99. labour Flashcards

1
Q

Stages of labour?

A

Latent phase: irregular contractions, mucous plug, cervix beginning to efface up to 4cm

First stage: strong regular uterine contractions, cervical dilatation up to 10cm

Second stage: from full dilation to delivery of baby

Third stage: from birth of baby to expulsion of placenta

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Define labour

A

regular painful contractions with cervical changes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

mechnism of labour

A

Descent
Flexion
Internal rotation
Extension
Restitution
External rotation
Delivery of body

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

cardinal movements of labour

A

Descent
Flexion
Internal rotation
Extension
Restitution
External rotation
Delivery of body

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Ideal foetal position

A

occiput anterior

left occiput anterior is ideal

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

entonox

A

gas and air

Gas and air contains a mixture of 50% nitrous oxide and 50% oxygen. This is used during contractions for short term pain relief.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

IM diamorphine vs IM pethidine

A

Pethidine shorter

Diamorphine longer and more powerful

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Braxton-Hicks Contractions

A

occasional irregular contractions of the uterus. They are usually felt during the second and third trimester.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Diagnosing labour

A

Show (mucus plug from the cervix) clear or bloody show

Rupture of membranes

Regular, painful contractions

Dilating cervix on examination

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Active management of the third stage

A

intramuscular oxytocin

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Rupture of membranes (ROM)

A

The amniotic sac has ruptured.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Spontaneous rupture of membranes (SROM)

A

The amniotic sac has ruptured spontaneously

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Prelabour rupture of membranes (PROM)

A

The amniotic sac has ruptured before the onset of labour.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Preterm prelabour rupture of membranes (P‑PROM)

A

The amniotic sac has ruptured before the onset of labour and before 37 weeks gestation (preterm).

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Prolonged rupture of membranes (also PROM)

A

The amniotic sac ruptures more than 18 hours before delivery.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

cervical length less than 25mm on vaginal ultrasound between 16 and 24 weeks gestation

A

vaginal progesterone to maintain pregnancy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

cervical length less than 25mm on vaginal ultrasound between 16 and 24 weeks gestation, who have had a previous premature birth or cervical trauma (e.g. colposcopy and cone biopsy)

A

Cervical cerclage

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Complications of PPROM

A

fetal: prematurity, infection, pulmonary hypoplasia

maternal: chorioamnionitis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Investigation PPROM (premature, prelabour)

A

A sterile speculum examination should be performed (to look for pooling of amniotic fluid in the posterior vaginal vault)

but digital examination should be avoided due to the risk of infection.

Ultrasound may also be useful to show oligohydramnios.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

Management PPROM prolonged

A
  1. admission
  2. regular observations to ensure chorioamnionitis is not developing
  3. oral erythromycin should be given for 10 days 250mg four times daily
  4. antenatal corticosteroids should be administered to reduce the risk of respiratory distress syndrome
  5. delivery should be considered at 34 weeks of gestation - there is a trade-off between increased risk of maternal chorioamnionitis with a decreased risk of respiratory distress syndrome as the pregnancy progresses
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

Preterm labour with intact membranes

A

regular painful contraction and cervical dilatation, without rupture of the amniotic sac.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

Tests if diagnostic doubt about PPROM premature prelabour

A

Insulin-like growth factor-binding protein-1 (IGFBP-1)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

Diagnosis of preterm labour with intact membranes

A

Less than 30 weeks gestation, clinical assessment alone is enough to offer management of preterm labour.

