 °C is warm; < 20°C cold

Cold water immersion will stimulate the diving reflex in young children (apnea, bradycardia, core-saving vasoconstriction)

Because water is an excellent conductor of body heat (cold water can cool the body 25 times faster than air in the same temperature), because evaporation further reduces an individual’s body heat and produces hypothermia

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10
Q

Clean Vs. Unclean Water

A

E.g. swamp, pond, sewage, mud, chlorine pool

May have aspirated pathogens and solid material

Risk of developing pneumonia and ARDS is much higher

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11
Q

FAVORABLE PROGNOSTIC FACTORS IN COLD WATER NEAR-DROWNING

Age

A

The younger the better outcome

Younger people tend to crash hard but easier to bring back

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12
Q

FAVORABLE PROGNOSTIC FACTORS IN COLD WATER NEAR-DROWNING

Submersion Time

A

The shorter, the better

60 min is the upper limit in cold water submersion

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13
Q

FAVORABLE PROGNOSTIC FACTORS IN COLD WATER NEAR-DROWNING

Water Temperature

A

The colder the better

Range 27-70oF

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14
Q

FAVORABLE PROGNOSTIC FACTORS IN COLD WATER NEAR-DROWNING

Water Quality

A

The cleaner the better

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15
Q

FAVORABLE PROGNOSTIC FACTORS IN COLD WATER NEAR-DROWNING

Other Injuries

A

None Serious

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16
Q

FAVORABLE PROGNOSTIC FACTORS IN COLD WATER NEAR-DROWNING

Amount of Struggle

A

Less struggle the better

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17
Q

FAVORABLE PROGNOSTIC FACTORS IN COLD WATER NEAR-DROWNING

Suicidal Intent

A

Lower survival rate among victims who attempted suicide than victims of accidental submersion

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18
Q

Pathophysiology

A

This is in regards to wet near-drownings, as dry near drownings will have normal lungs

  • Laryngospasm
  • Noncardiogenic pulmonary edema
  • Decreased surfactant
  • Alveolar shrinkage and atelectasis
    • Due to the pulmonary edema
  • Alveolar consolidation
  • Bronchospasm
  • Pulmonary hypertension
    • Secondary hypoxemia or inflammatory mediator release
19
Q

most important contributory factors to morbidity and mortality from drowning

A

The most important contributory factors to morbidity and mortality from drowning are hypoxemia and acidosis and the multiorgan effects of these processes.

20
Q

Central Nervous System Damage

A
  • May be caused by
    • A primary injury (prolonged hypoxemia during episode)
    • Secondary causes (reperfusion injury, cerebral edema, seizures, arrhythmias)
    • May be a concomitant head or spinal cord injury
  • CNS injury remains the major determinant of subsequent survival and long-term morbidity in cases of drowning. Two minutes after immersion, a child will lose consciousness. Irreversible brain damage usually occurs after 4-6 minutes.
21
Q

The Heart and Near Drowning

A

Can result in myocardial dysfunction

22
Q

Blood Volume Changes and Near Drowning

A

If more than 11 mL/kg of water is aspirated

Hypovolemia can result due to the fluid losses from increased capillary permeability

23
Q

Electroylte Changes and Near Drowning

A

If more than 22 mL/kg of water is aspirated

24
Q

CLINICAL MANIFESTATIONS

A

Apnea or tachypnea

Increased HR and BP

Cyanosis

Cough with frothy, white or pink sputum

Crackles on auscultation

Restrictive lung pathophysiology

25
ABG
Resp acidosis with hypoxemia Metabolic acidosis if lactic acid present Lactic Acid may be present if there is a lot of struggling and they used a lot of energy
26
CXR
Initial CXR may be normal; may deteriorate within the first 48-72 h Pulmonary edema and atelectasis Fluffy infiltrates; air bronchograms
27
Oxygenation Indices Qs/Qt
Increased Because the alveoli are filled with fluid
28
Oxygenation Indices DO2
Decreased
29
Oxygenation Indices VO2
Normal
30
Oxygenation Indices C(a-v)O2
Normal
31
Oxygenation Indices O2ER
Increased
32
Oxygenation Indices SvO2
Decreased
33
PRE-HOSPITAL MANAGEMENT
Conserve heat/maintain body temperature Remove wet clothes, cover **All drowning victims should get 100% O2** If in cardiac arrest: CPR; establish an airway
34
HOSPITAL MANAGEMENT For the Spontaneously Breathing Patient
Intubation and ventilation if PaO2 \< 60 mmHg on FiO2 of 0.50 or higher, or if respiratory acidosis present (\<7.25) NIPPV can be considered prior to intubation if patient is awake and alert
35
Hospital Management Mechanical Ventilaition
ARDS/lung protective strategy with permissive hypercapnia as necessary
36
Hospital Management
Bronchodilators (If bronchospasm is present) Manage Electroyle imbalance Surfactant Therapy Permissive Hypothermia
37
Hospital Management Warming of the Patient
Nearly all drowning victims are hypothermic to some degree Warm IV solutions, heated lavage, Active humidity on the ventilator Heating blankets (Bear hugger) (Rare) Cardiopulmonary bypass and blood warming \*\*Rewarming may be balanced by a goal of permissive hypothermia for neuroprotection…more research needed (Especially in victims that required CPR) Resuscitation should not end even if patient does not respond until a close approximation of normal body temperature is reached
38
Hospital Management Treatment for Fluid Depletion
Fluids, possibly inotropes
39
Hospital Management Bronchoscopy
To remove foreign material if present (debris, vomit)
40
Noncardiogenic Pulmonary Edema
Due to increased permeability of the membrane fluid from the pulmonary capillaries will move into the perivascular spaces, peribronchial space, alveoli, bronchioles, and bronchi (Interstitial edema) As a consequence of fluid movement, the alveolar wall and interstitial space swell, pulmonary surfactant concentration will decrease, and surface tension will increase Excessive fluid in the interstitial space causes lympathetic vessels to dilate and lymph flow to increase
41
Tachypnea in Near Drowning
Stimulation of peripheral chemoreceptors (hypoxemia) Decreased lung compliance- Increased ventilatory rate relationship Stimulation of J receptors Anxiety (conscious patient)
42
Cough with frothy, white or pink sputum in Near Drowning
the fluid that accumulates in the tracheobronchial tree is churned into a frothy white (sometimes blood tinged) sputum as a result of air moving into and out of the lungs (generally by means of mechanical ventilation)
43
What Happends to Virutally Every Near Drowning Patient
Virtually every near drowning victim suffers from hypoxemia, hypercapnia, and acidosis (acute ventilatory failure) Hypoxemia will generally persists after aspiration of fluids in the airway (wet drowning) because alveolar capillary damage and continued intrapulmonary shunting The degree of hypoxemia is directly related to the amount of alveolar capillary damage