ENT 1 OLDCARTS + Tx + Examples Flashcards

1
Q

Cerumen Impaction

A

O) Accumulation usually asymptomatic; impaction may cause Sx
L) EAC
D) Until it’s removed
C) Hearing loss, ear fullness, earache, itchiness, reflex cough, dizziness, and/or tinnitus
A) None
R) None
T) N/A
S) Not extreme

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2
Q

3 Tx for cerumen impaction

A

1) Cerumenolytic agents
-Patients without h/o ear infections, TM perforation, or otologic surgery
2) Irrigation
-If TM intact
3) Manual

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3
Q

Otitis Externa (aka “Swimmer’s ear”)

A

O) Rapid onset (~within 48 hrs in previous 3 wks)
-Water/swimming, mechanical trauma, allergic contact dermatitis, dermatologic conditions, devices (hearing aids, ear buds, diving caps), prior radiation therapy
L) EAC
D) N/A
C) Inflammation of EAC; Otalgia, pruritis, otorrhea, hearing loss
-May have purulent exudate (discharge), periauricular cellulitis, or TM erythema
A) Tenderness with tragal pressure or auricle manipulation
R) In severe cases, entire auricular region can be inflamed
T) Childhood predominant; more likely to occur in summer; induced by infectious, allergic, & dermatologic disease
S) Mild: minor discomfort, pruritis, minimal canal edema
-Moderate: intermediate pain, pruritis, + partial canal occlusion
-Severe: intense pain, complete canal occlusion; + fever, preauricular erythema, regional lymphadenopathy

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4
Q

Otitis externa Tx

A

1) Main components of treatment: Cleaning EAC (aural toilet)
-Treating inflammation & infection
-Pain control (NSAIDs or acetaminophen)
2) Topical therapy is mainstay
-Mild: Topical acidifying agent + glucocorticoid (i.e., acetic acid + hydrocortisone TID-QID) x 7 days
-Moderate: Topical antibiotic + glucocorticoid (i.e., Cipro HC BID, Cortisporin TID-QID) x 7 days
3) Severe/ immunocompromised cases: Topical antibiotic + glucocorticoid (i.e.,Cipro HC, Cortisporin)
-Some patients: add wick placement & systemic antibiotics (if evidence of deep tissue infection)
–Less severe: PO levofloxacin 500 mg daily x 7d
–More severe: IV vancomycin + IV cefepime
-Obtain cultures of ear drainage

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5
Q

Malignant (Necrotizing) External Otitis
(Invasive infection of EAC & skull base (osteomyelitis)

A

O) Nocturnal otalgia
L) EAC
D) Deep
C) Deep otalgia (nocturnal), persistent & foul otorrhea, EAC granulation
A)
R)
T) Diabetics over age 60, HIV, immunocompromised
S)

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6
Q

Malignant (Necrotizing) External Otitis Tx

A

Based on severity (4 wks-6 months of PO or IV antibiotics)

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7
Q

FBs in EAC

A

O) Sudden
L) More common in right ear (predominant handedness)
D) Not long; until FB is removed
C) Decreased hearing or ear pain, purulent or bloody ear drainage (rare), chronic cough or hiccups (rare)
A)
R)
T) Most common in children aged 6 & younger, esp with:
-Irritating conditions of the ear (i.e., cerumen impaction, otitis externa, otitis media), pica, or ADHD
S)

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8
Q

FBs in EAC Tx

A

1) Referral to otolaryngologist:
-Urgent removal: button batteries, live insects, penetrating FBs
-Removal within a few days: glass or other sharp FB, spherical or other FB wedged in medial EAC, & FB against TM
2) Non-specialists: Irrigation setup, alligator or Bayonet forceps, plastic or metal cerumen curette

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9
Q

AOM
(Acute otitis media: acute bacterial infection of middle ear (aka suppurative otitis media))

