Stolove Final - PNS

Question 1

What is the main source of peripheral neuropathy?

Answer 1

Occlusion of blood vessels to nerve tissue

Question 2

definitions of neuropraxia, myleinopathy, axonotmesis, and neurometesis

Answer 2

nerve compression
segmental damage
crush injury
complete severence

Question 3

what is disturbed during a myopathy?

Answer 3

nerve is normal, muscle units are not

Question 4

What is effected is a spinal motor nerve is damaged? dorsal root nerve?
what is a sign of a general neuropathy?
myopathy sign?

Answer 4

myotome
dermatome
stocking glove distribution(feet effected first)
proximal weakness

Question 5

what nerve is effected first during charcot marie tooth disease?
when does it first present?
signs?(gait)
treatment? (3)

Answer 5

peroneal
childhood
footdrop, steppage gate (winebottle)
orthotics, skin precautions, ROM

Question 6

carpal tunnel syndrome presents pain what time of the day?

what are the 3 clinical signs?

Answer 6

night

thenar weakness, atrophy, reduced grip strength

Question 7

main PT treatment for carpal tunnel syndrome? what does this do?

Answer 7

nerve and tendon gliding techniques to reduce scarring and adhesions

Question 8

Bells palsy presents with what main neuropathy?
3 ways one could attain bells palsy?
physical signs of BP (5)

Answer 8

unilateral facial nerve paralysis
latent herpes virus, inflammation of nerve, compression of nerve by acoustic neuroma
loss of taste, dry eye, thick saliva, mouth drop, things sound louder

Question 9

3 presentations associated with tardy ulnar palsy

what makes symptoms worse?

Answer 9

clawhand, flat hypothenar eminance, atrophy of interossei

elbow flexion

Question 10

2 signs use to detect mortons neuroma

Answer 10

mulders sign( palpate met head space as compression is appled)
lesegues sign(dorsiflex involved toe)

Question 11

Generalized Symmetric Polyneuropathy
Acute sensory neuropathy presents with what?
Chronic sensory neuropathy presents with what? (7)

Answer 11

allodynia (pain from prolonged exposure to non-painful stimuli (recoverable)
sensory loss, burning, parasthesias, impaired vibration, proprioception, diminished DTR
may have some distal motor weakness

Question 12

3 physical signs to look out in someone with generalized symmetric polyneuropathy (both chronic or autonomic)
treatment for this?

Answer 12

sweating,
orhostatic hypotension (falling)
resting tachycardia
control hyperglycemia

Question 13

signs of alcoholic neuropathy (5)

Answer 13

loss muscle bulk
decreased ankle DTRs, sensation in feet
aching calves
stocking glove paresthesias
footdrop/wristdrop

Question 14

3 parts of body paralyzed by guillian barre syndrome

Answer 14

face, trunk, extremities. (no DTRs)

Question 15

PT treatment of guillian barre syndrome involves what? (3)

Answer 15

prevent contracture, and decubiti (bed sores)
monitor vitals carefully
ROM and gentle stretching

Question 16

What kind of cell body is mainly effected by post polio syndrome?
2 clinical signs.

Answer 16

motor (degeneration of axons)

decline in strength (periods of stabilty) and excessive fatgue

Question 17

3 ways PTs treat post polio syndrome

Answer 17

non exhaustive general body conditioning
energy conservation
orthotics/gait aids

Question 18

chicken pox virus lies within the sensory ganglia of which 2 kind of nerves?
pain along where?
how treated? (2)

Answer 18

sensory and cranial
dermatome
oral anti virals, and analgesics

Question 19

tic douloreux effects which cranial nerve?
what triggers the effects?
how to treat? (2)

Answer 19

5th (trigeminal -maxillary divsion mainly)
chewing or smiling
tegretol or surgery-radio frequency rhizotomy

Question 20

what is paraneoplastic syndrome ?

3 types of pain associated with it

Answer 20

autoimmune response, directed at the cancer, attacks the neuron
burning, aching, stabbing

Question 21

lead toxicity affects both the CNS and PNS. what occurs to the PNS?
how to treat? (2)

Answer 21

initial demyelination followed by damage to cell body and axon
chelators, and cock-up spints

Question 22

motor endlplate summary of site of action pesticides/organophosphates affects which neurotransmitter the most?
is there too much or not enough of it?
how to treat?

Answer 22

Acetylcholine (ACh)
too much
increasing AChE

Question 23

how to treat pesticide and organophosphate toxicity (2)

Answer 23

wash skin and hair or emesis/lavage

inject pralidoxine, a cholinesterase activator

Question 24

Myesthenia Gravis pathogenesis
abnormality of which organ?
clincial signs (3)
how to treat?

Answer 24

decrease in ACH receptors
abnormality of thymus
muscle weakness, ocular muscles effected, cranial nerve muscles first to be affected
AChE inhibitor which increases ACh production

Question 25

pesicides/organophosphate what do they do to ACh in the body?

Answer 25

too much ACh production

treat by increasing AChE

Question 26

botulism results in what?

Answer 26

not enough ACh

Question 27

3 PT treatments of myesthenia Gravis

Answer 27

moniter breathing
energy conservation
isometrics

Question 28

clinical manifestations of botulism (2)

Answer 28

resirpatory failure, flaccid paralysis

Question 29

Complex Regional Pain Syndrome (CRPS) difference between 1 and 2

Answer 29

CRPS 1: occurs in absense of overt nerve injury

CRPS 2: occurs after peripheral nerve trauma

Question 30

reflex sympathetic (dystrophy Complex Regional Pain Syndrome (CRPS) ) pathogenesis
clinical signs (5)
treatment (2)

Answer 30

somatic injury triggers sympathetic efferent activity
reflex neurogenic inflammation
activation of nociceptors 
pain, edema, weakness, tremor spasm etc
TENS aquatic