Clinical Flashcards

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1
Q

What are the 3 types of Symptoms of Schizophrenia?

A
  • Positive
  • Negative
  • Cognitive
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2
Q

What is Psychosis?

A

A general term for disorders that involve a loss of contact with reality

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3
Q

What is Schizophrenia marked by?

A

•Periods of remission where the patient has no symptoms •Followed by relapses where their symptoms reoccur

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4
Q

What is Schizophrenia?

A
  • A psychotic disorder

* Where their ability to perceive, process + respond to environmental stimuli is impaired

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5
Q

What are Positive Symptoms in Schizophrenia?

A

Strange additions to normal behaviours

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6
Q

What are Negative Symptoms in Schizophrenia?

A

Loss of normal characteristics

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7
Q

What are Cognitive Symptoms in Schizophrenia?

A

Issues to do with information processing

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8
Q

What are the 4 Types of Positive Symptoms in Schizophrenia?

A
  • Delusions
  • Hallucinations
  • Disorganised thinking/speech
  • Abnormal motor behaviour/disorganised behaviour
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9
Q

What are Delusions as a Type of Positive Symptoms in Schizophrenia?

A

Bizarre beliefs which persist

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10
Q

What are the Different Types of Delusions as a Type of Positive Symptoms in Schizophrenia?

A
  • Delusions of reference
  • Delusions of grandeur
  • Delusions of persecution
  • Thought insertion
  • Thought broadcasting
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11
Q

What are Delusions of Reference as a Type of Delusion as a Type of Positive Symptoms in Schizophrenia?

A

Patient believes that other peoples behaviour is directed specifically at them

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12
Q

What are Delusions of Persecution as a Type of Delusion as a Type of Positive Symptoms in Schizophrenia?

A

Believes that one is being plotted or conspired against

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13
Q

What is Thought Broadcasting as a Type of Delusion as a Type of Positive Symptoms in Schizophrenia?

A

Belief that others can hear their thoughts

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14
Q

What is Thought Insertion as a Type of Delusion as a Type of Positive Symptoms in Schizophrenia?

A

Person believes that their thoughts have been implanted by some kind of external force

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15
Q

What are the Cultural Differences of Delusions as a Type of Positive Symptoms in Schizophrenia?

A
  • Delusions reflect pre-dominant themes + values of a persons culture
  • In industrially advanced countries (America) patients delusions focus on sinister uses of tech - e.g. patients may report that they’re being spied on by their TV
  • In Nigeria, mental illness is believed to be caused by evil spirits, so delusions may be in the form of witches
  • So delusions differ according to the patient’s culture
  • In some African cultures hallucinations are not seen as a sign of mental illness
  • So different cultures may diagnose disorders differently
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16
Q

What are Hallucinations as a Type of Positive Symptoms in Schizophrenia?

A
  • Perception of stimuli not actually present
  • It can be visual, auditory, or olfactory
  • Sometimes they will comment on the individuals character, usually in an insulting manner or they may give a command
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17
Q

What are the Cultural Differences of Hallucinations as a Type of Positive Symptoms in Schizophrenia?

A
  • People from different cultures may interpret the voices that they hear differently
  • Luhmann (2015) - found in some cultures the voices heard were harsh + critical, but in others they were seen as kind
  • So symptoms of schizophrenia may vary from cultures
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18
Q

What is Disorganised Thinking/Speech as a Type of Positive Symptoms in Schizophrenia?

A
  • Underlying with conscious thought that has an effect on a person’s language
  • Patient’s speech is jumbled due to loose associations in thoughts - where 1 idea constantly triggers another, so they jump from topic to topic
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19
Q

What is Abnormal Motor Behaviour/Disorganised Behaviour as a Type of Positive Symptoms in Schizophrenia?

A

Agitated movement - such as repeating movements over and over again

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20
Q

What are the 6 Types of Negative Symptoms in Schizophrenia?

A
  • Lack of energy + movement
  • Social withdrawal
  • Flatness of emotion
  • Not looking after appearance + self
  • Lack of pleasure in everyday things
  • Speaking little
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21
Q

How do the Negative Symptoms in Schizophrenia Link to Psychology as a Science?

A

They are more objective as they may be more directly observable

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22
Q

What are the 3 Types of Cognitive Symptoms in Schizophrenia?

A
  • Difficulties in concentrating + paying attention
  • Problems with working memory
  • Difficulties with executive functioning
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23
Q

What are the 3 Features of Schizophrenia?

A
  • Onset
  • Prevalence
  • Prognosis
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24
Q

What is Onset as a Feature of Schizophrenia?

A
  • Between late teens + mid 30s
  • Peak onset is early to mid 20s in males, and late 20s for females
  • The episodes develop gradually over time + may not be obvious at first
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25
Q

What is Prevalence as a Feature of Schizophrenia?

A
  • Refers to how common a disorder is
  • It is a universal illness
  • The likelihood of developing schizophrenia is between 0.7 and 1%
  • This is influenced by racial/ethnic background, residence + birth country
  • People who experience social problems (poverty + unemployment) are more likely to develop schizophrenia
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26
Q

What is Prognosis as a Feature of Schizophrenia?

A
  • Refers to likely course of the disease
  • 25% of people who had a schizophrenic episode recover
  • 50% of people have recurrent episodes
  • 25% of people experience schizophrenic symptoms continually
  • Life expectancy for person w/ schizophrenia is 10 years less than the average
  • Males show higher proportion of negative symptoms + have longer duration of the disorder - these factors are associated w/ a poor prognosis
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27
Q

How does Onset as a Feature of Schizophrenia Link to Developmental Psychology?

A

Schizophrenia is triggered by some aspect of development during the years of late teens and mid 30s - either biological or social

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28
Q

How does Prevalence as a Feature of Schizophrenia Link to Individual Differences?

A

People who experience social problems such as poverty and unemployment are more likely to develop schizophrenia

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29
Q

How does Prognosis as a Feature of Schizophrenia Link to Individual Differences?

A

Males show more negative symptoms + have a longer duration of the disorder - these factors are associated with poor prognosis

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30
Q

How does Prognosis as a Feature of Schizophrenia Link to Cultural Differences?

A

The WHO international pilot study of schizophrenia suggests - patients w/ this disorder in developing countries have more positive prognosis than do patients in Western industrialised societies

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31
Q

How does Prevalence as a Feature of Schizophrenia Link to Cultural Differences?

A

Prevalence varies according to countries

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32
Q

What is a Neurotransmitter?

A

Chemicals that allow neurons to communicate with one another which allows our brains to think and feel so that our bodies are able to do things

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33
Q

What is Dopamine?

A

Neurotransmitter associated with functions, including motivation and feeling pleasure

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34
Q

What is the Dopamine Hypothesis?

A

Suggest that schizophrenia can be explained by changes of dopamine functioning in the brain

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35
Q

How can Schizophrenia be Explained by High Levels of Dopamine?

A
  • Low levels of beta hydroxylase, so excess dopamine builds up in the synapses
  • Excess dopamine receptors at synapses in the brain
  • Due to hyper sensitivity of certain dopamine receptors
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36
Q

How are Low Levels of Serotonin Related to Schizophrenia?

A

Irregular serotonin activity = negative symptoms of schizophrenia

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37
Q

How are Low Levels of Dopamine Related to Schizophrenia?

A

Low levels of dopamine in mesocortical pathway = negative symptoms

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38
Q

How are High Levels of Dopamine Related to Schizophrenia?

A

High levels of dopamine in mesolimbic pathway = positive symptoms

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39
Q

What are Neurotransmitters (which aren’t serotonin or dopamine) Which have a Influence on Schizophrenia?

A
  • GABA

* Glutamate

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40
Q

What are the Strengths of the Theory of Neurotransmitters as an Explanation for Schizophrenia?

A
  • Explains positive + negative symptoms - increase of dopamine in mesolimbic pathway accounts for positive symptoms, decrease of dopamine in mesocortical pathway accounts for negative symptoms
  • Backed up by evidence from drug treatment - antipsychotic medications for work by blocking dopamine receptors
  • Testable evidence that excess dopamine plays a role in schizophrenia - people given Levodopa experience schizophrenic-like symptoms (e.g. hallucinations)
  • PET scans in investigating dopamine levels in patients provide an objective measure of neurotransmitter functioning in areas of the brain
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41
Q

What are the Applications of the Theory of Neurotransmitters as an Explanation for Schizophrenia?

A

Many antipsychotic medications used to treat schizophrenia work by blocking dopamine receptors

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42
Q

What are the Weaknesses of the Theory of Neurotransmitters as an Explanation for Schizophrenia?

A
  • It’s reductionist - Ignores other factors that may influence whether someone develops schizophrenia - biological, psychological + social factors
  • Cannot prove that excess dopamine causes schizophrenia, instead it may be a symptom of schizophrenia
  • Not all patients with schizophrenia respond to the drugs - Friedhoff (1980) found that some patients show no improvement after taking dopamine antagonists
  • PET scans show that blocking dopamine receptors doesn’t always remove the symptoms
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43
Q

What did Brown and Birley (1968) Find which Suggests the Theory of Neurotransmitters as an Explanation for Schizophrenia
is Reductionist?

A
  • Found that 50% of schizophrenic patients reported a major life event in the 3 weeks prior to the relapse
  • Suggesting that social conditions may trigger relapse
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44
Q

What did Owen (1978) Show which Supports the Theory of Neurotransmitters as an Explanation for Schizophrenia?

A
  • Post-mortem examinations of the brains of people with schizophrenia
  • Showed that they had a higher density of dopamine receptors in the cerebral cortex
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45
Q

What does it mean if Schizophrenia is Genetic?

A
  • People who are genetically related to schizophrenics should be more likely to have schizophrenia themselves
  • The chances of them having schizophrenia is higher
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46
Q

What are the 3 Types of Research that have Investigated the Role of Genetics in Schizophrenia?

A
  • Family studies
  • Adoption studies
  • Twin studies
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47
Q

What are Family Studies as a Type of Research that has Investigated the Role of Genetics in Schizophrenia?

A
  • Investigate whether close biological relatives also have schizophrenia
  • Problematic as the closer the relative the more likely they are to share the same environment
  • So it’s difficult to separate out the effects of nature with nurture
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48
Q

What are Adoption Studies as a Type of Research that has Investigated the Role of Genetics in Schizophrenia?

A
  • Useful as a genetic factor can be looked for in adopted children who have been apart from their biological parents
  • Allows effects of nature to be separated from nurture which controls extraneous variable of environment
  • The adopted children whose biological parents are schizophrenic have a higher risk of developing schizophrenia
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49
Q

What are Twin Studies as a Type of Research that has Investigated the Role of Genetics in Schizophrenia?

A
  • Schizophrenia in MZ and DZ twins can be compared
  • If a twin has schizophrenia then the concordance rate between MZ twins should be 100% whereas it should be lower in DZ twins as they only share 50% of their genetic material
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50
Q

What are Dizygotic (DZ) Twins?

A
  • Non-identical

* Share 50% of their genes

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51
Q

What are Monozygotic (MZ) Twins?

A
  • Genetically identical

* Share 100% of their genes

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52
Q

What were the Results from Gottesman and Shield’s (1966) Study which related to Twin Studies?

A
  • Concordance rates for schizophrenia were higher in females compared to males for MZ and DZ twins
  • Concordance rates higher for MZ+DZ twins for severe schizophrenia compared to mild
  • Concordance rates for severe schizophrenia was much higher in MZ twins (75%) compared to DZ twins (22%)
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53
Q

What were the Conclusions from Gottesman and Shield’s (1966) Study which related to Twin Studies?

A
  • Schizophrenia does have a biological basis, developing it is influenced by a persons gene
  • However, not totally caused by genes as concordance rate for MZ weren’t 100% •Therefore genes are a risk factor for developing it but environmental triggers are necessary
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54
Q

What did Gottesman and Shield’s (1966) Investigate which related to Twin Studies?

A

Whether schizophrenia has a genetic basis for looking at concordance rates for schizophrenia and MZ and DZ twins

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55
Q

What are the Strengths of Genes as an Explanation for Schizophrenia?

A
  • Evidence from Twin studies suggest that there is a significant genetic factor in schizophrenia (e.g. Gottesman + Sheild)
  • Biological approach to explaining schizophrenia is associated with the medical profession + has scientific status + credibility
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56
Q

What are the Weaknesses of Genes as an Explanation for Schizophrenia?

A
  • Research methods used to establish genetic links e.g. twin studies have flawed methodologies - as don’t take account that genes may function differently in different environments
  • Reductionist as environmental factors may be involved in the development of schizophrenia - e.g. concordance rate in MZ twins for Schizophrenia is only around 40-50%, so if the illness was purely down to genetics, we’d expect the rate to be 100%
  • Confusion as to which genes are responsible for predisposing a person to schizophrenia as there could be many, Wright (2014) suggested that 700 genes have been linked to schizophrenia
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57
Q

What are the Individual differences in the Genetic Explanation of Schizophrenia?

A
  • Symptoms of schizophrenia are very diverse and not the same for everyone
  • Biological view is unlikely to be a complete one as there are likely to be different factors associated with developing the disorder that may account for the various subtypes and presentation of symptoms
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58
Q

What is the Issue and Debate of Nature-Nurture in the Genetic Explanation of Schizophrenia?

A
  • Diathesis-stress model argue that schizophrenia develops in those who have a biological predisposition to developing illness due to genetic’s (neurochemical or neuroanatomical factors)
  • But who also have some sort of environmental trigger - suggest the cause is a combination of factors
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59
Q

What does the Diathesis-Stress Model of Schizophrenia Suggest about the Genetic Explanation?

A

A genetic disposition which becomes apparent when the individual becomes stressed by factors in their environment

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60
Q

How is Developmental Psychology Linked to the Genetic Explanation of Schizophrenia?

A
  • Schizophrenia can be explained through the process of development
  • Thomas (2010) suggests schizophrenia stems from abnormalities in early brain development
  • Prenatal exposure to infection or lack of nutrition have been linked with schizophrenia (Opler and Susser (2005))
  • Schizophrenia develops in late adolescence and early adulthood
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61
Q

What are the Similarities between the Neurotransmitter Explanation for Schizophrenia and the Genetic Explanation for Schizophrenia?

A
  • Both reductionist
  • Both are biological explanations in terms of the medical model
  • Both have research evidence to support their theories
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62
Q

What is the Cognitive Explanation of Schizophrenia?

A
  • Explain human behaviour in terms of information processing
  • So, explains the symptoms of schizophrenia in terms of the patients thought processes
  • Cognitive impairments play an important role in the development and maintenance of schizophrenia
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63
Q

What are the 2 Cognitive Factors which may Explain Schizophrenia?

A
  • Problems with attention

* Problems with memory

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64
Q

What are the Problems with Attention as a Cognitive Factor which may Explain Schizophrenia?

