Fish (KP) Flashcards
Skin response to Bacterial injury?
- Aeromonas & Vibrio → dermal localization → ulceration
- Flavobacterium → Yellow pigmented bacteria → ulceration, yellow skin
Which fish tissues are capable of extreme regeneration w/ little to no scarring?
TQ
Cariac
Muscle
Retina
Skin
CNS
How much of the Gills do you have to lose before you see clinical signs?
(TQ)
50%
What are the gills responsible for & can be effected during gill insult?
(TQ)
- Gas Exchange
- Acid/base balance
- Osmoregulation
- Excretion of ammonia & carbon dioxide
How do gills respond to chronic injury?
TQ
Hypertrophy of the epthelial cells & fusion of the lamaelli →
increases gas diffusion distance & turbulence over the gills
Where does inflammation occur in the gills?
Mostly in the central venous sinuses b/c of large # of inflammatory cells
What is a Chaetoceros?
How does it harm the gills?
TQ
- Filamentous diatom with backward pointing spines
- act like burrs & fish can’t remove on it’s own
- Cause physical damage & severe acute bronchitis
What is a common bacterial gill disease?
Where does it cause infection?
What drugs help?
- Flavobacterium branchiophilum
- Superficial infection of the surface of gills
- NSAIDs - b/c prostaglandin-mediated vasoconstriction in gills
What is the anatomy of the fish heart?
TQ
- Single chambered system
- Atrium
- Ventricle
- Bulbus arteriosus
What is the difference between fast swimming & slow swimming fish hearts?
TQ
- Fast swimming →ventricle has a compact outer layer→ coronary blood vessels ⇒ aerobic oxidative metabolism
- Slow swimming →Spongy layer only →no coronary blood vessels →glycolytic metabolism
In a fish what is CO more depenent on?
CO = SV * HR
TQ
Stoke Volume (SV)
What can cause cataracts in fish eyes?
TQ
- Toxins from bacteria
- Eye flukes - Diplostomum flexicaudatum
What are the factors that cause gas bubble disease in fish?
What does it do to the fish?
What can help prevent it?
TQ
-
Causes:
- Rapid increase in water temp or rapid decrease in pressure
- Drop in pressure experienced by blood passing across gills
- Gas bubbles cause emboli in the vessels of the gills, chorioid gland (eye), pseudobranch & elsewhere
- Prevented by aerating the water
What is most of the kidney made up of?
TQ
Hemopoietic tissue
(main hemopoietic organ)
What is different about the fish kidney as compared to mammals?
TQ
- Kidney in fish = liver in mammals
- Fish kidney has no cortex or medulla (so no LOH) → can’t concentrate urine like mammals
- Renal portal MØs → act to phagocytoses foreign substances
Which can fish tolerate better?
A rise or drop in water temp?
TQ
- Drop in water temp
Warm water holds less O2 than cold.
How can this be harmful to the fish?
TQ
- Increases in water temp → increase in metabolism → increased metabolic/developmental rate → increase in O2 demand
- Increased susceptibility to bacterial infections
- Once fish can no longer extract enough O2 ⇒ respiratory distress syndrome
What can happen to your eggs/fry kept in water that is too warm?
TQ
- Spinal abnormalities
- cardiac abnormalities
- increase suceptibility to bacterial diseases
NH3 + H2O ⇔ NH4+ + OH-
pH & Temperature determine which way this equation goes
What will increase the amount of free ammonia (NH3)?
What will increase the amount of ionized ammonia (NH4+)?
TQ
- Increases in pH shift the equation to the left = more free ammonia = danger
- Increases in Temp. shift the eqation to the left = more free ammonia = danger
But wait…gills secrete ammonia, how come it doesn’t hurt the fish?
Gills also secrete CO2
- CO2 +H2O → H2CO3 (carbonic acid) → Lowers pH = protective mechanism
Is an increased amount of ammonia secreted by the gills damaging to the fish?
Nope
Is an increase in ammonia in the body damaging to the fish?
Yep = interference with neurtransmitters = coma
What is the nitrification cycle?
How is it accomplished?
- Ammonium → Nitrite → Nitrate
- Bacteria in the environment
- Nitrosomonas converts Ammonium → Nitrite
- Nitrobactor converts Nitrite →Nitrate
What is the impact of too much CO2?
TQ
- Impacts the carbonic anhydrase system → metabolic acidosis → impacts gas excretion into swim bladder (distention) and eye (exophthalmos)
- STOP FEEDING THE GOLDFISH!!!
What is hemorrhagic septicemia and in what fish would you find it?
- Acute gram neg bacterial dz associated with high water temps
- Bacteria have long filamentous structure
- Fresh water fish
What will you see in a fish
with hemorrhagic septicemia?
- Hemorrhagic necrosis of hematopoetic tissue, kidney, spleen & intestinal mucosa
- Congestion at base of fins & around vent
- Intussesception
- Swollen soft kidney & spleen
What 3 things are responsible for hemorrhagic septicemia?
- Aeromonas hydrophilia → Zoonotic, FWF
-
Aeromonas samonicida
- Furunculosis in salmonids & goldfish
- Pasteurellosis→ “Yellow tail” in Japan
What bacteria is responsible for atypical bacterial dz in fish?
Why is it atypical?
Pseudomonas
Atypical because
- hemorrhagic septicemia more common in marine environment and low temps
- does not have long filamentous structures
- Causes lethargy, sleepiness & stumpy gills
Yellow pigmented bacterial Dz or myxobacterial dz is caused by what types of gram negative bacteria?
- Flavobacterium columnare (columnaris Dz)→ fresh water
- Tenacibaculum → saltwater (jellyfish mouths)
- Flavobacterium branchiophilum → Bacterial gill Dz
- Flavobacterium psychrophilum → BacT Cold water Dz
What is the most important yellow pigmented bacterial Dz of fish?
Columnaris Dz (Flavobacterium columnare)
- Necrotizing dermatitis @ > 18*C
- Saddle patch lesion around dorsal fin
- Cotton-wool mouth
- Gills