Famous People/Laws/Definitions Flashcards
Subluxations are from abnormal biomechanics caused by muscle imbalance in a weight bearing spine. Pelvic distortion model (Basic distortion of the spine starts in the pelvis)
Carver
Developed a protocol for the tx of lumbar disc protrusion, spondylolisthesis, facet syndrome, subluxation, & scoliotic curves
Cox/McManus (Flex/dist)
Developed SOT. CSF flow through the pumping action of the sacrum & the cranial dura mater
DeJarnette
Fixation theory of joint hypermobility
Gillet/Fave
Subluxations are all pos. w/ disc wedging
Gonstead
Upper cervical specific; dentate lig.
Grostic
Found that disc herniations, exostoses, or subluxations may produce pressure on the dorsal nerve root
Hadley
Father of homeopathy (tx’s pts w/ heavily diluted preparations which are thought to cause effects similar to the symptoms presented)
Hahnemann
Father of modern medicine; first to manipulate
Hippocrates
Joint hypermobility; described SI movement & pelvic dynamics. Discovered & tested SI ligs.
Illi
3 phase model of instability (dysfunction, unstable, stabilization); Breaking up spinal adhesions on an injured segment
Kirkaldy/Willis
Segmental Facilitation Theory. Established the concept of subluxation creating a hyperactive nervous system, rather than a decrease in nerve impulses. Muscle is central to his theory
Korr
Wrote the 1st chiro. textbook in 1906; people started to research & incorporate motion instead of “bone out of place” idea
Langworthy, Smith, & Paxson
Revised Faye’s concept of the VSC
Skip Lantz
Founder of technique where sacrum is keystone of spine, first to use heel lifts
Logan (Logan Basic Technique)
World renown scientists in the field of biomechanics of the spine
Panjabi & White
General Adaptation Syndrome; under optimum conditions the body can respond to stressors
Selye
Wrote chiro textbook. Identified 33 principles of chiro. & “safety pin” cycle
Stephenson
Founder of osteopathy in 1856; utilized the circulatory system
Andrew Taylor Still
Came up with name “chiropractic”
Samuel Weed
Steady deformation d/t sustained axial pressure
Creep
Energy loss from loading & unloading disc (heat)
Hysteresis
Long standing creep w/ hysteresis event
Buckling syndrome
Deformation increases in proportion to the load applied
Hooke’s Law
Opposing translation of two body parts
Sheer
Any recoverable deformation
Elastic deformation
Property of a material or structure that returns it to its original form following the removal of the deforming load
Elasticity
Nonrecoverable deformation
Plastic deformation
Property of a material to permanently deform when it is loaded beyond its elastic range
Plasticity
Property of returning to the former shape or size after distortion
Resilience
Property of a material showing sensitivity to the rate of loading or deformation, two basic components are viscosity & elasticity. Slowly deforms, slowly reforms
Viscoelasticity
Property of materials to resist loads that produce shear. Strain delay
Viscosity
Balance of compression & tensile forces on connective tissue
Tensegrity
Increase epiphyseal plate pressure = decreased growth
Decreased epiphyseal plate pressure = increased growth
Heuter Volkman
Bone (structure) responds to stress (function)
Wolff
Soft tissue responds to stress
Davis’ law
A nerve which supplies a joint, supplies the muscle, skin, & tissue around it (axoplasmic aberration)
Hilton’s law
Pain threshold, pain managment
Algometry
Nitrogen release during adjustment (joint capsule seal)
Cavitation
Skin rolling over spine for dx & tx
Roulement
Prox. neurological irritation causing peripheral neuropathies
Double Crush
CNS remapping, reorganizing, & brain expansion. PNS synapse (?)
Neuroplasticity
Opiates from peri-aqueductal grey descend to bathe dorsal horn & reduce incoming pain messages
Descending inhibition
Facilitation d/t acute nerve compression bombardment of dorsal horn, “endogenous opiates” (PAG) release shakes off pain for body to respond to immediate threat
Descending modulation
Gate control theory (Wall’s), fast conducting proprioceptor & mechanoreceptor messages close the gate of slow pain receptors to CNS
Inhibiting system
Distraction principle such as: rubbing elbow, TENS unit. Proprioception is faster than nociception
Afferent inhibition
Deep joint, thin, myelinate A-beta fibers
Group III nociceptors
Deep joint, unmyelinated, C fibers
Group IV nociceptors
Functional disturbance of pain pathway
Neuropathic pain
Reversible inappropriate neurological response to environment, errors in = errors out (safety pin cycle gone bad)
Dysponesis
Pain d/t stimulus which doesn’t normally provoke pain
Allodynia
Pressure on nerve (direct or indirect)
Neurothlipsis
Autoimmune attack on C1 transverse ligs (adjustments help)
Grisel syndrome
Distorted or impaired voluntary movement
Dyskinesia
Impairment of viscera d/t nervous system, can by measured by skin temp. differentiation
Dysautonomia
Neuropathic, radicular, deep pain, even felt on skin, but long lasting
Deep nociception
Fast, quick, short bursts of pain, not recognized in long standing conditions
Cutaneous nociception
Simultaneous pain in structures innervated by shared spinal segment
Referred pain
Brain interprets pain from multiple tissues. Rationale for referred pain, such as cervicogenic headache
Central convergence projection
Transection of nerve causing multiple synapse recovery
Traumatic neuroma
Chronic instability resulting in mechanoreceptors morphing into nociceptors. When the injury has healed & motion is restored, the pain will subside b/c nociceptors revert back to proprioceptors
Wide dynamic receiver (WDR)
What are autocoids?
Chemical mediators released by connective tissue that cause pain, joint morphology, & decrease motion