A
  • People with schizophrenia have attentional processes which work in a different way
  • They don’t have normal functioning of these filtering processes, leading them to pay too much attention to irrelevant stimuli
  • Preventing them from making sense of the world in the way that most people can
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65
Q

What does Frith (1979) Suggest which Supports the Problems with Attention as a Cognitive Factor which may Explain Schizophrenia?

A
  • Inability to filter out unnecessary cognitive noise created by internal information processing
  • Patient is unable to self monitor effectively, resulting in hallucinations or delusions as they don’t realise that their thoughts are self generated
  • Patient experiences their own internal thought as an external voice
  • Which explains the symptoms of hallucinations and delusions
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66
Q

What does Hemsley (1993) Suggest which Supports the Problems with Memory as a Cognitive Factor which may Explain Schizophrenia?

A
  • People with schizophrenia aren’t able to activate relevant schemas
  • They experience sensory overload, so they are unable to predict what will happen next, their concentration is poor + they attend to irrelevant aspects of the environment
  • Their poor integration of memory + perception leads to disorganised thinking and behaviour
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67
Q

What are the Problems with Memory as a Cognitive Factor which may Explain Schizophrenia?

A
  • There is a substantial breakdown in the relationship between memory and perception in schizophrenics
  • Which is linked to schemas, as relevant schemas are not triggered in people w/ schizophrenia
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68
Q

What is the Research by McGuigan (1966) which Supports the Cognitive Explanation for Schizophrenia?

A
  • Found that the larynx of patients with schizophrenia was active during the time they were experiencing auditory hallucinations
  • Suggesting that they mistook their own inner speech for that of someone else
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69
Q

What is the Research by McGuire (1996) which Supports the Cognitive Explanation for Schizophrenia?

A

Found schizophrenics to have reduced activity in the parts of the brain involved in monitoring inner speech

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70
Q

What is the Research from PET Scans which Supports the Cognitive Explanation for Schizophrenia?

A
  • Show under activity in the frontal lobe of the brain of people suffering from schizophrenia
  • Which is linked to self monitoring
  • Providing biological support for this explanation
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71
Q

What are the Strengths of the Cognitive Explanation for Schizophrenia?

A
  • It explains some of the key problems associated with schizophrenia, e.g. it explains the symptom of delusions + hallucinations + disorganised behaviour
  • It is supported by research evidence
  • Useful in developing effective treatments which have helped many people with mental disorders by alleviating the symptoms and improving their lives - e.g. Morrison (2014) found that cognitive therapy reduced psychiatric symptoms + is safe for people with schizophrenia
  • It is less reductionist as it includes the idea that delusions are triggered by strange sensory experiences that arise from biological problems, suggesting a complex interaction between biological and cognitive causes of schizophrenia
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72
Q

What are the Weaknesses of the Cognitive Explanation for Schizophrenia?

A
  • Difficult to find out whether the cognitive problems are a cause of their disorder or an affect, As they may play a role in causing schizophrenia or schizophrenia itself may cause the cognitive problems
  • It is reductionist as it underestimates genetic factors, stressful life events and social factors in the development of schizophrenia
  • Many brain-damaged patients have problems with attention or the relationship between memory and perception, but they failed to develop the symptoms of schizophrenia, which challenges the cognitive explanations
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73
Q

What are the Similarities between the Genetic Explanation and the Cognitive Explanation for Schizophrenia?

A
  • Both supported by research evidence
  • Both look at human body as part of the explanation, as 1 looks at memory and the other looks at genes
  • Both reductionist as they underestimate the other factors into schizophrenia - such as social and environmental
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74
Q

What are the Differences between the Genetic Explanation and the Cognitive Explanation for Schizophrenia?

A
  • Cognitive explanation can be more useful as it leads directly to treatment
  • Cognitive looks at memory + attention as part of the explanation, whereas genetic looks at the biological inherited genes as part of the explanation
  • Genetic can show cause + effect link between genes + schizophrenia, whereas the cognitive symptoms shown may be a symptom of schizophrenia rather than a cause
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75
Q

What is the Health and Care Professions Council (HCPC)?

A
  • Clinical practitioners must register with this

* It sets standards which practitioners must meet in order to remain registered with the HCPC

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76
Q

What are the 7 Standards Set Out by the HCPC for Clinical Practitioners?

A
  • Character
  • Health
  • Standards of proficiency
  • Standards of conduct, performance + ethics
  • Standard for continuing personal development
  • Standards of education and training
  • Standards for prescribing
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77
Q

What is Character as a Standard Set Out by the HCPC for Clinical Practitioners?

A
  • Registrants provide credible character references from people who have known them for at least three years
  • Considering any criminal convictions and their character traits
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78
Q

What is Health as a Standard Set Out by the HCPC for Clinical Practitioners?

A
  • Provide info every two years when they re-register about their general health
  • Provide info on any health issues that they have only if it’s likely to affect their ability to practice safely
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79
Q

What are the Standards of Profficiency as a Standard Set Out by the HCPC for Clinical Practitioners?

A
  • There are a set of specific expectations for practitioners psychologists
  • E.g. professional autonomy + accountability, delivery of strategies for meeting health + social care needs
  • Specific requirements within each standard to be demonstrated by practitioners in different areas of psychology
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80
Q

What are Standards of Conduct, Performance and Ethics as a Standard Set Out by the HCPC for Clinical Practitioners?

A
  • List of 14 guidelines that practitioners must adhere to in their clinical practice
  • Including points such as maintaining confidentiality in work with service users, referring on to others when necessary, and only acting within the limits of their own knowledge
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81
Q

What are Standards for Continuing Personal Development as a Standard Set Out by the HCPC for Clinical Practitioners?

A
  • Professionals expected to take part in + document regular training that they undertake to develop their own practice
  • Including training events, evidence on how they’ve changed their practice, and an evaluation of the effectiveness of these changes
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82
Q

What are Standards of Education + Training as a Standard Set Out by the HCPC for Clinical Practitioners?

A

•Set of minimum levels of qualification specified before people can register to practice in
different areas of health + care professions
•Registrants must be able to evidence at least a master degree with bps qualification in the area of practice they will be working in
•HCPC sets out standards for training courses

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83
Q

What are Standards for Prescribing as a Standard Set Out by the HCPC for Clinical Practitioners?

A
  • These standards set out safe practice for prescribing medication by health and care professionals
  • Including the required knowledge and training to be able to prescribe within professional practice
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84
Q

What are the 2 Treatments which can Help People with Schizophrenia?

A
  • Drug treatment

* Cognitive treatment

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85
Q

What is Drug Treatment for Schizophrenia Based on?

A
  • The medical model of mental disorder
  • Where mental disorders are seen as an illness + assumed they have biological causes
  • Treatments are physical in nature
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86
Q

What are the Drugs used in Treatment for Schizophrenia Known as?

A

Anti-psychotic drugs

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87
Q

How does the Dopamine Hypothesis Explains how Drug Treatments for Schizophrenia Work?

A

Schizophrenia results from abnormally high levels of dopamine

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88
Q

How did the 1st Anti-Psychotic Drug (Phenothiazine) Explain how Drug Treatments for Schizophrenia Work?

A
  • Blocking receptors for dopamine
  • So, effects of dopamine aren’t picked up by the Brain
  • However, there were side effects - shaking, muscle tremors, jerky movement
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89
Q

What are the New Drug Treatments for Schizophrenia and How do they Work?

A
  • Clozapine - acts by blocking serotonin receptors

* Olanzapine + risperidone - have fewer side effects + more effective - known as ‘atypical’ drugs

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90
Q

What are the Strengths of Drug Treatment for Schizophrenia?

A
  • Effective in reducing symptoms + more effective than any other form of therapy as reduces symptoms quickly
  • Useful w/ assistance of antipsychotic drugs
  • Allowed patients to live relatively normal lives - prior to phenothiazine, schizophrenia was considered untreatable
  • Appropriate as it has more of genetic basis - main drugs used to treat schizophrenia block dopamine receptors, so appropriate to use as they target parts of physiological system that function abnormally in Schizophrenia
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91
Q

What did Meltzer (2004) Find which Supports that Drug Treatment is Effective in Reducing Symptoms of Schizophrenia?

A

Haloperidol gave significant improvements in all areas of functioning compared to placebo

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92
Q

What are the Weaknesses of Drug Treatment for Schizophrenia?

A
  • It’s reductionist - ignores psychological + social factors
  • Effectiveness + usefulness of drug therapy is limited - it is palliative - it suppresses the symptoms of the disorder without addressing the underlying causal processes - patients have to take drugs for many years
  • Unethical - drugs often produce side effects + some patients unable to give consent + may lead to addiction + dependency
  • Relapse is a problem - drugs don’t offer a long term cure as they don’t address the cause of the problem
  • Some patients are resistant to drugs
  • Most common drugs aren’t effective in treating the negative symptoms of schizophrenia
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93
Q

How is Drug Treatment for Schizophrenia an Issue of Social Control?

A
  • Drug treatment can be looked down on in society as it’s considered that you need to alter their behaviours to become more ‘normal’ - social norms in society
  • Cannot assume everyone has same symptoms + severity of symptoms due to labelling them ‘schizophrenic’
  • Pharmaceutical drug company’s want money
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94
Q

Why is Drug Treatment the Most Common Biological Treatment for Schizophrenia?

A
  • Dopamine hypothesis is widely accepted explanation for schizophrenia
  • Suggests that schizophrenia may result from high levels of dopamine
  • So, it follows the most common form of biological treatment for schizophrenia is drug therapy
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95
Q

What does the Cognitive Model of Abnormality Assume?

A
  • Mental disorder is created by errors in thinking

* Thoughts influence emotions + feelings

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96
Q

What do Cognitive Behavioural Therapies Attempt to do?

A

Attempt to change maladaptive behaviour by changing the way people think

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97
Q

What does the Cognitive Behavioural Therapy Assume?

A
  • Patients have irrational thoughts + beliefs about themselves + the world
  • Thoughts + beliefs are negative, self-defeating + contribute to development of mental disorders
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98
Q

How do Therapists using Cognitive Behavioural Therapy Try to Change the Patients Behaviour?

A
  • Focusing on the present symptoms
  • Looking at how the person thinks about how an event affected them - what they felt + how they behaved
  • Challenge negative thoughts + change them for more realistic + positive thoughts
  • Person will feel better + behaviour will change
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99
Q

What is the Aim of Cognitive Behavioural Therapy when Treating Schizophrenia?

A

Reduce the stress felt by the patient with schizophrenia + allow them to help them mange + understand their symptoms

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100
Q

What are the 3 Techniques Used in Cognitive Behavioural Therapy for Schizophrenia?

A
  • Belief modification
  • Focusing + reattribution
  • Normalising the experiences of the person w/ schizophrenia
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101
Q

What is Belief Modification as a Technique Used in Cognitive Behavioural Therapy for Schizophrenia?

A

Delusional thinking is challenged

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102
Q

What is Focusing + Reattribution as a Technique Used in Cognitive Behavioural Therapy for Schizophrenia?

A
  • To help w/ hallucinations (auditory)

* Therapist aims to show that the voices are self-generated + don’t need to be feared

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103
Q

What is Normalising the Experiences of the Person with Schizophrenia as a Technique Used in Cognitive Behavioural Therapy for Schizophrenia?

A

Psychotic symptoms are looked at as more normal to reduce the fear related to them

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104
Q

What are the Strengths of Cognitive Behavioural Therapy as a Treatment for Schizophrenia?

A
  • Useful as effective in treating - patients show increase in coping skills + decreased hallucinations + delusions
  • Most ethical - empowers patients by educating them in self-help strategies + they have more control over the process - allows patients to be more independent
  • Strategies learned cab be incorporated into a patients life - so it’s a useful treatment
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105
Q

What are the Weaknesses of Cognitive Behavioural Therapy as a Treatment for Schizophrenia?

A
  • Reductionist - doesn’t address the underlying cause of the mental disorder as it overlooks biological causes
  • Not effective - patient suffering severe schizophrenia may lack problem solving skills + don’t have an insight into their condition
  • Effectiveness is hard to judge - control treatments are sometimes given by non-experts, so the use of inadequate control treatment conditions may explain some of the findings
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106
Q

What did Sensky (2000) Find which Supports that Cognitive Behavioural Treatment is Effective in Treating Schizophrenia?

A
  • He compared CBT w/ non-specific ‘befriending interventions’ for patients w/ schizophrenia
  • Found that CBT was more effective in reducing both positive + negative symptoms of schizophrenia
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107
Q

How does Cognitive Behavioural Therapy Link to the Issue of Social Control?

A
  • CBT can be seen as an agent of social control, so patients thoughts may be changed to fit into social norms
  • Superficial way of trying to get patients to act normally without actually addressing the problem
  • But, by alleviating the symptoms, it makes the patient much more independent
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108
Q

What are 2 Similarities Between Cognitive Behavioural Therapy and Drug Treatment as Treatments for Schizophrenia?

A
  • Both supported by research evidence - e.g. Sensky, Meitzar
  • Both are useful as they allow people to live independent lives
  • Both ignore social factors
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109
Q

What are 2 Differences Between Cognitive Behavioural Therapy and Drug Treatment as Treatments for Schizophrenia?

A
  • Drug Therapy is more reductionist than CBT, as it focuses purely on biological treatment , where CBT looks at cognitive + behavioural
  • CBT can be considered more ethical than drug therapy
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110
Q

What is a Review Article?

A

Summarises previously published studies

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111
Q

Why are Review Articles Useful?

A
  • Looks at recent major advances + discoveries
  • Looks at significant gaps in research
  • Looks at current debates
  • Looks at ideas of where research might go next
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112
Q

What are the Strengths of Review Articles?

A
  • Generate new ideas about where research should go next
  • Can close significant gaps in research
  • Draws together wide body of data
  • Includes recent major developments
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113
Q

What are the Weaknesses of Review Articles?

A

Has practical problems - validity and reliability may be interpretative if original studies had flaws

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114
Q

What is the Aim of the Contemporary Study for Schizophrenia by Carlsson (2000)?

A

Review research to investigate:
•Use info on psychosis + neurotransmitter functioning to produce new antipsychotic drugs that are more effective w/ fewer side-effects
•Show neurotransmitter functioning in specific brain areas
•Investigate neurotransmitter functioning + psychosis, beyond the dopamine hypothesis

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115
Q

What is the Procedure of the Contemporary Study for Schizophrenia by Carlsson (2000)?

A
  • Literature review
  • Methods + findings of studies to do w/ neurotransmitter interactions in schizophrenia are analysed
  • Including studies using rodents to test neurotransmitter functioning + related brain structure functioning, studies on people w/ acute schizophrenia + people with schizophrenia in remission, studies which used brain scans to investigate psychosis
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116
Q

What were the Key Points about Dopamine from the Literature Review of the Contemporary Study for Schizophrenia by Carlsson (2000)?

