Perio Swep 1.3 Flashcards

1
Q

Papillary-Marginal-Attachment (PMA)-Index

A

Background: “The number of units affected correlates with the severity of gingival inflammation”
Facial gingival surface is divided in 3 scoring units P - M - A
Gingival units affected with gingivitis are counted. Presence or absence of inflammation is counted as {1} and {0}, respectively
Severity component can be considered
Score computed for tooth subject population
First published by Schour & Massler (1947)

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2
Q

Gingival Index (GI)

A

The severity of inflammation is assessed in 4 distinct gingival areas: distofacial papilla, facial margin, mesiofacial papilla, lingual gingival margin.
Scores: 0 to 3; bleeding is considered. Presence of bleeding automatically leads to a score ≥2
Score for tooth subject population
Useful for the calculation of prevalence and severity in population and individual
Frequently used index in clinical trials
First published by Löe (1961) and Löe & Silness (1963)

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3
Q

The bleeding tendency is assessed upon probing a periodontal pocket using

A

standardized pressure
Periodontal probe is inserted to the bottom of the periodontal pocket. Bleeding is observed 15 seconds following retraction of probe.
Presence or absence of bleeding is scored as {1} and {0}, respectively.
Not to be confused with bleeding as scored in GI
B-o-P is a valid indicator for periodontal stability. However, it is a poor indicator of periodontal breakdown.

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4
Q

The Extent and Severity Index (ESI)

A

Agreement
Disease is defined as attachment loss >1mm
Extent
Proportion of tooth sites in a patient showing signs of destructive periodontitis
Severity
Amount of attachment loss at the diseased sites, expressed as a mean value

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5
Q

The Periodontal Index System (PI)

A

Score 0: Negative.
Score 1, 2: Gingivitis
Score 6: Gingivitis with pocket formation
Score 8: Advanced destruction with loss of masticatory function
All teeth are examined. The circumference of each tooth is inspected visually, and given a score.
Index computed for subject, population.

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6
Q

The Periodontal Disease Index System:

This system includes several components:

A
Gingival status
Crevicular measurements
Periodontal Disease Index
Plaque criteria
Calculus Criteria
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7
Q

The Periodontal Disease Index System

A

Score 1, 2, 3: Severity of gingivitis
Score 4: Initial attachment loss (£3 mm)
Score 5: Moderate attachment loss (>3 mm and
£6 mm)
Score 6: Advanced attachment loss (>6 mm)

4 areas per tooth are examined using a probe. The most severe score is tabulated and used for the calculation of the subject’s PDI
Index computed for subject, population

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8
Q

Community Periodontal Index of Treatment Needs (CPITN)

A

Primarily designed to assess periodontal treatment needs in under served parts of the world
Uses a specially designed probe
Epidemiology: 10 index teeth are examined and worst finding is recorded per sextant
For individual subjects: worst finding of all teeth in a sextant is recorded, resulting in 6 scores per subject. The worst score determines the treatment needs score
Published and promoted by the World Health Organization (1982).

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9
Q

Community Periodontal Index of Treatment Needs (CPITN)

Perio status

A
Periodontal Status
Score 1:   Bleeding on gentle probing
Score 2:   Calculus felt during probing,crevicular depth £3 mm
Score 3:   Probing depth 4 mm or 5 mm
Score 4:   Probing depth ³6 mm
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10
Q

Community Periodontal Index of Treatment Needs (CPITN): treatment status

A
Treatment Needs
Code 0:   No treatment
Code I:    Improved oral hygiene
Code II:   I + professional scaling
Code III:  I + II + complex treatment
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11
Q

Periodontal Screening & Recording® (PSR)

Purpose:

A

Periodontal Screening and Recording® (PSR®) is a rapid and effective way to screen patients for periodontal diseases and summarizes necessary information with minimum documentation.

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12
Q

Periodontal Screening & Recording® (PSR)

Endorsement:

A

The ADA and the AAP support the use of PSR® by dentists as a part of oral examinations.

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13
Q

PSR - Benefits: Early detection:

A

PSR® includes evaluation of all sites. For this reason, it is a highly sensitive technique for detecting deviations from periodontal health and a uniquely appropriate screening tool for periodontal diseases that are, by nature, site specific and episodic.

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14
Q

PSR - Benefits:

Speed:

A

Once learned, PSR® takes only a few minutes to conduct for each patient. It can be readily incorporated into routine oral examinations.

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15
Q

PSR - Benefits

Simplicity:

A

PSR® is easy to administer and comprehend. The simplicity of the scoring system aids in monitoring a patient’s periodontal status.

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16
Q

PSR - Benefits

Cost-effectiveness:

A

PSR® utilizes a simple periodontal probe designed specifically for use with this screening system. It does not require the use of expensive equipment.

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17
Q

PSR - Benefits:

Recording ease:

A

Documentation for PSR® requires the recording of six numerical scores, one for each sextant of the mouth. It does not require extensive charting or lengthy narrative explanation.

