Chapter 55-test 2 Flashcards
Acute gastritis
Exposure to local irritants
NSAIDs steroids
If muscle is damaged, hermmorhage occurs
Chronic gastritis
Chronic exposure
ETOH
Tobacco
Radiation
S/s of chronic gastritis
Patchy diffuse inflammation
Vague epigastric pain
Pernicious anemia-destruction of paritial cells
S/s of acute gastritis
Thick, red mucosa, rugae
Rapid onset
Epigastric pain
May vomit blood
Type A chronic gastritis
Non erosive
Autoimmune
Parietal cell destruction
Fundus
Type B chronic gastritis
Caused by h pylori infection
Antrum
Atrophic chronic gastritis
Elderly Toxic exposure H pylori Related autoimmune Destruction of deep glands
Atrophic gastritis may lead to
Gastric cancer
MALT lymphoma
Treatment of gastritis
Diet No ETOH Caution NSAIDs Avoid caffeine Food and water safe Stress Smoking
Treatment of gastritis medications
Treat h pylori with at least 2 antibiotics and PPI H2 blocker PPI Mucosal barrier fortifiers (karophate) B12 injections to prevent anemia
Most ulcers are caused by
H pylori
Gastric ulcers usually develop in the
Antrum
Peptic ulcer disease hostile factors
H pylori
Gastric acid
NSAIDs
Pepsin
Peptic ulcer disease protective factors
Bicarbonate
Prostaglandins
Blood flow to mucosa
Risk factors for peptic ulcer disease
Heredity
Smoking
Stress