A

O)
L) Middle ear (TM bulging, air-fluid levels, perforation, retraction pockets, & cholesteatoma)
D)
C) Young children/infants: fever, fussiness, disturbed or restless sleep, poor feeding/anorexia, vomiting, diarrhea
-Children: otalgia, ear rubbing, hearing loss, ear drainage, + fever
-Adults: otalgia, decreased/muffled hearing, purulent otorrhea (TM rupture)
A) Pneumatic otoscopy: painful in children with AOM
R)
T) Usually precipitated by viral URI or seasonal allergic rhinits (adults); most common in infants & children (ages 6-24 mos)

S)

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10
Q

AOM Tx in kids

A

1) Immediate treatment with antibiotics (at risk kids & <2))
-If NO antibiotics in prior month: amoxicillin
-If antibiotics in prior month: amoxicillin-clavulanate (Augmentin)
-PCN/beta-lactam allergy: If no severe reaction (i.e., rash), oral cephalosporin (i.e., cefdinir), if severe use a macrolide (i.e., clindamycin)
-Duration of therapy: 10 days for < 2 y/o & 5-7 days for > 2 y/o
OR:
2) Observation with initiation of antibiotic therapy if signs & symptoms worsen or fail to improve after 48-72 hours (different criteria depending on reference used)

-Pain control for both: PO ibuprofen or acetaminophen (alternate topical anesthetics), tympanocentesis, or myringotomy

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11
Q

AOM Tx in adults

A

Treat all adults with antibiotics! (and acetaminophen or ibuprofen for pain):
1) Patients with no PCN allergy: Amoxicillin-clavulanate
2) Alternate: cephalosporin (i.e., cefpodoxime, cefdinir)
-Duration: 7-10 days

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12
Q

AOM with TM perforation Tx

A

1) Children: PO antibiotics only
2) Adults: If topical antibiotic added, avoid topicals with ototoxicity (aminoglycosides) & treat for 7-10 days.

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13
Q

Chronic Otitis Media (Recurrent infection of middle ear and/or mastoid in presence of TM perforation)

A

O) Persistent (6-12 weeks) purulent otorrhea with perforated TM despite treatment
L)
D)
C) Hallmark = purulent aural discharge
-Hearing loss, aural fullness, otalgia, & occasionally vertigo
A) None
R)
T)
S)

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14
Q

Chronic Otitis Media Tx (adults)

A

-Removal of infected debris
-Earplugs
-Topical antibiotic drops (ciprofloxacin or ofloxacin) x 2-4 weeks
-Avoid aminoglycosides (ototoxicity)
-Consider PO ciprofloxacin
-Possible surgical reconstruction of TM

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15
Q

Otitis Media with Effusion (OME)/ Serous Otitis Media
(Presence of middle ear effusion without signs of acute infection)

A

O) Often occurs after AOM, but may occur with ETD (in absence of AOM)
L) Multiple air-fluid levels visible through a translucent, slightly retracted, nonerythematous tympanic membrane, impaired TM mobility
D)
C) Conductive hearing loss (predominant)
Other: feeling of ear fullness, tinnitus, balance problems
A)
R)
T) More common in children (pre-school) than adult:
-FHx of otitis media (otitis-prone parents), bottle feeding, male, daycare (or in-person school) attendance, adenoidal hypertrophy, exposure to tobacco smoke, low SE status
S)

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16
Q

OME Tx in kids

A

1) Watchful waiting (most common)
-Most cases spontaneously resolve in 3-6 months
-If not at risk for speech, language, or learning problems and otherwise have normal hearing (also an option for children with mild CHL)
-F/u: clinical eval & hearing tests q3-6 months
2) Myringotomy with T-tube placement (w/wo adenoidectomy)
-If at risk for speech, language, or learning problems
TM changes (retraction pockets), persistent OME-associated hearing loss (threshold > 40 dB), bilateral OME > 3 months, unilateral > 6 months, or recurrent episodes

