Urological emergencies Flashcards
acute urinary retention
the sudden inability to pass urine which causes significant pain
acute urinary retention is characterised as
spontaneous or precipitated
precipitated acute urinary retention is where
there is a triggering event i.e. non-prostate related surgery, catheterisation, urethral instrumentation, anaesthesia, medication with sympathomimetic or anti-cholinergic affects
causes of acute urinary retention in men
benign prostatic hyperplasia (most common), meatal stenosis, paraphymosis, prostate cancer
causes of acute urinary retention in woman
cysteocels (where the bladder bulges into the vagina), rectocele (where the rectum bulges into the vagina), pelvic mass (gynaecological malignancy, uterine fibrosis)
in both male and females acute urinary retention can be caused by
bladder calculi, blader cancer, faecal impacatation, GI or retro-peritoneal malignancy
infections and inflammatory causes of acute urinary retention in males
balanitits (inflammation of the glans penis), prostatitis, prostatic abscess
infections and inflammatory causes of acute urinary retention in females
acute vulvovaginitis, lichen sclerosus
infections and inflammatory causes of acute urinary retention which can occur in both males and females
schistosomiasis, cystitis, herpes simplex, peri-urethral abscess
drug related causes of acute urinary retention
anti-cholinergics, opioids, anaestheticis, alpha-adrenergic agonists, benzodiazepines
presentation of acute urinary retention
teder distended palpable bladder with inability to pass urine
management of acute urinary retention
immediate and complete bladder decompression using a catheter, if painful retention with less than 1 litre residue and normal serum electrolytes trial without cathetersitation is carried out during the same admission
what should be prescribed to increase success of trial without catheterisation
alpha blocker (tamsulosin)
how does tamsulosin work
it is a selective alpha blocker which causes relaxation of smooth muscles in the bladder neck and prostate
post obstructive diuresis
high urine output greater than 200ml per hour for more than 2 consecutive hours after an obstruction is relieved
post obstructive diuresis most often presents in patients with
chronic bladder outflow obstruction in associated with uraemia, oedema, congestive heart failure and hypertension
post-obstructive diuresis is a state of
polyuresis where there is excessive amount of water excreted in the urine after treatment of a urinary tract obstruction
diureisus is a normal physiological response
to help eliminate excessive volume and solutes which accumulated during the prolonged obstruction but the diuresis should resolve after solute and volume have been normalised but in POD the kidney continues to eliminated fluid after homeostasis has been achieved
in post-obstructive diuresis there is a risk of
dehydration, electrolyte imbalance and hypovolaemic shock
post-obstructive diuresis usually resolves after
24-48 hours but if severe requires IV fluids and sodium replacement
acute loin pain always consider what in your differentials
diagnosis outwith the urinary tract such as AAA
acute loin pain most commonly caused by
nephrolithiasis
the urerter has 3 sites of what
constriction where it contracts smooth muscle, narrowing can occur at these sites and calculi can get lodged here:
- pelvics-ureteric junction
- pelvic brim
- vesico-ureteric orifice
the cells lining the renal tubules are predominantly
cuboidal epithelial cells
within the renal tubules what can form
crystal like structures known as calculus
if the calculus are small enough
the pass out in urine without causing any problems, but if they are large they can cause obstruction
obstruction within the tubules causes
release prostaglandisng causing pain
presentation of calculi
- acute flank pain which can radiate to the groin
- nausea,vomiting, fever
risk factors for renal stones
- white caucasian
- obesity
- high sodium and protein diet
- carbonic anhydrase inhibitors
- sodium and calcium containing medication
some of the risk factors for renal stones
increase uurianry solute concentration (sodium, calcium, oxalate) and some risk factors reducing concentrations of salt forming inhibitors (citrate and magnesium)
these combined factors cause
urine super-saturation causing the formation of calculus
types of stones
- calcium oxalate (most common) and are radiopaque and more likely to form in acidic urine
- calcium phosphate are radiopaque and most likely to form in alkaline urine
- struvite are common in chronic UTIS and contain magnesium ammonia and phosphate and are radioopaue
- uric acid stones are radiolucent
- cystine stones are very rare and are radiopaque
investigations for renal stones
FBCS, CRP, urinalysis
- ULTRASOND IS INITIAL IMAGING OF CHOIDE AND THEN NON-CONTRAST CT
management of renal stones
- Intra-muscular diclofenace
- tamsulosin
- stones less than 5mm usually pass spontaneously within 4 weeks
indications to urgently treat renal stones
pain unrelieved, persistent causes, vomiting, high- grade obstruction
- ureteric obstruction
- renal abnormality such as horseshoe kidney
- previous renal transplant
URETERIC OBSTRUCTION CAUSED BY STONE COMBINDE WITH INFECTION IS A SURGICAL EMERGENCY options include nephrostomy tube insertion of ureteric catheter or ureteric stent
non-emergency treatment of renal stones
- shock wave lithotripsy
- ureteroscopy (indicated in individuals where shock wave lithotripsy is contra-indicated i.e. pregnant females and in complex stone disease). In most cases a stent is left in situ for 4 weeks after the procedure
- percutaneous nephroplithotomy