Glaucoma Flashcards
What NML IOP
10-21
Higher in the AM compared to PM
Aqueos humor is produced where?
Ciliary body
What are the two things you need to Dx glaucoma
optic nerve damage AND! visual field loss
What is the anatomical blind spot?
Optic disk
What are the two methods of doing a tonometry for glaucoma
-Non-contact = air puff
-Applanation = pressing on cornea with instrument
—Tonopen
!!Goldmann applanation = gold standard!!!
What is the gold standard to eval a tonometry
Goldman applanation
What is the only thing we can actually treat in glaucoma pts
IOP
If a pt has an IOP greater than 21, with a NML optic nerve, ant. Chamber, and no visual field loss
What is the approach
No Tx necessary, monitor annually
What are the RSK fx for developing glaucoma
Age
Large C/D ration
Thin central corneal thickness
Fm Hx
High Myopia
What is the patho phys of Primary open angle glaucoma
Loss of retinal nerve fiber layer (NFL) reduces vision
IOP greater than 28
A pt presents with a cc of tunnel vision
Think
Primary open angle glaucoma
What is the treatment approach to primary open angle glaucoma >?
Referal to ophthalmology/ optometry
And Rx that decrease the aqueous or increase the flow
What are the rx for primary open angle that decrease the production of aqueous
B-blockers
Alpha -2 adrenergics
(Brimonidine or apraclonidine)
Carbonic anhydrase inhibitors
(Dorso lamine, Brinzolamide, Acetazolamide)
What are the Rx for Primary open angle that increase the flow of aqueous
Sympathomimietics
(EPI, memantine)
Prostaglandin analogs
(-Prost)
Miotic agents
(Pilocarpine)
What are the ADE of using Alpha 2-adrenergics in POAG
Allergic conjunctivitis
Or contact dermatitis