Eyelids: Part 2 Flashcards

1
Q

characteristics:

dystrophic levator muscle

A

congenital ptosis

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2
Q

symptoms: visual loss, diplopia, headache, pain
signs: anisocoria, proptosis, ocular motility defects

-superior tarsal fold usually absent/poorly developed
-may have weak/no eyelid crease
-ptosis decreases in down-gaze
—compensates by looking in chin up position

which signs & symptoms are these for?

A

congenital ptosis

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3
Q

characteristics:
neurogenic: innervational defect
—eg, CN3 paresis, Horner’s syndrome

myogenic: myopathy of levator muscle or impairment of transmission of impulses at NMJ
—eg, myasthenia gravis, myotonic dystrophy, progressive external ophthalmoplegia

aponeurotic/involutional → levator aponeurosis
—eg, age-related — fibrous tissue takes over tendon

mechanical → gravitational effect of a mass or from scarring
—eg, hordeolum, chalazion

A

acquired ptosis

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4
Q

symptoms: visual loss, diplopia, headache, pain
signs: anisocoria, proptosis, ocular motility defects

  • crease appears higher than usual
  • ptosis in affected eye is level w/ or lower than normal lid in downgaze
  • bilateral frontalis contraction (raised eyebrows)
  • may have increases w/ stress, fatigue (diurnal variation)

what signs & symptoms are these for?

A

acquired ptosis

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5
Q

what is the management for (congenital/acquired) ptosis?

A

upneeq (oxymetazoline 0.1%): 1 drop per day in affected eye for 6 weeks

—elevates upper eyelid — direct-acting alpha adrenergic receptor → selectively targets Mueller’s muscle

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6
Q

characteristics:

false impression of ptosis caused by —
—common cause: TED (thyroid eye disease)
—lack of support of lids by the globe due to orbital volume deficit

A

pseudoptopsis: contralateral lid retraction

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7
Q

signs & symptoms:

  • abnormal PFH height & MRD1
  • droopy contralateral eyelid
  • positive lid lag
  • proptotic eye
A

contralateral lid retraction

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8
Q
if cause established & symptoms mild — 
—treatment of underlying condition
     — chalazion: warm compresses w/ massage
     — eyelid neoplasm: excision
—observation
—taping upper lids open
—eyelid crutches attached to glasses

if cause not established &/or symptoms not addressed w/ conservative management, include all findings & refer to —
—neuro-ophthalmology — neurogenic, myogenic
—oculoplastics — congenital (esp. important to prevent occlusion amblyopia), aponeurotic, mechanical

what management is this for?

A

pseudoptopsis: contralateral lid retraction

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9
Q

characteristics:

false impression of ptosis caused by —
—familial tendency
—middle to older age groups

A

pseudoptopsis: dermatochalsis

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10
Q

signs & symptoms:

  • usually bilateral
  • loose & redundant tissue fold
  • draping of superior lid tissue over septum or lid margin
A

dermatochalasis

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11
Q

what is the management for dermatochalsis? what happens if left untreated?

A

blepharoplasty (if creating problems)

if left untreated → may also cause mechanical ptosis

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12
Q

what are these characteristics indicative of?

infectious, involutional, cicatrical

A

ectropion

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13
Q

signs & symptoms:

out-wardly turned eyelid margins

A

ectropion

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14
Q

This management is indicative of what?
lubricating ointment & eyelid taping at night

  • identify underlying cause
  • artificial tears & cool compresses
  • bacitracin or erythromycin

referral to oculoplastics
—only if long-standing, stable
to address significant corneal exposure, permanently correct

A

ectropion

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15
Q

characteristics:

congenital, allergic, mechanical, paralytic (CN7 palsy)

A

entropion

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16
Q

in-wardly turned eyelid margin

A

entropion

17
Q

This management is indicative of what?

bandage soft CLs

  • identify underlying cause
  • artificial tears & cool compresses
  • bacitracin or erythromycin

referral to oculoplastics
—only if long-standing, stable
to address significant corneal exposure, permanently correct

A

entropion

18
Q

characteristics:
suspected etiology —
—abnormality of collagen & elastin fibers
—spontaneous eversion of upper eyelid during sleeping
—superior palpebral conjunctiva rubs against pillow/sheets

associations — 
—keratoconus
—skin hyperelasticity & joint hypermobility
—obstructive sleep apnea
      —normal tension glaucoma (NTG)
—diabetes
—mental retardation
—typically seen in obese males
A

floppy eyelid syndrome

19
Q

signs & symptoms:

  • usually bilateral, asymmetric
  • chronically red, irritated eyes
  • mild mucous discharge
  • symptoms worsen during awakening due to eyelid eversion at night
A

floppy eyelid syndrome

20
Q
  • avoid sleeping face down
  • eye shields or eyelid taping
  • lubricating ointment QHS
  • treatment for PEE (erythromycin or bacitracin TID)

refer to — internist to rule out OSA; oculoplastics

what is this management for?

A

floppy eyelid syndrome

21
Q

characteristics:
abnormal pauses in breathing/instances of abnormally low breathing
—excessive daytime sleepiness & fatigue
—may be recognized as a problem by others witnessing individual during episodes

A

sleep apnea

22
Q

what is the management for sleep apnea?

A

may create transient hypoxemia & increased vascular resistance
—consult w/ internist for sleep study
—seek treatment for sleep apnea if needed

23
Q

characteristics:
glaucoma (optic neuropathy) w/
—open, apparently normal anterior chamber angles
—glaucomatous optic nerve damage
—vision loss characteristic seen in pts w/ chronic glaucoma related to elevated IOP
—w/o a clearly abnormal IOP (within normal limits, around ≤21 mmHg)

A

normal tension glaucoma (NTG)

24
Q

what is the management for normal tension glaucoma (NTG)?

A

may create transient hypoxemia & increased vascular resistance
—consult w/ internist for sleep study
—seek treatment for sleep apnea if needed