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18
Q

PSR - Benefits:

Risk management:

A

Proper, consistent, and documented use of PSR® shows that the dentist is evaluating a patient’s periodontal status.

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19
Q

PSR - Limitations

A

PSR® is a screening system designed to detect periodontal diseases. It is not intended to replace a comprehensive periodontal examination when indicated.
Patients who have been treated for periodontal diseases and are in a maintenance phase of therapy require periodic comprehensive periodontal examinations.
In addition, PSR® is designed primarily for use with adult patients and has limited utility in screening children and adolescents.

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20
Q

Peri-implant Tissues and Indices

A

Modified PlI

Modified GI

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21
Q

Reliability of gingival indices

A

(Reliability of an index to measure a condition in the same subject repeatedly and obtain the same score results each time)

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22
Q

Validity of a diagnostic test

A

(Sensitivity and specificity of various diagnostic tools used to create an index)

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23
Q

Halo Effect (Thorndike 1920):

A

The examiner’s general impression of target distorts his/her perception of the target on specific dimensions

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24
Q

Leniency/Severity Error (Saal & Landy 1977):

A

The examiner’s tendency to be lenient or severe

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25
Q

Central Tendency Error (Korman 1971):

A

The examiner’s reluctance to rate at either the positive or negative extreme, so all scores cluster in the middle

26
Q

Calibration involves:

A
  • Several examiners at different experience levels
    - Subjects with various disease extend and severity
    - Follow-up appointments
    Main goal is to determine inter/intra-examiner variations.
27
Q

Sensitivity

=

A

The probability that a test result will be positive when the test is administered to people who actually have the disease in question
Sensitivity = Pr(T+/D+)

28
Q

Specificity

A

The probability that a test will be negative when administered to people who are free of the disease in question
Specificity = Pr(T-/D-)

29
Q

Predictive Value Positive (PVP)

A

The probability of disease in a subject with a positive test result
PVP = Pr(D+/T+)

30
Q

Predictive Value Negative (PVN)

A

The probability of not having the disease when the test is negative
PVN = Pr(D-/T-)

31
Q

Bone loss % =

A

CEJ-Crest - 2 mm divided by

CEJ-Apex - 2 mm

32
Q

Hemiseptal defect:

A

a vertical defect in the presence
of adjacent roots; thus half of a septum remains on
one tooth, AKA one-walled defect

33
Q

Bone Loss in Furcation AreasPathologic resorption of bone within a furcation.

Shows up in radiograph as

A

radiolucency

Furcation radiolucency

34
Q

When radiographs of maxillary molars are observed, a small, triangular radiographic shadow is sometimes noted over either the mesial or distal roots in the proximal furcation area. However,

A

absence of the furcation arrow image does not necessarily mean absence of a bony furcation involvement because the arrow was not seen in a large number of furcations with Degree 2 or 3 involvement.

35
Q

PDL appears as the periodontal space of

A

0.4 to 1.5 mm on radiographs, a radiolucent area between the radiopaque lamina dura of the alveolar bone proper and the radio opaque cementum.

36
Q

Vessels within Interdental Bone appear as

A

They appear as radiolucent lines bordered by thin radiopaque lines

37
Q

IF the maxillary sinus is close to or has invaginated among the roots of the maxillary teeth, there may be difficulties with

A

surgical treatment of the periodontal problems.

38
Q

Mandibular Tori

A

A bony exostosis on the lingual aspect of the mandible, generally in the premolar-molar region; commonly bilateral

39
Q

If the roots are less than 2.5 mm apart

A

Periodontal bone loss will affect the entire interproximal

40
Q

Internal Resorption:

A

Tooth resorption beginning from within the pulp.

41
Q

External Resorption:

A

Resorption of tooth structure beginning on the external surface.

42
Q

To standardize the projection and to ensure the reproducibility of serial radiographs,

A

customized bite blocks was fabricated using XCP film holder (Dentsply) with impression material of hydrophilic addition reaction silicone (Dentsply) and direct conventional radiographic images (Figure 3).

43
Q

Gingival lesions of specific bacterial origin

A

Infective gingivitis and stomatitis.

The lesions may be due to bacteria. 

The lesions may or may not be accompanied by lesions elsewhere in the body.

Bacteria involved:
Neisseria gonorrhea, Treponema pallidum, Streptococci, Mycobacterium chelonae or other org.s

Clinical presentation … Variations

Fiery red edematous painful ulcerations
Asymptomatic chancres
Mucous patches
Atypical non-ulcerated, highly inflamed gingivitis.

