Surgical Management of GI Tract Tutorial Flashcards

1
Q
Case 1 - 84M 
Bowels have not opened for 3 days
Steady weight
Feals nauseous and anorexic 
Previously normal daily bowel motions

On examination -
Nothing unexpected
Empty rectum with smooth enlargement of prostate
Gossly swollen abdomen

PMH = arthritis of both hips, mild prostatic symptoms 
DH = no medications
SH = ex-smoker

What is the likely differential?

A

Acute intestinal obstruction

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2
Q

Which is more likely - obstruction of small or large bowel? And why?

A

Large bowel or ‘low’ obstruction because:
No evidence of previous abdominal operation - previous abdominal surgery is the commonest cause of small bowel obstruction
No evidence of strangulated external hernia
No vomiting - vomiting is normally a symptom of small intestine obstruction or v. v. late sign of large bowel obstruction
Grossly distended abdomen - because of the size of the large bowel

Obstruction of small bowel = most likely to vomit up the fluids from the stomach, liver, etc.
Strangulated external hernia = common cause of small bowel obstruction
Small bowel obstruction unlikely as it is usually secondary to previous abdo operation
Distension of large bowel = greater than small bowel

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3
Q

What is the first lline of management of a suspected acute intestinal obstruction?

A

Morphine for pain
NGT - aspirated 300ml of green fluid
IV line inserted and started on IV fluids

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4
Q

What is the next suitable investigation for a suspected acute intestinal obstruction?

A

Erect and supine ABX

Erect = fluid level = points towards obstruction
Supine shows distended large bowel

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5
Q

What does the abdominal x-ray (ABX) demonstrate and what is the likely diagnosis?

A

Enormously distended oval gas shadow looped on itself to give a –> ‘best inner-tube sign’ OR ‘coffee bean sign’ = large bowel

Haustrae don’t extend across the width of the gas shadow, suggesting this is large intestine

Likely to be volvulus of sigmoid colon

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6
Q

What is the volvulus of sigmoid colon?

A

Loop of sigmoid colon gets twisted around each other - like turning a key, twists on each other

Forms closed loop - stops anything coming in our out of that section of the large bowel that has looped on itself and trapped itself

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7
Q

What conservative management is effective in treating the majority of patients with a sigmoid volvulus?

A

Sigmoidoscopy =sigmoidscope passed with the patient lying in the left lateral position with their knees up to their chest - scope can be flexible or rigid

Flatus tube = soft rubber rectal tube passed along sigmoidoscope

This usually untwists the volvulus = release of vast quantitied of flatus and liquid faeces

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8
Q

If conservative management using a sigmoidscope and flatus tube does not work, what is the risk of leaving it untreated?

A

Left untreated, the loop of sigmoid, with its blood supply cut off by the torsion, would undergo necrosis

Torsion = cut off blood supply = ischaemia and necrosis of that part of the large bowel

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9
Q

What is the next option if the rigid sigmoidscope and flatus tube did not work?

A

Flexible sigmoidoscopy = requires highly skilled endoscopists

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10
Q

What is the next option if flexible sigmoidoscopy fails?

A

Exploratory laparotomy and sigmoid colectomy (resection of necrotic dead bowel) + join colon with rectum OR end colostomy (Hartmann’s procedure - colon is pulled out through a cut in the left iliac fossa of the abdomen)

Iliostomy = distal ileum = pulled out of cut in the right iliac fossa of the abdomen

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11
Q

Why does Hartmann’s procedure exist? What are the complications of joining the colon with rectum after resection?

A

Can easily fall apart

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12
Q

Case 2 - 84M
PC = 1 day history of abdominal pain, nausea, breathlessness
PMH = COPD, ex-smoker for 3 years

On examination:
Pale and sweaty, BP 100/60, abdominal distension and periumbilical guarding, absent bowel sounds, eveything else normal

Blood tests show:
Low Hb, high WCC, high uea, high creatinine, high serum lactate and high CRP

What is the differential diagnosis and why?

A

AMI - acute mesenteric ischaemia

Ex-smoker = increased CVD risk
Short history - sudden onset of symptoms 
Central pain with guarding
No previous abdominal scar or hernia
No bowel sounds = not working bowel 
Turbulent flow = more likely to clot 
Poor general condition
Increased serum lactate = late sign of bowel ischaemia (metabolic acidosis)
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13
Q

What investigations should be ordered next for this patient’s suspected acute mesenteric ischaemia?

A

CT of abdo and pelvis with contrast

Because you can see:
Thrombus in arteries and veins
Abnormal enhancement of bowel wall = ischaemia
Presence of embolus or infarction of other organs = something is firing off emboli in the body

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14
Q

CT scan shows: normal aorta and SMA
However, as we go lower down the body, SMA at middle colic artery no longer lighting up with contrast = embolus or thrombus blocking the SMA

What does SMA supply?

A

SMA = small and large bowel around splenic flexure

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15
Q

Fluid filled ichaemic small bowel on CT

Bowel is not enhancing - contrast is not reaching that area of bowel so blood supply is not reaching that area of bowel

What is the management for this patient?

A

Emergency exploratory laparotomy = damage control = get to the bowel before necrosis OR if already necrotic, resect necrotic bowel

Restore blood flow via SMA to recover the ischaemic bowel

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16
Q

How do emergency exploratory laparotomies work and what do they aim to do?

A

Middle midline incision
Evaluate the abdominal viscera
If obvious intestinal necrosis - resection of the affected bowel looks

What happens after a damage control laparotomy - go back, check bowel, suually find other bits of necrotic bowel that needs removing

Restore blood flow in SMA e.g. embolectomy (blow up balloon and anticoagulate and pull out embolus), OR endovascular management of SMA thrombus (use catheter to inject thrombolytic material), OR arterial bypass of SMA (rare)

17
Q

What are the endovascular management aims of an emergency exploratory laparotomy?

A

Endovascular management aims =
- Relief of acute symptoms with restoration of venous patency, prevention of clot propagation, and subsequent pulmonary embolism

  • Aims for clot lysis through the use of catheter-directed thrombolytic therapy
18
Q

What are some arterial causes of of AMIs?

A

Embolism (50%) =

  • From left auricle - atrial fibrillation
  • A mural infarct
  • Atheroma from aorta or aneurysm
  • Endocarditis vegetations
  • Left atrial myxoma

Thrombosis (20–35 %) =

  • Blocks origin of superior mesenteric artery & can cause ischaemia of full length of small bowel.
  • Due to atherosclerosis = often all main splanchnic vessels—coeliac, superior & inferior mesenteric arteries

Nonocclusive (<5%)

  • Due to hypotension/hypoperfusion.
  • Due to vasospasm in shock—nonocclusive mesenteric ischaemia (NOMI).
  • Critically ill patients with vasopressor requirements
  • Those undergoing dialysis with large volume fluid removal
19
Q

Can acute mesenteric ischaemia happen due to venous reasons?

A

Yes - venous = 10-15%

Superior mesenteric vein thrombosis

20
Q

What are some risk factors that can cause mesenteric vein thrombosis?

A

Portal hypertension
Portal pyaemia
Sickle cell disease

Related to the presence of an underlying hypercoagulable state