A
  • PET scans provide evidence to show high levels of dopamine related to psychosis - e.g. Abi-Dargham (1998) + Breier (1997)
  • Supporting the dopamine hypothesis
  • However interactions with other neurotransmitters (e.g. noradrenaline, serotonin, acetylcholine, glutamate, + GABA) may be related w/ schizophrenia
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117
Q

What were the Key Points about Glutamate from the Literature Review of the Contemporary Study for Schizophrenia by Carlsson (2000)?

A
  • Low levels of glutamate play a role in schizophrenia
  • Lodge (1989) - shows that PCP can induce psychosis by inhibiting the action of glutamate in the brain
  • Glutamate failure in cerebral cortex may lead to negative symptoms of schizophrenia, •Glutaminergic in the basal ganglia could be responsible for positive symptoms
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118
Q

What were the Key Points about Dopamine and Glutamate from the Literature Review of the Contemporary Study for Schizophrenia by Carlsson (2000)?

A
  • Some research has found an interaction between dopamine and glutamate
  • Reduced levels of glutamate is associated with increased dopamine release
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119
Q

What were the Key Points about Serotonin from the Literature Review of the Contemporary Study for Schizophrenia by Carlsson (2000)?

A
  • Serotonin may be implicated in schizophrenia
  • Serotonin levels are related to dopamine
  • Clozapine works by reducing dopamine and serotonin levels in the brain
  • NMDA antagonists, which limit glutamate, seem to stimulate serotonin levels
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120
Q

What were the 2 Main Theories About the Causes of Schizophrenia from the Contemporary Study for Schizophrenia by Carlsson (2000)?

A
  • Whether high levels of dopamine (hyperdopaminergia) cause schizophrenia
  • Whether low levels of glutamate (hypoglutamatergia) cause schizophrenia
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121
Q

What was the Conclusions from the Contemporary Study for Schizophrenia by Carlsson (2000)?

A
  • Glutamate deficiency may explain increased dopamine responsiveness
  • Glutamate deficiency should be studied
  • Increased serotonin activity is found in people w/ schizophrenia - dopamine + serotonin contribute to the positive + negative symptoms
  • More focus on other neurotransmitters is needed (e.g. GABA, acetylcholine, etc)
  • May be different subpopulations of those w/ schizophrenia, where it’s caused by different abnormal levels of different neurotransmitters - this has implications for treatment
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122
Q

What are the Strengths of the Secondary Data from the Contemporary Study for Schizophrenia by Carlsson (2000)?

A
  • Provides an overview of key findings in the area
  • generate new ideas about where research should go next
  • Closes significant gaps in research
  • Draws together wide body of data - allowing a large amount of info to be brought together quickly
  • Because of the extensive nature of material covered, the conclusion drawn is likely to be valid
  • Ethical - no direct ethical issues
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123
Q

What are the Weaknesses of the Secondary Data from the Contemporary Study for Schizophrenia by Carlsson (2000)?

A
  • Studies rely on secondary data from many other studies
  • The validity and reliability of Carlsson’s study may be affected by any research issues in the original studies
  • So, any conclusions from his study may be problematic
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124
Q

What are the Strengths of the Original 33 Studies from the Contemporary Study for Schizophrenia by Carlsson (2000)?

A
  • High reliability - studies cited by Carlsson are lab experiments - many are animal + PET scans using standardised procedures + controlled
  • High validity - Carlsson considered glutamate as a possible contribute to schizophrenia (another hypothesis), he argues both hypotheses may be true + research should continue in both (evidence seems to suggest this), he questions the validity of the dopamine hypothesis he himself developed in 1950s, Sendt (2012) literature review supports Carlsson’s findings
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125
Q

What are the Weaknesses of the Original 33 Studies from the Contemporary Study for Schizophrenia by Carlsson (2000)?

A
  • Low generalisability - some findings come from animals - so may not generalise to humans, culture may influence the type of auditory hallucinations (Luhrman, 2015), may be time locked (1999/2000) so research may no longer be representative of state of scientific ideas
  • Unethical - issues of deception + risk as patients don’t know if they’re being given the real drug or placebo, studies use humans - with or without schizophrenia being given amphetamines or PCP or other drugs that increased psychotic symptoms
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126
Q

What are the Applications from the Contemporary Study for Schizophrenia by Carlsson (2000)?

A
  • Useful in developing antipsychotic drugs
  • Glutamate antipsychotics are still in development (Papanastasiou et al, 2013) could bring relief to people who don’t respond well to typical antipsychotics
  • Improved dopaminergic drugs that have fewer side-effects based on better understanding of dopamine pathways + new atypical drugs that affect other neurotransmitters, like serotonin and glutamate
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127
Q

What are the Issue and Debates about Psychology as a Science in the Contemporary Study for Schizophrenia by Carlsson (2000)?

A
  • Use of scientific methods- e.g. PET scans
  • High validity - looks at a wide range of studies
  • Uses animal studies which aren’t generalisable
  • Not valid data - review article uses secondary data which might have flaws
  • High validity + credibility- Breier et al (1997) produced objective data
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128
Q

What are the Issue and Debates about Practical Issues in the Design and Implementation of Research in the Contemporary Study for Schizophrenia by Carlsson (2000)?

A
  • Difficult to study functioning of neurotransmitter in the brain
  • So, neurotransmitters which are easiest to study (dopamine + serotonin) tend to be most researched and others which are important tend to be ignored
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129
Q

What are the Issue and Debates about Reduction in the Contemporary Study for Schizophrenia by Carlsson (2000)?

A
  • This research is purely biological as only focuses on neurotransmitters, so it ignores social + cognitive factors
  • But, research uses many different studies to draw its conclusions (rodent studies, PET scans), as it generates such a wide range of evidence it can be considered somewhat holistic, also looked at many different neurotransmitters
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130
Q

What are the Issue and Debates about an Understanding of How Psychological Understanding has Developed Over Time in the Contemporary Study for Schizophrenia by Carlsson (2000)?

A
  • Further research suggested by the article will lead to more changes
  • Methods of investigating neurotransmitters have become more sophisticated overtime
  • Further research found that there is an interaction between dopamine + glutamate, as reduced level of glutamate is associated with increased dopamine levels
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131
Q

What are the Issue and Debates about the Use of Psychological Knowledge Within Society in the Contemporary Study for Schizophrenia by Carlsson (2000)?

A
  • The research suggests areas for further research which can be used to inform further treatments for schizophrenia
  • Future research as different sub populations have abnormal levels in different transmitters, not just dopamine
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132
Q

What is the Aim of the Longitudinal Study for Schizophrenia by Sensky (2000)?

A

Compare cognitive behavioural therapy (CBT) w/ non-specific ‘befriending interventions’ for patients w/ schizophrenia in effectively reducing positive symptoms of schizophrenia

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133
Q

What is the Design of the Longitudinal Study for Schizophrenia by Sensky (2000)?

A
  • Randomised controlled design
  • Patients allocated to one of two groups: A cognitive behavioural therapy group or an non-specific befriending control group
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134
Q

What were the Participants of the Longitudinal Study for Schizophrenia by Sensky (2000)?

A
  • 90 patients - 57 from clinics in Newcastle, Cleveland and Durham and 33 from London
  • They had a diagnosis of schizophrenia that had not responded to medication
  • Aged 16 to 60 years
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135
Q

What were the Results of the Longitudinal Study for Schizophrenia by Sensky (2000)?

A
  • Both interventions resulted in significant reductions in positive and negative symptoms and depression
  • At the 9 month follow-up evaluation, patients who had received cognitive therapy showed greater improvements on all measures
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136
Q

What were the Conclusions of the Longitudinal Study for Schizophrenia by Sensky (2000)?

A

CBT is effective in treating negative + positive symptoms of schizophrenia that are resistant to standard antipsychotic drugs, w/ its efficiency sustained over 9 months of follow-up

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137
Q

What was the Procedure for the

Cognitive Behavioural Therapy Group of the Longitudinal Study for Schizophrenia by Sensky (2000)?

A
  • Normal routine of CBT used - engaging with patient, examining antecedents of psychotic disorder, developing a normalising rationale + treatment of other disorders
  • Specific techniques for positive symptoms of schizophrenia used
  • Patients helped to change their beliefs + taught coping strategies to deal with the voices
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138
Q

What was the Procedure for the Befriending Group of the Longitudinal Study for Schizophrenia by Sensky (2000)?

A
  • Patients had same time allocation at the same intervals as the patients in the CBT
  • Therapists were empathetic + non-directive
  • No attempt at therapy, the sessions focused on hobbies, sports + current affairs
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139
Q

What was the Procedure for Assessing the Patients of the Longitudinal Study for Schizophrenia by Sensky (2000)?

A
  • Assessed by blind raters at baseline, after treatment (lasting up to 9 months), and a 9 month follow-up evaluation
  • Assessed using comprehensive psychiatric rating scale, the scale for assessment of negative symptoms and a depression rating scale
  • Patients continued to received routine care throughout the study
  • Patients received a mean of 19 individual treatment sessions over 9 months
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140
Q

What does Diagnosis Involve?

A
  • Clinician assessing a patient •Decides whether they show evidence of mental disorder
  • Decides whether their symptoms match those in a checklist of the features + symptoms of a mental disorder
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141
Q

Why is it Hard to Define Abnormal Behaviour in an Objective and Scientific Way?

A
  • No clear dividing line separating normal + abnormal behaviour
  • Concept of abnormality can change over time
  • Concept of abnormality can change between societies/cultures
  • Many different types of behaviour can be considered abnormal
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142
Q

What are the 4 D’s of Diagnosis?

A
  • Deviance
  • Distress
  • Dysfunction
  • Danger
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143
Q

What is Deviance as 1 of the 4 D’s of Diagnosis?

A
  • Behaviours/emotions that are unusual in society

* Behaviour must be statistically rare + disapproved of by most in society

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144
Q

What is Distress as 1 of the 4 D’s of Diagnosis?

A

Extent to which the individual finds their behaviour and/or emotions upsetting

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145
Q

What is Dysfunction as 1 of the 4 D’s of Diagnosis?

A

Extent to which the behaviour interferes with the persons day-to-day life

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146
Q

What is Danger as 1 of the 4 D’s of Diagnosis?

A

Behaviour which could harm others or the individual

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147
Q

What is the Issue and Debate of Issues of Social Control in the Diagnosis of Mental Disorders?

A
  • Clinicians have a lot of power and influence in the diagnosing of mental health problems
  • This has implications for patients who are labelled as mentally ill
  • As it’s possible to treat patients against their will if their sectioned by the mental health act + considered at risk to themselves or others
  • But, antipsychotic medication is critical for alleviating the distress associated with schizophrenia + enabling quality of life to be restored
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148
Q

What are the Strengths of the 4 D’s of Diagnosis?

A
  • Useful - they have practical applications in helping professionals decide whether a patient symptoms need a clinical diagnosis
  • Holistic way to assess someone’s mental health - cover a wide range of symptoms
  • Davis (2009) - suggests a 5th D needs to be added - duration to increase validity
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149
Q

What are the Weaknesses of the 4 D’s of Diagnosis?

A
  • Low validity - as diagnosis may not be accurate
  • Low reliability - if used by 2 professionals, may not get same diagnosis - reducing scientific status of diagnosis
  • Subjectivity in the application of the 4 D’s - E.g. dysfunction is subjective as individual may not think they have a problem + their unusual behaviour may suit them
  • Distress + danger is subjective + difficult to measure - not all mental disorders are distressing
  • Model criticised for being incomplete - Davis (2009) suggests that duration needs to be added
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150
Q

What is the Issue and Debate of Practical Issues in the Design and Implementation of Research in the Diagnosis of Mental Disorders?

A
  • Issues of reliability + validity- diagnosis process conducted through unstructured or semi-structured interviews
  • When gathering highly sensitive data about someone’s mental health - there are problems with the self-report method
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151
Q

What is the Issue and Debate of Psychology as a Science in the Diagnosis of Mental Disorders?

A
  • Diagnosing disorders relies on unstructured/semi-structured interviews
  • Involving the clinicians subjective interpretation of data
  • Which reduces scientific status of the diagnosing of mental illness as it’s objective, lacks reliability and validity
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152
Q

Why are Classification Systems used by Practitioners?

A

To help them make diagnosis + establish appropriate treatment regimes

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153
Q

What is the Aim of Classification Systems?

A

Provide clear + measurable criteria for diagnosis, which can be used in the same way by all practitioners - increasing the reliability of diagnosis

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154
Q

What is the Medical Model of Abnormality?

A
  • Biological approach assuming that the major source of abnormal behaviour is a form of medical illness
  • Believes that the best treatment for mental disorders is biological - e.g. drug treatment
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155
Q

What are the Strengths of the Medical Model of Abnormality?

A
  • Scientific + objective approach to diagnosis - has a lot of scientific credibility
  • It group symptoms together + classifies them into a syndrome - so, their cause can be discovered + treated
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156
Q

What are the Limitations of the Medical Model of Abnormality?

A
  • Limited - criticised for just focusing on the symptoms + not causes of this disorder - e.g. the model was criticised by anti-psychiatry movement in the 1960s
  • Symptoms cannot be easily measured, so clinician must make a subjective judgement
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157
Q

What was the Anti-Psychiatry Movement in the 1960’s which Criticised the Medical Model of Abnormality?

A
  • Thomas Szasz + R.D. Laing suggested that psychiatric symptoms is an understandable reaction to coping with a sick society
  • They felt that mental hospitals were not designed to cure people + they function as a prison, where disruptive people could be removed from society
  • Concerned that diagnosis could be used as an agent of state control + diagnosis results in labelling + could be used to medicalise social problems
  • Concerned about controversial medical practices in psychiatry - e.g. use of lobotomies as a treatment
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158
Q

What are the 2 Major Classification Systems?

A
  • ICD 10

* DSM-5

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159
Q

What is the Purpose of the DSM-5 and the ICD 10?

A
  • Diagnostic tool is designed to enable practitioners to identify + treat particular disorders
  • To provide a standardised, criterion based system to allow for accurate diagnosis of mental disorders
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160
Q

Why are the DSM and ICD Regularly Reviewed and Updated?

A
  • To take into account new research
  • To take account of peoples changing cultural views
  • To show that psychological understanding changes and develops over time
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161
Q

What are the Similarities Between the DSM and ICD?

A
  • Both regularly updated
  • Both attempt to make diagnosis accurate across cultures
  • Both rely on checklists + can be criticised for being reductionist
  • Both criticised as use the medical model
  • Both designed to enable practitioners to identify + treat particular disorders
  • Both include categories of mental disorders based on characteristic patterns of symptoms
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162
Q

What are the Differences Between the DSM and ICD?

A
  • DSM less reductionist than ICD - so it’s more holistic than ICD
  • ICD used in many countries in the world, but DSM used in America
  • ICD is free + an open resource, but DSM generates profits for the American psychological Association
  • DSM only deals w/ mental conditions, but ICD also diagnoses physical illness
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163
Q

What is the Issue and Debate of Reductionist vs Holistic in the Classification Systems?

