The Dizzy Patient Flashcards

1
Q

which systems does the balance system rely on inputs from

A

inner ear

eyes

joints

heart brain

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2
Q

which cardiac problems can cause dizziness

A

arrhythmias and postural hypotension

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3
Q

how can DM cause dizziness

A

hypoglycaemia

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4
Q

how can arthritis cause dizziness

A

in the neck can occlude the vertebral arteries, resulting in dizziness

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5
Q

nystagmus

A

Eyes make repetitive, uncontrolled movements

These often result in reduced vision and depth perception and can affect balance and coordination.

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6
Q

vestibular nystagmus

A

VOR: activation of the vestibular system causes eye movement

This reflex functions to stabilise images on the retinas during head movement by producing eye movements in the opposite direction to head movement

impairement can cause vestibular nystagmus

Note: VOR is important for stabilising vision all the time, it may be hard to read small print

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7
Q

what are important things to elicit from the history of a dizzy patient

A
  • Triggers – movement, standing up
  • Time course
  • Associated symptoms
  • Precipitators
  • Alleviating factors
  • Medication
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8
Q

examination of a dizzy patient

A
  • Otoscopy
  • Neurological
  • BP - lying and standing
  • Balance system
  • Audiometry
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9
Q

what can cause postural dizziness

A

low Sodium, low blood pressure, anaemia (check Hb)

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10
Q

can stress cause dizziness

A

yes, also through hyperventilation

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11
Q

benign positional paroxysmal vertigo

A

very common

attacks of sudden rotational vertigo lasting >30 seconds that are provoked by head turning

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12
Q

symptoms of BPPV

A

usually just vertigo

rare to have other ontological symptoms eg hearing loss, aural fullness or tinnitus

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13
Q

aetiology of BPPV

A
  • Common after head injury
  • Ear surgery
  • Idiopathic
  • Middle ear disease
  • Otosclerosis
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14
Q

pathogenesis of BPPV

A

displacement of otoconias in urticle into semicircular canals

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15
Q

why is BPPV the most common cause of vertigo looking up

A

otoconia are most commonly displaced into posterior SCC, this causes vertigo on looking up

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16
Q

what may BPPV be confused with

A

vertebrobasilar insufficiency - poor blood supply in basilar and vertebral arteries due to atherosclerosis

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17
Q

how would BPPV be differentiated from vertebrobasilar insufficiency

A

diagnosis of vertebrobasilar insufficiency requires symptoms of impaired circulation in the posterior brain due in association with vertigo

eg bilateral visual disturbance, weakness and numbness, nausea and vomiting, slurred speech, loss of coordination etc.

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18
Q

when is vertigo particularly experienced in BPPV

A
  • Looking up
  • Turning in bed, often worse on one side
  • First lying down in bed at night
  • On first getting out of bed in the morning
  • Bending forward
  • Rising from bending
  • Moving head quickly, often only in one direction
19
Q

what negatives are important to establish in the diagnosis of BPPV

A
  • No persistent vertigo
  • No speech, visual, motor or sensory problems
  • No tinnitus, headache, facial numbness or dysphagia
  • No vertical nystagmus
20
Q

what test is used in the diagnosis of BPPV

A

Hallpike’s test: this rotates the posterior semicircular canal in the plane of gravity

The patient’s eyes must be open and they must look straight ahead. While supine, the head is held and turned 40 degrees to one side, then rapidly lowered to below couch level. Ask the patient if they feel dizzy and look for nystagmus.

This lasts for under a minute due to adaption and reoccurs on sitting. If any of these features are absent (or persisting nystagmus), seek a central cause. The test fatigues so can only be performed accurately once.

21
Q

name 2 important points about the Hallpike test

A

test fatigues - must be right first time

patient must not close eyes

22
Q

Epley manoeuvre

A

BPPV diagnosis - allows displaced otoconia to be relocated back into the urticle using gravity

23
Q

what exercise can be performed if patient cannot tolerate Epley or Hallpike

A

Brandt-Daroff Exercise

24
Q

treatment of BPPV

A

it is usually self-limiting in months

alcohol intake may help

drugs: vestibular sedatives and histamine analogues

25
Q

name a vestibular sedative used for the treatment of vertigo

A

prochloperazine

26
Q

name a histamine analogue

A

betahistine

27
Q

Vestibular Neuronitis

A

prolonged vertigo (days-weeks) with no associated tinnitus or hearing loss

tends to be worst on the first day and then improve

28
Q

cause of Vestibular Neuronitis

A

possible viral aetiology - preceded by viral prodromal symptoms (URT)

can also be caused by bacterial infection (meningitis, middle ear infection)

29
Q

labrynthitis

A

prolonged vertigo (days-weeks) that may have associated tinnitus or hearing loss

30
Q

aetiology of labrynthitis

A

It has a possible viral aetiology and is often preceded by viral prodromal symptoms.

Less commonly caused by systemic infections e.g. measles, mumps, infectious mononucleosis.

Rarely caused by bacterial infection.

31
Q

management of vestibular neuronitis and labrynthitis

A

usually self-limiting (days-weeks)

mainly managed supportively with vestibular sedatives (eg prochloperazine)

if prolonged/atypical may require further investigation

32
Q

Ménière’s Disease

A

Excess endolymph causes dilatation of the endolymphatic spaces of the membranous labyrinth causes vertigo for around 12 hours with prostration, nausea/vomiting ± a feeling of fullness in the ear.

There is associated uni or bi lateral tinnitus ± sensorineural deafness.

33
Q

describe the pattern of attacks in Ménière’s disease

A

occur in clusters (<20 a month)

tinnitus can appear/get louder as a warning sign to attack

34
Q

cause of Ménière’s disease

A

unknown

35
Q

audiogram of Ménière’s disease

A

uni or bi lateral sensorineural hearing loss

36
Q

treatment of Ménière’s disease

A

supportive treatment during episodes

tinnitus therapy eg prochloperazine and betahistine

Grommet insertion/Meniette

intratympanic Gentamicin/steroids

37
Q

Grommets and Meniette

A
  • Meniette gives out low pressure ear pulses that influence the fluid system and displace the excess endolymphatic fluid
  • In order to use this, the patient must have a Grommet inserted in their ear
38
Q

prevention of Ménière’s disease

A

salt restriction, betahistine

roles of caffeine, alcohol, stress have controversion preventative action

39
Q

ototoxic effects of Gentamicin

A

damages the vestibular system and can cause complete hearing loss, vertigo, dizziness, tinnitus

however, it can be used as a last resort therapy in Ménière’s disease to purposefully damage the inner ear to stop attacks

40
Q

migraine and vertigo

A

it is estimated that a quarter of migraine sufferers have spontaneous attacks of vertigo and ataxia

41
Q

describe the auditory symptoms of migraine

A

phonophobia

fluctuating hearing loss and acute permanent hearing loss

motion sensitivity with bouts of motion sickness

42
Q

criteria for definite migrainous vertigo

A
  • Episodic vestibular symptoms of at least moderate severity (interfere with but don’t impede daily activities)
  • Migraine according to International Headache Society criteria
  • At least 1 of the following during at least 2 attacks: migrainous symptoms during vertigo, migraine-specific precipitants of vertigo, response to anti-migrainous drugs
  • Other causes must be ruled out
43
Q

criteria for probable migrainous vertigo

A
  • Episodic vestibular symptoms of at least moderate severity (interfere with but don’t impede daily activities)
  • At least 1 of: migrainous headache, photophobia, phonophobia, visual or other aura
44
Q

compare the use of prochlorperazine and betahistine

A

prochlorperazine is used for more severe cases than betahistine