Week 11 to Week 14 Flashcards

1
Q

Supports & Subluxation :

What are reasons for prescription for slings/supports?

A

Weakness, subluxation, hypotonia, pain, neglect, decreased cognition, Pain and subluxation for orthoses

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2
Q

Supports & Subluxation :

Reasons for discontinued use:

A

Improvement in strength

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3
Q

Summarize Article Continued (Foongchomcheay, Ada, & Canning, 2005):

A

Summary:
Supportive devices commonly prescribed by Australian physiotherapists to prevent subluxation

Slings most commonly prescribed

Wheelchair/chair attachments also widely used

Strapping also common

Best for preventing subluxation: Lab tray, arm trough, triangular sling, and Harris sling (sling for standing only/temporary)

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4
Q

Taping for Subluxation:

Taping Overview

A

Beneficial for shoulder impingement, joint sprains, and multi-directional instability

Reposition humeral head with tape to that it is in a neutral position

Taping allows client to feel normal alignment

Can result in immediate pain relief and improved ROM

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5
Q

Taping for Subluxation:

Article: (Peters & Lee, 2003)

A

Case study of shoulder taping on individual with R hemiplegic UE following CVA

Taping significantly reduced pain and relieved tension

Taping method: tri pull method = 3 pieces of tape

1) Mid humerus deltoid tuberosity across the scapula near T3 spinous process
2) Deltoid tuberosity across clavicle to mid clavicle
3) Deltoid tuberosity over acromion process to the neck

Outcome measures:
Results:

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6
Q

CA tri pull method

position

A

1) Medial = 1 inch below deltoid tuberosity - 1 inch above acromial process
2) Posterior = 1 inch below deltoid tuberosity - 1 inch above spine of scapula
3) Anterior = 1 inch below deltoid tuberosity - 1 inch above coracoid process

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7
Q

E-stim & Subluxation

A

May improve muscle strength, joint alignment, spasticity, and sensory deficits

Rationale = allows muscle activity to maintain the glenohumeral joint

No significant evidence of pain reduction with e-stim, did improve pain free PROM

No negative effects found

Functional e-stim found to be effective in reducing shldr subluxation. Most effective early on

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8
Q

Orthotics: Principles

A

Orthotics: Principles
Used to maintain or increase the length of soft tissues by preventing or lengthening shortened tissues and preventing overstretching of the antagonist

Used to correct biomechanical malalignment and protecting joint integrity

Used to position the hand to assist in functional tasks

Used to promote independence in specific areas of occupation

Compensate for weakness by providing an external support

Limited Evidence
Evidence currently does not support one style of orthosis over another
(Gillen, 2016)

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9
Q

Considerations for Prescribing & Designing Orthotics

A

Spasticity
Orthosis may prevent painful contractures and loss of tissue length
Serve to provide a stretch to the distal UE
TX should begin before spasticity becomes severe

Those with severe spasticity should not use Orthosis - Why?

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10
Q

Considerations for Prescribing & Designing Orthotics: Soft Tissue Shortening

A

Evaluate Extrinsics:
Extend the wrist with digits flexed, wrist in ext. and attempt to extend the digits
Next flex the wrist palm upwards

Evaluate Intrinsics:
Normal: MCP are flexed and IP joints are extended
Normal: MCP are extended and IP are flexed
Hold MCP joint in ext and attempt to flex the PIP joint

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11
Q

Extrinsic extensor tightness:

A

full passive composite wrist and digit flexion is not obtainable.

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12
Q

Extrinsic flexor tightness:

A

full passive composite wrist and digit extension is not obtainable.

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13
Q

Intrinsic tightness:

A

PIPs & DIPs can be fully passively flexed when MCP’s are in a position of flexion (puts the interossei on slack)

PIPs & DIPs cannot be fully passively flexed when MCP’s are in a position of extension

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14
Q

Considerations for Prescribing & Designing Orthotics

A

Low Load Prolonged Stress (LLPS)

LLPS in when the tissue is held in a low lengthened position for a total end range time (TERT).

