Sweep 4.1 Flashcards
Aggressive Periodontitis secondary features:
phagocyte abnormalities
hyper-responsive macrophage phenotype (e.g., elevated levels of PGE2 and IL-1b
progression may be self-arresting
NPD- Other Findings
Fever and malaise. Moderate elevation of temperature can be observed
White membrane of desquamated cells, bacteria, saliva proteins.
Membrane can be easily removed
Pemphigus vulgaris
Formation of intraepithelial bullae in skin and
mucous membranes.
Strong genetic background (Jewish and Mediterranean)
Painful desquamative lesions, erosions or ulcerations. Chronic course with recurrent bulla formation. Typically in middle age or elderly
Acute myeloid leukemia associated with
gingival changes
Bone loss % =
CEJ-Crest - 2 mm
CEJ-Apex - 2 mm
O’Leary Index plaque
disclose, rinse, count and index teeth that have plaque.
Recurrent HSV diff diagnosis
Aphthous ulcers do not affect keratinized mucosa
Recurrent HSV
Treatment:
Limit bacterial superinfection
Careful plaque control
Anti virals in immunocompromised patients
Thrush
Candida albicans Acquired during birth Pseudomembranous / erosive lesions Predisposing conditions Antibiotics Immunosuppression Malnutrition HIV Diabetes
Lichen planus histopath
Subepithelial band-like accumulation of lymphocytes
characteristic of a type IV hypersensitivity reaction.
Fibrin in the basement membrane.
Deposits of IgM, C3, C4, and C5.
Acantholysis
Canthus layer- another name for stratum spinosum
Why? Because it has intercellular bridges or ‘canthae’
Acantholysis-breakdown of the spinous bridges
Canthus layer-
another name for stratum spinosum
Periapical cemental dysplasia:
Fibrous-osseous cemental lesions.
Differential diagnosis: Cemento-ossifying fibroma and fibrous dysplasia.
Recurrent herpetic infections
herpes labialis
Diagnosis:
-
generally considered an aphtous ulceration.
- ulcers in attached gingiva and hard palate
Hereditary gingival fibromatosis-
possible mechs
TGF-beta1 favor the accumulation of ECM.
May be located on chromosome 2 in human.
