Community Acquired Pneumonia Flashcards

1
Q

What is the typical history associated with community-acquired pneumonia (CAP)?

A
  • Acute onset of fever, chills, and productive cough
  • Pleuritic chest pain and shortness of breath
  • History of recent exposure to respiratory infections
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2
Q

What are the key physical examination findings in community-acquired pneumonia (CAP)?

A
  • Fever, tachypnea, and tachycardia
  • Dullness to percussion and decreased breath sounds over the affected area
  • Crackles and bronchial breath sounds on auscultation
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3
Q

What investigations are necessary for diagnosing community-acquired pneumonia (CAP)?

A
  • Chest X-ray showing infiltrates or consolidation
  • Sputum culture and Gram stain
  • Blood tests: CBC, blood cultures, CRP
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4
Q

What are the non-pharmacological management strategies for community-acquired pneumonia (CAP)?

A
  • Adequate hydration and rest
  • Use of a humidifier to ease breathing
  • Elevation of the head of the bed to improve ventilation
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5
Q

What are the pharmacological management options for community-acquired pneumonia (CAP)?

A
  • Empiric antibiotics based on local resistance patterns (e.g., macrolides, fluoroquinolones)
  • Analgesics and antipyretics for fever and pain
  • Oxygen therapy if hypoxemia is present
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6
Q

What are the red flags to look for in community-acquired pneumonia (CAP) patients?

A
  • Severe respiratory distress or cyanosis
  • Hemodynamic instability: hypotension, tachycardia
  • Signs of sepsis or multi-organ dysfunction
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7
Q

When should a patient with community-acquired pneumonia (CAP) be referred to a specialist?

A
  • Refractory symptoms or failure to improve with initial antibiotic therapy
  • Suspected or confirmed drug-resistant pathogens
  • Need for advanced diagnostic evaluation or surgical intervention
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8
Q

What is one key piece of pathophysiology related to community-acquired pneumonia (CAP)?

A
  • Infection of the lung parenchyma
  • Most commonly caused by bacteria (e.g., Streptococcus pneumoniae, Haemophilus influenzae)
  • Leads to alveolar inflammation and consolidation
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