Stroke Syndromes Flashcards
Ischemic Stroke
Occlusion of a blood vessel; more common than hemorrhagic
*Thrombotic are most common
Hemorrhagic Stroke
Rupture of an aneurysm (Anterior communicating arteries)
- Produces crescent shaped CT image
- Will see severe headache, nuchal rigidity, and decreased consciousness
Lacunar infarct
Small infarcted area due to occlusion of small end artery
Watershed infarct
Occurs b/w distribution of two mjor arterie
TIA
Transient Ischemic Attack
Normal fnxn returns in 30mins-24hrs
*Assoc. w/ increased risk for stroke; possibly within hours
Ischemic cascade
Death of neuronal cells causes excess release of glutamate
=>Ca2+ into other cells causing their death as well
Pnumbra
Area surrounding center of infarction in which damage is not yet irreversible
Lesion of optic chiasm
Produces a bitemporal hemianopsia
Lesion central to optic chiasm
Produces homonymous hemianopsia
-(Optic tracct, lateral geniculate nucleus of thalamus, Area 17)
ICA Stroke
- May have amaurosis fugax due to occlusion of CRA
- Global aphasia
- Eye deviation to side of lesion
- Tongue deviation away from lesion
Contralateral:
Spastic paralysis w/ Babinski (Primary Motor Cortex)
Paralysis of lower face (UMNs of VII)
Loss of fine touch, vibration, conscious proprioception (SI)
-Pt. cannot localize pain either
Ipsilateral:
Vision loss
MCA stroke
-Global aphasia if on left
Contralateral:
Spastic paralysis (Lateral part of Area 4)
Loss of fine touch, vibration, conscious proprioception (Lateral part of Areas, 3,1,2)
No pain localization (Lateral part of Areas 3,1,2)
*All upper body > lower body
Ipsilateral: Eye deviation towards lesion Tongue deviation away from lesion Neglect `
ACA stroke
-Abulia, akinetic mutism, urinary incontinence
Contralateral:
Spastic paralysis w/ Babinski (Medial part of Area 4)
Loss of fine touch, vibration, conscious proprioception (Medial Areas 3,1,2)
No localization of pain (Medial Areas 3,1,2)
*Upper body > Lower body
PCA stroke
-Contralateral homonymous hemianopsia w/ macular sparing
- Memory deficits
- Due to damaged hippocampus
Abulia
Loss of ability to act voluntarily
Akinetic mutism
Decreased thought, movement, speech, emotion