A
  • DSM and ICD rely on checklists
  • Can be criticised for being reductionist
  • But, DSM takes account of psychological factors + disability, so it’s less reductionist than the ICD
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164
Q

What is the Issue and Debate of How Psychological Understanding has Changed Over Time in the Classification Systems?

A
  • Late 19th Century - Kraeplin argued that pychiatric disorders are physical in nature + should be studied as a branch of medical science + it is possible to classify specific mental health disorders by their symptoms, diagnose them + predict their course
  • Both DSM + ICD are regularly updated
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165
Q

What is the Issue and Debate of Psychology as a Science in the Classification Systems?

A
  • Reliable - rely on standardised checklists of observable behaviour - used in the same way by all clinicians
  • But, there will always be an element of subjectivity when interpreting the diagnostic criteria
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166
Q

What is the Issue and Debate of Practical Issues in the Classification Systems?

A
  • DSM +ICD are regularly updated

* So, they show that psychological understanding changes and develops over time

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167
Q

What is the Issue and Debate of Cultural Differences in the Classification Systems?

A
  • DSM and ICD attempt to make diagnosis accurate across cultures
  • But, some argue that they still have a western bias
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168
Q

When was the DSM 5 Published?

A

May 2013

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169
Q

What does the DSM-5 Assess Individuals In Terms Of?

A
  • The type of disorder from which they suffer - e.g. psychotic disorders
  • Psychological + contextual factors - as it can influence the well-being of the individual - e.g. unemployment
  • Disability - looks at overall level of functioning of the individual
  • Co-existing factors - e.g. medical conditions
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170
Q

What does the DSM-5 Assessment of an Individual Involve?

A
  • Clinical interviews
  • Observations of the client
  • Medical records of the client
  • Focuses on assessing patients along the spectrum
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171
Q

What is the Issue and Debate of Developmental Psychology in the Classification Systems?

A
  • Latest version of DSM reflects developmental + lifespan considerations
  • Reflects developmental processes that occur early in life (e.g. schizophrenia) and those that are more commonly developed during adolescence (e.g. depression)
  • Highlighting understanding that some psychiatric illnesses occur during certain periods of our development
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172
Q

How does the DSM 5 Manual Divide into 3 Sections?

A
  • 1 - Intro to manual w/ instructions on its use
  • 2 - Contains classification of the main mental health disorders - e.g. OCD, schizophrenia, bipolar, anxiety, etc
  • 3 - Contains other assessment measures to aid diagnosis - cultural formulation interview guide to help diagnose individuals from a different culture from the clinician + includes other conditions that are being assessed for possible future diagnosis
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173
Q

When is Diagnosis is Considered to be Reliable?

A
  • If different practitioners arrive at same diagnosis of a patient
  • When clinicians agree on a diagnosis there is high inter-rater reliability for the system of diagnosis
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174
Q

What is the Evidence from Spitzer and Williams (1985) that Suggests that Diagnosis is Low in Reliability?

A
  • Reviewed process of diagnosis

* Suggested that experienced psychiatrists only agree on diagnosis about 50% of the time

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175
Q

What is the Evidence from Hiller et al (1992) that Suggests that Diagnosis is Low in Reliability?

A

Argued reliability is in doubt for some disorders related to schizophrenia

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176
Q

What is the Evidence from Brown (2002) that Suggests that Diagnosis is High in Reliability?

A
  • Tested reliability + validity of DSM-5 diagnosis for anxiety + mood disorders
  • Found them to be good to excellent
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177
Q

What is the Evidence from Jacobsen et al (2005) that Suggests that Diagnosis is High in Reliability?

A
  • Through a random sample of Danish in-patients + outpatient’s w/ a history of schizophrenia
  • Showed that ICD 10 is a reliable measure of schizophrenia + compares well in terms of reliability with the DSM-111-R
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178
Q

What is the Evidence from Pederson (2001) that Suggests that Diagnosis is High in Reliability?

A
  • Found 71% of psychiatrists agreed with the ICD 10 definition of depression when assessing 116 patients
  • This indicates high inter-rater reliability
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179
Q

What are the Improvements in Reliability of Diagnosis?

A
  • Improved w/ the use of standardised interview schedules specifying symptoms to ask about + giving instructions as to how to rate their severity
  • DSM + ICD have specific diagnostic operational criteria for diagnosis to increase objectivity + reliability - e.g. clear definitions are given for each disorder + a list of symptoms
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180
Q

What are the Possible Patient Factors that Impact on Reliability of Diagnosis?

A
  • Patients may give different psychiatrists slightly different info
  • E.g. lack of standardisation in assessment + interview techniques
  • Leading to different practitioners giving different diagnoses to the same person
181
Q

What are the Possible Clinician Factors that Impact on Reliability of Diagnosis?

A
  • Practitioners using the classification systems aren’t completely objective
  • Practitioner may not use the categories of the classification system correctly or they may gather insufficient info
182
Q

What is the Evidence from Ward (1962) that Suggests that Diagnosis is Low in Reliability?

A
  • Studied 2 psychiatrists diagnosing the same patient
  • Found that disagreement occurred because of the inconsistency of info provided by the patient (5%), psychiatrists interpretation of symptoms (32.5%), inadequacy of the classification system (62.5%)
183
Q

What does the Validity of Diagnosis Refer to?

A

The extent to which a diagnosis is accurate

184
Q

What is Concurrent Validity in Diagnosis?

A
  • Compares evidence from different diagnostic tests to see if they agree
  • The DSM + ICD seem to have good concurrent validity
185
Q

What is Aetiological Validity in Diagnosis?

A
  • Exists when the diagnosis reflects known causes

* E.g. cause of disorder may be due to a problem w/ damage to a particular area of the brain

186
Q

What is Predictive Validity in Diagnosis?

A
  • Future course of the disorder is known and can be applied to the person
  • So, the diagnosis can be checked against the outcome to see if it’s valid
187
Q

What are Possible Factors that Impact on Validity of Diagnosis?

A
  • Implicit bias in clinician as the beliefs of them mean that they arrive at an inaccurate diagnosis
  • Patient may not disclose all info as they may be embarrassed or they don’t remember all details - so, clinician not able to arrive at an accurate diagnosis
  • Interviews are subjective - producing findings that the clinician expects to find
  • If the classification system is biased then diagnosis will not be valid
188
Q

What is the Evidence from Cochrane et al (1995) that Suggests that Diagnosis is Low in Validity?

A
  • Blames classification systems used in Britain
  • Argues that they lead practitioners to take on a Eurocentric bias
  • So, they cannot take into account the normal behaviours of other cultures as they are based on European ideas
189
Q

What is the Evidence from Littlewood (1992) that Suggests that Diagnosis is Low in Validity?

A
  • Questions international validity of DSM-5

* Suggests that the assumptions it makes about nuclear family life aren’t applicable to all cultures

190
Q

What is Implicit Bias in Diagnosis?

A

Positive or negative attitudes that a person may hold at an unconscious level

191
Q

What did David Rosenhan Suggest about Diagnosis?

A
  • Diagnosis was not accurate
  • Being given a psychiatric diagnosis would negatively affect a persons life
  • As, they would be left with a psychiatric label which others could judge them negatively on + misinterpret their behaviour
192
Q

What was the Aim of the Classic Study 1 by Rosenhen (1973)?

A
  • See if psychiatrists could differentiate between sane + insane people
  • Investigate life for patients in psychiatric hospitals
  • Raise awareness about conditions in psychiatric hospitals
193
Q

Who were the Participants of the Classic Study 1 by Rosenhen (1973)?

A

Hospital staff and patients

194
Q

What was the Method of the Classic Study 1 by Rosenhen (1973)?

A
  • Field experiment with participant observation
  • Participant observation by 8 sane people (a student, 3 psychologists, a painter, housewife, psychiatrist, paediatrician)
195
Q

What was the Independent Variable of the Classic Study 1 by Rosenhen (1973)?

A

Symptoms (lack of symptoms)

196
Q

What was the Dependent Variable of the Classic Study 1 by Rosenhen (1973)?

A

Diagnosis they’re given

197
Q

What was the Procedure of the Classic Study 1 by Rosenhen (1973)?

A
  • Phoned 12 psychiatric hospitals giving a false name + address •All complained of hearing unclear voices saying “hollow, empty, thud” - chose these words as they aren’t associated with a particular disorder
  • Apart from hearing voices, the pseudo patients behaved normally
  • Once admitted, they stopped simulating any symptoms + took part in ward activities -took notes of their experiences in the hospital
  • They had daily visitors who found that they were acting normally
198
Q

What were the Results of the Classic Study 1 by Rosenhen (1973)?

A
  • All admitted to hospital + all but 1 diagnosed with schizophrenia
  • Normal behaviour was misinterpreted - writing notes described as “patient engaged in writing behaviour”, arriving early for lunch described as “oral acquisitive syndrome”
  • No staff suspected that they were sane
  • Average stay in hospital was 19 days, shortest stay = 7 days, longest stay= 52 days
  • Given a total of 2100 medication tablets
  • 35/119 real patient has voiced their suspicions about the pseudo patients
  • Nurses stayed in ward offices 90% of the time, each real patient spent less than seven minutes per day with psychiatric staff + patients treated with no respect
199
Q

What was the Conclusion of the Classic Study 1 by Rosenhen (1973)?

A
  • Psychiatrist couldn’t detect sanity
  • The psychiatric hospitals weren’t useful for people with mental disorders
  • Diagnostic labels change the perception of the person so that all their behaviour was in interpreted within the context of the label - lead to self-fulfilling prophecy as the person is treated in a way that is consistent with the label which confirms the validity of the label
200
Q

What was the Aim of the Classic Study 2 by Rosenhen (1973)?

A

See if the hospitals who had been told they were going to be approached by pseudo patients would be able to tell the sane from insane people

201
Q

What was the Procedure of the Classic Study 2 by Rosenhen (1973)?

A
  • A teaching + psychiatric hospital was informed that over the next 3 months 1 or more pseudo patients were trying to get admitted to their hospital
  • Each staff member was asked to write the likelihood that the patients who sought to be admitted were pseudo patience
  • In reality, no pseudo patients were admitted
  • Staff members rated new patients on a scale of 1 to 10 as ‘how likely to be a fraud’, where 193 patients were assessed
202
Q

What were the Results of the Classic Study 2 by Rosenhen (1973)?

A
  • 41/193 rated as pseudo patient by at least 2 members of staff
  • 23/193 rated as pseudo patient by at least 1 psychiatrist
  • 19/193 rated as pseudo patient by a psychiatrist + 1 other member of staff
203
Q

What were the Conclusions of the Classic Study 2 by Rosenhen (1973)?

A
  • The staff were unable to detect insanity

* This confirmed Rosenhan’s initial results that there is poor validity in the diagnosis process

204
Q

What was the Aim of the Classic Study 3 by Rosenhen (1973)?

A

Investigate patient/staff contact

205
Q

What was the Procedure of the Classic Study 3 by Rosenhen (1973)?

A
  • In 4 of the hospitals, pseudo patients asked a member of staff when they will be eligible for ground privileges
  • Avoided asking the same member of staff more than once a day
  • Rosenhan compared responses given to the patients to those given to a student at Stanford university
206
Q

What were the Results of the Classic Study 3 by Rosenhen (1973)?

A
  • 71% psychiatrists ignore + 4% stop and talk
  • 88% nurses ignore + 0.5% stop and talk
  • But, 0% faculty at the Uni ignore + 100% stop and talk
207
Q

What was the Conclusion of the Classic Study 3 by Rosenhen (1973)?

A
  • Psychiatric patients are treated differently to non-psychiatric patients
  • Patients are powerless while on the mental ward
  • Lack of eye contact between staff + patients depersonalises the patients
208
Q

What is Depersonalisation?

A

Where people aren’t treated as unique individuals, worthy of respect

209
Q

How was the Classic Study by Rosenhen (1973) High in Generalisability?

A
  • Study took place in a range of hospitals
  • 12 hospitals across the USA
  • So, the results are generalisable to the USA of 1960s
210
Q

How was the Classic Study by Rosenhen (1973) Low in Generalisability?

A
  • Sample was very small and was unrepresentative - only 12 hospitals
  • May not generalise outside the USA as there may be cultural differences
  • May not generalise to current times
211
Q

How was the Classic Study by Rosenhen (1973) High in Reliability?

A
  • Certain aspects were standardised
  • E.g. All pseudo patients presented with the same single symptom of hearing voices saying “hollow, empty thud”
  • All pseudo patients behaved normally as soon as they’re admitted to hospital + said they didn’t have symptoms
  • Questions asked by pseudo patients and controls were standardised
212
Q

How was the Classic Study by Rosenhen (1973) Low in Reliability?

A
  • It was a field experiment
  • Variables such as patient-staff interactions couldn’t be standardised
  • So, research couldn’t be repeated in a consistent way
  • Study used participant observation so this is low reliability - as participant observers find it difficult to observe all details + may get distracted or over-involved
213
Q

What were the Applications for the Classic Study by Rosenhen (1973)?

A
  • Highlighted danger of labelling
  • People recognise that hospitals may not be the best place to treat mental illness
  • Reduce abuse of power in mental institutions by staff (e.g. CCTV)
  • Number of criteria used to diagnose mental illness increased - diagnosis now more accurate/valid, as a diagnosis of schizophrenia cannot rely on only 1 symptom
214
Q

How was the Classic Study by Rosenhen (1973) High in Validity?

A
  • High ecological validity
  • Pseudo patients able to gather 1st-hand data about the treatment of the patients
  • Setting (hospital) + tasks (hospital activities, life as a patient) are true to life
  • Process of being admitted to hospital, interactions with staff, discharge + experience of life in a hospital was true
  • Covert observation increases validity as reduces demand characteristics - nurses + doctors likely to act as they normally would
215
Q

How was the Classic Study by Rosenhen (1973) Low in Validity?

A
  • Low ecological validity
  • Lack of control is due to nature of research (field experiment) - so, impossible to standardise things such as patients/staff interactions or reactions of other patients
  • Observer bias may reduce validity - Rosenhan had strong opinions about psychiatry + the medical model - his opinions may have influenced his observations
216
Q

How was the Classic Study by Rosenhen (1973) Ethical?

A
  • Raised awareness of poor conditions in psychiatric hospitals
  • Confidential - names of individual doctors/nurses were not published
217
Q

How was the Classic Study by Rosenhen (1973) Unethical?

A
  • Hospital staff deceived
  • No consent given
  • No right to withdraw
  • Participants didn’t know they were part of the study
  • No protection of other patients - staff are wasting time on pseudo patients instead of looking after genuinely ill people
  • No protection - undermine confidence of doctors + nurses
218
Q

What is the Issue and Debate of the Use of Psychology in Social Control in the Classic Study by Rosenhen (1973)?

