exam 4: learning and memory Flashcards

1
Q

what does memory consolidation through rehearsal lead to?

A

leads to long-term memory

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2
Q

when ___ (RNA or DNA) is blocked, it decreases memory

A

RNA

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3
Q

working memory does not equal ____ _____ memory

A

long term

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4
Q

what are two things seen in alzheimer’s patients?

A

neurofibrillary tangles and amyloid beta proteins

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5
Q

t or f: every disruption in your environment disrupts the loop of the hippocampal formation, making it less efficient/effective

A

true

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6
Q

give examples of priming in real life:

A

housing prices, politics, advertising

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7
Q

t or f: priming is part of the story telling brain

A

true

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8
Q

are both implicit and explicit memory part of long term memory?

A

yes

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9
Q

why do neurons change shape in the basis of learning and memory?

A

to get more effective (allows them to get more efficient at communicating)

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10
Q

the repetitive loop of actions potential looping through the __________ __________ ___________ is what equals learning

A

hippocampal formation pathway

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11
Q

what is a flashbulb memory?

A

vivid and long-lasting memory of a surprising or shocking event

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12
Q

if something were to go wrong in the ______________ you would have some sort of amnesia

A

hippocampus

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13
Q

what are the two types of memory that sometimes work interchangeably in explicit memory?

A

semantic and episodic

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14
Q

what are AMPA receptors?

A

chemical receptors, connect to Na+ ion channels

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15
Q

t or f: when we recall a memory it is common that we change it

A

true

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16
Q

what does Ach, 5-HT have to to do with memory?

A

improves memory process

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17
Q

process of __________ _________:

  • action potential enters through entorhinal cortex
  • goes through perforant pathway and dentate dyrus
  • then forms synapse, forming another pathway (mossy fiber pathway)
  • goes through CA3 and fornix
  • makes connections in limbic system and cortex around it and synapses with other neurons
  • schaffer collateral pathway formed and axons synapse with neurons in CA1 area
  • exit through entorhinal cortex
A

hippocampal formation

18
Q

what is anterograde amnesia?

A

difficulty making new memories after a traumatic event (to hippocampus)

19
Q

what are neurofibrillary tangles?

A

something has changed within the neurons themselves

20
Q

what does NE/Epi have to do with memory?

A

improves emotional memories (short-term) through the amygdala

21
Q

what happens to nucleotide arrangement during learning or memory?

A

RNA/DNA changes

22
Q

what could H.M. no longer learn?

A

new people, conversations, rules, locations

23
Q

what is episodic memory?

A

personal experiences in your life

24
Q

________ memory uses constant effort

25
person's ability to observe, analyze and understand visual information in their environment
visuospatial learning
26
what are NMDA receptors?
chemical receptors blocked by Mg++, voltage receptors activated by long term potentiation can move Mg++, connect to Na+ and Ca++ ion channels
27
t or f: calcium allows learning to occur
true
28
what is retrograde amnesia?
loss of memory prior to the traumatic event (to hippocampus)
29
t or f: our personal perception of an event changes how we remember it
true
30
explain what happened to H.M. and severe epileptic patient
he had severe epilepsy and life threatening seizures, had his medial temporal lobe removed and had severe damage to his learning capabilites
31
what are amyloid beta protein?
plaques in the brain
32
what is semantic memory?
learning factual knowledge
33
what is an emotional memory?
a memory of an event that evoked a strong emotional response
34
what do the amygdala and hippocampus have to do with memory?
epinephrine and cortisol (from SAM and HPA) leads to flashbulb and emotional memories
35
____ (NMDA or AMPA) antagonists block memory
NMDA
36
what is the hippocampal formation?
cortex that is immediately around the hippocampus and some of the limbic system; brings about learning and memory
37
how does cellular modification in memory work?
calcium must be present to modify shape
38
what could H.M. still learn?
more automatic memories/skills (implicit learning) what shown fragments of an elephant picture until it created a full elephant then was shown same picture a few days later and knew it was an elephant from the few fragments in the beginning but didn't know why he knew
39
how are NMDA and AMPA receptors affected by sleep loss?
negative effects, decreases synaptic connections and brain plasticity
40
t or f: brain damage to the hippocampal formation will result in troubles with memory and learning
true
41
what are parts of the cellular basis of learning and memory?
nucleotide rearrangement, long-term potentiation, AMPA and NMDA glutamate receptors, cellular modification