stroke Flashcards

1
Q

Stroke/CVA

A

brain attack
A stroke, also known as a cerebrovascular accident (CVA), is the rapid loss of brain function(s) due to disturbance in the blood supply to the brain. This can be due to ischemia (lack of blood flow) caused by blockage (thrombosis, arterial embolism), or a haemorrhage (leakage of blood).

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2
Q

Classification

A

Haemorrhagic stroke

Ischaemia stroke

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3
Q

Haemorrhagic stroke

A

bleeding from a cerebral artery

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4
Q

Subarachniod haemorrhage

A

bleeding into subarachnoid space
due to rupture of congenital anerysm or trauma
sudden intense headache, vomiting, neck stiffness and loss of consciousness
10% die within two hours
40% die within two weeks

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5
Q

Aneurysm

A

saccular
fusiform
ruptured

treated by surgical clipping

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6
Q

Intracerebral haemorhage

A
bleeding in deeper part of brain
longstanding hypertension
arterial wall weaken
micro-aneurysm develop 
rupture and bleed
sever headache and vomitting
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7
Q

Ischaemic stroke

A

80% strokes due to occulsion
atherom of cerecral artery
emboli from atheromatous plaque in hear/neck vessel

Most common vessel: MCA > PCA > ACA> brainstem (more serious)

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8
Q

Embolic stroke

A

completed stroke -tia-
sudden onset

blood clot that forms elsewhere in the body (embolus) breaks loose and travels to the brain via the bloodstream.
lodges in cerebral artery distruptiung the flow of blood

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9
Q

artrial fibrilation

A

Atrial fibrillation is irregular heartbeat,blood clots are more likely to form in your heart, increasing stroke risk

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10
Q

thrombotic stroke

A
completed stroke-tia-
 overnight
develops over several days
early full recovery
20% risk of full CVA with a month
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11
Q

Bamford classification

A

TACS – Total anterior circulation stroke
PACS – Partial anterior circulation stroke
POCS– Posterior circulation stroke
LACS – Lacunar stroke(deep penetrating arteries)
(TAC)S=stroke
(TAC)I=infarct
(TAC)H=haemorrhage

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12
Q

Tramatic brain injury

A

injury to the brain caused by external physical force,
altered state of consciousness
impairment of cognitive or physical abilities
disturbances of behaviour or emotional functioning

temporary or permanent

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13
Q

Mechanism of injury

A

Penetrating injury-risk of infection

Compression - inner cerebral traume

Deceleration- outer cerebral injury discrete cognitive deficit

Diffuse axonal injury– poor communication between brain structures, reduces processing speed, global impairment

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14
Q

Extradural haemorrhage

A

bleeding into extradural space

cause by severe trauma

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15
Q

subadural haemorrhage

A

bleeding in subdural space
usually cause by severe trauma
maybe be delayed symptons

headache
drowsiness 
stupor- altered state of conciousness
hemiparesis
coma
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16
Q

Secondary damage of TBI

A
raised ICP (intercranial pressure)
infection
ischaemic changes
vasospasm
complication of systemic dysfunction
17
Q

Transient ischaemic attack TIA

A

A TIA is a sign that part of the brain is not getting enough blood, and there is a risk of a more serious stroke in future

18
Q

Hemiplegia

A

paralysis at one side of the body

19
Q

Clinical features of stroke

MOTOR

A

Alterations in tone
Low tone - flaccidity
High tone – spasticity

Ataxia
Weakness 
Asymmetry
Loss of normal movement patterns,postural adjustments and balance	
Compensations
20
Q

Pattern of spasticity

A
Flexor pattern in upper limb
Shoulder elevation & retraction
Internal rotation & adduction
forearm pronation
elbow worst and and finger flexion
Extensor pattern in lower limb
Hip retraction
Hip extension
Knee extension
Ankle plantarflexion & inversion
21
Q

Clinical features of stroke

SENSORY

A

impaired cutaneous sensation
stereognosis
proprioceptive impairment
Visual field loss- Homonymous Hemianopia

22
Q

Clinical features of stroke

SPEECH

A

Expressive dysphasia
Damage to Brocas area
Loose ability to produce speech

Receptive dysphasia
Damage to Wernikes area in temporal lobe
Lose ability to understand speech

global aphasia

23
Q

Clinical features of stroke

COGNITIVE

A

Perceptual Problems

Astereognosis
Visual Agnosia
Auditory Agnosia
Depth perception
Apraxia
Anosagnosia 
Inattention/neglect
24
Q

Agnosia

A

Inability to recognise

25
Q

Neglet

A

more common with left hemiplegia
patients fails to attend to stimuli from the one side
poor prognosis if persists
results in many functional problems

26
Q

Clinical features of stroke

A
Disorders of emotion 
Delay / absent swallow reflex
Incontinence 
Secondary musculoskeletal problems
Gait problems
Functional difficulties
Social problems
27
Q

Physiotherapy treatment

A

prevent complications,
minimise impairments and to maximise function

Assessment
Identification of problems
Clinical reasoning
Goal setting
Objective outcomes
Choice of Rx method
Interdisciplinary input
28
Q

Factors affecting recovery

A

Extent and nature of lesion
Integrity of collateral circulation
Capacity of Nervous System to re-organise

Environment
Pre-morbid status
Age
Co-exisiting conditions
Nutrition/Hydration
Medication

Motivation participation and Attitude of patient

29
Q

Medical management

A
Treat as medical emergency
MRI/CT scan
Ischaemic stroke (80%)– aspirin, anticoagulants - thrombolysis
SAH – surgery or endoplastic procedures
SDH/EDH/ICH – treat hypertension
TIA – aspirin, prevention work
Other tests – Blood tests, angiography, echocardiology
Other medication – osmotic agents
Surgery – carotid endarterectomy
30
Q

National Stroke startergy

A

Treat Stroke as a medical emergency
Effective assessment and treatment of vascular risk factors
TIAs assessed and scanned within 24hrs
Suspected stroke transferred to specialist stroke unit with MDT treatment and assessment
Range of services available locally to support long-term needs
Opportunity to return to work

31
Q

lacunar stroke

A

??