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Pharmacology Test 4 > Ch. 50 > Flashcards

Ch. 50 Flashcards

1
Q

Types of Acid-Controlling Drugs

A

Antacids
H2 Antagonists
Proton Pump Inhibitors

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2
Q

Aluminum carbonate

A

Aluminum Salt

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3
Q

Hydroxide salt

A

Aluminum Salt

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4
Q

Gaviscon

A

Combination aluminum and magnesium

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5
Q

Maalox

A

Combination aluminum and magnesium

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6
Q

Mylanta

A

Combination aluminum and magnesium

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7
Q

Di-Gel

A

Combination aluminum and magnesium

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8
Q

hydroxide salt

A

Magnesium Salt

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9
Q

carbonate salt

A

Magnesium Salt

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10
Q

Tums

A

calcium salt (Ca Carbonate)

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11
Q

Highly soluble. Buffers the acidic properties of HCL. Quick onset, but short duration. May cause metabolic alkalosis. Sodium content may cause problems in patients with
HF, hypertension, or renal insufficiency

A

Sodium Bicarbonate

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12
Q

activated charcoal

A

Antiflatulant

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13
Q

simethicone

A

Anitflatulant

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14
Q

Alters elasticity of mucus-coated bubbles, causing

them to break

A

simethicone

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15
Q

Adverse effect of aluminum and calcium

A

constipation

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16
Q

Adverse effect of magnesium

A

diarrhea

17
Q

Adverse effects produces gas and belching; often combined with
simethicone

A

calcium carbonate

18
Q

Chemical binding, or inactivation, of another drug. Produces insoluble complexes. Result: reduced drug absorption

A

Chelation

19
Q

Drug Interations of Antacids include: Increased absorption of basic drugs. Decreased absorption of acidic drugs as a result of:

A

Increased stomach pH

20
Q

Interations of Antacids include: Increased excretion of acidic drugs
and decreased excretion of basic drugs as a result of:

A

Increased urinary pH

21
Q

cimetidine

A

H2 Anatagonist

22
Q

nizatidine

A

H2 Anatagonist

23
Q

famotidine

A

H2 Anatagonist

24
Q

ranitidine

A

H2 Anatagonist

25
Q

MOA: block receptors of acid-producing parietal cells, therefore production of hydrogen ions is reduced,
resulting in decreased production of HCl

A

H2 Antagonists

26
Q

Indications: GERD, PUD, erosive esophagitis, adjunct therapy in control of upper GI
bleeding, pathologic gastric hypersecretory conditions

A

H2 Antagonists

27
Q

May induce impotence and

gynecomastia

A

cimetidine

28
Q

MOA: Irreversibly bind to H
+
/K
+
ATPase enzyme, this bond prevents the movement of hydrogen ions
from the parietal cell into the stomach. Result: gastric acid secretion is
temporarily blocked

A

Proton Pump Inhibitors

29
Q

lansoprazole

A

Proton Pump Inhibitor

30
Q

omeprazole

A

Proton Pump Inhibitor

31
Q

rabeprazole

A

Proton Pump Inhibitor

32
Q

pantoprazole

A

Proton Pump Inhibitor

33
Q

esomeprazole

A

Proton Pump Inhibitor

34
Q

Indications: GERD maintenance therapy, erosive esophagitis, short-term treatment of active duodenal and
benign gastric ulcers, Zollinger-Ellison syndrome, treatment of
H. pylori
–induced ulcers (administered with an antibiotic)

A

Proton Pump Inhibitors

35
Q

Cytoprotective drug. Used for stress ulcers, PUD. Attracted to and binds to the base of ulcers and
erosions, forming a protective barrier over these
areas. Protects these areas from pepsin, which normally
breaks down proteins (making ulcers worse

A

sucralfate

36
Q

A synthetic prostaglandin analog. Protects gastric mucosa from injury by enhancing
local production of mucus or bicarbonate. Promote local cell regeneration. Help to maintain mucosal blood flow

A

misoprostol

37
Q

Used for prevention of NSAID-induced gastric

ulcers

A

misoprostol

38
Q

Antiflatulent drug. Used to reduce the discomforts of gastric or intestinal
gas (flatulence). Alters elasticity of mucus-coated gas bubbles,
breaking them into smaller ones

Decreases gas pain and increases expulsion via
mouth or rectum

A

simethicone

Pharmacology Test 4 (10 decks)

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