test 2: 19 developmental orthopedic disease Flashcards

1
Q

Any orthopedic disorder related to abnormal growth, maturation or structural development of bone, cartilage, tendon or ligament

A

DOD
developmental orthopedic disease

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2
Q

OCD vs OC

A

osteochondritis
* manifestation of osteochondrosis

Osteochondritis dissecans (OCD)
* Dissecting flaps of abnormal cartilage at articular surface

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3
Q

— is failure of endochondral ossification

A

Osteochondrosis

usually at secondary physis (ephiphyseal)

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4
Q

what happens during osteochondrosis

A

failure of endochondral ossification

bone failed to form at ephiphyseal (secondary physis)

retained cartilage core are weak and can cause necrosis, fissues/flaps

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5
Q

what can cause OCD

A

rapid growth
nutrition: over fed, abnormal glucose levels, mineral imblances
genetics
biomechanical: joint built wrong

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6
Q

what are the predilection sites for OCD in large animals

A

stifle
tarsus
fetflock
shoulder
cervical articular process joints

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7
Q

clinical signs of OCD

A

+/- Joint effusion
+/- Lameness

often bilateral

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8
Q

in the stifle where is OCD common

A
  • Lateral trochlea ridge (LTR) of the femur
  • Medial trochlea ridge (MTR) of the femur
  • Patella
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9
Q

where is OCD common in the tarsus

A
  • Distal intermediate ridge of the tibia (DIRT)
  • Lateral trochlear ridge (LTR) of talus
  • Medial trochlear ridge (MTR) of talus
  • Medial malleolus of tibia
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10
Q

conservative treatment of OCD

A

time: may heal over time in first year (bone becomes ossified)
rest
dietary restrictions: prevent fast growth
NSAIDS if needed for lameness

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11
Q

when to do surgery on OCD

A

after year 1
want to remove loose or free fragments to prevent further injurt/OA down the line

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12
Q

prognosis of OCD after treatment in stifle and tarsus

A

stifle: good
tarsus: excellent

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13
Q

prognosis of OCD after treatment in fetlock, shoulder and cervical articular process joints

A

fetlock: respond to conservative treatment, but if extensive lesion: poor
shoulder: poor
cervical articular process joints: can lead to OA and type II CVSM

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14
Q

what causes subchondral cyst-like lesions

A

Likely due to trauma to articular cartilage + subchondral bone leading to progressive cyst formation

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15
Q

predilection sites of subchondral cyst-like lesions

A

Medial femoral condyle
phalanges
proximal radius
MC3/MT3

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16
Q

how to treat subchondral cyst-like lesions

A

Stimulate bony filling of cyst to restore biomechanical integrity to joint surface
* Arthroscopic cyst debridement +/- filling cavity
* Translesional screw placement
* Intra-lesional corticosteroid or orthobiologic injection

17
Q

Abnormal endochondral ossification in metaphyseal
physis

A

physitis

18
Q

physitis causes

A

metaphyseal flaring causing visible, painful enlargment

often affects multiple long bones

need to differentiate septic and non-septic

19
Q

how to treat physitis

A

Conservative management
Dietary restriction to limit growth
Exercise restriction
NSAIDs for pain

20
Q

what can cause angular limb deformity

A

Physeal asymmetry

Incomplete ossification of cuboidal bones

21
Q

valgus

A

later deviation

physeal asymmetry that leads to angular limb deformity

22
Q

varsus

A

medial deviation

physeal asymmetry that leads to angular limb deformity

23
Q

predilection sites for angular limb deformities

A

carpus (valgus- lateral)
fetlock (varus- medial)
tarsus (valgus- lateral)

24
Q

conservative treatment of angular limb deformity

A

trimming or shoeing

increase hoof on side you want hoof to grow toward

exercise restriction

25
Q

surgical treatment of angular limb deformities

A

Growth Acceleration
* Periosteal transection (stripping)
* Little evidence of efficacy

Growth Retardation
* Transphyseal bridge
* Transphyseal screw
* Corrective osteotomy / ostectomy
* Try to avoid due to increased difficulty compared to small animals

26
Q

Shoeing: Place extension on — side from which hoof is deviating

A

opposite

Ex. turned out = apply medial extensio

27
Q

Trimming: — the wall toward which hoof is deviating

A

Lower

Ex. turned out = lower outside wall

Create hoof with more hoof on side you want hoof to grow towards

28
Q

flexural limb deformities causes persistent —

A

hyperflexion
tendons too short for bone

can be congenital or acquired

29
Q

prediliction sites of flexural limb deformities

A

coffin: clubbed foot

30
Q

conservative treatment for flexural limb deformities

A

Dietary changes to slow growth Controlled exercise
Encourage stretching without making foal too painful
Toe extension shoes
Oxytetracycline
NSAIDs

31
Q

surgical treatment of flexure limb deformities

A

increase length of ligament

inferior check ligament desmotomy: increase DDFT (coffin joint)

superior check ligament desmotomy: increase SDFT (fetlock, carpus)

32
Q

— is wobblers syndrome

A

Cervical Vertebral Stenotic Myelopathy

compression of spinal cord that leads to compression and ataxia

33
Q

conservative treatment for wobblers

A

conservative: exercise restrictions, NSAIDs, dietary restriction

Cervical Vertebral Stenotic Myelopathy : compression of spinal cord

34
Q

surgical treatment of wobbler’s

A

cervical ventral stabilization

Cervical Vertebral Stenotic Myelopathy : compression of spinal cord