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17
Q

OME Tx in adults

A

1) Mild Sx: no treatment/reassurance
2) More Sx: intermittent auto-insufflation
3) Moderate Sx due to seasonal allergic rhinitis: short-term(< 12 wks) antihistamines, PO decongestants, and/or nasal steroids
4) Moderate Sx due to URI: short-term (6-10 wks) nasal saline, PO decongestants, and/or nasal steroids (topical decongestant for air travel)

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18
Q

Cholesteatoma (Abnormal accumulation of squamous epithelium within the middle ear & mastoid)

A

O)
L) Congenital: white mass behind intact TM, deep retraction pocket + granulation, TM surface granulation
Acquired: deep retraction pockets, white mass behind TM, granulation at periphery of TM, new onset hearing loss, chronic drainage
D) Continued growth and/or infection if not surgically excised
C) Asymptomatic or chronic ear drainage/ persistent otorrhea or HL
-HL: Progressive, unilateral, conductive
A)
R)
T) History of recurrent AOM and/or chronic OME, 1st T-tubes placed at older age, cleft palate, craniofacial anomalies, turner syndrome, down syndrome, FHx of chronic middle ear disease and/or cholesteatoma
S)

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19
Q

Tx of Cholesteatoma in kids and adults

A

1) Early kids: ventilation & retraction pocket reduction or surgery
-Advanced: surgery
2) Adults: surgery with tympanoplasty + mastoidectomy & + ossicular reconstruction

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20
Q

Eustachian Tube Dysfunction (ETD)
(failure of functional valve of eustachian tube to open or close properly)

A

O)
L) possible retraction of the TM with decreased mobility on pneumatic otoscopy
D)
C) Aural fullness, +/- mild hearing impairment, popping/crackling with swallowing or yawning
A)
R)
T) Viral UI, allergies, etc
S)

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21
Q

(ETD) Eustachian tube dysfunction Tx (for 6 causes)

A

1) Rhinosinusitis: nasal steroids, intranasal/sinus rinses, nasal decongestants (pseudoephedrine, oxymetazoline), antibiotics (bacterial), & pain medications
2) Allergic & non-allergic rhinitis: trigger avoidance, PO & topical antihistamines, topical intranasal steroids, smoking cessation
3) Laryngopharyngeal reflux: lifestyle & dietary modifications, PPIs
4) Mass lesions: surgery for adenoid hypertrophy & nasopharyngeal carcinoma
5) ET insufflation: modified Valsalva maneuver
6) Surgery for failure of medical management: T-tubes, eustachian tuboplasty, balloon dilation of eustachian tube

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22
Q

Acoustic Neuroma/ Vestibular Schwannoma
(Schwann cell-derived tumor that commonly arise from the vestibular portion of the eighth cranial nerve)

A

O)
L) CN8: Unilateral (95%), benign lesion (among the most common intracranial tumors); 5% associated with hereditary syndrome neurofibromatosis type 2 (bilateral tumors)
D) Lasts hours
-gradually grow to involve the cerebellopontine angle, eventually compressing the pons & resulting in hydrocephalus
C) Progressive Unilateral SNHL, deterioration of speech discrimination, continuous disequilibrium
A)
R)
T)
S)

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23
Q

Tx for acoustic neuroma

A

Observation, microsurgical excision, or sterotactic radiotherapy

NOTE: Anyone with unilateral or asymmetric SNHL should be evaluated for intracranial mass lesion!