More than 30 weeks gestation, a transvaginal ultrasound can be used to assess the cervical length. When the cervical length on ultrasound is less than 15mm, management of preterm labour can be offered. A cervical length of more than 15mm indicates preterm labour is unlikely.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

Tocolysis

A

involves using medications to stop uterine contractions. Nifedipine, a calcium channel blocker, is the medication of choice for tocolysis. Atosiban is an oxytocin receptor antagonist that can be used as an alternative when nifedipine is contraindicated.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
When is tocolysis used
Tocolysis can be used between 24 and 33 + 6 weeks gestation in preterm labour to delay delivery and buy time for further fetal development, administration of maternal steroids or transfer to a more specialist unit (e.g. with a neonatal ICU). It is only used as a short term measure (i.e. less than 48 hours).
26
Bishop score
scoring system used to determine whether to induce labour
27
Score of 8 or more on bishop score
predicts a successful induction of labour. A score below this suggests cervical ripening may be required to prepare the cervix.
28
Options for induction of labour
1. membrane sweep 2. vaginal prostaglandin E2 3. Cervical ripening balloon 4. Artificial rupture of membranes
29
complication of induction of labour with vaginal prostaglandins?
Uterine hyperstimulation
30
Criteria for uterine hyperstimulation
Individual uterine contractions lasting more than 2 minutes in duration More than five uterine contractions every 10 minutes
31
Management uterine hyperstimulation
Removing the vaginal prostaglandins, or stopping the oxytocin infusion Tocolysis with terbutaline
32
Indications for continuous CTG monitoring
Sepsis Maternal tachycardia (> 120) Significant meconium Pre-eclampsia (particularly blood pressure > 160 / 110) Fresh antepartum haemorrhage Delay in labour Use of oxytocin Disproportionate maternal pain
33
5 key features to look for in CTG
Contractions – the number of uterine contractions per 10 minutes Baseline rate – the baseline fetal heart rate Variability – how the fetal heart rate varies up and down around the baseline Accelerations – periods where the fetal heart rate spikes Decelerations – periods where the fetal heart rate drops
34
Reassuring baseline rate and variability
110-160 5-25
35
Non-reassuring base line rate and variability
100-109 or 160-180 <5 for 30-50 mins >25 for 15-25 mins
36
Abnormal baseline rate and variability
<100 or >180 Less than 5 for over 50 minutes or More than 25 for over 25 minutes
37
what are accelerations a sign of?
Accelerations are generally a good sign that the fetus is healthy, particularly when occurring alongside contractions of the uterus
38
Early decelerations
gradual dips and recoveries in heart rate that correspond with uterine contractions. The lowest point of the declaration corresponds to the peak of the contraction. Early decelerations are normal and not considered pathological. They are caused by the uterus compressing the head the fetus, stimulating the vagus nerve of the fetus, slowing the heart rate.
39
Late decelerations
are gradual falls in heart rate that starts after the uterine contraction has already begun. There is a delay between the uterine contraction and the deceleration. The lowest point of the declaration occurs after the peak of the contraction. Late decelerations are caused by hypoxia in the fetus, and are a more concerning finding. They may be caused by excessive uterine contractions, maternal hypotension or maternal hypoxia. urgent fetal blood sampling
40
Variable decelerations
abrupt decelerations that may be unrelated to uterine contractions. There is a fall of more than 15 bpm from the baseline. The lowest point of the declaration occurs within 30 seconds, and the deceleration lasts less than 2 minutes in total. Variable decelerations often indicate intermittent compression of the umbilical cord, causing fetal hypoxia. Brief accelerations before and after the deceleration are known as shoulders, and are a reassuring sign that the fetus is coping.
41
Prolonged decelerations
last between 2 and 10 minutes with a drop of more than 15 bpm from baseline. This often indicates compression of the umbilical cord, causing fetal hypoxia. These are abnormal and concerning.
42
4 types of decelerations
