CVA treatment considerations Flashcards
Stage 1 motor recovery following stroke:
initial flaccidity, no voluntary movement
Stage 2 motor recovery following stroke::
emergence of spasticity, hyperreflexia synergies (mass patterns of movements)
Stage 3 motor recovery following stroke::
voluntary control of movement synergies
spasticity increased and may become severe
Stage 4 motor recovery following stroke::
movement combinations that do not follow the paths of either synergy are mastered
spasticity begins to decline
Stage 5 motor recovery following stroke::
difficult movement combinations are learned
Stage 6 motor recovery following stroke::
disappearance of spasticity, individual joint movements become possible and coordination approaches normal
Expressive aphasia
non fluent aphasia
Broca’s motor aphasia
A central language disorder in which speech is typically awkward, restricted, interrupted and produced with effort
Expressive aphagia results from what?
lesion involving the 3rd frontal convolution of the left hemisphere
Verbal apraxia:
Impairment of volitional articulatory control secondary to a cortical, dominant hemisphere lesion
Dysarthria
Impairment of speech production resulting from damage to the central or peripheral nervous system;
What does dysarthria cause?
causes weakness, paralysis or incoordination of the motor-speech system (respiration, articulation, phonation and movements of jaw and tongue)
Receptive aphasia
fluent aphasia
Wenicke’s aphasia
A central language disorder in which spontaneous speech is preserved and flows smoothly, while auditory comprehension is impaired
What is receptive aphagia a result of?
lesion in the posterior first temporal gyrus of the left hemisphere (Wernicke’s area)
Global aphasia
Severe aphasia
Examine for marked impairments in comprehension and production of language
Pelvis Postural Alignment Deviations:
asymmetrical weight-bearing majority on stronger side
sacral sitting
fear of weight shifting to stronger side
in standing, unilateral retraction and elevation on stronger side
Trunk Postural Alignment Deviations:
with sacral sitting, flattened lumbar curve with exaggerated thoracic curve and forward head
lateral flexion with trunk shortening on more affected side
Shoulders Postural Alignment Deviations:
unequal heights with more affected shoulder depressed
humeral subluxation
winging
Head/Neck Postural Alignment Deviations:
protraction with lateral trunk flexion
lateral flexion of head with rotation away from affected side
Upper Extremities Postural Alignment Deviations:
more affected UE flexed, adducted with IR, flexion, pronation, wrist and finger flexion
stronger UE used for postural support
Lower Extremities Postural Alignment Deviations:
more affected LE hip abduction and ER with hip and knee flexion
unequal weight bearing on feet
Common hip gait deficits:
poor hip position (retracted, flexed, trendelenberg, scissoring, insufficient pelvic rotation)
Weak hip flexors during swing
Circumducted gait
External rotation with adduction
Backward leaning of trunk
Exaggerated flexion synergy
Weak knee extensors
Compensatory locking of knee in hyperextension
May be caused by spastic knee extensors
Ankle
Foot drop
Equinus gait (heel does not touch down)
Varus foot
Equinovarus position
What causes pushing?
lesions in the area of the posterolateral thalamus