A
  • Labelling behaviours as abnormal leads to diagnosis, which pathologies the behaviour
  • Those in anti-psychiatry movement would argue that this forces people to conform to the standards set by society + if they don’t then they must receive treatment that will normalise their behaviour
  • Argued that if we don’t label the behaviour as abnormal than people would be free to behave how they like
  • E.g. in Rosenhan’s research - pseudo patients were treated for schizophrenia with tablets for their ‘symptoms’
219
Q

What is the Issue and Debate of Psychology as a Science in the Classic Study by Rosenhen (1973)?

A
  • Field experiment so lack of controls - e.g. standardisation reduces reliability
  • Low validity - participant observation means that there is observer bias - e.g. Rosenhan had strong opinions about medical model
  • Mainly qualitative data (pseudo patients experience) reduces scientific status
  • Covert observations increase validity as demand characteristics are reduced
220
Q

What is the Issue and Debate of How Psychological Understanding has Changed over Time in the Classic Study by Rosenhen (1973)?

A
  • When Rosenhan’s pseudo patients were incorrectly diagnosed with schizophrenia, the classification system in use was the DSM 11, which didn’t use a criterion based system for diagnosis
  • Currently the DSM-5 + ICD 10 give very specific symptoms which means that diagnosis should be both more reliable (consistent between clinicians) and more accurate
221
Q

What is the Issue and Debate of the Use of Psychological Knowledge in Society in the Classic Study by Rosenhen (1973)?

A
  • More respect for patients
  • More validity of diagnosis - criterion based system
  • Awareness of the dangers of labelling
  • Recognition that hospitals aren’t necessarily the best place to treat mental illness - move to care in the community
222
Q

How can Individual Differences be Linked to the Diagnosing of Mental Disorders?

A
  • Different people may react differently to practitioners
  • Different people may show different symptoms - e.g. different hallucinations, different levels of distress + dysfunction
223
Q

How can Developmental Psychology be Linked to the Diagnosing of Mental Disorders?

A

Schizophrenia usually develops in a persons late teens/early 20s, after an apparently normal childhood

224
Q

What is the Issue and Debate of Socially Sensitive Research in the Classic Study by Rosenhen (1973)?

A
  • Studying mental health issues is socially sensitive research
  • Involving labelling people with an illness in order to investigate treatment
  • Rosenhan able to provide an account of the quality of care in psychiatric hospitals
  • But, Spitzer criticised the study as sensationalist + causing harm to the psychiatric profession by casting doubt on the treatment of mental health
225
Q

What is Obsessive Compulsive Disorder (OCD)?

A
  • An anxiety disorder
  • Causing distress and has a major impact on the patient’s functioning
  • Characterised by the presence of obsessions and/or compulsions
226
Q

What are Obsessions in OCD?

A

Persistent, irrational, unwanted thoughts

227
Q

What are Examples of Common Obsessions in OCD?

A
  • Afraid of hurting themselves
  • Afraid of hurting others
  • Unwanted sexual thoughts, images, or urges
  • Contaminated or contaminating someone else
  • Fear of not having done a specific act that could result in harm
228
Q

What are Compulsions in OCD?

A
  • Tasks that people do to relieve themselves of the obsessions
  • The person feels driven to perform these tasks
  • This makes them feel less anxious or distressed
  • They can be mental or physical
  • They are extreme beyond reason or they are not realistically connected with what they are meant to stop
229
Q

What are Examples of Common Compulsions in OCD?

A
  • Cleaning or washing
  • Checking that you haven’t made a mistake
  • Ordering or arranging things in a particular way
  • Repeated checking behaviours
230
Q

People with OCD may have a Tic Disorder, What are Examples of this?

A
  • Eye blinking
  • Facial grimacing
  • Shoulder shrugging
  • Head jerking
  • Repeated clearing of the throat, sniffing, or grunting sounds
231
Q

What is the OCD cycle?

A
  • Starts with obsessions
  • Which causes strong feelings of anxiety - the person feels compelled to act + remove the discomfort
  • So, compulsions occur as a result
  • Leading to a feeling of relief from the anxiety
  • But, the obsessive response has been strengthened for the future
232
Q

What is the 5 Diagnostic Criteria for OCD listed by the DSM-5?

A
  • Obsessions cannot be ignored/suppressed + cause anxiety
  • Obsessions and compulsions are time-consuming - they take up more than 1 hour per day
  • Symptoms cannot be explained by substance use, another medical condition or mental disorder
  • Clinician determine if individual has fair or poor insight into the disorder, recognising that their obsessive compulsive beliefs are not true or if they have poor insight, true
  • Find out if the individual has a current or past history of a tic disorder
233
Q

What are the Onset Features of OCD?

A
  • The age of onset is late teens + early 20s

* But, in 25% of male sufferers, it is before the age of 10

234
Q

What are the Prevalence Features of OCD?

A
  • The prevalence rate is between 1.1 and 1.8% of the population
  • In adults, OCD is more common in females
  • In children, OCD is more common in males
235
Q

What are the Prognosis Features of OCD?

A
  • Symptoms develop gradually but they can be extreme
  • 70% of people experience a chronic and lifelong course
  • 5% have episodic symptoms
  • But, the content of obsessions doesn’t determine prognosis
236
Q

What are Factors Associated with a Good Prognosis in OCD?

A
  • Milder symptoms
  • Brief duration of symptoms
  • Good functioning before full onset
237
Q

What are the Risk Factor Features of OCD?

A
  • Family history - having a family member w/ the disorder can increase the risk of developing OCD
  • Stressful life events - people who have experienced traumatic or stressful events have an increased risk of getting OCD
238
Q

How does the Issue and Debate of Individual Differences Link to the Features of OCD?

A
  • Everyone doesn’t necessarily develop the same symptoms - cultural differences, males + females differ in symptoms/features
  • Not everyone responds to treatment in the same way
  • Genetic + environment factors can affect a persons risk of developing OCD
239
Q

How does the Issue and Debate of Culture + Gender Link to the Features of OCD?

A
  • Females + males are diagnosed differently depending whether they are a child or adult
  • OCD occurs across cultures at a similar rate, show a similar age at onset + show similar structure of symptoms
  • But, there are cultural differences in how some of these symptoms can be expressed
240
Q

What are Examples of the Symptoms of OCD?

A
  • Current or past history of tic disorder can affect severity of disorder
  • Obsessions cannot be ignored or suppressed
  • Obsessions + compulsions consume at least 1 hour per day + have a great impairment to daily life
241
Q

What are Examples of the Features of OCD?

A
  • Risk is higher for people with higher negative emotionally
  • In adults, OCD is more common in females
  • The prevalence rate is between 1.1 and 1.8% of the population
242
Q

What did OCD-UK Suggest which provides Evidence for the Features of OCD?

A

OCD affects about 1.2% of the population

243
Q

What did Grohol (2005) Suggest which provides Evidence for the Features of OCD?

A

Suggest about 2.3% of US population has OCD in one year

244
Q

What did Sasson et al (1994) Suggest which provides Evidence for the Features of OCD?

A

Estimates that about 2% of people have OCD worldwide

245
Q

What are the 2 Explanations of the Causes of OCD?

A
  • Biological explanation

* Cognitive explanation

246
Q

What does the Biological Explanation for the Causes of OCD Suggest?

A

Neuro-circuitry in the brain is not working correctly

247
Q

What is the Thalamus which Relates to the Biological Explanation for the Causes of OCD Suggest?

A
  • Contains primitive checking + cleaning behaviours hardwired in the brain
  • If this is overactive, it triggers a compulsion to engage in these behaviours
248
Q

What is the Orbitofrontal Cortex which Relates to the Biological Explanation for the Causes of OCD Suggest?

A
  • Alerts the brain to potential worries in the environment

* If overactive, the person would experience anxiety

249
Q

What is the Cingulate Nuclei which Relates to the Biological Explanation for the Causes of OCD Suggest?

A

Connect the orbitofrontal cortex to the thalamus

250
Q

How do the Brain Structures Relate to the Biological Explanation for the Causes of OCD Suggest?

A
  • Basal ganglia malfunctions
  • So, thalamus becomes overactive, causing compulsions to occur - so cleaning + checking behaviours are more likely
  • Overactive thalamus causes a cascade reaction in the orbitofrontal cortex, then this becomes overactive
  • Orbitofrontal cortex alerts the brain when something seems odd, causing anxiety which leads to the obsessions in OCD
251
Q

What is the Basal Ganglia which Relates to the Biological Explanation for the Causes of OCD Suggest?

A
  • Aims to inhibit the thalamus
  • If not working properly, it can’t inhibit the thalamus
  • Thalamus becomes overactive, causing compulsions to occur
252
Q

What are the Strengths for the Biological Explanation for OCD?

A
  • A lot of supporting evidence
  • Brain scans show differences in brain structures + functioning between people who have OCD + healthy controls
  • High scientific credibility - brain scans are a scientific way of investigating OCD + many studies of brain activity have come up with similar findings
  • Surgical reasoning of the cingulate gyrus has been used as treatment for OCD - so, it’s correct that OCD result from faulty feedback loop between thalamus + orbitofrontal cortex - since breaking the loop can cure OCD
253
Q

What are the Limitations for the Biological Explanation for OCD?

A
  • It’s reductionist - doesn’t fully explain OCD -surgery sometimes doesn’t work as OCD is more complex than this theory suggests
  • Difficult to show cause+effect - differences in brain activity in people with OCD may be a symptom of their OCD rather than the cause
  • Brain activity + thoughts are related, so can’t say if brain activity altered the thoughts or the thoughts altered the brain activity as they cannot be separated
  • Support for the biological link comes from genetic link as it suggests that physiological factors underlie the development of the disorder in many people - e.g. twin studies
254
Q

How does the Issue and Debate of Reductionism Relate to the Biological Explanation of OCD?

A
  • Isolating mental health to biological processes in the brain simplifies a complex behaviour
  • This may not be an appropriate way to view mental disorders
255
Q

How does the Issue and Debate of Psychology as a Science Relate to the Biological Explanation of OCD?

A
  • Use of brain imaging helps investigate relationship between brain structures + behaviour
  • It’s very scientific + adds reliability to the findings
  • This increases the scientific credibility of the theory
256
Q

How does the Issue and Debate of Individual Differences Relate to the Biological Explanation of OCD?

A
  • Some people have a family heredity risk, but some don’t
  • Some people respond to surgery, but some don’t
  • People suffering from OCD can develop different individualised symptoms
257
Q

How does the Issue and Debate of Nature vs Nurture Relate to the Biological Explanation of OCD?

A
  • Supports the nature side of the debate - as it looks at structure + functioning of the brain and is supported by genetic factors
  • But, the concordance rate in MZ twins was not 100% for OCD, so it is not a full exclamation
258
Q

How does the Evidence from Menzies (2007) Support the Biological Explanation for OCD?

A
  • Brain scans show differences in brain structure
  • Found that people suffering from OCD had a different amount of grey matter in orbitofrontal cortex to those without OCD
259
Q

How does the Evidence from Whiteside et al (2004) Support the Biological Explanation for OCD?

A
  • Brain scans show differences in brain functioning between people who have OCD + healthy controls
  • In patients with OCD, the cingulate gyrus, basal ganglia + orbitofrontal cortex are active when at rest - they become more active as OCD symptoms are stimulated
260
Q

How does the Evidence from Salloway + Duffy (2002) Support the Biological Explanation for OCD?

A

Found that PET scans of OCD patients had increased activity in the prefrontal cortex

261
Q

How does the Evidence from McGuire et al (1994) Support the Biological Explanation for OCD?

A
  • When people w/ OCD are shown objects that bring on their symptoms
  • There is an increase in activity in the orbitofrontal cortex + caudate nucleus
262
Q

How does the Evidence from Feng (2007) Support the Biological Explanation for OCD?

A
  • He bread mice to show symptoms consistent with OCD (excessive grooming + anxious behaviour) when a targeted gene is missing
  • This gene is associated with planning + initiation of action
  • Showing a link to formation of compulsions
  • Genetic explanation for OCD can be used to support the wider biological explanation in general
263
Q

How does the Evidence from Van Grootheest et al (2005) Support the Biological Explanation for OCD?

A

Found from a review of 70 studies that the concordance rates for MZ twins was between 27 to 47%, which is higher than for DZ twins

264
Q

How does the Evidence from Carey + Gottesman (1981) Support the Biological Explanation for OCD?

A

Found 87% concordance rate in MZ twins for obsessive symptoms + features compared to 47% for DZ twins

265
Q

How does the Evidence from Kireev et al (2013) Challenge the Biological Explanation for OCD?

A
  • OCD is more complex than this theory suggests

* He suggests that the functions usually performed by the cingulate gyrus can be taken over by other areas of the brain

266
Q

How does the Cognitive Explanation Explain Behaviour in General?

A
  • Thoughts lead to emotions

* Which in turn trigger behaviours

267
Q

What does the Cognitive Explanation for OCD Look at?

A

The role that thoughts play in the disorder

268
Q

What does the Cognitive Explanation for OCD Suggest?

A
  • It is the thought processes of people that explain OCD

* As, people who suffer from OCD have obsessive thoughts

269
Q

What are the 5 Beliefs that Researchers Suggest are Important in the Development and Maintenance of Obsessions in the Cognitive Explanation for OCD?

A
  • Tendency to overestimate the likelihood of danger
  • Exaggerated responsibility that 1 is responsible for preventing misfortunes
  • Belief that certain thoughts are very important + should be controlled
  • Belief that someone having a thought or urge will increase the chances that it will come true
  • Belief that 1 should always be perfect + mistakes are unacceptable
270
Q

What are the 3 Ways that the Cognitive Explanation Explains OCD?

A
  • People with OCD misinterpret their thoughts, due to false beliefs
  • OCD is due to memory problems
  • OCD sufferers may be hypervigilant
271
Q

How can the Cognitive Explanation Explain OCD in terms of Misinterpreted Thoughts due to False Beliefs?

A
  • Individuals prone to developing OCD exaggerate the thought + respond as if it was a real threat
  • Causing high level of anxiety + guilt
  • People interpreting intrusive thoughts as catastrophic will believe that thinking holds truth + will be distressed, so they practice ritual behaviours
  • People who fear their own thoughts attempt to neutralise feelings by avoiding situations and engaging in rituals
  • The anxiety produced by the thoughts lead to the compulsive behaviours seen in OCD
272
Q

Why do People Attach Exaggerated Danger to Their Thoughts in the Cognitive Explanation for OCD in terms of Misinterpreted Thoughts due to False Beliefs?

A

Because of false beliefs learned earlier in life

273
Q

How can the Evidence from Sher et al (1989) Show that the Cognitive Explanation Explains OCD in terms of Memory Problems?

A

Found that people with OCD had poor memories for their actions

274
Q

How can the Evidence from Trivedi (1996) Show that the Cognitive Explanation Explains OCD in terms of Memory Problems?

