Diseases and Conditions of the Avian Respiratory System Flashcards

1
Q

How do birds with acute respiratory disease tend to present?

A
  • Dyspnoea with open mouth breathing
  • Whole body effort in inspiration or expiration
  • Exaggerated ‘tail bobbing’
  • Acute change in vocalisation noise
    • Wheeze, squeak, rasp, gurgle etc
    • Glottis, trachea, syrinx
  • Bottom of the cage and gasping
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2
Q

How may birds with chronic respiratory disease present?

A
  • Sinusitis (often with conjunctivitis)
    • Soft swellings
    • Hard ‘abscesses’
  • Chronic airsaculitis
  • Rhinitis – (nasal discharge)
  • Oculanasal discharge
  • Stained/matted feathers around the nares
  • Sneezing
  • Dyspnoea – (+/- above and weight loss)
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3
Q

How should normal respiration in a bird appear?

A
  • Should not be noticeable
  • The mouth should remain closed
  • An increase in abdominal effort or head movement may be recognised in association with increased respiration following exercise but this should return to normal within minutes of ceasing activity
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4
Q

Why should you observe an avian patient from a distance first?

A
  • Subtle changes in posture, wing position
  • Respiratory rate and respiratory pattern that may indicate an abnormality (tail-bobbing)
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5
Q

Signs of upper respiratory tract disease in birds?

A
  • Change in voice
  • Sneezing
  • Periorbital swellings - Sinus swelling
  • Rhinorrhoea
  • Plugged nares - Nasal granuloma
  • Exercise intolerance
  • Head-shaking
  • Mucopurulent nasal discharge
  • Inflamed swollen cere
  • Stretching the neck - Yawning
  • Epiphora
  • Open-mouthed breathing – Dyspnoea – but not stressful
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6
Q

Signs of lower respiratory tract disease in birds?

A
  • Dyspnoea – with distress
  • Open-mouthed breathing
  • Tail-bobbing
  • Loss of voice
  • Change in vocalization
  • Laboured respiration
  • Exercise intolerance
  • Coughing
  • Sounds on auscultation
  • Depression
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7
Q

What are the clinical signs of rhinitis?

A
  • Unilateral or bilateral
  • Occluded nares
  • Rhinorrhoea – inflammatory exudate
    • Mucous
    • Purulent
  • Sneezing +/- discharge
  • A growth or change in size of the nasal opening
    • Chronic inflammation - Rhinolith
    • Knemidocoptes
  • Crusted soiled feathers around the nares
  • Grooves in the maxilla
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8
Q

How would you diagnose rhinitis?

A
  • Cytology of nasal discharge (flush)
  • Culture and sensitivity of rostral choana
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9
Q

What pathogens would you tend to find in swabs from the rostral choana/nares?

A
  • Culture of nares yields mainly contaminates
  • Normal choanal flora + Gm+ve
    • Lacto, Streps, Staph epidermidis, Corynebact
  • Potential pathogens = Gm –ve
    • Staph aureus (inc MRSA), Staph spp, Fungus
  • Special pathogens
    • Chlamydophila, Mycoplasma
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10
Q

How would you treat rhinitis?

A
  • Parental antibiotics (C&S)
  • Nasal flushes and intranasal antibiotics
  • Nebulisation
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11
Q

What are the clinical signs of sinusitis?

A
  • Periocular swelling and reddening
  • Distended sinus may soft, tense or firm
  • +/- nasal discharge
  • Sunken eye – chronic sinusitis in macaws
  • Chronic problems in Amazons and Af Greys
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12
Q

What are the causes of sinusitis?

A
  • Same organisms as with rhinitis
  • Hypovitaminosis A
    • Hyperkeratosis
    • Squamous metaplasia
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13
Q

How would you diagnose sinusitis?

A
  • Appropriate wash
  • Culture and sensitivity
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14
Q

How would you treat sinusitis?

A
  • Isolate any causative organisms and treat accordingly
    • Sinus flush
    • Surgical removal of ‘abscesses’
  • Correct the underlying malnutrition
  • Look at environmental humidity (S Am species)
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15
Q

How is the trachea different in avian species?

A
  • The trachea consists of complete cartilaginous rings in most avian species
  • The tracheal rings can calcify with age
  • The anterior trachea can be wider than the lower trachea (Macaw)
  • The length, configuration and anatomic position of the trachea vary widely
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16
Q

What diseases might affect the glottis, trachea and syrinx?

