cell biology Flashcards

1
Q

what cell cycle determines cell cycle length?

A

G1 (duplication). affected by growth factors.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

when is G0?

A

after G1 variably.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

where are ribosomes made?

A

nucleolus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

double membrane nucleus?

A

outer is contiuous with RER.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

where does steroid hormone bind its receptor?

A

in the cytoplasm, then enters nucleus to act as transcription factor (directly binds DNA).

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

where does thyroid hormone bind its receptor?

A

in the nucleus; acts as transcription factor

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

purines?

A

guanine
adenine

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

pyrimidines?

A

cytosine
thymidine
uricil (R)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

how many H bonds in GC and AT/U

A

3 in GC
2 in AT AU

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

in glycolysis, how much ATP and pyruvate made from 1 glucose molecule?

A

2 ATP and 2 pyruvate.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

where does Krebs cycle occur?

A

inner matrix of mitochondria (2 membrane).

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

products of Krebs cycle?

A

NADH and FADH2 from 2 pyruvate (from glucose)
go on to form total 36 ATP from 1 glucose

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

what else can enter the Krebs cycle?

A

amino acids, ketones, short chain fatty acids.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

gluconeogenesis reactants and products?

A

lactic acid (cori cycle) and amino acids (#1 alanine) make GLUCOSE

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

why can’t fat/lipids be available for GNG (in setting of starvation)?

A

because acetyl CoA (breakdown of fat metabolism) cannot be converted back to pyruvate.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

cori cycle products?

A

lier converts muscle lactate into new glucose (using pyruvate)

17
Q

how does golgi apparatus modify proteins?

A

carbohydrates then transport to cellular membrane/secrete/lysosomes

18
Q

what organ has a lot of RER?

A

pancreatic acinar cells.

19
Q

what organ has a lot of SER?

A

liver and adrenal cortex (lipids and steroid synthesis)

20
Q

phagosomes (large) and endosomes (small) have to fuse with what to degrade particules?

A

lysosomes.

21
Q

three major signaling pathways?

A

phospholipase C, protein kinase A, MAPK/ERK

22
Q

phospholipase C

A

PIP2 into DAG and IP3; increase Ca from SER.

23
Q

protein kinase C

A

Ca and DAG activates…. phosphorylates other proteins.

24
Q

protein kinase A

A

cAMP acrivates… phosphorylates other proteins.

25
Q

myosin vs actin

A

myosin THICK filmaments;
actin THIN filaments

26
Q
A