A
  • Found people suffering from OCD had low confidence in their memory ability
  • Also, their non-verbal memory was impaired
275
Q

How can the Evidence from Woods et al (2002) Show that the Cognitive Explanation Explains OCD in terms of Memory Problems?

A
  • He conducted a meta-analysis

* Found that patients with OCD had slightly worse memories for recalling stimuli

276
Q

How can the Cognitive Explanation Explain OCD in terms of Hypervigilance?

A
  • They have attentional bias, so they’re overly sensitive to threat
  • They use rapid eye movement to scan the environment + attend selectively to threat related stimuli
  • So, they feel very anxious
  • The threat perceived become the basis of their obsessions
  • The compulsive behaviours are designed to reduce their anxiety
277
Q

How can the Case Study from Rachman (2004) Show that the Cognitive Explanation Explains OCD in terms of Hypervigilance?

A
  • Female patient had severe fear of diseases
  • Had catastrophic thoughts about the probability of harm
  • She overestimated the seriousness of contact with anyone
  • When in public she rapidly scanned the environment + people on the lookout for evidence of blood
  • Her hypervigilant scanning meant that she could recall the blood related items she had encountered over many years
278
Q

How can the Evidence from Williams et al (1997) Show that the Cognitive Explanation Explains OCD in terms of Hypervigilance?

A

Suggested that OCD sufferers suffer from hypervigilance, so they have an attentional bias + become very anxious

279
Q

How can the Evidence from Van Balkom et al (1996) Support the Cognitive Explanation for OCD?

A
  • Found that therapy based on the cognitive explanation was as affective as drug treatment
  • This supports the cognitive theory because if OCD were purely biological, then the biological treatment should work better than the cognitive treatment
280
Q

What are the Strengths of the Cognitive Explanation for OCD?

A
  • Backed up with supporting evidence - which gives it scientific credibility
  • Therapy based on the cognitive explanation has been successful as a treatment for OCD
  • Cognitive biases (hypervigilance) give a good account of individual differences in susceptibility to OCD - the cognitive approach is easily adapted to individuals’ unique symptoms
281
Q

What are the Limitations of the Cognitive Explanation for OCD?

A
  • Reductionist - it ignores the role of biology + learning in the development of faulty cognitions
  • Doesn’t prove a cause+effect link - doesn’t show that the faulty cognitions are the cause of OCD, instead they could be a symptom - If the cognitive model was complete, treatments would always be useful
282
Q

How does the Issue and Debate of Comparisons Between Ways of Explaining Behaviour Using Different Themes Relate to the Cognitive Explanation for OCD?

A
  • OCD can be considered from a biological point of view + also a cognitive point of you
  • Both approaches have different strengths and weaknesses
283
Q

How does the Issue and Debate of Nature vs Nurture Relate to the Cognitive Explanation for OCD?

A
  • Everyone may have similar biological structures in common with people, but no one is exactly the same
  • Menzies (2007) provides biological support
  • Trivedi (1996) provides cognitive support
  • Interactive effect which causes OCD, as the brain is shaped by its environment + the environment is shaped by the brain of the person experiencing it
284
Q

What are the 2 types of treatment for OCD?

A
  • Drug treatment

* Cognitive behavioural therapy (CBT)

285
Q

When is drug treatment most commonly used as a type of treatment for OCD?

A

When cognitive behavioural therapy doesn’t work

286
Q

How do Antidepressants as a type of drug treatment work to treat OCD?

A
  • They raise serotonin levels
  • By blocking it’s reuptake from the synapse back into the releasing neurone
  • So, more serotonin is available for longer
287
Q

What are Examples of Antidepressants as a type of drug treatment that work to treat OCD?

A
  • Fluoxetine

* Sertraline (SSRI - selective serotonin re-uptake inhibitor)

288
Q

How long does it take for Antidepressants as a type of drug treatment to work to treat OCD?

A
  • Up to 12 weeks for drugs to be effective

* But, some people don’t respond to medication

289
Q

How do Anti Anxiety Drugs as a type of drug treatment work to treat OCD?

A
  • Increasing the effectiveness of GABA in regulating anxiety

* GABA lowers physiological arousal + returns body to a resting state

290
Q

What are Examples of Anti Anxiety Drugs as a type of drug treatment that work to treat OCD?

A
  • They’re called benzodiazepines

* E.g. Valium

291
Q

How do Beta Blockers as a type of drug treatment work to treat OCD?

A
  • Block the stress hormone that are released into the bloodstream by adrenal glands
  • They prevent this physiological response (e.g. increased heart rate) from occurring
  • They feel less stressed, so they have fewer obsessional thoughts + less compulsive behaviour
292
Q

What are Examples of Beta Blockers as a type of drug treatment that work to treat OCD?

A

Propranolol

293
Q

What are the Strengths of Drug Treatment for OCD?

A
  • It’s useful - as it can be used to treat cases of OCD which have not responded to CBT
  • There’s empirical evidence to show that drug treatment can be effective in OCD - e.g. Soomro (2007) - there’s scientific credibility
  • Drug treatment can be combined with CBT which may raise the effectiveness of CBT
  • Modern drug therapy with psychological therapy has reduced the need to resort to neurosurgery which was unethical
294
Q

What are the Weaknesses of Drug Treatment for OCD?

A
  • Drug treatments alone cannot treat most people with OCD
  • Lasts for 12 months before medication can be reduced or discontinued
  • May increase the patient’s anxiety
  • Can cause side-effects (e.g. nausea + headaches) - limiting the usefulness of drugs as people don’t want to take them
  • Can relapse if treatment is stopped
  • There’s individual differences in the way that people respond to drugs
  • Have to be sensitive to individual differences
295
Q

How does the Finding Soomro et al (2007) of Support the use of Drug Treatment for OCD?

A

Found that antidepressants were more effective than placebo in reducing the symptoms of OCD

296
Q

How does the Finding of POTS (2004) Support the use of Drug Treatment for OCD?

A

Drug treatment can be combined with CBT and has been shown to raise the effectiveness of CBT

297
Q

How does the Finding of Koran et al (2002) Support the use of Drug Treatment for OCD?

A
  • Antidepressant medication did have long-term affects compared to a placebo
  • It was effective at preventing relapse over an 80 week trial
298
Q

How does the Finding of Ravizza et al (1995) Challenge the use of Drug Treatment for OCD?

A

SSRIs were not effective for 40% of people

299
Q

How does the Finding of Brody et al (1998) Challenge the use of Drug Treatment for OCD?

A

Differences in the metabolism in the right compared to the left orbitofrontal cortex predicts whether a person will respond better to drugs or CBT

300
Q

How does the Finding of Goodman et al (1993) Challenge the use of Drug Treatment for OCD?

A
  • Prescribing drugs to patients, needs to be tailored to individual needs
  • Especially important with patients with refractory OCD
  • E.g. may be given a combination of SSRI drug with an antipsychotic drug
301
Q

What is Refractory OCD?

A

OCD that is very difficult to treat

302
Q

What are the 2 components of CBT as a type of treatment for OCD?

A
  • Cognitive component

* Behavioural component

303
Q

What does the Cognitive Component of CBT focus on as a type of treatment for OCD?

A
  • Changing thought processes

* Helping people to deal with obsessions found in OCD

304
Q

What does the Behavioural Component of CBT focus on as a type of treatment for OCD?

A

Changing actions/behaviours

305
Q

What are people encouraged to do in CBT as a type of treatment for OCD?

A

Focus on their thoughts, and emotional + behavioural responses to those thoughts

306
Q

What is the aim of the cognitive component in CBT as a type of treatment for OCD?

A

Change the beliefs that they trigger

307
Q

How does the Cognitive component in CBT start as a type of treatment for OCD?

A
  • Start with thoughts that are least anxiety provoking

* Client encouraged to test the beliefs that the thoughts activate until they don’t generate anxiety

308
Q

How is cognitive distortion of Catastrophing addressed in CBT as a type of treatment for OCD?

A
  • Person predicts a negative outcome

* Then, jump to the conclusion that if the negative outcome did happen, it would be a catastrophe

309
Q

What is Habituation Training (Franklin et al 2000) in the Cognitive component of CBT as a type of treatment for OCD?

A
  • Client thinks repeatedly about their obsessive thoughts
  • By deliberately thinking about obsessions, they’ll become less anxiety provoking
  • With the consequence that compulsive behaviour is not required to reduce high levels of anxiety
310
Q

What is the most common Behavioural therapy used as a part of CBT as a type of treatment for OCD?

A

Exposure and response prevention therapy (ERPT)

311
Q

What does ERPT focus on in the Behavioural component of CBT as a type of treatment for OCD?

A

The compulsions found in OCD

312
Q

What is ERPT in the Behavioural component of CBT as a type of treatment for OCD?

A
  • Exposes clients to objects/situations that cause anxiety
  • Requires the client to resist performing the compulsive behaviour
  • The therapist helps the person develop ways in which they can resist performing the compulsive behaviours
313
Q

What are the steps involved in ERPT in the Behavioural component of CBT as a type of treatment for OCD?

A
  • Informing clients about exposure + response prevention, and what therapy involves
  • Using exposure hierarchy - starting with mildly anxiety raising situations + goes to the highest level of anxiety
  • Repeated exposure to situations that cause high anxiety, until anxiety reduces
  • Getting the client to resist from performing the compulsive behaviour
314
Q

What are Clients in ERPT in the Behavioural component of CBT asked to do outside of therapy as a type of treatment for OCD?

A

Practice exposing themselves to feared situations + reframing from the compulsive behaviour

315
Q

How does the finding from Franklin et al (2005) support ERPT in the Behavioural component of CBT as a type of treatment for OCD?

A
  • After ERPT, clients showed between 55% and 75% improvement

* The improvement lasts for 5 to 6 years

316
Q

What are the Strengths of CBT as a type of treatment for OCD?

A
  • Supported by evidence which shows that it’s effective in reducing the symptoms of OCD
  • Doesn’t have any side-effects
  • Has lower relapse rates than medication
  • Ethical form of treatment - patients are empowered as they are taught techniques which they can use on their own
317
Q

How does the Issue and Debate of Social Control link to CBT as a type of treatment for OCD?

A

CBT empowers a patient - implying that they are less controlled by others in society

318
Q

How does the finding by NICE (2006) support CBT as a type of treatment for OCD?

A
  • CBT is endorsed by the National Institute for health and clinical excellence
  • So, it’s effective in reducing symptoms of OCD
319
Q

What are the Weaknesses of CBT as a type of treatment for OCD?

A
  • Limited treatment which should ideally be used in combination with other forms of therapy
  • Difficult to disentangle which of the benefits from CBT are due to the cognitive or behavioural component
  • Individual differences means that therapy needs to be tailored to the needs of the particular person - can be ineffective
  • ERPT wouldn’t be effective for clients that don’t have compulsions
320
Q

How does the finding of Masellis et al (2003) not support CBT as a type of treatment for OCD?

A
  • Found that up to 44% of clients only suffer from obsessions
  • Up to 75% had co-morbid depression, which lessons effects of ERPT
  • So, ERPT may not be effective
321
Q

What is Relapse Rate?

A

Recurrence of their disorder/disease

322
Q

What are the Similarities between CBT and Drug Treatment as treatments for OCD?

A
  • Both useful and effective as treatment for OCD

* Both have supporting evidence to show treatment is effective - drug treatment = Soomro (2007), CBT = NICE (2006)

323
Q

What are the Differences between CBT and Drug Treatment as treatments for OCD?

A
  • CBT more ethical - it empowers patients as they’re taught techniques which they can use on their own + has lower relapse rate
  • Individual differences
  • Drug treatment with CBT combined as effective - POTS (2004)
324
Q

What is the Aim of the Contemporary Study by POTS (2004) for OCD?

A

Compare 3 treatments to see which is most effective in treating young people w/ OCD:
•CBT on its own
•An SSRI (sertraline) on its own
•CBT + sertraline combined

325
Q

What was the Type of Sample used in the Contemporary Study by POTS (2004) for OCD?

A
  • Volunteer sample
  • All diagnosed using DSM-IV
  • Study ran across 3 centres in the USA
326
Q

What were the Participants in the Contemporary Study by POTS (2004) for OCD?

A
  • 112 participants
  • All American
  • 92% white
  • Aged 7-17 years - average age of 11
327
Q

How was the Severity of the Participants’ Symptoms Measured in the Contemporary Study by POTS (2004) for OCD?

A
  • A standardised test - the Children’s Yale-Brown Obsessive Compulsive Scale
  • Only those with a score of 16 or above on the scale were included
328
Q

Which Children were Excluded from the Contemporary Study by POTS (2004) for OCD?

A
  • Children who were comorbid w/ other disorders (e.g. Tourette’s)
  • This would avoid interaction effects - to control confounding variables
329
Q

Why were children with ADHD required to be on stimulant medication + stable in the Contemporary Study by POTS (2004) for OCD?

A

To ensure that their ADHD wouldn’t affect the treatment for OCD

330
Q

Why were no children on anti-obsessional medication at the start of the Contemporary Study by POTS (2004) for OCD?

A
  • To ensure that any in the Ps was due to the treatments offered in the study
  • Controlling confounding variables
331
Q

What were the 4 Conditions of the Contemporary Study by POTS (2004) for OCD?

A
  • Drugs only
  • Placebo pill only
  • CBT only
  • CBT + drug treatment
332
Q

How were the children allocated to the Conditions in the Contemporary Study by POTS (2004) for OCD?

A

Randomly, using a computerised system

333
Q

How were the children assessed at the start of treatment of the Contemporary Study by POTS (2004) for OCD?

A
  • They were interviewed

* The baseline measure was taken using the CY-BOCS using independent evaluators

334
Q

How long did the Contemporary Study last by POTS (2004) for OCD?

A

12 weeks

335
Q

Why did each child have a specialist psychiatrist assigned to them for the duration of the Contemporary Study by POTS (2004) for OCD?

A
  • To monitor their progress

* To provide them with support

336
Q

What was the Procedure for the Drugs only and Placebo conditions of the Contemporary Study by POTS (2004) for OCD?

A
  • Children attended weekly sessions for 6 weeks, then attended every other week (9 sessions in total)
  • Dose established +changed during sessions
  • Parents would monitor that children took their medication + kept a medication diary
  • Any adverse reactions would result in the medication being changed or stopped
337
Q

How many sessions did the CBT group have in the Contemporary Study by POTS (2004) for OCD?

A

14 clinical sessions over the 12 week period

338
Q

What were the 4 Components in the CBT group of the Contemporary Study by POTS (2004) for OCD?

A
  • Psychological training
  • Cognitive training
  • Mapping OCD target symptoms
  • Exposure + response ritual prevention
339
Q

What did each CBT session consist of for the CBT group in the Contemporary Study by POTS (2004) for OCD?

