HNS33 Swallowing And Speech Flashcards

1
Q

Stages of swallowing

A
  1. Oral stage
  2. Pharyngeal stage
  3. Esophageal stage
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2
Q

Oral stage

A

Chewing (voluntary)
—> Integrity of hard palate (e.g. cleft palate: food can go into nose)
—> Tongue mobility
—> Food bolus loaded onto tongue
—> **Palatoglossal arch closed (Posterior oral cavity is closed)
—> Tongue push bolus backward to oropharynx
—> **
Palatoglossal arch opened
—> **Closure of nasopharynx by soft palate (pump theory: prevent leakage of pressure into nasal cavity)
—> **
Elevation of larynx (+ forward)
—> Mechanoreceptors activated
—> Pharyngeal plexus (CN9, 10)
—> Swallowing reflex

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3
Q

Pharyngeal stage

A
  • Involuntary
  • Shortest stage (~seconds)
  • Complex muscle contractions
    —> Soft palate and Posterior pharynx appose tongue
    —> Food bolus enter pharynx

Components:
1. Pharyngeal contractions: Suprahyoid muscles
—> ***Larynx move upward + forward
—> Forced into epiglottis

  1. Epiglottis pushed down by base of tongue base (pushing back —> Major pump)
    —> ***Folding of epiglottis over airway
  2. ***Closure of vocal cords / glottis
  3. Suprahyoid muscle pulled larynx upward and outward
    —> ***Upper esophageal sphincter opened at the same time
  4. Food enter pharyngeal wall
    —> Contraction of constrictor muscles (accessory muscle)
    —> Pharyngeal peristalsis begins
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4
Q

Esophageal stage

A
  • Involuntary (don’t aware of it happening)
  • Continuation of esophageal peristalsis
    —> UES remains open until bolus completely passes into esophagus
    —> Food bolus empties into esophagus
    —> UES closes
    —> Peristalsis of esophagus to push food down
    —> LES relaxes + Airway re-opens + Larynx down
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5
Q

***Neural control of swallowing

A

Oral stage:

  1. Mastication (CNV3)
  2. Tongue movement (CN12 + Pharyngeal plexus)
  3. Sensation (CNV2, CNV3, CN9)
  4. Special sensation (CN7, CN9)

Oropharyngeal stage:

  1. Tongue movement (CN12 + Pharyngeal plexus)
  2. Sensation (CN9)
  3. Soft palate movement (CNV3 (Tensor veli palatini), CN10 Pharyngeal plexus (Others))

Pharyngeal stage:
1. Sensation
—> Superior (CN9)
—> Hypopharynx (CN10 Superior laryngeal nerve)

  1. Laryngeal elevation
    —> **Geniohyoid (C1 via CN12 / Cervical plexus (Ansa cervicalis) via CN12)
    —> **
    Stylohyoid (CN7)
    —> **Mylohyoid, Anterior belly of digastric (CNV3)
    —> **
    Palatopharyngeus (Pharyngeal plexus)
  2. Closure of glottis / vocal cords (CN10 Recurrent laryngeal nerve)
  3. Opening of UES / Cricopharyngeus: (CN10 Pharyngeal plexus)
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6
Q

***Prevention of aspiration

A
  1. Elevation of larynx + Folding of epiglottis —> close Laryngeal inlet
  2. Closure of false cords (Most important mechanism)
  3. Closure of glottis / true cords (Most important mechanism)
    - vocal cord palsies —> aspiration
  4. Generation of positive subglottic pressure (exhale a bit against closed glottis during swallowing)
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7
Q

Clinical significance of swallowing

A

Symptoms

  1. Globus (lump in throat)
  2. Dysphagia
  3. Choking
  4. Cough
  5. Silent aspiration due to sensation deficit —> recurrent aspiration pneumonia
  6. Pneumonia

Causes

  1. Intra-luminal (tumour)
  2. Intra-mural (muscle problem)
  3. Extra-luminal (thyroid tumour)
  4. CNS pathologies

Investigations

  1. Endoscopy
  2. VFSS (videofluoroscopic swallowing study)
  3. Manometry (measurement of pressure within various parts of the gastrointestinal tract —> see if muscles working properly)
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8
Q

2 Components of Speech

A
  1. Phonation
    - generation of sound by forcing ***air through larynx —> cause vocal fold to vibrate
  2. Articulation
    - change of quality of sound
    - generation of sound by movements of ***mandible, lips, larynx, soft palate, tongue
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9
Q

