Pulselessness – causes?
**best initial management of all forms of pulselessness is CPR
Asystole – management
Ventricular Fibrillation – therapy?
(shock, drug, shock, drug, shock, drug, & CPR at all times in between shocks)
When should cardioversion be UNsynchronized?
VF & pulseless VT are only indications for unsynchronized cardioversion.
There is no organized electrical activity to synchronize with.
Hemodynamically stable VT – management?
Amiodarone, then Lidocaine, then Procainamide.
If medications fail, then cardiovert the patient.
Hemodynamically Unstable VT – management?
Electrical cardioversion several times, followed by medications such as amiodarone, lidocaine, or procainamide
How is Hemodynamic Instability defined?
Varying P waves – what does this indicate?
That the impulse may be coming from different sites, such as with a wandering pacemaker rhythm, irritable atrial tissue, or damage near the SA node.
Absent P waves – what does this indicate?
May signify conduction by a route other than the SA node, as with a junctional or atrial fibrillation rhythm
PR interval
– from beginning of P wave to beginning of QRS complex
– 0.12 - 0.20 seconds
Short PR intervals – what does this indicate?
That the impulse originated somewhere other than the SA node, such as with junctional arrhythmias or pre-excitation syndromes
Prolonged PR intervals – what does this indicate?
Conduction delay through the atria or AV junction
– due to digoxin toxicity or heart block
Heart block = slowing related to ischemia or conduction tissue disease
QRS complex
– duration?
0.6 - 0.10 seconds.
Considered abnormal if > 0.12 seconds
Normal ECG Axis
-30 to +90 degrees
so from ~2 O’clock to 6 o’clock
What to consider when you think you see a Premature Ventricular Contraction?
(aka when you see random QRS complexes that’re different from those of the normal rhythm)
Ventricular Escape beats – these will appear as a late QRS complex, rather than premature
Normal beats with aberrant ventricular conduction – these will have a P wave, whereas a PVC does not.
(they’re caused by a supra ventricular impulse taking an abnormal pathway through the ventricular conduction system)
How to recognize Premature Atrial Complexes?
– Narrow smooth complexes
– QRS in early beat looks the same as other QRS complexes
– PR is constant for sinus beats; P wave morphology is different for the premature beat, since it originates from an ectopic focus in the atrium
What is Ventricular Bigeminy & Trigeminy?
V. Bigeminy = PVCs that occur every other beat
V. Trigeminy = PVCs that occur every third beat
How to recognize Multifocal Atrial Tachycardia?
How to recognize Wandering Atrial Pacemaker?
Rhythm = May be irregular Rate = Normal (60-100 bpm) P Wave = Changing shape and size from beat to beat (at least three diffferent forms) PR Interval = Variable QRS = Normal (0.06-0.10 sec)
** T wave is normal. If heart rate exceeds 100 bpm, then rhythm may be Multifocal Atrial Tachycardia (MAP)
What does the differential diagnosis of an irregularly irregular rhythm include?
What arrhythmia is most specific for Digitalis toxicity?
Atrial Tachycardia with AV block
(increases ectopy in atria/ventricles – A. tachycardia. Also increases vagal tone – AV block)
** Atrial Tachycardia is different from Atrial Flutter based on heart rate. AT is slower at 150-250 BPM, wheres A. Flutter is 250-350 BPM.