Equine Dysrythmias Flashcards

1
Q

How do myocardial disease and endocardial disease differ in how they manifest?

A
  • Endocardial disease
    • Valvular regurgitation, Jet lesions
    • MANIFEST AS CARDIAC MURMURS
  • Myocardial disease
    • Disruption to action potential propagation, Abnormalities in contraction
    • MANIFEST AS CARDIAC DYSRHYTHMIAS
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2
Q

How does myocardial disease manifest?

A
  • No clinical signs
  • Poor performance (AF, VPD’s)
  • Collapse (Multiple VPD’s and VT)
  • V rarely death (VT to VF)
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3
Q

How are Cardiac Dysrrhythmias diagnosed?

A
  • Diagnosis
    • ECG
  • Evaluation of underlying cause
    • Blood tests, echocardiogram, other
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4
Q

What is Myocarditis?

A

Inflammation of the myocardium

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5
Q

Causes of myocarditis?

A
  • Bacterial
    • Staph aureus
    • Strept equi
    • Clostridium chauvoei
    • Mycobacterium spp
    • Secondary to sepsis, pericarditis, endocarditis
  • Borrelia burgdorferi – (Lyme disease)
  • Viral
    • FMD, EIA, EVA, EIA, AHS
  • Parasitic
    • Lge strongyles, Toxoplasma, Sarcocystis
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6
Q

Techniques used for myocardial evaluation?

A
  • Echocardiography
  • Dobutamine-atropine stress echocardiography
    • Can evaluate the heart at increasing heart rates
  • Myocardial biopsies
    • Ultrasound guided
  • ECG
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7
Q

Which is the most important cardiac dysrhythmia in the horse?

A
  • Atrial fibrillation
  • Lack of coordinated atrial electrical activity
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8
Q

What can trigger atrial fibrillation?

A

Can be triggered by electrolyte/acid-base imbalances, anaesthetic and drug administration (that causes bradycardia), exercise

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9
Q

Clinical signs of atrial fibrillation?

A
  • None
  • Exercise intolerance/poor performance
  • Epistaxis
  • Weakness/syncope
  • Myopathy
  • Colic
  • CHF
  • Cattle - primary GI disease (also foot rot and pneumonia). Also see anorexia and decreased milk production
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10
Q

Diagnosis of atrial fibrillation?

A
  • Auscultation and physical examination
  • Resting ECG
    • No p wave
    • Normal qrs complex and t waves
    • f (fibrillation) waves
  • Further diagnostics PRIOR to attempting treatment
    • Assessment of electrolytes and acid-base abnormalities
    • Echocardiography to assess for underlying cause/chamber enlargement
    • Exercising ECG
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11
Q

What is paroxysmal atrial fibrillation?

A
  • lasts less than 24-48 hrs and spontaneously convert
  • Sometimes associated with K+ depletion (Furosemide) and administration of bicarbonate
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12
Q

Treatment of atrial fibrillation?

A
  • Pharmacological: Quinidine sulphate (Quinidine gluconate) (Negative inotrope)
  • Monitor toxicity by assessing prolongation of QRS complex
  • DC Cardioversion
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13
Q

Side effects of Quinidine sulphate (Quinidine gluconate)?

A
  • Fatal dysrhythmias
  • Colitis – drug very irritant to mucosa
  • Laminitis /nasal oedema/ataxia
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14
Q

Prognosis of paroxsymal atrial fibrillation?

A

Excellent to good – unless keeps recurring

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15
Q

Prognosis of sustained atrial fibrillation with no underlying cardiac disease?

A
  • < 3months – good either technique
  • >3 months – better with DC conversion
  • Risk of re-fibrillation – risk factors there
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16
Q

Prognosis of sustained atrial fibrillation with underlying cardiac disease?

A
  • Average with DC conversion
  • More likely to re-fibrillate than if no underlying cardiac disease
17
Q

Prognosis of sustained atrial fibrillation with heart failure?

A

Poor to grave