Diagnosis:
Biopsy
Microbiologic examination
44
Q

Herpes virus infections-

Herpes simplex viruses type 1 and 2

A

Varicella-zoster virus

Herpes simplex 1 usually causes oral manifestations.
45
Q

Primary herpetic gingivostomatitis-

A

Symptoms:

- painful severe gingivitis with redness 
- ulcerations with serofibrinous exudate 
- edema accompanied by stomatitis
Characteristics:
	- Incubation period is one week.
	- Formation of vesicles, which rupture, coalesce 
	  and leave fibrin-coated ulcers.
	- Healing within 10 to 14 days.
46
Q

Herpes virus can stay latent in trigeminal ganglion for years.

Found in
A

gingivitis, Necrotizing Ulcerative Diseases (NUG/NUP) and periodontitis.

More primary infections occur at older ages in 
industrialized society.
47
Q

Primary herpetic gingivostomatitis may be asymptomatic in childhood, but may also give rise

A
severe gingivostomatitis (painful severe gingivitis with redness, ulcerations with serofibrinous exudate and edema.
The incubation period is 1 week
A characteristic feature is formation of vesicles, which rupture, coalesce, and leave fibrin coated ulcers. 
Fever and lymphadenopathy are other classic features.
48
Q

Recurrent herpetic infections

A

herpes labialis

Vermilion border and/or the skin adjacent to it.

20-40% of individuals with primary infection.

Trauma, UV light exposure, fever, menstruation 

Diagnosis:

- generally considered an aphtous ulceration.
- ulcers in attached gingiva and hard palate.
49
Q

Recurrent infections occur in general more than once a

A

year, usually at the same location on the vermillion border and/or skin adjacent to it, where neural endings are known to be clustering.
Factors that trigger recurrent infections are listed as trauma, UV light exposure, fever, menstruation and others.

50
Q

Herpes zoster-

- Virocella-zoster virus causes varicella (chicken pox).
A
  • Small ulcers usually on the tongue, palatal and gingiva.
    • Latent in the dorsal root ganglion.
    • Unilateral lesions.
    • 2nd and 3rd branch of the trigeminal ganglion.
51
Q

Gingival lesions of fungal origin

A

Candidosis
Linear gingival erythema
Histoplasmosis

52
Q

Candidosis-

Candida species isolated from the mouth:

A

C.albicans, C.glabrata, C.krusei, C.tropicalis, C. parapsilosis and C. guillermondii.

Oral carriage of C.albicans in healthy adults: 3-48%.

Reduced host defense posture

C.albicans is frequently isolated from the subgingival flora of patients with severe periodontitis.

53
Q

Linear gingival erythema-

A
  • Distinct linear erythematous band limited to the free gingiva.
  • Lack of bleeding.
    Positive for C.albicans by culture:
    50% of HIV associated gingivitis sites,
    26% of unaffected sites of HIV seropositive patients, 3% of healthy sites of HIV negative patients.
54
Q

Histoplasmosis-

A
  • Granulomatous disease caused by Histoplasma capsulatum *
    • Acute and chronic pulmonary histoplasmosis and a disseminated form… Immunocompromised patients.
    • Any area of the oral mucosa.
    • Nodular or papillary and later may become ulcerative type of lesions with pain.
    Diagnosis: Clinical view and histopathology
55
Q

Hereditary gingival fibromatosis-

A
  • Diffuse gingival enlargement.
    • Disease entity or a part of a syndrome.
      (example: hypertrichosis, mental retardation, epilepsy, hearing loss, growth retardation, abnormalities of extremities)
    • May interfere with or prevent tooth eruption.
    • Possible mechanism(s):
      TGF-1 favor the accumulation of ECM.
      May be located on chromosome 2 in human.
56
Q

Gingival lesions of systemic origin

A

Allergic and traumatic reactions
Other gingival manifestations
Mucocutaneous disorders

57
Q

Type I reactions (immediate Type),mediated by

A

IgE,

or

58
Q

Type IV reactions (delayed type) mediated by

A

T-cells.

Allergies to:
Dental restorative materials (type IV, contact allergy)

Oral hygiene products, chewing gum and food 
[generally flavor additives or preservatives]

A diffuse fiery red edematous gingivitis sometimes with ulcerations or whitening.

59
Q

Chemical traumatic lesions-

A

Surface etching by various chemical products with toxic properties.

Examples: chlorhexidine-induced mucosal desquamation, acetylsalicylic acid burn, cocaine burn.

Incorrect use of caustics by the dentist.
60
Q

Physical traumatic lesions-

A

Hyperkeratosis, a white leukoplakia-like, frictional keratosis.

Gingival laceration resulting in gingival recession.

Traumatic ulcerative gingival lesion.
(brushing and flossing techniques)
61
Q

Thermal injury-

A

Minor burns from hot beverages.
Mostly seen on palatal and labial mucosa.
Painful, erythematous lesions.
Vesicles may develop.

62
Q

Foreign body reactions-

A

Epithelial ulceration that allows entry of foreign material into gingival connective tissue.
Foreign body can be generally detected via X-rays.
(Examples: amalgam tattoo, abrasives, toothpick etc.)