early late variable prolonged
43
A CTG is normal with respect to decelerations when...
no decelerations, early decelerations or less than 90 minutes of variable decelerations with no concerning features.
44
Four categories of CTG?
Normal Suspicious: a single non-reassuring feature Pathological: two non-reassuring features or a single abnormal feature Need for urgent intervention: acute bradycardia or prolonged deceleration of more than 3 minutes
45
Management of prolonged fetal bradycardia
3 minutes – call for help 6 minutes – move to theatre 9 minutes – prepare for delivery 12 minutes – deliver the baby (by 15 minutes)
46
Sinusoidal CTG
A sinusoidal CTG is a rare pattern to be aware of, as it can indicate severe fetal compromise. It gives a pattern similar to a sine wave, with smooth regular waves up and down that have an amplitude of 5 – 15 bpm. It is usually associated with severe fetal anaemia, for example, caused by vasa praevia with fetal haemorrhage.
47
Patient Controlled Analgesia
Patient-controlled intravenous remifentanil. This involves the patient pressing a button at the start of a contraction to administer a bolus of this short-acting opiate medication. Patient-controlled analgesia requires careful monitoring. There needs to be input from an anaesthetist, and facilities in place if adverse events occur. This includes access to naloxone for respiratory depression, and atropine for bradycardia.
48
adverse effects of epidural
Headache after insertion Hypotension Motor weakness in the legs Nerve damage Prolonged second stage Increased probability of instrumental delivery
49
What is cord prolapse
The umbilical cord descends below the presenting part of the fetus and through the cervix into the vagina, after rupture of the fetal membranes. There is a significant danger of the presenting part compressing the cord, resulting in fetal hypoxia.
50
Most significant risk factor for cord prolapse
abnormal lie after 37 weeks gestation (i.e. unstable, transverse or oblique)
51
When should you suspect cord prolapse
Where there are signs of fetal distress on the CTG
52
Management of cord prolapse
1. oragnise emergency CS 2. Don't handle cord or push it back in 3. Push presenting part upwards to prevent it compressing the cord 4. woman can lie in the left lateral position (with a pillow under the hip) or the knee-chest position (on all fours), using gravity to draw the fetus away from the pelvis and reduce compression on the cord. 5. Tocolytic medication (e.g. terbutaline) can be used to minimise contractions whilst waiting for delivery by caesarean section.
53
Management of shoulder dystocia
HELPERR(R) H- call for help E - evaluate for episiotomy L - legs in McRoberts P - Suprapubic pressure E - Enter pelvis R - rotational manoeuvre R - remove posterior arm (R - replace head and CS)
54
What medication is given after instrumental delivery?
single dose of co-amoxiclav
55
Indictions for instrumental delivery
Failure to progress Fetal distress Maternal exhaustion Control of the head in various fetal positions
56
Risks to baby with instrumental delivery
Cephalohaematoma with ventouse Facial nerve palsy with forceps Rarely: Subgaleal haemorrhage (most dangerous) Intracranial haemorrhage Skull fracture Spinal cord injury
57
Which of the mothers nerves may be compressed with forceps delivery
Femoral nerve Obturator nerve
58
First-degree tear
Injury limited to the frenulum of the labia minora (where they meet posteriorly) and superficial skin
59
Second degree tear
including the perineal muscles, but not affecting the anal sphincter
60
Third degree tear
including the anal sphincter, but not affecting the rectal mucosa 3A = <50% of external anal sphincter 3B = >50% of the external anal sphincter 3C = external and internal sphincters affected
61
Fourth degree tear
including the rectal mucosa
62
Management first degree tears
First-degree tears usually do not require any sutures.
63
Management of secondary degree tears
sutures on ward
64
Management of third and fourth degree tears
sutures in theatre
65
Preventing tears
Perineal massage is a method for reducing the risk of perineal tears. It involves massaging the skin and tissues between the vagina and anus (perineum). This is done in a structured way from 34 weeks onwards to stretch and prepare the tissues for delivery.
66
When is active management of the 3rd stage indicated
