w5- sgl Flashcards
what are the two forms of vtach
increased automaticity –> purkinje with reduced membrane potentials
scarring slows conduction and causes reentry circuits in boder zones
what is the source of troponin from in a person with the flu or COPD
•Supply/demand mismatch
differentiate typical angina, atypical and non cardiac chest pain
what is Prinzmetal’s angina
coronary vasopasm
coronary dissection often occurs after what
pregnancy
what is cardiogenic shock
Loss of enough myocytescan result insevere reduction in contractility and thus cardiac output –> hypotension
what are some of the mechanical complications of MI
- VSD
- Free wall rupture
- Papillary muscle rupture from mitral regurg
- Aneurysm/Pseudoaneurysm formation (more dangerous)
- LV thrombus
what supplies the Anterolateral and posteromedial papillary muscle
anteriolateral is supplied by LAD and LCx
Postromedial pap muscule supplied by RCA.
what would you see with nonischemic transmural ischemia (including prinzmetal angina)
transient ST elevations or
T wave inversion
when woul the q wave begin
when would the t wave inversion begin
within hours
within days and will disappear within a week
outline the TIMI score
ID the acute manegment for MI
- Anticoagulation
- Parenteral
- Dual anti-platelet therapy
- Aspirin
- P2Y12
- High intensity statin
- Beta-blocker
- Revascularization
- IIb/IIIa
- Nitroglycerin
- ACEi/ARB
why do you include a beta blocker in MI tx
reduce o2 demand of the heart
differentiate the primary vs secondary Tx for MI
