Cardiology Flashcards

1
Q

Mobitz type II heart block is usually the result of dysfunction in?

A

the His-Purkinje system

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2
Q

Mitral valve prolapse vs regurgitation

A

MVP: the mitral leaflets bulge into the left atrium every time the ventricles contract.
Late systolic crescendo murmur at the apex with mid-systolic click.

Mitral regurgitation the most common cause is a complication from mitral valve prolapse.
Murmur: holosystolic (pansystolic) murmur, heard best at the apex.

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3
Q

The most common cause of aortic stenosis in a young patient is?

A

a bicuspid aortic valve.

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4
Q

T waves and central nervous system pathology

A

Diffuse, deep, symmetrically inverted T waves may be seen in a severe central nervous system trauma or pathology.
These are called cerebral T waves: Ex, ischemic stroke, intracranial bleeding, and traumatic brain injury.
This could be explained by autonomic dysfunction and alterations in ventricular repolarization.

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5
Q

Cardiac Index (CI)

A

is a hemodynamic parameter that relates the cardiac output (CO) of a patient to their body surface area (BSA). It is expressed in liters per minute per square meter (L/min/m²).
The normal range: 2.5to4.0L/min/m²

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6
Q

The Pulmonary Capillary Wedge Pressure (PCWP)

A

Estimate the pressure in the left atrium of the heart.
6–12mmHg
Increased PCWP: left ventricular dysfunction or left-sided heart failure, can also suggest fluid overload or pulmonary congestion.
Decreased PCWP: may indicate hypovolemia or reduced preload.

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7
Q

Cardiac shock and positive pressure mechanical ventilation?

A

In patients who are in shock or have compromised cardiac output, initiation of positive pressure mechanical ventilation can result in a profound decrease in preload and worsen systemic perfusion.

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8
Q

Cholesterol emboli syndrome

A

Often follows angiography and is the result of a disrupted plaque moving down the blood stream and lodging in tiny vessels throughout the body.
Acute renal failure (eosinophiluria), pancreatitis, gastrointestinal symptoms, and a characteristic “blue toe,” due livedo reticularis.

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9
Q

Clinical manifestation of a right bundle branch block (RBBB)?

A

Widely split S2 heart sound.

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10
Q

For hemodynamically stable patients with ventricular tachycardia, the most appropriate next step in management is?

A

initiation of an amiodarone infusion.

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11
Q

Digoxin toxicity

A

Symptoms of digoxin toxicity include nausea, vomiting, blurry yellow vision, electrolyte abnormalities, and cardiac arrhythmias, increased PR intervals, decreased QT intervals, and T wave inversion.

Hypokalemia is a risk factor for digoxin toxicity due to competitive binding at the potassium site of the Na/K-ATPase pump.

Management: slow normalization of potassium and magnesium levels and administration of antibody fragments (Fab) targeting digoxin.

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12
Q

Medications that could cause Torsade de pointes

A

Erythromyicin, Clarithromycin, lexogloxacin, clindamycin, class 1 A and 3 antiarrhythmics, some antihistamines (astemizole, terfenadine), cocaine, methadone.

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13
Q

Wolff Parkinson White syndrome ekg descripction

A

Short PR interval with a slow upstroke of the QRS complex.

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14
Q

The most common cause of grade III systolic murmur in children with Wolff Parkinson white syndrome is?

A

Ebstein anomaly

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15
Q

A classic radiologic sign found with Coarctation of the aorta

A

Figure 3 sign, enlarged and distorded aortic knob and subclavian artery forming the number 3.

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16
Q

In tetralogy of Fallot what is correlated with the presence or absence of cyanosis after birth?

A

The degree of right ventricular outflow tract obstruction

17
Q

Obstructive shock

A

an extracardiac physical obstruction to the flow of the blood into or out of the heart.
for example, cardiac taponade, massive pulmonary embolism.

18
Q

Why do we administer calcium gluconate to patients with hyperkalemia?

A

To prevent global depolarization of the myocardium while correcting his electrolyte abnormalities.

19
Q

Large pulmonary embolism effects on the right ventricle

A

Increased right ventricular size, increased right ventricular wall tension, and decreased right ventricular perfusion.