Active management is routinely offered to all women to reduce the risk of postpartum haemorrhage. It is also initiated if there is: - Haemorrhage - More than a 60-minute delay in delivery of the placenta (prolonged third stage)
67
Steps of active management of the third stage
1. IM oxytocin 10IU 2. Controlled cord traction 3. After placenta delivered, massage uterus until firm
68
Definiton PPH
bleeding after delivery of the baby and placenta 500ml after a vaginal delivery 1000ml after a caesarean section
69
Severity classifications PPH
Minor PPH – under 1000ml blood loss Moderate PPH – 1000 – 2000ml blood loss Severe PPH – over 2000ml blood loss
70
Priamary vs secondary PPH
Primary PPH: bleeding within 24 hours of birth Secondary PPH: from 24 hours to 12 weeks after birth
71
Causes of PPH
T – Tone (uterine atony – the most common cause) T – Trauma (e.g. perineal tear) T – Tissue (retained placenta) T – Thrombin (bleeding disorder)
72
Most common cause PPH
uterine atony
73
Preventing PPH
Treating anaemia during the antenatal period Giving birth with an empty bladder (a full bladder reduces uterine contraction) Active management of the third stage (with intramuscular oxytocin in the third stage) Intravenous tranexamic acid can be used during caesarean section (in the third stage) in higher-risk patients
74
Management PPH
1. stabilise 2. mechanical: - rubbing the fundus - catherterisation 3. medical: - oxytocin slow IV injection --> continuous infusion IV - ergometrine slow IV or IM (contraindicated in hypertension) - carboprost IM (CAUTION IN ASTHMA) - misoprostol sublingual - tranexamic acid 4. surgical: - Intrauterine balloon tamponade - B-Lynch suture - Uterine artery ligation - Hysterectomy is the “last resort” the RCOG state that the intrauterine balloon tamponade is an appropriate first-line ‘surgical’ intervention for most women where uterine atony is the only or main cause of haemorrhage
75
Investigations secondary PPH
Ultrasound for retained products of conception Endocervical and high vaginal swabs for infection
76
Causes secondary PPH
RPOC infection
77
Management secondary PPH
Surgical evaluation of retained products of conception Antibiotics for infection
78
What PPH drug should you be cautious of in HTN
Ergometrine
79
What PPH drug should you be cautious of in asthma
Carboprost
80
Category 3 CS
Delivery is required, but mother and baby are stable.
81
category 4 CS
This is an elective caesarean, as described above.
82
Category 2 CS
There is not an imminent threat to life, but caesarean is required urgently due to compromise of the mother or baby. Decision to delivery time is 75 minutes.
83
Category 1 CS
There is an immediate threat to the life of the mother or baby. Decision to delivery time is 30 minutes.
84
Specific risks of spinal
Nerve damage (spinal anaesthetic) Haematoma (spinal anaesthetic)
85
Why are H2 receptor antagonists (e.g. ranitidine) or proton pump inhibitors (e.g. omeprazole) given before caeserean section?
risk of aspiration pneumonitis during caesarean section, caused by acid reflux and aspiration during the prolonged period lying flat. H2 receptor antagonists (e.g. ranitidine) or proton pump inhibitors (e.g. omeprazole) are given before the procedure to reduce the risk of this happening.
86
What drugs are used in CS to prevent complications
H2 receptor antagonists (e.g. ranitidine) or proton pump inhibitors (e.g. omeprazole) before the procedure Prophylactic antibiotics during the procedure to reduce the risk of infection Oxytocin during the procedure to reduce the risk of postpartum haemorrhage Venous thromboembolism (VTE) prophylaxis with low molecular weight heparin
87
Sepsis 6
Three tests: Blood lactate level Blood cultures Urine output Three treatments: Oxygen to maintain oxygen saturations 94-98% Empirical broad-spectrum antibiotics IV fluids
88
Two key causes of sepsis in pregancy
Chorioamnionitis Urinary tract infections
89
In maternal sepsis, if there was fetal distress, how would the baby be delivered
Emergency caesarean section may be indicated when there is fetal distress, guided by a senior obstetrician. General anaesthesia is usually required for women with sepsis, as spinal anaesthesia is avoided.
90
Management maternal sepsis
Very heavy-hitting antibiotics are required, needing to cover gram-positive, gram-negative and anaerobes. There are also significant consequences of inadequate treatment. Example regimes include piperacillin and tazobactam (tazocin) plus gentamicin, or amoxicillin, clindamycin and gentamicin.