A
  • Goal setting
  • Review of the previous week
  • Therapist assistant practice
  • Homework
  • Monitoring
340
Q

What was the Combined condition of the Contemporary Study by POTS (2004) for OCD?

A
  • Drugs and CBT in conjunction

* Sessions were time linked and provided simultaneously

341
Q

When were the Participants Assessed in the Contemporary Study by POTS (2004) for OCD?

A

At baseline, 4 weeks, 8 weeks, 12 weeks

342
Q

Who Assessed the Participants in the Contemporary Study by POTS (2004) for OCD?

A

Independent evaluators trained to a reliable standard

343
Q

How did the authors ensure the assessment was reliable in the Contemporary Study by POTS (2004) for OCD?

A
  • Evaluators carefully trained

* Evaluation process was strictly supervised + reviewed

344
Q

How many of the original participants completed the Contemporary Study by POTS (2004) for OCD?

A

97 out of 112

345
Q

What is CY-BOCS in the Contemporary Study by POTS (2004) for OCD?

A

Children’s Yale-Brown obsessive compulsive scale

346
Q

How was the progress of participants measured in the Contemporary Study by POTS (2004) for OCD?

A
  • Looked at how much the participants improved on the CY-BOCS over the 12 week period
  • Gathered quantitative data
347
Q

What does Remission mean?

A

Where the person has no signs + symptoms of the disorder

348
Q

How did the authors define ‘entering remission’ in the Contemporary Study by POTS (2004) for OCD?

A

A drop below 10 on the CY-BOCS

349
Q

What were the Results of the Contemporary Study by POTS (2004) for OCD?

A
  • All conditions showed improvement at 12 weeks as measured on CY-BOCS, but placebo improvement wasn’t significant
  • In CBT alone condition, 39.3% entered remission, compared to 21.4% in the drugs alone condition
  • Combination treatment (drugs + CBT) was most effective in reducing symptoms
350
Q

What was the Conclusion of the Contemporary Study by POTS (2004) for OCD?

A
  • CBT lead to more improvements than drugs - so, should be the 1st line of treatment
  • Drugs may compensate for less effective therapy, but minimal gain if CBT is effective
  • Drugs require careful monitoring, as SSRI’s are linked to suicidal ideation in young people
  • But, drug treatment was well tolerated
  • Early intervention effective for children and young people with OCD
351
Q

How was the Contemporary Study by POTS (2004) for OCD High in Generalisability?

A
  • Large sample - 112 children + low dropout rate
  • Representative of target population - young people w/ OCD
  • Analysis of sample showed there was no difference in groups - so, any difference in group would be down to the IV
352
Q

How was the Contemporary Study by POTS (2004) for OCD Low in Generalisability?

A
  • Excluded children who were co-morbid
  • Not generalisable to adults - Ps only aged 7-17 years
  • Conducted in USA - may not be generalisable to non-western societies - as, other societies may respond differently to CBT and other medication
353
Q

How was the Contemporary Study by POTS (2004) for OCD High in Reliability?

A
  • The evaluators who assessed the children symptoms had been trained to a reliable standard
  • The scale used to assess their symptoms was a standardised measure
  • All participating centres (3) used the CBT manual in order to maintain consistency in treatment
354
Q

How was the Contemporary Study by POTS (2004) for OCD Low in Reliability?

A
  • This study was conducted over 3 centres, but 1 centre had better results on the CBT only condition
  • This may be due to the therapist effect, as 1 therapist may have been more proficient at CBT than the others
355
Q

How did the Contemporary Study by POTS (2004) for OCD have Useful Applications?

A
  • Provides strong evidence about the most effective treatment for OCD in children + young people
  • Shows value of clinical treatments - cause + effect
  • Show usefulness of psychological therapies
  • SSRI’s can lead to suicidal ideation in some individuals which needs to be taken into account
  • Findings have shown effectiveness of CBT + sertraline combined - but individual differences mean therapies need to be tailored to the person
356
Q

How was the Contemporary Study by POTS (2004) for OCD High in Ecological Validity?

A
  • Study conducted over a long period of time - 12 weeks

* Studying people in their real life

357
Q

How was the Contemporary Study by POTS (2004) for OCD High in Validity?

A
  • The assessment scale (CY-BOCS) had been validated as an accurate measurement of OCD symptoms
  • Ps randomly allocated into conditions - can infer cause-and-effect
  • The assessors were blind to the condition that the Ps were in - so, they couldn’t be biased in the evaluation of improvements
  • Ps in placebo condition were unaware that the drug wasn’t active, as was the therapist - this controlled for demand characteristics
358
Q

How was the Contemporary Study by POTS (2004) for OCD Ethical?

A
  • All Ps + 1 of their parents gave full written consent
  • Volunteer sample - Ps wanted to do the study
  • Each participant was assigned psychiatrist to monitor and support them
  • Those in the drug treatment condition were regularly checked + dosages changed as necessary - protecting them from harm
359
Q

How was the Contemporary Study by POTS (2004) for OCD Unethical?

A
  • Use of deception in placebo condition
  • But, this was necessary in order to ensure validity of the study
  • Everyone in placebo condition was offered therapy on completion of the study
360
Q

How does the Issue and Debate of Practical Issues Relate to the Contemporary Study by POTS (2004) for OCD?

A
  • Could have problems with attrition - but, there is actually a low dropout rate
  • Training of assesses to a reliable standard is costly and time-consuming
  • Longitudinal study - carried out over 12 weeks = time-consuming + expensive
  • There are difficulties in recruiting Ps w/ similar symptoms of OCD - but when tested, all groups are homogeneous
  • Volunteer sample - Ps may be more motivated, so CBT results may be better than in the general population + didn’t have co-morbid Ps, so don’t know how effective for children with multiple issues
361
Q

How does the Issue and Debate of Reductionism Relate to the Contemporary Study by POTS (2004) for OCD?

A
  • Less reductionist as it looks a different treatments to explain OCD - cognitive, behavioural + medication
  • So, more holistic
  • But, it ignore social + environmental factors
362
Q

How does the Issue and Debate of Comparisons Between Ways of Explaining Behaviour using Different Themes Relate to the Contemporary Study by POTS (2004) for OCD?

A
  • Comparison of explanations + treatment for OCD
  • CBT assumes faulty thinking
  • Drug treatment assumes faulty brain functioning
  • The study took account of different explanations
363
Q

How does the Issue and Debate of Psychology as a Science Relate to the Contemporary Study by POTS (2004) for OCD?

A
  • Drug treatment is objective - controls (e.g. evaluators + ADHD medication) - controlled confounding variables
  • The children’s version of the Yale-Brown obsessive compulsion scale is standardised to give quantitative data that is objective - therefore, it’s scientific
364
Q

How does the Issue and Debate of Culture Relate to the Contemporary Study by POTS (2004) for OCD?

A
  • OCD occurs in most cultures, but POTS study can only be applied to children in the USA
  • E.g. children were 92% white
  • So, lacks cultural variety
365
Q

How does the Issue and Debate of Issues of Social Control Relate to the Contemporary Study by POTS (2004) for OCD?

A
  • OCD is distressing for the sufferer, so change may be in their best interest
  • However, drug treatment may be criticised for trying to correct someone’s behaviour to go along with social norms
366
Q

How does the Issue and Debate of the Use of Psychological Knowledge within Society Relate to the Contemporary Study by POTS (2004) for OCD?

A
  • POTS gives treatment for OCD that’s effective
  • This helps patients that suffer
  • Leading to the relief of symptoms + helped them lead relatively normal lives
367
Q

How does the Issue and Debate of Issues Relating to Socially Sensitive Research Relate to the Contemporary Study by POTS (2004) for OCD?

A
  • Mental health is a sensitive issue
  • Issues of abnormality - once labelled, it’s hard to get rid of that label
  • From POTS - CBT is the 1st treatment that should be used + its socially acceptable
368
Q

How does the Issue and Debate of an Understanding of How Psychological Research has Developed Overtime Relate to the Contemporary Study by POTS (2004) for OCD?

A
  • Practice changed as a result of POTS study
  • Before 2004, CBT wasn’t routinely used for children - but, this study has helped change this
  • So that CBT is now the 1st line of treatment that OCD, + shows that CBT and drug treatment are very effective in combination
  • So, we are using less drastic measures like drugs with side-effects or brain surgery to help relieve OCD
369
Q

What is the Aim of Valentine et al (2010) study which uses clinical interviewing?

A

Study the usefulness of psycho education within group work for offender patients in a high security forensic hospital setting

370
Q

What were the Participants of Valentine et al (2010) study which uses clinical interviewing?

A
  • 42 males
  • Detained in Broadmoor high security hospital
  • Most received a diagnosis of schizophrenia
  • They were part of a program that aimed at helping them understand and cope with their illness
371
Q

What is the Method of Valentine et al (2010) study which uses clinical interviewing?

A
  • Participants interviewed using a semistructured interview technique
  • This was to understand their experience better + get info as to how the group could be improved in the future
  • Group run for 20 sessions over a 3-year period
  • Psycho education program considered symptoms, treatment options and coping mechanisms
  • Then, a content analysis was conducted on the data gathered to pick out key themes in the responses
372
Q

What were the Key Themes Identified in the Data of Valentine et al (2010) study which uses clinical interviewing?

A
  • What participants valued + why
  • What was helpful about the group
  • Clinical implications
  • What was difficult/unhelpful about the group
373
Q

What were the Results of Valentine et al (2010) study which uses clinical interviewing?

A
  • Patients valued knowing and understanding their illness
  • Group sessions allowed them to understand their symptoms + how other peoples experiences were similar
  • Increased confidence in dealing with illness, making them more positive about the future
  • Patients valued the group + would recommend it
374
Q

What were the Conclusions of Valentine et al (2010) study which uses clinical interviewing?

A
  • There were positive + negative changes in measures taken after psycho education group
  • Qualitative data showed how Ps valued knowledge about their illness which gave them power
  • So, semi-structured interviews are useful in gathering detailed rich data which can be used to inform future practice
375
Q

How is Valentine et al (2010) study which uses clinical interviewing Low in Generalisability?

A
  • Ps very severe + had complex needs
  • This is shown as they were in a high security institution
  • So, can’t generalise the findings to another population
376
Q

How is Valentine et al (2010) study which uses clinical interviewing Low in Reliability?

A
  • Semi structured interviews used - so, questionnaires are not standardised
  • E.g. some questions weren’t standardised
  • So, difficult to replicate
377
Q

How is Valentine et al (2010) study which uses clinical interviewing High in Reliability?

A
  • Some questions were standardised- e.g. pre-post test scores
  • Researchers recorded their interviews to allow them to play back + check accuracy of data
  • So, they can also get other researchers to check the data, which increases interrater reliability
378
Q

What are the Applications for Valentine et al (2010) study which uses clinical interview?

A
  • Data can inform future practice

* Results showed that Ps did benefit from psycho educational program, as valued knowledge about illness

379
Q

How is Valentine et al (2010) study which uses clinical interviewing High in Validity?

A
  • Semi structured interviews allowed more detailed data to be gained from patients
  • Info from patients interviews allowed them to fully express their own point of view, helping the researchers to understand their perspective more clearly
380
Q

How is Valentine et al (2010) study which uses clinical interviewing Ethical?

A
  • Ps allowed to withdraw from the study/request for their data not be included
  • Ps gave consent
381
Q

How is Valentine et al (2010) study which uses clinical interviewing Unethical?

A
  • Ps still vulnerable + not easy for them to refuse consent/withdraw from study
  • Given confinement
382
Q

How do Individual Differences Relate to Valentine et al (2010) study which uses clinical interviewing?

A
  • Different insight into their illness
  • So, may respond differently to the programme from each other
  • Usefulness of programme may not apply to everyone
383
Q

What is the Inductive Approach in Qualitative Data?

A
  • Observing or analysing something

* Then, drawing a set of principles from it or a model of how things might be from the data

384
Q

What is the Deductive Approach in Qualitative Data?

A
  • Researcher specifies themes they’ll search for
  • Going from a theory to predict what might happen
  • Using the data to test against the theory
  • Starts with a hypothesis
385
Q

What is the Grounded Theory?

A
  • A way of analysing qualitative data
  • Finding theory from the data, the theory is grounded in the data
  • This theory is analysed using the inductive method
386
Q

What is the Aim of the Grounded Theory?

A
  • Retain the richness of the data
  • Produce coding that captures those meaning in a manageable form
  • Show new ways of understanding behaviour, including shared meanings between people
387
Q

What are the Stages of Grounded Theory?

A
  • Coding is done
  • Codes collected into concepts, which groups data together
  • Concepts grouped into similarities and differences, theory starts to develop
  • Theory comes from collecting the categories together, it’s about forming a model that can explain the data
388
Q

What are the Strengths of Grounded Theory?

A
  • Rich + detailed data collected - showing new ways of understanding behaviours
  • Able to develop a theory to explain the data - uses specific terms to explain how it’s done and is explicit in its guidance
  • Theory should have high validity - as, evidence is integrated into the theory
389
Q

What are the Weaknesses of Grounded Theory?

A

•Low validity - if the data gathered to develop the theory was problematic
•Time-consuming to gather + analyse data
•Theory may be based on a subjective opinion - researchers may be biased in the way they interpret data
•Researchers may force data to support the theory - so, may miss crucial evidence that contradicts the concepts they believe
- as, they selectively sampling data as a theory begins to emerge

390
Q

What are the Strengths of Thematic Analysis?

A
  • Flexible way of analysing qualitative data + doesn’t always need a theory to drive the analysis
  • Maintains richness in data + summarising a large amount of qualitative data in a manageable way
  • Allows insight into data which isn’t always anticipated by the researcher + yield more detailed and meaningful info than quantitative data
391
Q

What are the Weaknesses of Thematic Analysis?

A
  • Hard to find themes in data that aren’t driven by the questions asked by the researcher in the interview
  • Unscientific - themes are highly dependent on the subjective opinion of the researcher - so, can lead to researcher bias
392
Q

How does Thematic Analysis Take Place?

A
  • Familiarise with data
  • Generate codes
  • Look for themes in codes
  • Review the themes
  • Define + name the themes
  • Produce the report
393
Q

What are the different methods for researching mental health?

A
  • The use of longitudinal methods
  • The use of cross-sectional methods
  • The use of cross-cultural methods
  • The use of meta-analysis
  • The use of primary and secondary data
394
Q

What is a way to explore the causes of mental health problems?

A
  • Investigate impact of different treatment methods

* Find out how disorders progress in patients

395
Q

What is the use of longitudinal methods as a method for researching mental health?

A
  • Takes place over a long period
  • Involves comparing a sample group with their own performance overtime
  • So, developmental changes can be seen through patterns of measurement
396
Q

What would the use of longitudinal methods as a method for researching mental health allow?