A
  • Fungal - Aspergillosis
  • Bacterial - Chlamydophila
  • Parasitic
    • Syngamus trachea (gape worms) – See Parasite notes
    • Sternostoma tracheacolum
  • Viral
    • Amazon tracheitis virus
    • Pox
    • Herpes
  • Malnutrition
    • Granuloma
    • Metaplasia
  • Fumes and toxins
  • Acute obstruction
17
Q

What diagnostics would you use for diseases affecting the trachea, glottis and syrinx?

A
  • Tracheal wash
  • Auscultation
  • Endoscopy
  • Transillumination (tracheal mites)
18
Q

How would you treat diseases of the trachea, glottis and syrinx?

A
  • Nebulisation
  • Ivermetin (mites and worms)
  • Fenbendazol (worms)
  • For obstruction – placement of an air sac tube
19
Q

What changes in the air sacs may indicate disease?

A
  • All air sacs are thin-walled and lack vascularity.
  • The air sacs of a normal bird are completely transparent (appear similar to clear plastic wrap)
  • Any alteration in transparency should be considered abnormal (Endoscopy)
  • The presence of blood vessels in the air sacs may be an indication of early inflammation
20
Q

How useful is ascultation in air sac disease?

A
  • The sinuses, trachea, lung, thoracic air sacs and abdominal air sacs can be auscultated using a paediatric stethoscope
  • Audible sounds on inspiration generally correlate with upper respiratory tract disease
  • While sounds on expiration are more commonly associated with lower respiratory tract diseases
21
Q

What diagnostic techniques can be used for disease in the air sacs or lungs?

A
  • Ascultation
  • Endoscopy
    • Laparoscopic – biopsy, surgical removal
    • Tracheal – wash/biopsy
  • Radiography
  • Blood profile
  • Culture and Cytology
    • Sinus wash
    • Choanal swab
    • Tracheal wash
    • Endoscopic sample
22
Q

What are the primary sites of Aspergillosis?

A
  • Skin
  • Beak and sinus
  • Respiratory system
    • Peracute allergic
    • Acute
    • Chronic granuloma formation
23
Q

How would you diagnose Aspergillosis?

A
  • CBC – Marked leucocytosis ( can be 100,000)
  • Radiography
  • Endoscopy
  • Serology?
  • (Post mortem)
24
Q

How would you treat Aspergillosis?

A
  • Antifungals
    • Itraconizole; Amphotericin B
  • Nebulisation
    • Antifungals/F10
  • Supportive treatment
  • Surgical removal
  • Nutrition and immune stimulants
25
Q

Symptoms of mycoplasma?

A
  • Rhinitis, sinusitis, conjunctivitis
  • Bubbles in conjunctiva
  • Mucopurulent sinus swellings
  • Dyspnoea
26
Q

Diagnosis and treatment of mycoplasma?

A
  • Diagnosis
    • Culture not easy as mycoplasma spp unstable
  • Treatment
    • Oral abs
    • Eye ointment
    • Sinus flushing
27
Q

What is the source and presentation of teflon poisoning?

A
  • Presenting signs
    • DEAD birds
    • Acute respiratory dyspnoea
  • Source
    • Overheated pans and some heat lamps
28
Q

How would you treat teflon poisoning?

A
  • Oxygen
  • Bronchial dilators by nebulisation
  • ?Dexamethasone?
29
Q

How would you stabilise the dyspnoeic bird?

A
  • Oxygen therapy
  • Fluids (Intraosseous)
  • For obstructive dyspnoea restoration of a patent airway (air sac tube)
  • For asthmas nebulisation - bronchial dilators – clenbuterol
30
Q

How would you carry out a nasal flush?

A
  • Sterility important
  • Warm saline (39C)
  • 10ml -35ml syringe
  • Wrap bird in towel head pointing down
  • Flush each sinus
  • Collect fluid as it appears from hard palate
  • Centrifuge – cytology and culture
  • Treat with appropriate medication – 2x daily for 10 days +/- . Leave 4 days post treatment re-culture
31
Q

How would you carry out a sinus flush?

A
  • Similar protocol to nasal flush
  • Insert needle into infraorbital sinus between nares and eye
  • Insert small amount of fluid
  • Ensure that the globe of the eye does not bulge!!
  • Slowly insert more fluid and complete wash/treatment
32
Q

When is an air sac tube indicted?

A
  • As an immediate ‘life-saving’ procedure
  • To relieve acute obstructive dyspnoea
  • NOT for restrictive respiration (1st consideration O2/Centisis)
  • To allow tracheal/oral surgery (FB removal)
33
Q

What is the most common cause of subcutaneous emphysema?

A

Due to damage to any air sac system but most common with damage to the cervicocephalic, abdominal or caudal thoracic air sacs