TERT = 1-2 hrs, 3 -4 hours, ideally progresses to 6 - 8 hrs

Stretching manually is not enough and must be followed up with orthotic devices

Orthotic must be readjusted (weekly) to ensure prolonged stretch is occurring

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15
Q

Orthotics: Application Consideration: Normal Posture of the Distal UE

A

Forearm neutral (midway between supination and pronation)

Wrist 10 - 15 degrees of ext

Thumb slight ext and abd, MCP and IP flexxed 15 - 20 degrees

2nd metacarpal aligned w/ radius

Digits: all joints in slight (10-20 degrees) flexion

Palmar arch maintained

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16
Q

Orthotics: Application Consideration: Typical Deviations after Stroke

Wrist Flexion =

A
Wrist Flexion = 
flattened palmer arches
passive digit extension
shortened collateral ligaments @ MPs 
narrowed thumb web space
decreased grip
decreased wrist deviation ROM
Edema
shortening of wrist & extrinsic digit flexors
lengthening of wrist & extrinsic digit extensors
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17
Q

Orthotics: Application Consideration: Typical Deviations after Stroke

Wrist & digit flexion

A
shortened extrinsic flexors
lengthened extrinsic extensors
decreased normal tenodesis action
contractures and deformity
skin maceration risk
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18
Q

Orthotics: Application Consideration: Typical Deviations after Stroke

Extreme ulnar deviation

A

decreased wrist /
shortened tissues ulnar FA
lengthened tissues radial FA
shift of carpal rows

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19
Q

Orthotics: Application Consideration: Typical Deviations after Stroke

Loss of palmar arches

A

decreased grip

decreased dexterity

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20
Q

TBI

A

The most common cause of death and disability among young people

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21
Q

TBI: Open

A

penetrating injury or perforating

Injury depends on shape, mass, direction, and velocity of the object

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22
Q

TBI: Closed

A

Closed direct or indirect impact without penetration

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23
Q

TBI: Blast injuries =

A

= can occur in conjunction with open or closed

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24
Q

Pathology: Focal

A

Caused by a direct blow to the head with an external object or fall, penetrating injury from a weapon, collision of the brain with the inner tables of the skull

Common findings from falls = intracerebral and brain surface contusions (inferior and dorsal-lateral frontal lobes, anterior and medial temporal lobes, and less common inferior cerebellum)

