Chapter 21 Patho Flashcards

1
Q
  1. What is Thrombus?
  2. Lodges at the bifurcation of the pulmonary artery and is often fatal.
  3. a blood clot that forms and remains in the viens
  4. a blood clot that becomes dislodged and travels to another part of the body.
A
  1. a blood clot that forms and remains in the viens
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2
Q
  1. What is Embolus?
  2. Lodges at the bifurcation of the pulmonary artery and is often fatal.
  3. a blood clot that forms and remains in the viens
  4. a blood clot that becomes dislodged and travels to another part of the body.
A
  1. a blood clot that becomes dislodged and travels to another part of the body.
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3
Q
  1. What is Saddle emboli?
  2. Lodges at the bifurcation of the pulmonary artery and is often fatal.
  3. a blood clot that forms and remains in the viens
  4. a blood clot that becomes dislodged and travels to another part of the body.
A
  1. Lodges at the bifurcation of the pulmonary artery and is often fatal.
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4
Q
  1. The major pathological or structural changes of the lungs associated with pulmonary embolism.
  2. Most common originate from deep venous thrombosus (DVT) in the lower part of the body.

Or

  • Blockage of the pulmonary vascular system,
  • Pulmonary infarction,
  • Alveolar atelectasis,
  • Alveolar consolidation,
  • Bronchospasm
A
  • Blockage of the pulmonary vascular system,
  • Pulmonary infarction,
  • Alveolar atelectasis,
  • Alveolar consolidation,
  • Bronchospasm
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5
Q
  1. Where is the Pulmonary Embolism originates from?
  2. Most common originate from deep venous thrombosus (DVT) in the lower part of the body.
  3. Venous stasis (stagnate blood flow)
  4. Hypercongoagulation.
  5. Injury to the endothelial cells that line the vessels
A
  1. Most common originate from deep venous thrombosus (DVT) in the lower part of the body.
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6
Q
  1. What are the 3 primary factors known as Virchow’s triad are associated with formation of DVT
  2. Fat.
  3. Air.
  4. Bone Marrow
  5. Tumor fragments

Or

  1. Venous stasis (stagnate blood flow)
  2. Hypercongoagulation.
  3. Injury to the endothelial cells that line the vessels
A
  1. Venous stasis (stagnate blood flow)
  2. Hypercongoagulation.
  3. Injury to the endothelial cells that line the vessels
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7
Q
  1. What are the other factors that cause emboli?
  2. Fat.
  3. Air.
  4. Bone Marrow
  5. Tumor fragments

Or

  1. Venous stasis (stagnate blood flow)
  2. Hypercongoagulation.
  3. Injury to the endothelial cells that line the vessels
A
  1. Fat.
  2. Air.
  3. Bone Marrow
  4. Tumor fragments
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8
Q
  1. Where do the Emboli travels in the heart?
  2. Venous stasis (stagnate blood flow)
  3. Hypercongoagulation.
  4. Injury to the endothelial cells that line the vessels

Or

Emboli travels through the right side of the heart and into the pulmonary artery, where is eventually becomes lodge/wedged when the diameter the vessel get small enough

A

Emboli travels through the right side of the heart and into the pulmonary artery, where is eventually becomes lodge/wedged when the diameter the vessel get small enough

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9
Q
  1. What is Diagnosis and Screening show in CXR?

Emboli travels through the right side of the heart and into the pulmonary artery, where is eventually becomes lodge/wedged when the diameter the vessel get small enough

Or

  1. Does not show P.E.
  2. Best for ruling out other conditions that mimic symptoms of P.E (Pneumothorax etc)
A
  1. Does not show P.E.

2. Best for ruling out other conditions that mimic symptoms of P.E (Pneumothorax etc)

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10
Q
  1. Spiral (helical) CT scan

Not Best and quickest second line test for P.E

Or

Best and quickest first line test for P.E

A

Best and quickest first line test for P.E

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11
Q
  1. What is EKG?
  2. Does not show P.E.
  3. Best for ruling out other conditions that mimic symptoms of P.E (Pneumothorax etc)

Or

  1. May see ST-T wave changes that are nonspecific.
  2. As with CXR, best for ruling out other conditions that mimic P.E (MI pericarditis)
A
  1. May see ST-T wave changes that are nonspecific.

