Headache Flashcards

1
Q

What is primary headache disorder?

A

Non life or sight threatening headache

Mainly chronic or recurrent

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2
Q

What headaches fall under primary headache disorder?

A
  • Tension headache
  • Migraine
  • Cluster headache
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3
Q

What is secondary headache disorder?

A

Headache due to another condution

Some are life or sight threatening

Many are ‘acute’

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4
Q

Gives some examples of things that can cause a secondary headache?

A
  • Subarachnoid/ intracranial haemmorhage
  • Tumour/ Space occupying lesion
  • Hydrocephalous
  • Meningits / intra cranial infections
  • Temporal giant cell arteritis
  • Acute, closed angle glaucauma
  • Medication related
  • Systemic hypertension/ pre-eclampsia
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5
Q

What are some of the flags for potentially life or sight threatening headaches?

A

SNOOP mneumonic

  • Systemic signs e.g. meningitis fever, neck stiffness. Pregnant, cancer?
  • Neurological symptoms e.g. SoL, ICH, glaucoma (visual)
  • Onset (new or changed & patient >50 years) e.g. Malignancy, closed angle glaucoma
  • Onset thunderclap e.g. vascular haemorrhage
  • Papilledema, pulsatile tinnitus, positional provication, precipitated by exercise → raised ICP
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6
Q

How may a patient desribe a tension type headache?

A
  • Generalised to the frontal / occipital regions
  • Tight, band like +/- radiating to the neck
  • Mild- moderate intensity
  • Worse at the end of the day
  • Recurrent (30mins-1 hr)
  • Aggrevated by stress, poor posture, lack of sleep
  • Often responds to simple analgesics
  • Few associated symptoms
  • Clinical examination is normal
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7
Q

What is the pathophysiology behind a tension type headache?

A

Thought to be due to tension in the muscles of the head and neck

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8
Q

What demographics do tension type headaches affect?

A
  • More common Female > Male
  • Young adults and teens
  • First onset >50 years unusual
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9
Q

What are the demographics of a migraine patient?

A
  • More common females > males (1 in every 5 females)
  • Common (15 in 100)
  • Presents early- mid life
  • Most have had first attack by age 30
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10
Q

How may a patient with migraine present?

A
  • Unilateral (temporal or frontal lobes)
  • Throbbing/ pulsating
  • Moderate- severe, often disabling
  • Prolonged headache, between 4-72 hours
  • Triggers; certain foods, menstrual cycle, stress, lack of sleep, often have family history
  • Can respond to simple analgesics
  • Associated symptoms; photophobia, nausea + vomiting, aura, neurological symptoms
  • Clinical examination is normal
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11
Q

Explain what occurs to cause a medication over use headache

A
  • Regular use of analgesics to treat preexisting headache and headache not responding
  • Most common caused by cocodamol
  • Co-exists with depression and sleep disturbance
  • Female > Male
  • Headache present on at least 15 days/month
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12
Q

How do you treat a medication over use headache?

A

Stop medication

Heache will worsen before it improves

Typically resolved completely by 2 months

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13
Q

What are the demographics of a cluster headache?

A
  • Affect Males > Females
  • Rare 1 in 1000
  • Usually begin age 30-40 years
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14
Q

What is thought to be the underlying pathophysiology behind a cluster headache?

A

Possibly due to hypothalamic activation with secondary trigeminal and autonomic involvement

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15
Q

How would a patient with cluster headache present?

A
  • Headache around/ behind the eye, unilaterally
  • Sharp, stabbing, penetrating pain often at night
  • Severe, intense, often disabling, agitated
  • Last 15mins- 3 hours
  • Occur in clusters with periods of remission
  • Triggers; alcohol, cigarettes, volatile smell, warm temp, lack of sleep
  • Ipsilateral autonomic symptoms; red watery eye, runny nose, ptosis, constricted pupil
  • Simple analgesics often ineffective → need O2 and triptans
  • Will have evidence of autonomi symptoms on examination (only if having headache at time)
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16
Q

How does headache due to a space occupying lesion present?

A
  • Gradual, progressive
  • Dull, variably described
  • May be mild in severity, worse in mornings/ on waking
  • Worsened by leaning forward, cough, valsalva manoeuvre
  • Simple analgesics may be affective in early stages
  • May be associated with nausea & vomiting, focal neurological or visual symptoms
17
Q

What is the pathophysiology of trigeminal neuralgia?

A
  • Mainly caused by compression of trigeminal nerve due to a loop of blood vessel
  • 5% are due to tumours/ skull base abnormalities or AV malformations
18
Q

What are the demographics of trigeminal neuralgia?

A
  • Affecfs females > males
  • 25/ 100,000 UK
  • 50-60 years peak incidence
19
Q

How does a patient with trigeminal neuralgia usually present?

A
  • Unliateral, pain felt in >1 divisions of the CN V
  • sharp, shooting pain - electric shock/ burning
  • Severe, lasts 2s-2mins
  • Sudden Onset
  • Triggers; light touch to face/ scalp, eating, cold wind, combing hair
  • Simple analgestics not effective, difficult to treat
  • May get preceding symptoms; tingling, numbness, pain can radiate to CN 5 distribution
  • Clinical examination is usually normal
20
Q

What is temporal arteritis?

A

Vasculitis involving small and medium sized arteries of the head

  • Females > Males
  • <50 years (most common in > 75 years
  • Abrupt onset headache, visual disturbance or jaw claudication
21
Q

Which artery is most commonly affected by temporal (giant cell) arteritis?

A

Superficial temporal artery

22
Q

What is the main risk of temporal (giant cell) arteritis)?

A

Risk of irreversible loss of vision due to ischemia of cranial nerve 2