the management of deep caries Flashcards

1
Q

give an example of a basic treatment plan

A
  1. emergency relief of pain
  2. establish a healthy oral environment
  3. corrective therapy
  4. replace missing teeth
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2
Q

what treatment can be involved in establishing a healthy oral environment

A
diet diary 
PFS 
preventative advice for caries
OHI/scaling 
caries stabilisation
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3
Q

what steps can be included in corrective therapy

A

permanent restorations
RSD
endodontic treatment
crowns

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4
Q

what is caries

A

a disease of the hard tissues of the teeth characterised by demineralisation and proteolytic destruction of the tissues by acids produced by bacteria in dental plaque feeding on dietary carbohydrates

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5
Q

what structures are included in the pulp

A
odontoblasts 
blood vessels 
nerves
lymphatic system 
stem cells 
connective tissue
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6
Q

which nerves are found in the pulp

A

a delta nerves and c fibre nerves

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7
Q

describe a delta nerves

A

myelinated fast response
short sharp pain
tooth sensitivity described like an electric shock or sharp pain

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8
Q

describe c fibres

A

unmyelinated slow response timing

and is a dull throbbing pain

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9
Q

what components cause demineralisation of the tooth surface

A

the fermentable carbohydrates and cariogenic bacteria producing lactic acid and this causes demineralisation of the tooth surface

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10
Q

what causes demineralisation of the tooths surface

A

fluoride and saliva flow can help remineralise early white spot lesions

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11
Q

what can we use to help diagnose caries

A

radiographs

3-in-1 will show WSL as chalky

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12
Q

when does a white spot lesion form

A

when there is more demineralisation than demineralisation occurring at the tooth surface but this is still reversible

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13
Q

what is a brown spot lesion

A

occurs when there is repeated demineralisation and remineralisation leading to pigments from blood or food being incorporated into the enamel

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14
Q

how deep is a BSL

A

still only superficial will not progress into a cavity if kept clean and fluoride varnish applied

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15
Q

how does the tooth respond when a lesion starts to form

A
  1. arterial flow to the pulp increases
  2. causes an increase in dentinal tubular fluid
  3. fluid acts to flush out bacterial toxins and carries anti bacterial components such as IgE and lactoferrin
  4. peritubular dentine gets deposited and the tubules become narrower and creating a barrier to the bacteria(sclerosis)
  5. affected odontoblasts produce tertiary dentine
  6. can be arrested at this stage
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16
Q

what happens if demineralisation continues of the WSL

A

secondary dentine is laid down and cavitation may occur- then starts to spread laterally at the ADJ

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17
Q

when carrying out cons treatment what do we need to remove

A

infected enamel and infected dentine we can leave the affected dentine as it protects the pulp

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18
Q

what happens when caries gets close to the pulp

A

the pulp becomes inflamed and a delta fibres are triggered and there’s pain- this is reversible pulpitis which can be reversed

19
Q

what happens when we have a very deep cavity

A

the bacteria approach the pulp and inflammation becomes irreversible- removing the caries can’t reverse the pulpitis but can treat painful toothache

20
Q

which fibres are the last to die in pulp necrosis

A

c fibres are the last to die

21
Q

what happens once the pulp is dead

A

the bacteria thrive on the dead tissue and there is no more defence

22
Q

what does caries management depend on

A

the status of the pulp

23
Q

what do we do if there is vital pulp

A

try to keep the tooth alive

24
Q

what do we do if the tooth has irreversible pulpitis or pulpal necrosis

A

RCT or extraction

25
Q

how do we test if the tooth is alive or not

A

symptoms
clinical appearance
special tests
radiographs

26
Q

what are sensitivity tests

A

such as ethyl chloride or electric pulp test (EPT)

27
Q

what is the issue with sensitivity tests

A

no 100% reliable and many false positives and false negatives

28
Q

what do the numbers of the electric pulp test mean

A

shouldn’t be inferred as meaning something-eg any number under 80 means probably alive

29
Q

what can help with diagnosing whether the pulp is dead or alive

A

evidence of periodical changes

sometimes a dead tooth looks darker

30
Q

how do we maintain pulp vitality when carrying out a treatment

A

Use rubber dam
Remove caries from the walls of the cavity first
Never use a high speed for removing caries
On the floor of the cavity only use excavators, gently scrape away anything soft, never use force
Leave affected dentine over the pulp
Use RMGIC liner to seal affected dentine

31
Q

what kills the pulp

A

bacteria and dentists-therefore wanna keep bacteria out

32
Q

how do we protect the pulp if the caries is nearing closer to it

A

create a sela with RMGIC liner-fuji liner and only place it on the dentine

33
Q

what size are the tubules near the pulp

A

tubules are wider nearer the pulp

34
Q

what are the three types of pulpal exposure

A

iatrogenic-the dentist exposes the pulp during caries removal
carious
traumatic-– trauma causes a fracture of the tooth involving the pulp

35
Q

how do we manage iatrogenic pulpal exposure

A

if perviously asymptomatic then

  1. rubber dam
  2. wash cavity
  3. dry with cotton wool
  4. then get tutor
36
Q

what do we do if the dentine around the exposure is carious

A

if carious RCT required

37
Q

what do we od if the size of the exposure is greater than 2mm

A

then RCT

38
Q

what do we do if the size of the exposure is less than 2mm

A

place Ca(OH)2 over the exposure and dress the tooth and monitor

39
Q

what is setting calcium hydroxide also known as in clinic

A

drycal
MTA( mineral trioxide aggregate)
biodentine( bio silicate material )

40
Q

give examples of non setting calcium hydroxide

A

hypocal or calsept

41
Q

what are the properties of calcium hydroxide

A

Very high pH (>pH 11)
Creates an alkaline environment i.e. bactericidal (highly toxic to bacteria)
Stimulates odontoblasts to lay down new reparative dentine in the dentinal tubules
Stimulates stem cells in the pulp tissue to create new odontoblast-like cells to create dentine bridges across pulpal exposures

42
Q

when is pulp cap most successful

A

Absence of signs or clinical symptoms of pulpitis before you start opening the tooth up
Normal response to vitality tests by the tooth
Younger patients without previous history of restorative work on the tooth in question
Tooth is isolated (Rubber dam) and there is no salivary contamination
Small exposure <2mm in diameter
Exposure is not probed, or blown dry (damaging pulp tissue directly)

43
Q

what are clinical procedures to avoid exposure of the pulp

A

step wise excavation
remove caries until near the pulp and stop
fill with GIC
reevaluate after 6 months and replace restoration if symptom free and vital

44
Q

what is the stepwise excavation technique

A
  1. Establish sound enamel margins & caries free ADJ
  2. Remove “infected” dentine and leave a layer of “affected” dentine
  3. Place layer of calcium hydroxide liner over very deep affected dentine
  4. If cavity cannot be restored in a single visit then:
    Restore the whole cavity with RMGIC (RMGIC marginal seal is critical
    to prevent any micro-leakage at the & review 6 months later)
  5. If cavity can be restored in 1 visit, then use RMGIC to restore the
    “dentine depth, leaving 2-3 mm coronally for your definitive composite
    or amalgam restoration