41. Change in hearing Flashcards

1
Q

most common causes of hearing loss

A

ear wax, otitis media and otitis externa.

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2
Q

differentials hearing loss

A

Conductive hearing loss
(otitis media)
Glue ear
Otosclerosis
Impacted wax
Eustachian tube dysfunction

Sensorineural hearing loss
Presbycusis - Age related
Occupational
(Meniere’s disease)
(labyrinthitis) (Meningitis → labyrinthitis)
Drug toxicity
(Acoustic neuroma)

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3
Q

history taking hearing loss

A
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4
Q

examination hearing loss

A
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5
Q

causes conductive hearing loss

A

(otitis media)
Glue ear
Otosclerosis
Impacted wax
Eustachian tube dysfunction

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6
Q

causes sensorineural hearing loss

A

Presbycusis - Age related
Occupational
(Meniere’s disease)
(labyrinthitis) (Meningitis → labyrinthitis)
Drug toxicity
(Acoustic neuroma)

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7
Q

what is primary tinnitus

A

no identifiable cause and often occurs with sensorineural hearing loss

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8
Q

what is secondary tinnitus?

A

identifiable cause:

Impacted ear wax
Ear infection
Ménière’s disease
Noise exposure
Medications (e.g., loop diuretics, gentamicin and chemotherapy drugs such as cisplatin)
Acoustic neuroma
Multiple sclerosis
Trauma
Depression

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9
Q

what systemic conditions can cause tinnitus

A

Anaemia
Diabetes
Hypothyroidism or hyperthyroidism
Hyperlipidaemia

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10
Q

red flags tinnitus

A

Unilateral tinnitus
Pulsatile tinnitus
Hyperacusis (hypersensitivity, pain or distress with environmental sounds)
Associated unilateral hearing loss
Associated sudden onset hearing loss
Associated vertigo or dizziness
Headaches or visual symptoms
Associated neurological symptoms or signs (e.g., facial nerve palsy or signs of stroke)
Suicidal ideation related to the tinnitus

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11
Q

Invetsigation tinnitus

A

Full blood count (anaemia)
Glucose (diabetes)
TSH (thyroid disorders)
Lipids (hyperlipidaemia)

Audiology can be used to assess the hearing in detail and help establish the underlying cause.

Imaging (e.g., CT or MRI) may be rarely required to investigate for underlying causes such as vascular malformations or acoustic neuromas.

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12
Q

management tinnitus

A

Tinnitus tends to improve or resolve over time without any interventions.

Underlying causes of tinnitus can be treated, such as impacted ear wax or infection.

Several measures can be used to help improve and manage symptoms:

Hearing aids
Sound therapy (adding background noise to mask the tinnitus)
Cognitive behavioural therapy

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13
Q

newborn hearing test

A

Otoacoustic emission test

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14
Q

what test is done if otoacoustic emission test is abnormal

A

Auditory Brainstem Response test

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15
Q

hearing 6-9 months

A

Distraction test

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16
Q

heairng testing > 3 years

A

Pure tone audiometry

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17
Q

complications of hearing impairment/unrecognised hearing impairment in children

A

Poor speech development
Impaired learning
Mental health
Learning difficulties

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18
Q

causes of hearing loss in chidlren

A

Congenital:
Maternal rubella or cytomegalovirus infection during pregnancy
Genetic deafness can be autosomal recessive or autosomal dominant
Associated syndromes, for example Down’s syndrome

Perinatal:
Prematurity
Hypoxia during or after birth

After birth:
Jaundice
Meningitis and encephalitis
Otitis media or glue ear
Chemotherapy

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19
Q

audiogram interpretation

A

20dB
anything above the 20dB line is normal

sensioneural = bone and air impaired so both below line

conduction = only air is impaired so air below line and bone above line

20
Q

what is glue ear

A

otitis media with effusion. The middle ear becomes full of fluid, causing a loss of hearing in that ear.

21
Q

ototscopy glue ear

A

dull tympanic membrane with air bubbles or a visible fluid level, although it can look normal.

22
Q

investigation glue ear

A

Referral for audiometry to help establish the diagnosis and extent of hearing loss.

