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Cell Pathology > Chronic Inflammation > Flashcards

Chronic Inflammation Flashcards

1
Q

What is chronic inflammation?

A

Lecture 4, slide 7

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2
Q

What are the histological features of chronic inflammation?

A

Lecture 4, slide 8

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3
Q

Why may an inflammatory response be chronic?

A

Lecture 4, slide 9

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4
Q

What are the different types/categories of chronic inflammation?

A

Lecture 4, slide 13

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5
Q

What is chronic osteomyelitis? What are its risk factors, pathogenesis and treatment?

A

Lecture 4, slide 15

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6
Q

What is alcoholic hepatitis histology? What does continued heavy drinking cause? What does ceasing alcohol use cause?

A

Lecture 4, slide 17

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7
Q

What causes silicosis? What is silicosis?

A

Lecture 4, slide 19

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8
Q

What is the pathogenesis of silicosis?

A

Lecture 4, slide 20-21

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9
Q

What is primary infection in TB infection (when is it usually, how is it transmitted, what is its pathogenesis, how is this revealed?)?

A

Lecture 4, slide 23

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10
Q

What are the sequelae/consequences of primary TB infection?

A

Lecture 4, slide 24

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11
Q

When is secondary TB seen?

A

Lecture 4, slide 25

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12
Q

What is the pathogenesis and histology of TB?

A

Lecture 4, slide 26, 33

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13
Q

What is a granulomata?

A

Lecture 4, slide 27

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14
Q

What can a TB granulomata be accompanied by?

A

Lecture 4, slide 28

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15
Q

What is delayed type hypersensitivity? How is TB screened for?

A

Lecture 4, slide 32

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16
Q

How else may TB be screened for?

A

Lecture 4, slide 34-35

17
Q

What is rheumatoid arthritis (RA)?

A

Lecture 4, slide 41

18
Q

What are the current treatments for RA?

A

Lecture 4, slide 42-44

19
Q

What are some therapeutic targets in RA?

A

Lecture 4, slide 45

20
Q

Why may therapeutic antibodies not be continuously effective?

A

Lecture 4, slide 46

21
Q

Give examples of new and future treatments of chronic inflammatory diseases.

A

Lecture 4, slide 48

22
Q

What are inflammasomes? What are the different signals in inflammasomes?

A

Lecture 4, slide 50-53

23
Q

Discuss a study investigating the relationship between gout, MSU and inflammasomes.

A

Lecture 4, slide 56-59

24
Q

How may gout be treated with therapeutic antibodies?

A

Lecture 4, slide 61-62

Cell Pathology (10 decks)

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