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25
Pathology: Diffuse vs. Focal: Focal
Caused by a direct blow to the head with an external object or fall, penetrating injury from a weapon, collision of the brain with the inner tables of the skull Common findings from falls = intracerebral and brain surface contusions (inferior and dorsal-lateral frontal lobes, anterior and medial temporal lobes, and less common inferior cerebellum)
26
Pathology: Diffuse vs. Focal: Focal
Caused by a direct blow to the head with an external object or fall, penetrating injury from a weapon, collision of the brain with the inner tables of the skull Common findings from falls = intracerebral and brain surface contusions (inferior and dorsal-lateral frontal lobes, anterior and medial temporal lobes, and less common inferior cerebellum)
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Pathology: Diffuse vs. Focal: Coup
(the site of direct injury)
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Pathology: Diffuse vs. Focal: | Countercoup
(site of indirect injury)
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Epidural hematomas (EDHs
associated with skull fractures and disruption of meningeal arteries
30
Subdural hematomas (SDHs) =
occur between the dura and brain surface due to tearing of bridging veins
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Location of trauma dictates symptoms:
Pre-frontal & anterior temporal areas: impaired memory, emotion and motivation Orbitofrontal area: impulsivity Frontolateral cortex:impassivity, hemiparesis, impaired attention and mental flexibility
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Multifocal & Diffuse Brain injury
Often caused by sudden deceleration of the body and head with variable forces and deeper portions of the brain
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Intracerebral hemorrhage (ICH)
nearly always present with missile wounds and common after falls and assault
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Subarachnoid hemorrhage (SAH) & Intraventricular hemorrhage (IVH)
occur when the pia or arachnoid is torn.
35
Diffuse axonal injuries (DAIs)/ Traumatic axonal injury (TAI) =
= prototypic lesions caused by rapid deceleration and rotation of the brain in the skull
36
Possible symptoms include:
``` Ataxia Diplopia Dysarthria Impulsivity, irritability Apathy, poor initiative Decreased mental processing speed & efficiency Impaired attention Impaired abstract reasoning, planning, problem solving ```
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Primary injury =
= occurs at the time of trauma
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Secondary injury=
Occur as a result of the effects of brain swelling in a closed space, loss of perfusion, and decreased delivery of oxygen The American Association of Neurological Surgeons has developed guidelines for management of severe TBI to minimize secondary injury Resuscitation of blood pressure and oxygenation, management of elevated intracranial pressure, nutrition after acute trauma and seizure prophylaxis Secondary effects (Radomski, 2008): hypoxia, hypotension, hypothermia, and hyperthermia (most common)
39
State of Consciousness
TBI typically results in an altered level of consciousness From Coma to conscious awareness Progression along this continuum varies depending on age, previous health, severity of injury, and medical/therapeutic/environmental management.
40
Coma =
As coma resolves client is either partially aware (minimally conscious) or if no awareness = vegetative
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Vegetative State =
- Wakefulness without awareness - Characteristics: no awareness of self or environment, inability to interact with others, no sustained or voluntary behavioral responses, no language comprehension, sleep wake cycle varies, ability to regulate temp, breathing, and circulation with medical care, incontinence of bowels and bladder, variably preserved cranial nerves and spinal reflexes
42
Minimally Conscious State (MCS)
- Evidence of awareness of self and/or environment. - Must have 1 of the following: ability to follow commands, gesture or verbal yes/no responses, intelligible verbalization, and purposeful movement
43
Post Traumatic Amnesia (PTA)
- Single best measurable predictor of functional outcomes - Length of time from the injury to the moment the individual regains ongoing memory of daily events - Longer PTA = poorer outcomes (cognitive and motor abilities, and function)
44
Intracranial Pressure
When the brain is injured, it reacts like other parts of the body that may be injured…it swells. Unfortunately, the cranial vault where the brain sits inside the skull has a limited volume of about 1,400 milliliters. When the brain swells, it can damage and kill neurons by squeezing them and stopping oxygen from reaching the cells. If the (ICP) pressure gets too much, the brain can be forced into the hole at the base of the brain, the foramen magnum, and compress the brainstem. The brain stem is  where the consciousness, breathing, and heart rate are controlled. Damage here can result in coma or death.
45
Second Impact Syndrome (SIS)
Occurs when an individual suffers a second head injury before the initial injury has fully healed…leads to diffuse cerebral swelling Can potentially result in death within minutes Rare enough condition that its frequency of occurrence is debated Kevin Pearce (snowboarder)
46
Chronic Traumatic Encephalopathy
A neurodegenerative disease associated with repeated head trauma Results in generalized global atrophy of the brain, ventricular dilation, thinning of the corpus collosum, and neurofibrillary bundles similar to AD Symptoms include headaches, difficulties with attention and memory, mood disorders, motor dysfunction, and dementia First identified in 1954 and linked to boxing, it has gained more attention in recent years due to the suicides of NFL football players and a recent movie about Dr. Bennet Omalu