91
Acute onset, around time of delivery Shortness of breath Hypoxia Hypotension Coagulopathy Haemorrhage Tachycardia Confusion Seizures Cardiac arrest
amniotic fluid embolisation
92
Management amniotic fluid embolisation
supportive ABCDE CPR immediate caeserean
93
What is uterine rupture?
the muscle layer of the uterus (myometrium) ruptures. With an incomplete rupture, or uterine dehiscence, the uterine serosa (perimetrium) surrounding the uterus remains intact. With a complete rupture, the serosa ruptures along with the myometrium, and the contents of the uterus are released into the peritoneal cavity.
94
Presentation of uterine rupture
Uterine rupture presents with an acutely unwell mother and abnormal CTG. It may occur with induction or augmentation of labour, with signs and symptoms of: Abdominal pain Vaginal bleeding Ceasing of uterine contractions Hypotension Tachycardia Collapse
95
Management uterine rupture
1. stabilise/resus 2. transfusion? 3. emergency Caesarean and repair
96
Risk factors for uterine rupture
Vaginal birth after caesarean (VBAC) Previous uterine surgery Increased BMI High parity Increased age Induction of labour Use of oxytocin to stimulate contractions
97
Presentation of uterine inversion
Uterine inversion typically presents with a large postpartum haemorrhage. There may be maternal shock or collapse. An incomplete uterine inversion may be felt with manual vaginal examination. With a complete uterine inversion, the uterus may be seen at the introitus of the vagina.
98
why shouldn't you pull too hard on the cord during active management of the 3rd stage of labour
risk of uterine inversion
99
Management uterine inversion
1. Johnson manoeuvre (pushing it back up) 2. hydrostatic measures 3. surgery
100
VBAC
planned VBAC is an appropriate method of delivery for pregnant women at >= 37 weeks gestation with a single previous Caesarean delivery around 70-75% of women in this situation have a successful vaginal delivery contraindications include previous uterine rupture or classical caesarean scar
101
GBS prophylaxis in labour
benzylpenicillin
102
what to do if late decelerations
fetal blood sampling A pH of >7.2 in labour is considered normal. Urgent delivery should be considered if there is fetal acidosis.
103
Layers you cut through in c-section
Skin Subcutaneous tissue :Superficial fascia, Deep fascia Rectus sheath Rectus abdominis muscle (not cut, rather pushed laterally following incision of the linea alba) Parietal peritoneum Uterus
104
Indications for continuous CTG monitoring
As per NICE guidelines; the following would warrant continuous CTG monitoring if any of the following are present or arise during labour; suspected chorioamnionitis or sepsis, or a temperature of 38°C or above severe hypertension 160/110 mmHg or above oxytocin use the presence of significant meconium fresh vaginal bleeding that develops in labour - this was a new point added to the guidelines in 2014
105
Management of failure to progress
Amniotomy, also known as artificial rupture of membranes (ARM) for women with intact membranes Oxytocin infusion Instrumental delivery Caesarean section
106
what is considered delay in the 1st stage?
Less than 2cm of cervical dilatation in 4 hours Slowing of progress in a multiparous women
107
what is a parotogram
Women are monitored for their progress in the first stage of labour using a partogram. Recorded on a partogram are: Cervical dilatation (measured by a 4-hourly vaginal examination) Descent of the fetal head (in relation to the ischial spines) Maternal pulse, blood pressure, temperature and urine output Fetal heart rate Frequency of contractions Status of the membranes, presence of liquor and whether the liquor is stained by blood or meconium Drugs and fluids that have been given
108
alert and action lines partogram
Alert line is an indication for amniotomy (artificially rupturing the membranes) and a repeat examination in 2 hours. Crossing the action line means care needs to be escalated to obstetric-led care and senior decision-makers for appropriate action. theres an o in action and obsteric
109
delay in second stage?
2 hours in a nulliparous woman 1 hour in a multiparous woman
110
delay in 3rd stage?
More than 30 minutes with active management More than 60 minutes with physiological management