bactierology Flashcards

1
Q

how do you treat LRT infections

A

systemic therapy +- nebulization therapy, hydration for mucociliary elevator, SC

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2
Q

in a TTW, what should cytology look like

A

low total number of cells and very few neuts

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3
Q

three ways to distinguish URT from LRT

A

clinical signs (RR), physical exam ascult, rads/CBC/US

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4
Q

what nonimmunological things prevent respiratory infections

A

anatomy and horizontal head position, normal flora, sneezing and coughing, mucociliary clearance

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5
Q

what is the major non immunological mechanism of the respiratory tract

A

mucociliary clearance defense

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6
Q

major defense mechanism of the alveoli

A

pulmonary alveolar macrophages that help clear out bacti that make it to the alveoli

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7
Q

why is GA a risk factor for bacterial respiratory infections

A

abnormal positioning

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8
Q

two ways bacti enter the respiratory tract

A

inhalation or hematogenous: diffuse lesion without a clear relationship to the airway

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9
Q

septic lymphadenitis in a horse is consistent with what bacterial infection

A

S. equi equi

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10
Q

where are strep equi located in carrier animals

A

gutteral pouch chondroids

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11
Q

treatment for strangles

A

drain abscess and SC unless its extremely severe case or extremely early then antibiotics

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12
Q

cervical lymphadenitis in pigs, horses and cats can be caused by what

A

streptococcus

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13
Q

bacteria likely responsible for the destruction of sneezing, nasal turbinates in young pigs

A

B. bronchiseptica damages mucosa then p. multocida D causes bone resorption

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14
Q

common tracheobronchial infections in dogs

A

kennel cough (bord. bronch)

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15
Q

what are the most important contagious diseases in horses

A

strangles

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16
Q

most important contagious disease in canine

A

canine infectious respiratory disease

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17
Q

unique features about mycobacterium

A

strict anaerobes, acid fast, intracellular in macrophages (hard to treat), zoonotic

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18
Q

what does M. bovis cause

A

tuberculosis in bovine and sometimes humans

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19
Q

bovine with cough, ADR, on and off fever, decreased weight, caseous necrosis of lung and lymph node

A

mycobacterium bovis

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20
Q

what specific histological diseases does M. bovis cause

A

granuloma with central necrosis with giant epitheloid cells

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21
Q

T/F we are working to erradicate m. bovis

A

false. wild pops keep it active

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22
Q

what to do with reactor positive cow to m. bovis

A

cull and isolate herd from other animals.

23
Q

T/F pneumonia in equine is a staged process

A

most of the time true. starts with initial bacterial compromise then the more serious bacti continue to move in as damage continues

24
Q

when do the strict anaerobes infect equine

A

around a week after initial pathogens infect. and they indicate poor prognosis.

25
what two bacteria responsible for septicemia can cause pneumonia
H. somni in cattle and salmonella dublin in calves
26
in the bloodstream where do we have macrophages that recognize bacteria
spleen and liver
27
T/F transient bacteriemia occurs with acute conditions that are cleared in a few hours
F they are acute infections that are cleared within 30 mins. very fast
28
what animals are the most susceptible
neonatal that have lost their mothers antibodies or that have FPT
29
two main routes bacteria enter the blood stream
direct inoculation or spread from initial site through lymphatics or vessels
30
what are the signs of traumatic reticulopericarditis
consistent with R CHF. distended jugular, brisket edema, bottle jaw
31
what is navel ill
septicemia from an umbilical infection. can seed into joints, lungs etc
32
what is the primary cause of death from an infection
getting septic shock or sepsis
33
what do you need to diagnose sepsis
suspicion or confirmed presence, then 2-3 of the SIRS criteria
34
gram stain and defining shape of bacillus anthracis
G+ rod that is huge and shaped very rectangular
35
how is anthrax transmitted
spores that are ingested, in haled or through the vegetative form (mostly human thing)
36
what helps B. anthracis cause disease?
capsule resists phagocytosis, and toxins either cause edema or death
37
who is most susceptible to b. anthracis and what toxin is major reactor
cattle and sheep most susceptible and the lethal toxin plays a major role. pigs are the exact opposite.
38
most common presenting signs in ruminants with b. anthracis
sudden death with tarry blood coming out all orifices
39
what are the three forms of anthrax in humans
cutaneous, pulmonary and GI
40
how to dispose of an anthrax positive body?
call state vet. bury deep with quick lime or incinerate
41
what are our obligate intracellular CV bacteria
anaplasma marginale, rickettsia rickettsii, ehrlichia canis, chlamydia psittici, mycoplasma haemofelis,
42
what cells does RMSF vs E. canis target?
endothelial cells and monocytes
43
what animals does anaplasmosis cause the most disease? icteric MM, weakness, fever, hemolytic anemia
older animals. calves are resistant
44
what is the treatment for RMSF vs E. canis
doxycycline, longer duration for erlichia (28 days)
45
dog vascular bacteria that causes fever, lymphadenopathy, petechial hemorrhage, edema and maybe ocular hemorrhage
RMSF and E. canis.
46
reservoir host for RMSF
wild rodents
47
reservoir host for E. canis
canids only domestic and wild
48
R. ricketsii vs E. canis. who can have subclinical carriers if they are not treated acutely or properly
E. canis.
49
how do you treat chlamydia psittici in pet birds vs food flocks
pets can have doxycycline but all food animal birds can get chlortetracycline
50
how does mycoplasmosis haemofelis infect
vectors or transplacental. is very superficial on RBC and can have variable shape (contrast anaplasma)
51
what is cat scratch fever and what is actually causing it?
bartonella henselae. in the fecal material of fleas. cats get flea dirt under their claws and get it into a human when they scratch you.
52
what is the importance of bartonellosis
it is an important ddx in dogs with valvular endocarditis. cats don't typically show any signs with infection
53