Famous People/Laws/Definitions Flashcards

1
Q

Subluxations are from abnormal biomechanics caused by muscle imbalance in a weight bearing spine. Pelvic distortion model (Basic distortion of the spine starts in the pelvis)

A

Carver

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2
Q

Developed a protocol for the tx of lumbar disc protrusion, spondylolisthesis, facet syndrome, subluxation, & scoliotic curves

A

Cox/McManus (Flex/dist)

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3
Q

Developed SOT. CSF flow through the pumping action of the sacrum & the cranial dura mater

A

DeJarnette

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4
Q

Fixation theory of joint hypermobility

A

Gillet/Fave

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5
Q

Subluxations are all pos. w/ disc wedging

A

Gonstead

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6
Q

Upper cervical specific; dentate lig.

A

Grostic

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7
Q

Found that disc herniations, exostoses, or subluxations may produce pressure on the dorsal nerve root

A

Hadley

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8
Q

Father of homeopathy (tx’s pts w/ heavily diluted preparations which are thought to cause effects similar to the symptoms presented)

A

Hahnemann

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9
Q

Father of modern medicine; first to manipulate

A

Hippocrates

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10
Q

Joint hypermobility; described SI movement & pelvic dynamics. Discovered & tested SI ligs.

A

Illi

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11
Q

3 phase model of instability (dysfunction, unstable, stabilization); Breaking up spinal adhesions on an injured segment

A

Kirkaldy/Willis

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12
Q

Segmental Facilitation Theory. Established the concept of subluxation creating a hyperactive nervous system, rather than a decrease in nerve impulses. Muscle is central to his theory

A

Korr

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13
Q

Wrote the 1st chiro. textbook in 1906; people started to research & incorporate motion instead of “bone out of place” idea

A

Langworthy, Smith, & Paxson

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14
Q

Revised Faye’s concept of the VSC

A

Skip Lantz

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15
Q

Founder of technique where sacrum is keystone of spine, first to use heel lifts

A

Logan (Logan Basic Technique)

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16
Q

World renown scientists in the field of biomechanics of the spine

A

Panjabi & White

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17
Q

General Adaptation Syndrome; under optimum conditions the body can respond to stressors

A

Selye

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18
Q

Wrote chiro textbook. Identified 33 principles of chiro. & “safety pin” cycle

A

Stephenson

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19
Q

Founder of osteopathy in 1856; utilized the circulatory system

A

Andrew Taylor Still

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20
Q

Came up with name “chiropractic”

A

Samuel Weed

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21
Q

Steady deformation d/t sustained axial pressure

A

Creep

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22
Q

Energy loss from loading & unloading disc (heat)

A

Hysteresis

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23
Q

Long standing creep w/ hysteresis event

A

Buckling syndrome

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24
Q

Deformation increases in proportion to the load applied

A

Hooke’s Law

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25
Q

Opposing translation of two body parts

A

Sheer

26
Q

Any recoverable deformation

A

Elastic deformation

27
Q

Property of a material or structure that returns it to its original form following the removal of the deforming load

A

Elasticity

28
Q

Nonrecoverable deformation

A

Plastic deformation

29
Q

Property of a material to permanently deform when it is loaded beyond its elastic range

A

Plasticity

30
Q

Property of returning to the former shape or size after distortion

A

Resilience

31
Q

Property of a material showing sensitivity to the rate of loading or deformation, two basic components are viscosity & elasticity. Slowly deforms, slowly reforms

A

Viscoelasticity

32
Q

Property of materials to resist loads that produce shear. Strain delay

A

Viscosity

33
Q

Balance of compression & tensile forces on connective tissue

A

Tensegrity

34
Q

Increase epiphyseal plate pressure = decreased growth

Decreased epiphyseal plate pressure = increased growth

A

Heuter Volkman

35
Q

Bone (structure) responds to stress (function)

A

Wolff

36
Q

Soft tissue responds to stress

A

Davis’ law

37
Q

A nerve which supplies a joint, supplies the muscle, skin, & tissue around it (axoplasmic aberration)

A

Hilton’s law

38
Q

Pain threshold, pain managment

A

Algometry

39
Q

Nitrogen release during adjustment (joint capsule seal)

A

Cavitation

40
Q

Skin rolling over spine for dx & tx

A

Roulement

41
Q

Prox. neurological irritation causing peripheral neuropathies

A

Double Crush

42
Q

CNS remapping, reorganizing, & brain expansion. PNS synapse (?)

A

Neuroplasticity

43
Q

Opiates from peri-aqueductal grey descend to bathe dorsal horn & reduce incoming pain messages

A

Descending inhibition

44
Q

Facilitation d/t acute nerve compression bombardment of dorsal horn, “endogenous opiates” (PAG) release shakes off pain for body to respond to immediate threat

A

Descending modulation

45
Q

Gate control theory (Wall’s), fast conducting proprioceptor & mechanoreceptor messages close the gate of slow pain receptors to CNS

A

Inhibiting system

46
Q

Distraction principle such as: rubbing elbow, TENS unit. Proprioception is faster than nociception

A

Afferent inhibition

47
Q

Deep joint, thin, myelinate A-beta fibers

A

Group III nociceptors

48
Q

Deep joint, unmyelinated, C fibers

A

Group IV nociceptors

49
Q

Functional disturbance of pain pathway

A

Neuropathic pain

50
Q

Reversible inappropriate neurological response to environment, errors in = errors out (safety pin cycle gone bad)

A

Dysponesis

51
Q

Pain d/t stimulus which doesn’t normally provoke pain

A

Allodynia

52
Q

Pressure on nerve (direct or indirect)

A

Neurothlipsis

53
Q

Autoimmune attack on C1 transverse ligs (adjustments help)

A

Grisel syndrome

54
Q

Distorted or impaired voluntary movement

A

Dyskinesia

55
Q

Impairment of viscera d/t nervous system, can by measured by skin temp. differentiation

A

Dysautonomia

56
Q

Neuropathic, radicular, deep pain, even felt on skin, but long lasting

A

Deep nociception

57
Q

Fast, quick, short bursts of pain, not recognized in long standing conditions

A

Cutaneous nociception

58
Q

Simultaneous pain in structures innervated by shared spinal segment

A

Referred pain

59
Q

Brain interprets pain from multiple tissues. Rationale for referred pain, such as cervicogenic headache

A

Central convergence projection

60
Q

Transection of nerve causing multiple synapse recovery

A

Traumatic neuroma

61
Q

Chronic instability resulting in mechanoreceptors morphing into nociceptors. When the injury has healed & motion is restored, the pain will subside b/c nociceptors revert back to proprioceptors

A

Wide dynamic receiver (WDR)

62
Q

What are autocoids?

A

Chemical mediators released by connective tissue that cause pain, joint morphology, & decrease motion