47
Decerebrate rigidity =
humans results from a midbrain lesion and is manifested by an exaggerated extensor posture of all extremities.
48
Decorticate rigidity =
Decorticate posture is an abnormal posturing in which a person is stiff with bent arms, clenched fists, and legs held out straight. ... This type of posturing is a sign of severe damage in the brain
49
Retrograde amnesia =
s a loss of memory-access to events that occurred, or information that was learned, before an injury or the onset of a disease.
50
Anterograde amnesia =
Anterograde amnesia is a loss of the ability to create new memories after the event that caused amnesia, leading to a partial or complete inability to recall the recent past, while long-term memories from before the event remain intact.
51
Symptoms: Visual & Perceptual: Visual
Blurred vision, convergence insufficiency, reduced blink rate……. Also damage to oculomotor nerve
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Symptoms: Visual & Perceptual: Perceptual
Often result of high right hemisphere damage Visual perception = Right left discrimination, figure ground, position in space Body schema = anosognosia , unilateral neglect Speech & Language = aphasia, dyslexia, dysprosody
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Symptoms: Psychosocial & Behavioral: Psychosocial
``` Self concept Social roles Independent living Dealing with loss Affective changes ```
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Symptoms: Psychosocial & Behavioral: Behavioral
Common RLA Level IV (agitated/confused) = yelling, swearing, grabbing, biting
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Assessment: | Glasgow Coma Scale (GCS)
Assesses 3 behavioral areas: motor responses, verbal responses, and eye opening
56
Assessment: Glasgow Coma Scale (GCS): Motor
1) pain, no motor response to pinch, 2) body becomes rigid in extension with pinch, 3) flexes body inappropriately to pain, 4) pulls away with pinch, 5) pulls examiner’s hand away with pinch, 6) follows simply commands
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Assessment: Glasgow Coma Scale (GCS): Verbal:
1) no noise to 5) carries on conversation correctly
58
Assessment: Glasgow Coma Scale (GCS): Eyes:
1) does not open eyes to 4) opens eyes on own/spontaneous
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Ranchos Los Amigos Levels (RLA)
Measurement of levels of awareness and cognitive function Typical progression is linear although some with very severe injury may skip a level (typically level IV = agitated and confused)
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The GCS measures the following functions: Eye Opening (E) ``` 4 = 3 = 2 = 1 = NT = ```
``` 4 = spontaneous 3 = to sound 2 = to pressure 1 = none NT = not testable ```
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The GCS measures the following functions: Verbal Response (V) ``` 5 = 4 = 3 = 2 = 1 = NT = ```
``` 5 = orientated 4 = confused 3 = words, but not coherent 2 = sounds, but no words 1 = none NT = not testable ```
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The GCS measures the following functions: Motor Response (M) ``` 6 = 5 = 4 = 3 = 2 = 1 = NT = ```
Motor Response (M) ``` 6 = obeys command 5 = localizing 4 = normal flexion 3 = abnormal flexion 2 = extension 1 = none NT = not testable ```
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The Rancho Los Amigos Scale ``` Level 1 Level 2 Level 3 Level 4 Level 5 Level 6 Level 7 Level 8 Level 9 Level 10 ```
Level 1= No reaction - The brain-injured person is unconscious Level 2= Generalized reaction - The brain-injured person will react but inconsistent and without purpose. Level 3= Level 3: Localized reaction - The brain-injured person is improving. Level 4= Level 4: Confused/Agitated - The brain-injured person has become very active but they are not yet able to understand what's going on Level 5= Level 5: Confused/Inappropriate - The brain-injured person has become less agitated. Level 6= Confused/Appropriate - Things are looking up. The brain-injured person is motivated but still depends on others to lead the way. Level 7= Automatic/Appropriate - The brain-injured person seems to act appropriately in the hospital and at home. Level 8= Purposeful/Appropriate - At last! The brain-injured person remembers how the past fits with the future Level 9= Purposeful-appropriate, goes through daily routine aware of need fr stand-by-assistance, depression may continue Level 10= Purposeful-Appropriate/Modified Independent, goes through daily routine but may require more time or compensatory strategies, periodic depression may occur
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Evaluating TBI: Lower Level
``` Evaluate: Level of arousal - attend, follow commands, communicate, awake Vision - scan, attend, eye contact Sensation - pain, temp, movement ROM Motor Control - tone, reflexes Dysphagia Emotional and behavioral factors ``` Interventions: Sensory stim, w/c positioning, bed positioning, splinting/casting, dysphagia, behavioral, family/caregiver edu
65
Evaluating TBI:Intermediate to Higher Level
RLA IV to VIII = client is alert, confused, agitated, and inappropriate with responses, may follow 2-3 step commands, easily distracted Same as Low level with addition of ADLS, work readiness, and reintegration into the community, IADLS Physical status, dysphagia, cognition, vision Interventions: Neuromuscular (NDT, PNF), Ataxia, Cognition, Vision, Behavioral, dysphagia & self feeding, Functional mobility
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Interventions: Positioning Wheelchair Positioning ``` Effective seating = Pelvis = Trunk = LE = Head = ```
