exam 4: learning and memory Flashcards

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1
Q

what does memory consolidation through rehearsal lead to?

A

leads to long-term memory

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2
Q

when ___ (RNA or DNA) is blocked, it decreases memory

A

RNA

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3
Q

working memory does not equal ____ _____ memory

A

long term

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4
Q

what are two things seen in alzheimer’s patients?

A

neurofibrillary tangles and amyloid beta proteins

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5
Q

t or f: every disruption in your environment disrupts the loop of the hippocampal formation, making it less efficient/effective

A

true

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6
Q

give examples of priming in real life:

A

housing prices, politics, advertising

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7
Q

t or f: priming is part of the story telling brain

A

true

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8
Q

are both implicit and explicit memory part of long term memory?

A

yes

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9
Q

why do neurons change shape in the basis of learning and memory?

A

to get more effective (allows them to get more efficient at communicating)

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10
Q

the repetitive loop of actions potential looping through the __________ __________ ___________ is what equals learning

A

hippocampal formation pathway

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11
Q

what is a flashbulb memory?

A

vivid and long-lasting memory of a surprising or shocking event

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12
Q

if something were to go wrong in the ______________ you would have some sort of amnesia

A

hippocampus

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13
Q

what are the two types of memory that sometimes work interchangeably in explicit memory?

A

semantic and episodic

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14
Q

what are AMPA receptors?

A

chemical receptors, connect to Na+ ion channels

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15
Q

t or f: when we recall a memory it is common that we change it

A

true

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16
Q

what does Ach, 5-HT have to to do with memory?

A

improves memory process

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17
Q

process of __________ _________:

  • action potential enters through entorhinal cortex
  • goes through perforant pathway and dentate dyrus
  • then forms synapse, forming another pathway (mossy fiber pathway)
  • goes through CA3 and fornix
  • makes connections in limbic system and cortex around it and synapses with other neurons
  • schaffer collateral pathway formed and axons synapse with neurons in CA1 area
  • exit through entorhinal cortex
A

hippocampal formation

18
Q

what is anterograde amnesia?

A

difficulty making new memories after a traumatic event (to hippocampus)

19
Q

what are neurofibrillary tangles?

A

something has changed within the neurons themselves

20
Q

what does NE/Epi have to do with memory?

A

improves emotional memories (short-term) through the amygdala

21
Q

what happens to nucleotide arrangement during learning or memory?

A

RNA/DNA changes

22
Q

what could H.M. no longer learn?

A

new people, conversations, rules, locations

23
Q

what is episodic memory?

A

personal experiences in your life

24
Q

________ memory uses constant effort

A

explicit

25
Q

person’s ability to observe, analyze and understand visual information in their environment

A

visuospatial learning

26
Q

what are NMDA receptors?

A

chemical receptors blocked by Mg++, voltage receptors activated by long term potentiation can move Mg++, connect to Na+ and Ca++ ion channels

27
Q

t or f: calcium allows learning to occur

A

true

28
Q

what is retrograde amnesia?

A

loss of memory prior to the traumatic event (to hippocampus)

29
Q

t or f: our personal perception of an event changes how we remember it

A

true

30
Q

explain what happened to H.M. and severe epileptic patient

A

he had severe epilepsy and life threatening seizures, had his medial temporal lobe removed and had severe damage to his learning capabilites

31
Q

what are amyloid beta protein?

A

plaques in the brain

32
Q

what is semantic memory?

A

learning factual knowledge

33
Q

what is an emotional memory?

A

a memory of an event that evoked a strong emotional response

34
Q

what do the amygdala and hippocampus have to do with memory?

A

epinephrine and cortisol (from SAM and HPA)
leads to flashbulb and emotional memories

35
Q

____ (NMDA or AMPA) antagonists block memory

A

NMDA

36
Q

what is the hippocampal formation?

A

cortex that is immediately around the hippocampus and some of the limbic system; brings about learning and memory

37
Q

how does cellular modification in memory work?

A

calcium must be present to modify shape

38
Q

what could H.M. still learn?

A

more automatic memories/skills (implicit learning)

what shown fragments of an elephant picture until it created a full elephant then was shown same picture a few days later and knew it was an elephant from the few fragments in the beginning but didn’t know why he knew

39
Q

how are NMDA and AMPA receptors affected by sleep loss?

A

negative effects, decreases synaptic connections and brain plasticity

40
Q

t or f: brain damage to the hippocampal formation will result in troubles with memory and learning

A

true

41
Q

what are parts of the cellular basis of learning and memory?

A

nucleotide rearrangement, long-term potentiation, AMPA and NMDA glutamate receptors, cellular modification