2. As with CXR, best for ruling out other conditions that mimic P.E (MI pericarditis)

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12
Q
  1. What is D-dimer?
  2. Venous stasis (stagnate blood flow)
  3. Hypercongoagulation.
  4. Injury to the endothelial cells that line the vessels

Or

  1. Blood test that looks for increased level of protein fibrinogen, a component of the blood-clotting process.
  2. Fast and very accurate.
  3. If the D-dimar level is normal, it essential rules out the possibility of blood clot.
A
  1. Blood test that looks for increased level of protein fibrinogen, a component of the blood-clotting process.
  2. Fast and very accurate.
  3. If the D-dimar level is normal, it essential rules out the possibility of blood clot.
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13
Q
  1. What is Factor V (Leyden) deficiency?
  2. Fast acting anticoagulant.
    - Prevents clot from growing.
    - Prevents new formations.
    - Given via I.V

Or

Genetic hypercoagulation.
1. May require lifelong anticoagulation therapy.

A

Genetic hypercoagulation.

1. May require lifelong anticoagulation therapy.

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14
Q
  1. What does heparin do to PE?
  2. Slow acting maintenance anticoagulant.
    - Need to periodical check blood clotting factor to adjust dose.

Or

  1. Fast acting anticoagulant.
    - Prevents clot from growing.
    - Prevents new formations.
    - Given via I.V
A
  1. Fast acting anticoagulant.
    - Prevents clot from growing.
    - Prevents new formations.
    - Given via I.V
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15
Q
  1. What is Coumadin?
  2. Fast acting anticoagulant.
    - Prevents clot from growing.
    - Prevents new formations.
    - Given via I.V

Or

  1. Slow acting maintenance anticoagulant.
    - Need to periodical check blood clotting factor to adjust dose.
A
  1. Slow acting maintenance anticoagulant.

- Need to periodical check blood clotting factor to adjust dose.

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16
Q
  1. What is Thrombolytic agents?
  2. Does not show P.E.
  3. Best for ruling out other conditions that mimic symptoms of P.E (Pneumothorax etc)

Or

  1. Actually dissolve blood clots.
    - Strptokinase.
    - Urokinase.
    - Referred to as “Clot Busters”
    - High risk of bleeding after given
A
  1. Actually dissolve blood clots.
    - Strptokinase.
    - Urokinase.
    - Referred to as “Clot Busters”
    - High risk of bleeding after given
17
Q

What is Preventive Measures?

  1. Actually dissolve blood clots.
    - Strptokinase.
    - Urokinase.
    - Referred to as “Clot Busters”
    - High risk of bleeding after given

Or

  1. Walking to promote venous circulation
  2. Exercises of lower body
  3. Drink fluids to promote hydration.
  4. Compression stockings
  5. Vein filter.
  6. Pneumatic compression.
  7. Pulmonary embolectomy (Surgical removal)
A
  1. Walking to promote venous circulation
  2. Exercises of lower body
  3. Drink fluids to promote hydration.
  4. Compression stockings
  5. Vein filter.
  6. Pneumatic compression.
  7. Pulmonary embolectomy (Surgical removal)
18
Q
  1. Most pulmonary emboli are from:

A. Blood emboli
B. Fate emboli
C. Air emboli
D. Malignant neoplasms

A

A. Blood emboli

19
Q
  1. Which of the following is a thrombolytic agent?

A. Heparin
B. Coumadin
C. Lasix
D. Streptokinase

A

D. Streptokinase

20
Q
  1. Most pulmonary emboli originate from thrombi in the:

A. Lungs
B. Right side of the heart
C. Leg and pelvic veins
D. Pulmonary veins

A

C. Leg and pelvic veins

21
Q
  1. The lab test used to identify hypercoagulation and supports the diagnosis the diagnosis of the pulmonary embolism is:

A. CBC
B. COHgb
C. D-dimer
D.Tropinin

A

C. D-dimer