A

Psychologists to see if there is any reduction in the symptoms to help them to assess how effective the treatment is

397
Q

How are measurements taken in the use of longitudinal methods as a method for researching mental health?

A

How the symptoms present themselves over a time period at certain intervals

398
Q

What is the study of POTS (2004) as an example of the use of longitudinal methods as a method for researching mental health?

A
  • Compared the use of CBT with the use of sertraline in children with OCD
  • Study lasted for 12 weeks, patients assessed at weeks: 4, 8 and 12
  • Showed change and development in OCD symptoms overtime
399
Q

What is the study of Sensky (2004) as an example of the use of longitudinal methods as a method for researching mental health?

A
  • Compare that CBT vs befriending for schizophrenia
  • Treatment lasted 9 months + patients assessed 9 months after treatment ended
  • Found that CBT is effective in treating negative and positive symptoms in schizophrenia that are resistant to drugs
400
Q

What is the study of Meltzer (2004) as an example of the use of longitudinal methods as a method for researching mental health?

A
  • A 6 week study that compared placebo, 4 new drugs + haloperidol
  • Found that haloperidol was better than placebo in improving positive + negative symptoms of schizophrenia
  • So, study showed change + development in schizophrenia
401
Q

What are the Strengths of the longitudinal method as a method for researching mental health?

A
  • High validity - participant variables are controlled for - as, same Ps used
  • High reliability - reliable way to measure the effect of time on behaviour - enabling clinicians to evaluate the effectiveness of treatments in terms of improving the quality of a patient’s life
402
Q

What are the Weaknesses of the longitudinal method as a method for researching mental health?

A
  • Low validity- Ps may drop out, reducing the sample size
  • May be factors which affect individuals development, making it hard to draw conclusions
  • Take a long time + can be expensive
  • By the time data can be used to draw conclusions in the study, the data may be irrelevant
403
Q

What is the use of cross-sectional methods as a method for researching mental health?

A
  • Useful when researchers want to take a quick snapshot of behaviour in a given population in a set period of time
  • Uses a large sample
  • Conclusions are drawn from the data gathered
404
Q

What is the study of Luhrmann (2015) as an example of the use of cross-sectional methods as a method for researching mental health?

A
  • The aim was to compare auditory hallucinations in the USA, India and Ghana
  • It was an interview based study
405
Q

What are the Strengths of the cross-sectional method as a method for researching mental health?

A
  • Data gathered quickly - so, conclusion can be drawn + acted on more rapidly
  • High validity - results more likely to be valid, as they will be reported at the same time when they have most application
  • Can be economical - as, it requires less commitment in terms of time from a researcher compared to the longitudinal design
406
Q

What are the Weaknesses of the cross-sectional method as a method for researching mental health?

A
  • Not good for finding out the cause of something like a mental disorder as they’re descriptive research
  • There might be a cohort effect - as, the study looks at different people at the same moment in time + those people will belong to a different cohort
  • They’re unlikely to include any historical info about a patient, or info about the future - this is because they’re a snapshot
407
Q

What is Cohort Effect?

A

The difference in social and cultural groups that change with age and time

408
Q

What is Culture?

A

The way a group of people share their decisions + behaviour, such as sharing norms in society

409
Q

What is the use of cross-cultural methods as a method for researching mental health?

A
  • Carried out by researchers who want to compare some behaviour or attitudes in different cultures - similarities + differences
  • Universality can be studied - if a result is found in many different cultures, then it may be in our nature
410
Q

What are the Strengths of the cross-cultural method as a method for researching mental health?

A
  • It identifies elements of abnormal behaviour that can be attributed to biological factors
  • They aid clinicians understanding of the cultural factors they should consider when diagnosing + treating patients from different cultural groups
  • Allows researcher to gain an understanding of how culture plays a role in the validity and reliability of diagnosis in clinical psychology
411
Q

What are the Weaknesses of the cross-cultural method as a method for researching mental health?

A
  • In conducting research across cultures, there is likely to be a conflict between the cultures
  • So, conclusions drawn may lack validity if the interpretation of the patient’s behaviour doesn’t consider their cultural background
412
Q

What is the study of Luhrmann (2015) as an example of the use of cross-cultural methods as a method for researching mental health?

A
  • Found that Ps in the USA were more likely to report negative commands
  • Whereas, patients from India + Ghana were more likely to report rich relationships with their voices
413
Q

What is the use of meta-analysis as a method for researching mental health?

A
  • Using the findings of different studies - secondary data
  • Findings are analysed
  • Focuses on effect sizes
414
Q

How have meta-analysis as a method for researching mental health been used in clinical psychology?

A

To find out about the effectiveness of therapies and treatments across different patient groups

415
Q

What are the Strengths of the meta-analysis as a method for researching mental health?

A
  • Conclusions drawn from a huge sample and different areas
  • Results can be generalised to a larger population due to the large sample
  • More data used - so, there is likely to be more precision in the analysis
  • Quick and cost-effective
  • Research generally focuses on using peer-reviewed publications - so, data has been scrutinised
416
Q

What are the Weaknesses of the meta-analysis as a method for researching mental health?

A
  • Researchers don’t gather data directly - so, may be on identified issues of reliability + validity in the methods of data gathering
  • Low validity due to publication bias
  • E.g. research that produce null effects may not be published + therefore would be ignored by meta analysis
417
Q

What is the study of Carlsson (2000) as an example of the use of meta-analysis as a method for researching mental health?

A
  • Used meta analysis in combining the results are various neurotransmitters studies
  • To investigate the role of neurotransmitters including dopamine, serotonin and glutamate in schizophrenia
418
Q

What is the use of primary data as a method for researching mental health?

A

Gathered first-hand from source directly by researcher

419
Q

What is the use of secondary data as a method for researching mental health?

A

Data already gathered by others to use for future research

420
Q

What is the study by Rosenhan (1973) as an example of the use of primary data as a method for researching mental health?

A

Gathered primary data from first-hand observations

421
Q

What is the study by Lavarenne et al (2013) as an example of the use of primary data as a method for researching mental health?

A

Used data from one of the group sessions, which were primary data

422
Q

What is the study by Carlsson (2000) as an example of the use of secondary data as a method for researching mental health?

A
  • The use of meta analysis/secondary data for neurotransmitter functioning
  • E.g. studies w/ rodents, studies that used brain scans, studies on people w/ acute schizophrenia + people w/ schizophrenia in remission
423
Q

What is the study by Gottesman and Shields (1966) as an example of the use of secondary data as a method for researching mental health?

A
  • Used secondary data for their twin studies

* E.g. hospital notes

424
Q

What are the Strengths of the primary data as a method for researching mental health?

A
  • More credible than secondary data - as, they’re gathered for the purpose with chosen research method, design etc
  • Operationalisation is done with research in mind, so there’s likely to be validity with regard to the aim
425
Q

What are the Strengths of the secondary data as a method for researching mental health?

A
  • Cheap - as, data is already collected
  • Can be large quantities of data, so there might be detail
  • Can be from different sources - so, there is a possibility of comparing data to check for reliability + validity
426
Q

What are the Weaknesses of the primary data as a method for researching mental health?

A
  • Expensive - as, data gathered from the start

* Limited to time, place and number of participants

427
Q

What are the Weaknesses of the secondary data as a method for researching mental health?

A
  • When analysed originally, there may have been subjectivity
  • Likely to be gathered to suit another aim, so may not be valid for the study
  • May have been gathered some time before, so not in a relevant time period
428
Q

What are case studies as a method for researching mental health?

A
  • Studying individuals or small groups with a unique characteristic or experience
  • Often evidence gathered will be qualitative, allowing an in-depth analysis of the group
  • Researchers triangulate the data to draw conclusions
429
Q

What are case studies in relation to clinical psychology as a method for researching mental health?

A
  • Case studies may be of people with rare symptoms or individual taking part in a specific therapy
  • A full understanding of the patient’s problems can be assessed + all the factors that may have an affect on them can be taken into account
430
Q

What was the aim of the study by Bradshaw (1998) as an example of the use of case studies as a method for researching mental health?

A
  • Investigate how CBT can be used to treat a woman with schizophrenia
  • An attempt to use psychotherapy to treat schizophrenia where drug treatment had previous been preferred
431
Q

What was the procedure of the study by Bradshaw (1998) as an example of the use of case studies as a method for researching mental health?

A
  • Carol had 3 years of CBT + follows up her progress 6 months and a year after therapy
  • Carol symptoms were measured on 4 scales: roll functioning scale (RFS), goal attainment scale (GAS), hospitalisation, global pathology index (GPI)
  • Carol had three stages of treatment
432
Q

What was the 1st stage of Carols treatment in the procedure of the study by Bradshaw (1998) as an example of the use of case studies as a method for researching mental health?

A
  • Development of rapport - honest + trusting communication
  • CBT sessions lasted for 15mins to 1hour
  • Carol + therapist often went for a walk
  • They shared their love of baseball
433
Q

What was the 2nd stage of Carols treatment in the procedure of the study by Bradshaw (1998) as an example of the use of case studies as a method for researching mental health?

A
  • Understanding CBT - took 2months
  • Carol was educated in CBT, schizophrenia and treatment
  • The focus was on improving ways of coping with stress
  • The ABC model (Ellis, 1957) was used
434
Q

What was the treatment part in the procedure of the study by Bradshaw (1998) as an example of the use of case studies as a method for researching mental health?

A
  • 1st year - focus on managing anxiety + therapy was on faulty attributions
  • A weekly activity schedule gave her structure after leaving the hospital - Carol recited what she did during the day to help therapist recognise signs of stress
  • Middle phase - 16months of building more strategies to cope with stressful situations - e.g. the use of a ‘stress thermometer’ + the use of meditation
  • End phase - 3months - Carol developed plants to maintain the treatment without therapist - e.g. she wrote cue cards w/ coping strategies
435
Q

What were the Results of the study by Bradshaw (1998) as an example of the use of case studies as a method for researching mental health?

A
  • Carol improved in psychosocial functioning, achievement of goals, reduction of symptoms, little distress + never re-hospitalised
  • After 1 year - RFS was stable at 27, whereas at the start she scored 6
  • At end of study - GPI score of 1 indicated that there were few symptoms present
  • GAS increased from 19.85 to 80.15 - shows that treatment goals had been attained - goals were: starting a college course, taking a volunteer job + going out once a week w/ friends
436
Q

What was the Conclusion of the study by Bradshaw (1998) as an example of the use of case studies as a method for researching mental health?

A
  • Improved in Carols functioning in all 4 measures after a 3 year course of CBT - so, CBT can be successful in treating schizophrenia
  • Bradshaw + Roseborough (2004) carried out a meta-analysis of case studies of 22 patients - they suggested that 86% improved their psychosocial functioning + 82% had reduced severity of symptoms - this supports CBT as an effective treatment for Schizophrenia
437
Q

How was the study by Bradshaw (1998) as an example of the use of case studies as a method for researching mental health Low in Generalisability?

A
  • Unusual case - Carol is much younger than most females with schizophrenia, comes from a supportive background + has no family history of mental illness
  • She is female + the disorder presents differently in males
  • Her type of schizophrenia (undifferentiated type) isn’t the only type of schizophrenia - CBT may not be affective with other types
438
Q

How was the study by Bradshaw (1998) as an example of the use of case studies as a method for researching mental health Low in Reliability?

A
  • Many details of the study are hard to replicate
  • E.g. the rapport between Carol + her therapist
  • Case study - can’t replicate the study in the exact same way
439
Q

How was the study by Bradshaw (1998) as an example of the use of case studies as a method for researching mental health High in Reliability?

A
  • CBT follows standard procedure by Aaron Beck
  • The measures used (e.g. GAS, GPI, RFS) are well established tools by psychiatrists
  • Carols symptoms were checked against DSM-IV
  • The same measures used at start of study, 3x during therapy + 2x afterwards = test- retest reliability
440
Q

What were the Applications in the study by Bradshaw (1998) as an example of the use of case studies as a method for researching mental healthy?

A
  • Promotes the use of CBT to help treat patients with schizophrenia
  • So, clients lower their dependency on antipsychotic drugs (biological treatment/side-effects/compliance)
  • But, treatment of CBT is only available to 10% of sufferers in the UK
441
Q

How was the study by Bradshaw (1998) as an example of the use of case studies as a method for researching mental health High in Ecological Validity?

A
  • Real patient receiving real treatment was used

* E.g. CBT and coping methods

442
Q

How was the study by Bradshaw (1998) as an example of the use of case studies as a method for researching mental health High in External Validity?

A
  • Supported by other case studies in the meta analysis - Bradshaw + Roseborough (2004)
  • 86% improved psychosocial functioning and 82% had to reduce symptoms
443
Q

How was the study by Bradshaw (1998) as an example of the use of case studies as a method for researching mental health High in Internal Validity?

A
  • Use of scales to accurately measure her progress increases internal validity - e.g. GAS, GPI, RFS
  • Gained quantitative data - e.g. her scores on RFS, GPI and GAS
  • Gained qualitative data -e.g. her walks, volunteer job, change in self-esteem + view of herself with someone in the future
  • Real changes are seen over a period of 4 years
444
Q

How was the study by Bradshaw (1998) as an example of the use of case studies as a method for researching mental health Low in Internal Validity?

A
  • All the clients were taking antipsychotic medication alongside CBT
  • So, this could be the real reason there was improvement in the study which supported the case study of Carol
445
Q

How was the study by Bradshaw (1998) as an example of the use of case studies as a method for researching mental health Ethical?

A
  • Bradshaw respected the young woman’s dignity + privacy by concealing her real identity + using the pseudonym ‘Carol’ instead
  • At the end of study, Carol was much healthier + could give full informed consent for her data to be used
  • Her therapist developed rapport by being genuine + having empathy
  • CBT helped her live a less stressful life as improvement was still in evidence a year after therapy
  • Carol was empowered - using a stress thermometer + a pat on the back technique - allowed her to feel in control of her treatment
446
Q

How was the study by Bradshaw (1998) as an example of the use of case studies as a method for researching mental health Unethical?

A
  • There are issues of consent
  • As, Carol was in too bad a state at the start of study to consent to share details of her progress with the researcher
  • So, consent was obtained from family and therapist instead
447
Q

How was Carol described in the study by Bradshaw (1998) as an example of the use of case studies as a method for researching mental health Ethical?

A

•She was a good student, shy but with several friends
•But, when she started to experience hallucinations, she withdrew from people + acted in a bizarre manner
•She was hospitalised + diagnosed with
undifferentiated type schizophrenia using the DSM-IV

448
Q

How are Peer Reviews Effective?

A
  • It helps to maintain standards within that area of research
  • It can attract funding
449
Q

How can Peer Reviews be Criticised?

A
  • The actual findings may not be relevant by the time it’s published
  • There are practical issues - peer review can involve many amendments, so may be costly + take a long time
  • Some research is never published - as, research with null findings isn’t published - there is a publication bias for positive outcomes, leading to a distorted view of a subject area