Allows interaction with the environment, prevents skin breakdown/ joint contractures, facilitates normal muscle tone, inhibits primitive reflexes, increases sitting tolerance, enhances respiration and swallowing, promotes function
67
Interventions: Positioning Bed Positioning
Crucial early on to prevent sores, facilitate normal tone/ ROM/ mobility Barriers: spasticity, splints, IVs, tubes, medical precautions Side lying or semiprone = abnormal tone or posturing
68
Interventions: Splinting & Casting
Similar to CVA When spasticity impacts function When ROM is limited When there are soft tissue contractures Contraindications: uncontrolled hypertension, open wounds, unhealed fx, impaired circulation, acute inflammation
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Interventions: Sensory Stimulation Goal =
Goal = increase arousal/awareness with sensory stim Responses measured (Radomski, 2008): respiration, pulse, blood pressure, head movements, eye opening/movements, eye fixation, mimic responses, aimed and non aimed motor responses, and articulations
70
Interventions: Cognition | Generation effect =
content that is self generated is remembered better than content that is provided/given
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Interventions: Cognition= 2 cooking and 2 financial management tasks
- 1 task of each done with provided conditions the other done with generated conditions - Provided information:   “Scramble two eggs in a bowl. Pour into hot pan on stove. Let cook three minutes.  Flip.” - Self-generated information: “Scramble two    in a bowl.   into hot pan on stove. Let cook    minutes. Flip.”
72
Interventions: Cognition= 2 cooking and 2 financial management tasks
- 1 task of each done with provided conditions the other done with generated conditions - Provided information:   “Scramble two eggs in a bowl. Pour into hot pan on stove. Let cook three minutes.  Flip.” - Self-generated information: “Scramble two    in a bowl.   into hot pan on stove. Let cook minutes. Flip.”
73
Interventions: Behavioral
Post traumatic agitation occurs in 33 - 50% of those with TBI Clinicians need to determine factors contributing to problem behaviors Client factors, Social context and environment, & Physical environment One on one coaching = 24/7 Psychotropic meds = regulate sleep and minimize agitation Environmental modifications = alarm system, helmet, quiet room
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Interventions: Behavioral Environmental vs Interactive
- Environmental = alter objects or environment to facilitate behavior, inhibit unwanted, maintain safety - Interactive = used to interact with client, use consistent implementation by all
75
Interventions: Community Integration Article (Kim & Colantonio, 2010)
Systematic review of 10 articles to determine best practice for OT in community integration Assessment: Reintegration to Normal Living Index Community Integration Questionnaire (most widely used) Findings:
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Interventions: Community Integration Article: Giving Second Chances: The Brain Injury Wellness Program (Klymasz, 2013)
Patient's and caregivers report frustration with being discharged from therapy before they feel ready Brain Injury Wellness Program: 6, 1-hour sessions that focus on community integration, wellness, function, and QOL Results: increased community participation, success in implementation of compensatory strategies, gained confidence
77
Interventions: Family & Caregiver Education
May have unrealistic expectations or interpret information differently than medical staff (unresponsiveness may be laziness, deafness…. When it is cognitive status) Provide family with concise information Involved throughout the rehab process Educated on ROM, positioning, transfers……. Substance Use Drug rehab services
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D/C Planning
Home safety Complete home eval to determine needed home modifications Equipment Eval and Ordering May refer to driver training/eval May refer to work/vocational training
79
What is Parkinson’s Disease?
ICD 10: G20 Chronic progressive condition Loss of dopamine-producing cells in the basal ganglia Cause is unknown. Recent studies point to environmental and genetic factors
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PD Prevalence
PD is the second most common neurodegenerative disorder in the US Effects approximately 1 million in the US
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PD Symptoms: Motor
``` Bradykinesia Hypokinesia Rigidity Tremor Disturbed postural reflexes ```
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Bradykinesia
slowness of movement and is one of the cardinal manifestations of Parkinson's disease.
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Hypokinesia
partial or complete loss of muscle movement due to a disruption in the basal ganglia.
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Rigidity
inability to be to bent or be forced out of shape.
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Tremor
involuntary, somewhat rhythmic, muscle contraction and relaxation involving oscillations or twitching movements of one or more body parts.
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PD Symptoms: Non-Motor
``` Mental Functions Sleep Disorders Voice and Speech Depression Psychosis Anxiety Fatigue Smell disorders Constipation ```
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PD Symptoms: Progression
Motor symptoms often precede non-motor symptoms Motor symptoms often begin unilaterally Less than 5% end up w/c or bedridden Life expectancy is almost average with medical advances Slower progression: Faster progression:
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PD OT Interventions: Physical Exercise