***Intrinsic muscles of larynx

A
  1. Adductors —> Close glottis (move cord towards midline)
    - Lateral Cricoarytenoid
    - Transverse + Oblique Arytenoid
  2. Abductors —> Open glottis
    - ***Posterior Cricoarytenoid (ONLY abductor that open airway for inspiration, 2 vocal cords normally closed due to tension of ligaments)
  3. Shortening of vocal cord —> Thicken cord —> Lower pitch
    - Thyroarytenoid / Vocalis muscle
  4. Lengthening of vocal cord —> Pull thyroid cartilage anteriorly —> Increase tension of cord —> Higher pitch
    - Cricothyroid (supplied by ***Superior laryngeal nerve)
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10
Q

Superior laryngeal nerve vs Recurrent laryngeal nerve

A

ALL laryngeal muscles: Recurrent laryngeal nerve (branch of CN10)

EXCEPT

Cricothyroid muscle: External laryngeal nerve (branch of Superior laryngeal nerve —> branch of CN10)

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11
Q

Histology of vocal cord

A
  1. Mucosal layer
    - **Pseudostratified squamous epithelium (superior + inferiorly)
    - **
    Non-keratinising squamous epithelium (contact surface of medial cord)
  2. Lamina propria / Subepithelial tissue
    - 3 layers
    - Superficial layer (**Superficial lamina propria / **Reinke’s space)
    —> **Loose areolar tissue
    —> allow vocal cord epithelium to vibrate freely
    —> pathology at this place —> impede vibration of epithelium —> hoarseness
    - Intermediate layer + Deep layer —> **
    Vocal ligament
  3. Vocalis and Thyroarytenoid muscle
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12
Q

Phonation

A

Action of intrinsic **Adductor laryngeal muscles (Lateral Cricoarytenoid, Transverse + Oblique Arytenoid)
—> Adduct / close vocal cord
—> Exhale
—> **
Generate subglottic pressure
—> Pressure >3-5 cm H2O
—> Force vocal cords apart
—> Immediate falling of subglottic pressure
—> Vocal cords close again
—> Quick repetition
—> Vibration of vocal folds generate sounds
—> Male - 120Hz, Female - 230Hz

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13
Q

Loudness and Pitch

A

Loudness

  1. Force of expiration
  2. Lung function, Respiratory muscles

Pitch
1. Tension, “Dimentions” / Length of vocal folds (by Intrinsic laryngeal muscle)
—> higher pitch: longer and thinner vocal folds
—> lower pitch: shorter and thicker vocal folds
2. Size of larynx

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14
Q

Clinical significance of phonation

A

Symptoms
1. Hoarseness

Causes

  1. Air: COPD (air generation —> force generation)
  2. Neurological: Recurrent laryngeal nerve palsy (surgical complication, neurological problem) —> cannot close cord tightly
  3. Structure: Vocal cord pathologies (Polyps, Cancer, Scarring of SLP)

Investigations

  1. Cross-sectional imaging (CT, MRI)
  2. Endoscopy
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15
Q

Articulation

A

Change in shape of **upper aero digestive tract
—> Change **
resonance chamber
—> Generate **modification of tone
—> Different **
harmonics of tone e.g. nasal speech

  1. Mandible (CN5)
  2. Lips (CN7)
  3. Larynx (CN10)
  4. Soft palate (CN10)
  5. Tongue (CN12)

Pathologies:

  1. ***Higher centre —> poor coordination of muscles —> poor articulation e.g. Cerebellum dysfunction —> Scanning speech
  2. Tongue-tie —> Dysarthria (痴脷筋)
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16
Q

Higher centre for speech

A
  • Left frontal cortex (Broca’s area) (Premotor cortex)
  • Storage of “neural circuits” for words
  • Expressive aphasia (諗到講唔到)
    —> loss of the ability to produce language (spoken / manual / written) despite comprehension remaining intact
    —> effortful speech
17
Q

Clinical significance of articulation

A

Symptoms

  1. Dysarthria
  2. Expressive aphasia

Causes

  1. Tongue tie
  2. Oral pathologies (cancer, mass in back of throat —> hot potato voice)
  3. CNS pathologies (stroke, CNS tumour) —> damage to Broca’s area

Investigations

  1. Endoscopy
  2. CT brain