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24
Q

Peripheral vertigo Sx

A

Vertigo onset is sudden, often with tinnitus & hearing loss, usually horizontal nystagmus

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25
Q

Central vertigo Sx

A

Onset is gradual, not associated with auditory symptoms

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26
Q

Benign Paroxysmal Positioning Vertigo (BPPV)
(Due to small calcified otoliths moving around loose in the inner ear (posterior canal))

A

O) Recurrent episodes of vertigo lasting < 1 minute & provoked by specific types of head movements
L) Inner ear (posterior canal)
D) <1 min episodes that occur in clusters lasting several days; episodes recur periodically for weeks to months without therapy
-Dix-Hallpike maneuver usually provokes paroxysmal vertigo & nystagmus when affected ear turned downward
C) Vertigo (short episodes), + nausea & vomiting
-Typically NO hearing loss & other Sx.
-Nystagmus + vertigo during DH maneuver = usually appear with latency of a few seconds & lasts < 30 seconds, nystagmus in opposite direction upon sitting up
A) Specific types of head movements provoke; DH maneuver vertigo/ nystagmus intensity & duration diminish with repetition to same side
R)
T) Idiopathic or prior head trauma; residual effect of Meniere disease, vestibular neuronitis, ear surgery, herpes zoster oticus, or inner ear ischemia
S) Adaptive

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27
Q

Tx for BPPV

A

Particle repositioning via Epley maneuver

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28
Q

Labyrinthitis (when with unilateral hearing loss), aka vestibular neuritis/neuronitis, neurolabyrinthitis, acute peripheral vestibulopathy

A

O) Rapid onset of continuous vertigo
L)
D)
C) Vertigo with nausea, vomiting, & gait instability
-On exam: spontaneous unilateral, horizontal, or horizontal-torsional nystagmus suppressed with visual fixation
A)
R)
T) Several days to a few weeks
-Possible viral origin
S) Usually severe vertigo

29
Q

Labyrinthitis Tx

A

1) Supportive: vestibular suppressants during acute phase (antiemetics, antihistamines, anticholinergics, benzodiazepines)
2) Glucocorticoids may help during acute period (10-day prednisone taper)
3) Vestibular rehabilitation

30
Q

Define traumatic vertigo

A

Labyrinthine concussion secondary to head injury (peripheral cause), may result in chronic posttraumatic vertigo

31
Q

Describe perilymphatic fistula (rare)

A

Peripheral vertigo, fluctuating hearing loss from injury (barotrauma, valsalva)

32
Q

What triggers cervical vertigo (a peripheral cause)?

A

Neck movement to certain positions

33
Q

Migrainous vertigo

A

O) Episodic
L)
D)
C)
A) Vertigo (peripheral), ; visual & motion sensitivity, auditory sensitivity, photosensitivity
-similar to Meniere’s but NO HL or tinnitus
R)
T) Assoc. with migraines
S)

34
Q

Describe HL in Meniere’s:

A

Initially fluctuating, initially affecting lower frequencies; later in course: progressive, affecting higher frequencies. Sensorineural.

35
Q

Closed EET (External Ear Trauma)

A

O) During amateur wrestling, rugby, boxing, mixed martial arts
L)
D)
C) Blunt trauma may cause hematoma of pinna
A)
R)
T)
S) Untreated may result in necrosis & chronic scarring or further cartilage formation & permanent deformity (“cauliflower ear”)

36
Q

Auricular (pinna) hematoma Tx

A

1) I&D or large-needle aspiration
2) Pressure dressing
3) Anti-staph antibiotics
4) ENT(or plastic surgery) referral

37
Q

External ear laceration Tx

A

Careful eval for cartilage involvement:
1) No cartilage: primary closure with 6-0 suture (or surgical glue), pressure dressing
2) Cartilage: copious irrigation, closure, & PO antibiotics
3) Complex lacerations & avulsions: usually require ENT or plastic surgery consultation.

38
Q

1) Define barotrauma
2) Causes?
3) Sx?

A

1) Injury to middle & inner ear structures due to impact injury/blunt trauma or explosive acoustic trauma (barotrauma)
-TM rupture, hemotympanum, round & oval membrane rupture, ossicular disruption
2) Flying, diving, decompression chambers, & hyperbaric oxygen chambers
3) Pressure, then pain; CHL, SNHL, vertigo, tinnitus

39
Q

Barotrauma Tx

A

1) Most injuries heal/resolve spontaneously with time
2) Emergent surgery for peri lymphatic fistula
3) Tympanoplasty or patching of round or oval window

40
Q

A pt presents with an earache, a sense of ear fullness, itchiness, and tinnitus. What’s your next step? What may they have?