Progressive resistive, joint mobilization, postural stability/balance training, gait training, aerobic activities More likely to improve performance skills and not task performance itself Encourage clients to engage in regular physical activity
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PD OT Interventions: Physical Exercise
Progressive resistive, joint mobilization, postural stability/balance training, gait training, aerobic activities More likely to improve performance skills and not task performance itself Encourage clients to engage in regular physical activity
90
PD OT Interventions: Physical Exercise
Progressive resistive, joint mobilization, postural stability/balance training, gait training, aerobic activities More likely to improve performance skills and not task performance itself Encourage clients to engage in regular physical activity
91
PD OT Interventions: Environmental Cues
Auditory rhythmic cues stronger than visual and tactile cues for regulating walking Moderate evidence to support client-preferred external cues during performance of ADLs had positive effects on motor control Provide Targeted external cues
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PD: OT Interventions: Self Management | Cog behavioral:
education, goal setting, practice, and feedback to incorporate into daily life Plan for and manage the progression of the disease
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PD: General Interventions
``` Optimizing daily schedule Dealing with stress and time pressure Practicing arm/hand motor skills Attention to Task Cognitive movement strategies Minimize dual task Use Cues Rhythmic and single Auditory, Visual, Tactile/Proprioceptive Environmental modifications Caregiver support and education ```
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PD Early & middle/Late stages Stages 1 to 5
Stage 1: minimal symptoms; usually tremors; symptoms don't affect daily routine Stage 2: Diseases starts to affect whole body; routine activities may take longer to complete Stage 3: loss of balance and coordinaton; routine activities may become difficult Stage 4: nearly impossible to live on your own; routine activities should not be performed alone Stage 5; final & most severe stage; confined to bed; dementia; confusion; and hallucinations begin
95
General Taping Information KT is good for..?
: pain, swelling, inflammation, postural malalignment, muscle imbalance, trigger points
96
What are the benefits of K-tape?
May encourage realignment of joint structures Offers proprioceptive feedback Beneficial for shoulder impingement, joint sprains, and multi- directional instability Reposition humeral head with tape to that it is in a neutral position Taping allows client to feel normal alignment Can result in immediate pain relief and improved ROM May help with inflammation by encouraging lymphatic drainage
97
Cuts of K-Tape Y-tape I-tape X-tape Donut Web/Fan
Y-tape Used to surround a muscle To facilitate or inhibit muscle stimuli, should be 2 inches longer than muscle I-tape - most commonly used Used for more acute injuries Edema and pain management, correct alignment X-tape Used when origin and insertion change depending on movement Donut Specifically for edema Overlapping strips and the center is cut our over areas of focus Web/Fan Primarily used for edema
98
Application of KT Insertion to Origin is used to.....? Origin to Insertion is used to....?
Insertion to Origin Used to inhibit overused or stretched muscles Light stretch required (15 – 25%) Origin to Insertion Used to facilitate weak or under performing muscles Light to moderate stretch required (50-75%)
99
CA Tri Pull Method
``` 1) Identify the 3 landmarks on your client Deltoid Tuberosity Coracoid process Acromial Process Spine of scapula ``` 2) Measure out and cut the 3 pieces of tape 3) Clean and prepare the skin 4) Apply Tape 1) Medial = 1 inch below deltoid tuberosity - 1 inch above acromion process (ends at neck) 2) Posterior = 1 inch below deltoid tuberosity - 1 inch above spine of scapula 3) Anterior = 1 inch below deltoid tuberosity - 1 inch above coracoid process
100
Kinesio Taping in Stroke Origin to insertion or insertion to origin? What are the 5 steps to placing tape onto your ct?
Origin to insertion method as the deltoid is noted to be weakened or paralyzed 1) Pt positioned with head, trunk, and scapula in best alignment 2) Abduct shoulder to 90 degrees 3) Cut Y tape and place anchor on the acromion process with no stretch 4) Move shldr to Extension and apply first tail to anterior deltoid as it is stretched (end at insertion) with no stretch in tape 5) Move shoulder in horizontal abduction and apply second tail over posterior deltoid ending at insertion
101
Definition:ALS
A group of progressive, degenerative neuromuscular diseases:
102
ALS: Progressive bulbar palsy
Destruction in corticobulbar tracts and brain stem Symptoms: speech, swallowing, breathing may be affected
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ALS: Progressive spinal muscular atrophy (Lower Motor Neuron)
Destruction in LMN in the spinal cord (sometimes brainstem) Symptoms:weakness or muscle atrophy of extremities, cervical extensor weakness, fasciculation, muscle cramps, and loss of reflexes
104
ALS: Primary lateral sclerosis (Upper Motor Neuron)
Destruction of cortical motor neurons Symptoms: results in: general weakness. Spasticity, and hyperreflexia
105
Clinical Picture:ALS- Early
- Focal weakness in arms, legs, or bulbar muscles - Trip or drop things - Slurred speech - Abnormal fatigue - Uncontrollable laughing/crying
106
Clinical Picture: ALS- Progresses
Progresses Marked muscle atrophy Weight loss Spasticity Muscle cramping Fasciculation Difficulty walking, dressing, fine motor, swallowing, breathing
107
OT’s Role: ALS What are the 6 stages?
