force, velocity, and power Flashcards

1
Q

what is the shape of the length tension curve

A

inverted U shape

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2
Q

why does the length tension curve have that shape

A

at contractile end of ROM
- muscle produces less force because there’s not much room to move

at tensile end of ROM
- muscle produces less force because there’s not enough overlap for cross bridge cycling

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3
Q

what affects the force velocity characteristics of muscle contraction

A

number of cross bridges forms
environmental conditions

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4
Q

what are the sarcomere properties during isometric hold

A

amount of filament overlap depends on the length at which the muscle is held prior to activation

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5
Q

what are the sarcomere properties during shortening (concentric)

A
  • sliding movement allows myosin filaments to become overlapped by actin
  • tension in muscle increases while the muscle is getting shorter
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6
Q

what are the sarcomere properties during lengthening (eccentric)

A
  • cross bridges generate less tension than the external stretching force applied to the muscle (lengthen)
  • opposing acting filaments in the sarcomeres are pulled away from each other
  • tension in muscle increases when the muscle is stretched
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7
Q

what causes active vs passive tension

A

active = actin and myosin
passive = titin
- exponential increase in titin activity as muscle length increases

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8
Q

how does total tension change through movement

A
  • increases (active)
  • decreases (dip between active and passive tension)
  • increases again (passive - titin)
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9
Q

what is the popping sarcomere hypothesis

A

uncontrolled extension of individual half sarcomeres in each myofibril

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10
Q

how do sarcomeres “pop”

A

sarcomeres are stretched until its passive tension is unable to support the force and the half sarcomere pops
- occurs one at a time from weakest to strongest
- actin and myosin aren’t overlapping but titin maintains structure

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11
Q

what causes DOMS

A

after eccentric contraction
- half sarcomeres popped
- sarcomere length increases (stretched)
- metabolites released into muscle and accumulate
- activates free nerve endings

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12
Q

what do group III/IV (mostly III) afferents respond to

A

mechanical deformation and intramuscular substances that cause pain
- project to the CNS and promote sensation of pain

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13
Q

what is the shape of the force-velocity curve

A

non linear inverse relationship
- less force = higher velocity
- more force = lower velocity
(zero velocity = highest force)

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14
Q

what is the shape of the power curve

A

inverted U
- with optimal F and V values below 50% of their max values

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15
Q

what are the values of P and V at F max

A

P and V = 0

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16
Q

what are the values of P and V at F <50% max

A

P = max
V <50% max

17
Q

what are the values of P and V at F = 0

A

P = 0
V = max

18
Q

when are there more sarcomeres in series

A

when the muscle is less pennated

19
Q

when are there more sarcomeres in parallel

A

when the muscle is more pennated

20
Q

what is the function of sarcomeres in series

A

more energy efficient in terms of displacement
- high speed
ex: hamstrings and dorsiflexors (longer muscles with less force but higher speed)

21
Q

what is the function of sarcomeres in parallel

A

more efficient in force production
ex: plantar flexors, and quads (short muscles with large force and slow velocity)

22
Q

what causes more sarcomeres in parallel

A

hypertrophy

23
Q

what values are equal and unequal between the two types (series and parallel)

A

equal = work and power potential
unequal = force, displacement, velocity

24
Q

what is ACSA

A

largest CSA along the length of the muscle (girth only)

25
Q

what is PCSA

A

total CSA of all fibres within the muscle (perpendicular to fibres)
- depends on muscle pennation

26
Q

what is the effect of pennation on muscle force production

A

more pennated = stronger muscle
- can become more pennated with high resistance training - more sarcomeres in parallel

less pennated = faster muscle - lower force production