the management of deep caries Flashcards
give an example of a basic treatment plan
- emergency relief of pain
- establish a healthy oral environment
- corrective therapy
- replace missing teeth
what treatment can be involved in establishing a healthy oral environment
diet diary PFS preventative advice for caries OHI/scaling caries stabilisation
what steps can be included in corrective therapy
permanent restorations
RSD
endodontic treatment
crowns
what is caries
a disease of the hard tissues of the teeth characterised by demineralisation and proteolytic destruction of the tissues by acids produced by bacteria in dental plaque feeding on dietary carbohydrates
what structures are included in the pulp
odontoblasts blood vessels nerves lymphatic system stem cells connective tissue
which nerves are found in the pulp
a delta nerves and c fibre nerves
describe a delta nerves
myelinated fast response
short sharp pain
tooth sensitivity described like an electric shock or sharp pain
describe c fibres
unmyelinated slow response timing
and is a dull throbbing pain
what components cause demineralisation of the tooth surface
the fermentable carbohydrates and cariogenic bacteria producing lactic acid and this causes demineralisation of the tooth surface
what causes demineralisation of the tooths surface
fluoride and saliva flow can help remineralise early white spot lesions
what can we use to help diagnose caries
radiographs
3-in-1 will show WSL as chalky
when does a white spot lesion form
when there is more demineralisation than demineralisation occurring at the tooth surface but this is still reversible
what is a brown spot lesion
occurs when there is repeated demineralisation and remineralisation leading to pigments from blood or food being incorporated into the enamel
how deep is a BSL
still only superficial will not progress into a cavity if kept clean and fluoride varnish applied
how does the tooth respond when a lesion starts to form
- arterial flow to the pulp increases
- causes an increase in dentinal tubular fluid
- fluid acts to flush out bacterial toxins and carries anti bacterial components such as IgE and lactoferrin
- peritubular dentine gets deposited and the tubules become narrower and creating a barrier to the bacteria(sclerosis)
- affected odontoblasts produce tertiary dentine
- can be arrested at this stage
what happens if demineralisation continues of the WSL
secondary dentine is laid down and cavitation may occur- then starts to spread laterally at the ADJ
when carrying out cons treatment what do we need to remove
infected enamel and infected dentine we can leave the affected dentine as it protects the pulp