Module 5 - Burns Flashcards
Burns result in
cute inflammatory response & release chemicals with in the body
As severity of burns increase tissue and underlying organs have an increased risk at being …
destroyed
thermal burns
result from direct contact with a heat source (fire, sun, water)
nonthermal burns
result from exposure to an electric or chemical source, radiation, light, or friction (fug burns, radiation therapy, etc)
Pediatric and geriatric burns are more concerning because
their skin is thinner and more susceptible to heat
Burns are classified based on
Burns are classified based on depth and how much surface area the burn effects on the body
Partial thickness
(first degree) - affects the epidermis and part of the dermis - often with no scarring
Deep partial thickness
(second degree) - that destroy the epididymis and parts of the dermis - area is red, swollen, blistered, and cause a lot of pain
Full thickness burns
(Third degree)- destroys all skin layers and underlying tissue - wound appears hard and dry and charred (eschar)
Blisters
Frequently seen in deep partial thickness burns
Surrounding skin may be red
Area is often hypersensitive and painful
Edema
Seen in deep partial thickness and full thickness burns
Results from fluids shifting
Dead skin slowly sloughs off with healing occurring when cells along the edges regenerate
Dark coloring
Seen in full thickness burns; often painless initially, but pain increases as inflammation continues
As eschar shrinks, pressure is placed on tissues beneath, which could further compromise circulation
If on a limb, escharotomy (surgical cuts through the charred area) may be required to ease pressure and restore circulation
Requires skin graft for healing
Hypovolemic shock
Chemical mediators release and cause fluid shifts when the inflammatory response is invokes, meaning that if a burn is larger the shift can cause too much water, protein, and electrolytes to move into the surrounding tissues leading to edema, decreased blood pressure, and hypovolemic shock
Hematocrit increases due to the concentration of RBCs in circulating fluid in a decreasing circulating fluid volume deficit (FVD), or hemoconcentration
Albumin also moves out of the capillaries lowering osmotic pressure, making it difficult to correct and maintain circulating blood volume until the inflammation subsides
Inflammation process and healing of burns
Burns cause coagulation necrosis (an alteration in cell proteins).
Neutrophils and monocytes collect along the injury to destroy and remove microorganisms and destroy foreign material.
Fibroblasts and new collagen fibrils appear and start wound repair within 6-12 hours of the initial injury.
Immunologic Changes Resulting from a Burn Injury
Burns destroy the body’s first layer of defense—the skin barrier.
The bone marrow function becomes suppressed leading to a decrease in the amount of immunoglobulins circulating in the blood.
As the bone marrow function is suppressed, the white blood cells (WBCs), which are formed in the bone marrow, do not function adequately.
The inflammatory process affects the functioning of lymphocytes, monocytes, and neutrophils, placing the patient at an increased risk for infection.