1) Ambulatory, no ADL problems, mild weakness How would you treat? 2) Ambulatory, moderate weakness in certain muscles How would you treat? 3) Ambulatory, severe weakness in certain muscles How would you treat? 4) Wheelchair confined, almost independent, severe weakness in legs How would you treat? 5) Wheelchair confined, dependent, pronounces weakness in legs, severe in arms How would you treat? 6) Bedridden, Dep ADLs, Max A - Dep in all tasks How would you treat?
108
Huntington’s Disease
Fatal degenerative neurologic disorder Genetic, transmitted in an autosomal dominant pattern Damage to the corpus striatum which is responsible for motor control Damage to the caudate nucleus is linked to cognitive and emotional function
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Huntington’s Disease: Presymptomatic Stage
Decreased speed of finger tapping | Unified Huntington's Disease Rating Scale
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Huntington’s Disease: Early Stage
Alterations in behavior, changes in cognitive functioning, choreiform movements of the hands
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Huntington’s Disease: hands | Middle Stage
Memory and decision making skills Help them maintain meaningful habits and routines Better with familiar and routine tasks Gait and balance disturbances
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Huntington’s Disease: Late Stage
``` Verbal comprehension Dysarthria Depression worsens Bradykinesia and akinesia Increasing difficulty with handwriting Slowed saccadic and ocular eye movements Dysphagia and choking hazard ```
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Huntington’s Disease: Clinical Picture: Medical treatment for HD
Medical Treatment Can address the symptoms but not stop the progression of the disease Antidepressants Goal: manage symptoms, reduce burden of symptoms, maximize function, and provide pt and family education
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OT’s Role: HD (early/middle/end?)
Early stages Address memory and concentration: How? Assist with strategies for employment: How? Address anxiety, depression, and irritability: How? Address the effects of Chorea Modifications for fine motor control: How? ``` Middle Stages Provide visual cues to prompt actions Watch for signs of Suicide: How? Address fatigue: How? Address increase chorea: How? Address dysphagia: How? ``` Final Stages Address chorea replaced with rigidity: How?
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Definition MS
Progressive, inflammatory neurologic disease Damage to the myelin sheath in the CNS Onset between the ages of 20 - 45 Etiology: unknown, suspected combination of environmental and genetic factors
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Clinical Picture: MS (early symptoms and advance stages?)
Early symptoms: Paresthesia, diplopia, visual loss in one eye, fatigue, emotional lability, sensory loss in extremities Trigeminal neuralgia, symptoms exacerbated with increased body temperatures Advance stages: Varying degrees of paralysis, dysarthria, dysphagia, severe visual impairment, ataxia, spasticity, nystagmus, neurogenic bladder, impaired cognition
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Clinical Picture: MS 1) Relapsing and Remitting 2) Secondary Progressive 3) Primary Progressive
Patterns of symptoms: 1) Relapsing and Remitting 85% of cases Slow, step like progression as deficits accumulate 2) Secondary Progressive Begins with relapsing and remitting that progresses into primary progressive 50% of those with relapsing/remitting progress to secondary progressive 3) Primary Progressive 10% of MS population Downward slope with little recovery after an exacerbation Become non ambulatory, incontinent, dysphagia, dysarthria, severely compromised LE function, varying UE function
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Clinical Picture:MS Life expectancy
Life expectancy Favorable Prognosis: (chart page 937) Minimal disability after 5 years of onset complete/rapid remission of initial symptoms Age of onset less than 40 y/o Only 1 symptom the first year Onset with sensory symptoms or mild optic neuritis ``` Poor Prognosis: Progressive course Age of onset greater than 40 Cerebellar involvement Polysymptomatic Male sex ```
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OT’s Role: MS Evaluation:
Assess motor and praxis, sensory perceptual, emotional regulation, cognitive, communication skills As fatigue is often an issue assessments results may vary depending on time of day Self report often inaccurate Assess sleep and sleep patterns Assess visual tracking, scanning, and acuity Assess cognition
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OT’s Role: Treatment:
Treatment: MS Society resources for clinicians: Articles: Systematic Reviews Part I and Part II (Yu & Mathiowetz, 2014a&b) Part 1 = activity and participation -Goal directed interventions, health promotion programs, and fatigue management programs -Direct training in functional performance -Group fatigue management delivered face to face or long distance ``` Part 2 = Impairment Cognition Emotional Regulation Exercise Motor Training ```
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Guillain-Barre Definition
A rare autoimmune disease with no known cure, no established treatment, requiring a long period of recovery, no clear cause Acute, Inflammatory disorder in which the body’s immune system attacks the peripheral nervous system Damage to the myelin sheath prevents nerve conduction that leads to muscle weakness, pain, and/or paralysis of the entire body
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Guillain-Barre Clinical Picture
Rapidly progressive weakness of bilateral extremities distal to proximal Brain does not receive signals from the body Body does not receive signals from the brain to move Often first symptom is tingling, crawling skin, painful sensations that begin in the hands and feet If demyelination continues may impact breathing, speaking, swallowing, blood pressure, and/or heart rate