A

Otoscopy; cerumen impaction

41
Q

A pt with cerumen impaction (verified via otoscopy) reports that they had a myringotomy as a child. What should you do next?

A

Refer to a specialist (refer if Hx of surgery, current chronic cerumen impaction, or current perforated TM)

42
Q

You diagnose a pt with otitis externa. You know that this condition is most likely due to what?

A

Acute bacterial infection: Pseudomonas aeruginosa, S. epidermidis, & S. aureus

43
Q

A pt presents with minor discomfort, pruritis, and minimal canal edema upon examination.

1) You diagnose them with what? What stage?
2) How do you treat them?

A

1) Mild otitis externa
2)
Cleaning EAC (aural toilet)
Treating inflammation & infection
Pain control (NSAIDs or acetaminophen)
Topical acidifying agent + glucocorticoid (i.e., acetic acid + hydrocortisone TID-QID) x 7 days

44
Q

A pt presents with intermediate pain, pruritis, and partial canal occlusion

1) You diagnose them with what? What stage?
2) How do you treat them?

A

1) Otitis externa; moderate
2) Cleaning EAC (aural toilet)
Treating inflammation & infection
Pain control (NSAIDs or acetaminophen)
Topical antibiotic + glucocorticoid (i.e., Cipro HC BID, Cortisporin TID-QID) x 7 days

45
Q

A pt presents with intense pain, complete canal occlusion, fever, preauricular erythema, regional lymphadenopathy.

1) You diagnose them with what? What stage?
2) How do you treat them?

A

1) Severe otitis externa
2) Clean EAC, NSAIDs, Topical antibiotic + glucocorticoid (i.e., Cipro HC, Cortisporin)
-Some patients: add wick placement & systemic antibiotics (if evidence of deep tissue infection)
–Less severe: PO levofloxacin 500 mg daily x 7d
–More severe: IV vancomycin + IV cefepime
-Obtain cultures of ear drainage

46
Q

A pt with HIV presents with minor discomfort of the ear, pruritis, minimal canal edema. How do you treat them?

A

The same as you would a severe pt:
-Clean EAC, NSAIDs, Topical antibiotic + glucocorticoid (i.e., Cipro HC, Cortisporin)
-Some patients: add wick placement & systemic antibiotics (if evidence of deep tissue infection)
–Less severe: PO levofloxacin 500 mg daily x 7d
–More severe: IV vancomycin + IV cefepime
-Obtain cultures of ear drainage

47
Q

Malignant external otitis is a potential complication of what common condition?

A

Otitis externa

48
Q

Jimothy, aged 69, with a Dx of diabetes and HIV presents with the complaint of nocturnal deep ear pain and persistent and foul otorrhea.

1) Your first guess should be that he has what condition?
2) What is the most likely cause of this condition you suspect?
3) How do you diagnose him?
4) How do you treat him?

A

1) Malignant (Necrotizing) External Otitis
2) Pseudomonas aeruginosa
3) CT and/or MRI; bone scan
4) 4 wks-6 months of PO or IV antibiotics depending on severity

49
Q

A parent brings their 4 year old in who is complaining of ear pain. The parent notes that they don’t seem to be responding when called as quickly.

What should you first suspect?

A

FB in their EAC

50
Q

Pts with AOM or OME are at risk of developing what?

A

Middle ear effusion

51
Q

A 2 year old, who just got over a viral URI or seasonal allergic rhinits (their parent really isn’t sure since the Sx are similar), presents with fever, fussiness (maybe due to otalgia), disturbed or restless sleep, poor feeding, and vomiting. Upon examination, you note that their TM is bulging, opaque, and with reduced mobility.