23
Q

management glue ear

A
  1. active observation: the management for a child with a first presentation of otitis media with effusion is active observation for 3 months - no intervention is required
  2. grommet insertion - to allow air to pass through into the middle ear and hence do the job normally done by the Eustachian tube. The majority stop functioning after about 10 months
  3. adenoidectomy
24
Q

Presentation eustachain tube dysfunction

A

PC: reduced or altered hearing, popping noises/sensations in ear, fullness in ear, pain/discomfort, tinnitus

25
pathophysiology eustachian tube dysfunction
When the Eustachian tube is not functioning correctly or becomes blocked, the air pressure cannot equalise properly and fluid cannot drain freely from the middle ear. The air pressure between the middle ear and the environment can become unequal. The middle ear can fill with fluid.
26
invetsigation eustachain tube dysfunction
If typical history and trigger: clinical diagnosis If persistent: Tympanometry, audiometry, nasopharyngoscopy, CT
27
management eustachian tube dysfunction
Will resolve spontaneously with no treatment Valsalva manoeuvre (holding the nose and blowing into it to inflate the Eustachian tube) Decongestant nasal spray Antihistamine and a steroid nasal spray for allergic rhinitis Otovent is an over the counter device where the patient blows into a balloon using a single nostril, which can help inflate the Eustachian tube, clear blockages and equalise pressure. Surgical Treating any other pathology that might be causing symptoms, for example, adenoidectomy (removal of the adenoids) Grommets Balloon dilatation Eustachian tuboplasty
28
what is otosclerosis
Otosclerosis describes the replacement of normal bone by vascular spongy bone. It causes a progressive conductive deafness due to fixation of the stapes at the oval window.
29
inheritance otosclerosis
Otosclerosis is autosomal dominant and typically affects young adults
30
presentation otosclerosis
Onset is usually at 20-40 years - features include: conductive deafness tinnitus tympanic membrane the majority of patients will have a normal tympanic membrane 10% of patients may have a 'flamingo tinge', caused by hyperaemia positive family history
31
management otosclerosis
hearing aid stapedectomy
32
management impacted ear wax
olive oil sodium bicarbonate 5% almond oil
33
how may impacted ear wax present
pain conductive hearing loss tinnitus vertigo
34
sudden onset sensorineural hearing loss plan
urgent referral to ENT. The majority of SSNHL cases are idiopathic. High-dose oral corticosteroids are used by ENT for ALL cases of SSNHL.
35
what type of HL does otosclerosis cause
conductive
36
what type of HL does presbycusis cause
SN
37
what type of noise is difficult to hear presbycusis
high-frequency hearing is affected bilaterally, which can lead to conversational difficulties, particularly in noisy environments.
38
what type of noise is difficult to ehar in otoscleorsis
It tends to affect the hearing of lower-pitched sounds more than higher-pitched sounds. Female speech may be easier to hear than male speech (due to the generally higher pitch). This is the reverse of the pattern seen in presbycusis.
39
pathophysiology presbycusis
sensory hair cells and neurons in the cochlea atrophy over time The precise cause is unknown however is likely multifactorial Arteriosclerosis: May cause diminished perfusion and oxygenation of the cochlea, resulting in damage to inner ear structures Diabetes: Acceleration of arteriosclerosis Accumulated exposure to noise Drug exposure (Salicylates, chemotherapy agents etc.) Stress Genetic: Certain individuals may be programmed for the early ageing of the auditory system
40
presentation presbycusis
chronic, slowly progressing history of: Speech becoming difficult to understand Need for increased volume on the television or radio Difficulty using the telephone Loss of directionality of sound Worsening of symptoms in noisy environments Hyperacusis: Heightened sensitivity to certain frequencies of sound (Less common) Tinnitus (Uncommon)
41
investigations presbycusus
Audiometry
42
what frequencies are particualrly affected occupational hearing loss
3000-6000 Hz
43
what drugs are ototoxic
aminoglycosides (e.g. Gentamicin), furosemide, aspirin and a number of cytotoxic agents
44
What is the nerve found within the middle ear?
facial nerve
45