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Guillain-Barre 1) Initial/Acute Phase: 2) Plateau Phase: 3) Recovery Phase:
Initial Phase: Passive ROM, positioning, splinting to prevent contractures/deformity Passive activities like watching tv Address anxiety, fear, and panic Recovery Phase: Precautions: prevent muscle belly tenderness, fatigue, and further damage to nerves Address proximal joints first as they recovery then move to distal Introduce activities as they are able to tolerate them, just right challenge, opportunities for success AE, compensatory strategies, energy conservation, joint protection Lifestyle redesign
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Postpolio Syndrome Definition (PPS)
Peak of Polio in the US was 1952 After 30 - 40 years 25% - 40% of these adults deal with new muscle pain, weakness, and/or paralysis = postpolio syndrome PPS
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6 criteria to diagnose Postpolio Syndrome (PPS):
1) Pervious paralysis due to polio 2) Period of partial or complete recovery 3) Gradual or sudden onset of progressive muscle weakness or fatigue 4) New difficulties with breathing and swallowing 5) A year or more of the above symptoms 6) Other causes have been ruled out
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PPS: Clinical Picture
Slow progression with periods of stability (plateaus) Fatigue is found to be the most debilitating symptom Risk of muscle atrophy, scoliosis, osteoporosis, fractures, contractures, and depression Difficulty with ADLs, IADLs, ambulation, stairs, home mang, transfers, driving, eating and swallowing, bladder and bowel control While symptoms greatly impact quality of life there are rarely life threatening
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PPS: Role of OT
Work simplification, pacing and energy conservation, adaptive equipment, passive and active ROM, muscle re-education, proper posture and body mechanics, joint protection Connect with support groups for feelings of denial, anger, hopelessness, feel a burden Introduce changes gradually with just right challenge to provide confidence and success Caution with exercise: In general body weight reduction, work with dietician
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Define - Feeding - Eating - Dysphagia
Feeding: “the process of setting up, arranging, and bringing food (fluids) from the plate or cup to the mouth, sometimes called self feeding” OTPF Eating: “The ability to keep and manipulate food/fluid in the mouth and swallow it; eating and swallowing are often used interchangeably” OTPF Dysphagia: inability to swallow or difficulty with swallowing Clients who may have difficulty feeding or eating: ???
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Preparatory Phase & Stage 1 of Swallowing
Preparatory Phase Before the 4 stages of eating Begins when client enters the dining area Considers variety of foods, presentation of food, seating during meals, meal time atmosphere, mealtime habits Sensory quality of the food and items, appetite & hunger 1) Oral Preparatory Phase Action: Visual and olfactory information stimulates saliva production Jaw opens and lips close around food with musculature creating a seal to prevent spillage Chewing: Time to form bolus is shorter for softer foods
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Stage 2 & 3 of Swallowing
Stage 2 & 3 of Swallowing 2) Oral Phase Begins when tongue initiates movement of bolus towards the pharynx Tongue elevates, pushing bolus against the hard palate and guides the bolus back Thicker foods require more pressure of tongue on the palate This is a voluntary phase, person must be alert and involved 3) Pharyngeal Phase Closure of the larynx, the laryngeal entrance, and epiglottis to prevent material from entering airway Bolus moves through the pharynx towards the esophagus, passes through pharynx divided in half at the valleculae and down each side of the pyriform sinuses Upper esophageal sphincter relaxes and opens allowing material to enter the esophagus
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Final Stage of Swallowing
4) Esophageal Phase Begins when bolus enters the esophagus through the cricopharyngeal juncture or upper esophageal sphincter (UES) Esophagus is a straight tube 10 inches long that connects the pharynx to the stomach UES separates pharynx from esophagus and LES separates esophagus from the stomach Muscles of esophagus contract and push bolus down = peristaltic wave contractions Epiglottitis returns to a relaxed state to allow airway to open, return to breathing
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Dysphagia & Aspiration | Symptoms of Dysphagia:
``` Difficulty shaping food into bolus Loss of food from mouth or nose Coughing, throat clearing Wet or gurgling voice after Changes in mealtime behavior Food residue in mouth Delayed or absent swallow Weak cough Reflux of food Aspiration ```
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Interventions & Goals
``` Trunk and head positioning -Improve swallow -Reduce risk of aspiration Rehabilitative: Exercises ``` Compensatory: Oral hygiene Modify food texture 1) facilitate appropriate positioning during eating 2) Improve motor control at each stage of swallowing 3) Maintenance of adequate hydration and nutrition 4) Prevention of aspiration 5) Reestablishment of oral eating to the safest/optimum level on least restrictive diet
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Diet selection for Dysphagia Food: 4 Levels of Dysphagia Dysphagia Level 1: Dysphagia Level 2: Dysphagia Level 3: Dysphagia Level 4:
Dysphagia Level 1: Dysphagia Puree Dysphagia Level 2: Mechanical Soft Dysphagia Level 3: Dysphagia Advanced ``` Dysphagia Level 4: Normal Liquids Thin Nectar like Honey like Spoon thick ```