1) What do they likely have?
2) Is this more likely to be bacterial or viral?
3) Do they need urgent treatment? Explain.
4) What condition often precedes this child’s condition?

A

1) Acute Otitis Media (AOM)
2) Bacterial
3) No; they are not < 6 months, immunocompromised, toxic-appearing, and are without craniofacial abnormalities (ie, cleft palate), so you can give the pt the option of observation before Tx.
4) Middle ear effusion

52
Q

A pt has had 3 episodes of AOM in the past 4 months. What should you do?

A

Refer to a specialist

53
Q

Recurrent infection of middle ear and mastoid in presence of TM perforation is likely what?

A

Chronic Otitis Media

54
Q

A pt with recurrent purulent aural discharge and HL and aural fullness for the past 7 wks presents to your clinic. Upon examination, you note their TM is perforated.

What is your Dx?

A

Chronic Otitis Media

55
Q

A pt with a cholesteatoma is at risk for what?

A

Chronic Otitis Media

56
Q

A 3 year old with ETD or AOM presents with CHL and balance problems. Upon examination you note their TM is nearly immobile, and their TM is either retracted or neutral. Their parent reports concern of speech delays.

1) What do they likely have?
2) What do you do?

A

1) Otitis Media with Effusion (OME) (aka Serous Otitis Media)
2) Recommend T-tubes

57
Q

An adult with a URI presents with OME and moderate symptoms. What do you recommend for Tx?

A

Short-term (6-10 wks) nasal saline, PO decongestants, and/or nasal steroids (topical decongestant for air travel)

58
Q

A white mass behind TM, with an intact TM, is really only likely with what condition?

A

Congenital cholesteatoma

59
Q

A pt with ETD has a cholesteatoma. You know that this is a _______________ cholesteatoma.

A

Primary

60
Q

A pt complains of chronic ear drainage and new HL. Upon examination you note TM perforation, deep retraction pockets, a white mass behind TM, granulation at periphery of TM.

What do they likely have? What type?

A

Secondary acquired cholesteatoma

61
Q

A pt presents with aural fullness, mild HL, and popping/crackling with swallowing or yawning. Their TM is retracted. They report that their allergies have been bad lately.

This is characteristic of what?

A

Eustachian Tube Dysfunction secondary to allergic rhinitis

62
Q

A pt presents with unilateral HL and complains they can’t understand what people are saying. They report a sense of disequilibrium that is constant.

1) What is your next move?
2) Assuming imaging is consistent w your suspicions, what is your Dx?

A

1) MRI or CT
2) Acoustic Neuroma/ Vestibular Schwannoma

63
Q

A pt presents with vertigo, but they explain that it started extremely suddenly. You breathe a sigh of relief. Why?

A

Central causes of vertigo typically have a gradual onset

64
Q

A pt presents with HL, tinnitus, and sudden vertigo. What should you be thinking they have?

A

A peripheral cause of vertigo

65
Q

A pt presents with a sudden case of vertigo; their episodes last <1 min, and they occur in clusters lasting several days. Sometimes they get nausea with these episodes. They’ve been having this issue for 2 months now with no relief.

You think they have what?

A

Benign Paroxysmal Positioning Vertigo (BPPV)

66
Q

Dix-Hallpike maneuver usually provokes paroxysmal vertigo & nystagmus (for <30 seconds) when affected ear turned downward in what circumstance?

A

If the pt has BPPV

67
Q

A pt has a rapid onset of continuous & usually severe vertigo with nausea, vomiting, & gait instability, and unilateral HL. They tough it out expecting it to get better, but its been 2 weeks with no improvement. What do you suspect?

A

Labyrinthitis

68
Q

Your pt with a TM perforation is excited for their upcoming trip to the Bahamas. What do you need to tell them?

A

Absolutely DO NOT go diving