Shock: General Overview and Approach Flashcards

1
Q

definition of shock

A

inadequate tissue perfusion to meet cellular requirements of O2 and nutrtients

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2
Q

outline the changes to the vascular system that occurs during stress to ensure oxygen delivery

A
  • increased HR
  • increased contractility
  • vasoconstriction and vasodilation to enhance blood flow.
  • if unable to meet metabolic requirements, leads to end organd dysfunction and clinical evidence of shock.
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3
Q

how to blood pressure change in someone who is hsock

A

usually hypotensive (but not always).

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4
Q

how is preload clinically evaluated

A

JVP or catheter pressures

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5
Q

4 broad categories of shock

A
  1. hypovolemic
  2. cardiogenic
  3. obstructive
  4. distributive.
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6
Q

primary problem of hypovolemic shock

A
  • reduced preload due to tauma (ex/ traumatic amputation, massive GI bleed)
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7
Q

what clinical/physical exam findings are altered when a person has hypovolemic shock

A

hypovolemic shock is caused by decreased preload, which results in low JVP

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8
Q

hypovolemic shock is caused by decreased preload, which results in low JVP. What mechanisms are in place to compensate for the preload reduction?

A
  1. afterload– results in cool extremities
  2. contractiliy –>increased CO
  3. rate–> tachycardia
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9
Q

primary problem causing cardiogenic shock

A
  1. contractilty –>> decreased CO
  2. rate –> rachycardia
  3. rhythm –>pulse irregularity
    - all cause an elevated JVP.

this can be caused due to MI ex

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10
Q

cardiogenic shock is due to decreased CO, tachycardia, and pulse irregulairty causing an elevated JVP and poor system circulation and perfusion. What systems are in place to compensate for the measures of cardiogenic shock?

A
  1. afterload –> cool extremities
  2. rate –> tachycardia
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11
Q

causes of distributive shock and primary problem of DS.

A
  • usually septic or anaphylaxis

primary problem is reduced afterload. –> warm extremities

compensation is elevated contractiltiy causing increased CO and tachycardia

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12
Q

6 main mechanisms of how inadequate perfusion may occur

A
  1. afterload changes
  2. changes in rate
  3. changes in rhythm
  4. changes in preload
  5. changes in contractility
  6. changes in blood content.
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13
Q

causes of obstructive shock and primary problems of obstructive shock

A
  • can be due to PE or pulmonary hypertension
  • primary problem is due to reduced contractility (decreased CO) or elevated preload (elevated JVP)

compensated due to afterload (cool extremities) and rate (tachy

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14
Q

two types of shock that result in high JVP elevation

A
  • cardiogenic
  • obstructive.
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15
Q

type of shock that has warm extremities

A

distributive shock

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16
Q

two types of shock with low JVP

A
  • hypovolemic shock
  • decompensated distributive.
17
Q

initial workup for shock

A
  1. history
  2. physical exam
  3. investigations; abg, cbc, lytes, creatinine, glucose, ECG, CXR
18
Q

clinical manifestations of shock

A
19
Q

Six Physiologic Factors to Optimize in the Management of Shock (PRRCAC)

A

Preload

Rate

Rhythm

Contractility

Afterload
Content

20
Q

adequad preload is essential for cardiac output. You must assess JVP or CVP, or if necessary use echo or PAC.

too little preload –> administer ___

too much preload –> administer ___,___

A

too little: administer fluids

too much preload ; administer diuretics or venodilators

21
Q

how should you give fluids to someone in preload?

A

BOLUS– not infusion.

22
Q

Rate
 Too fast: -usually need to treat the
underlying cause -may need rate-controlling agent
or cardioversion
 Too slow: -administer __, ___, pacing
(transcutaneous or transvenous)

A

Rate
 Too fast: -usually need to treat the
underlying cause -may need rate-controlling agent
or cardioversion
 Too slow: -atropine, epinephrine, pacing
(transcutaneous or transvenous)

23
Q

contractility can be assessed with ___.

inadequate contractility can be treated by:

A

assessed with echo

  • trated by inotropes, optimize oxygen delivery to myocardium, correct metabolic derangements
24
Q

Afterload
 Assess through clinical exam
 Too much: ___, typically in
combination with fluids +/- inotropic
agent
 Too little: ___

A

Afterload
 Assess through clinical exam
 Too much: vasodilators, typically in
combination with fluids +/- inotropic
agent
 Too little: vasoconstrictors

25
Q
A