Cardiac Arrest Flashcards

1
Q

SCA = sudden, termination ___ activity associated
LOC, __ ___ and ____

A

SCA = sudden, termination cardiac activity associated
LOC, spontaneous breathing and circulation

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2
Q

T/F all unresponsive persons should be assumed to have cardiac arrest and CPR should be started immediately

A

true. it takes too long to confirm on the scene. Don’t be fooled by the pulse or agnoal repsiration

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3
Q
A
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4
Q

Outline some cardiovascular and non cardiac causes of SCA

A

cardiovascular: acute MI, ischemica, v fib, valvular heart disease, WPW, long QT, brugada, aneusym/dissection or aorta, cardiac tampnate or trauma.

non cardiac: PE, pneumothorax, drugs, varices, peripartum, severe hypoxia, hypoglycemia, hypothermia, hypo/hyperkalemia, acidosis.

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5
Q

outline the 3-phase time-sensitive model.

A
  1. electrical; defibrillation is paramount
  2. circulatory; chest compressions, vasopressors and defibrillation
  3. metabolic: new therapies: PCI, ECLS, TTM
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6
Q

the main cause of SCA is ____ resulting in ____, but arrythmias like ___ is increasing in proportion

A

the main cause of SCA is CAD resulting in Vfib, but arrythmias like PEA or Asystole is incerasing in proportion over time. probably cause we are treating CAD with beta blockers and stuff which also prevent Vfib.

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7
Q

2 main meds used in SCA

A

Amiodarone is an antiarrhythmic medication used to treat and prevent a number of types of irregular heartbeats. This includes ventricular tachycardia, ventricular fibrillation, and wide complex tachycardia, as well as atrial fibrillation and paroxysmal supraventricular tachycardia

EPINEPHRINE 1mg

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8
Q

H’s and T’s of causes of cardiac arrest

A

hypokalemia, (or hyperkalemia), hypothermia, hypovolemia, hypoxia

tamponade, tension pneumothorax, thrombosis, toxins

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9
Q

CA resuscitation overal goals

A

chest compressions, oxygenation/ventrilation, early defibrillation, immediate attention to reversible causes like hypoxia or hyperkalemia

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10
Q

possible etiologies to this rhythm

A

this is Vfib. could be ischemic heart disease, severe hypoxia, electrocution, drug toxicity, anti-arrhthmic drugs, trauma, valve problem, congenital heart disease, heart surgery

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11
Q

when you are performing CPR what intervals should you give epinephrrin

A

epinephrine every 3-5 minutes. Consider advanced airway

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12
Q

CABDD initial management of Vfib

A

chest compressions

airway

breathing

defibrillation

diagnosis

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13
Q

___ ___ ___ is a syndrome characterized by an absent pulse, unconsciousness, and organized electrical activity on the ECG

A

pulseless electrical actibity

  • definining pathophysiology is challenging given the heterogeneity and lack of valid models.
  • 50% may be ischemic, 1/3 undergo PCI
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14
Q

5 H’s and 5 T’s of pulseless electrical activity.

A
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15
Q

NOTE:

A
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16
Q

PEA management

A

• Priorities:
– C-A-B’s
Epinephrine 1 mg asap and then q3-5 min
– Early identification of treatable conditions (see if you can overturn any H’s or T’s)

17
Q

T/F Resuscitation guidelines since 2010 have de-
emphasized advanced airway/ETT

A

TRUE. CPR most important. if you are competant, then sure do an airway. FOR SHOCKABLE RHYTHMS< they don’t need time taken to intubate them. SHOCK!

18
Q
A
19
Q

epinephrine is a strong ___ agonist. What are it’s effects

A

strong Alpha1 agonist. thus, it promotes contractility of the heart, increases heart rae, and increases SVR. increasing SVR is a negative aspect of it when you are trying to do CPR though

20
Q

in addition to epinephrine, what two other drugs have been proven useful in cardiac arrest setting?

A

amiodarone and lidocaine.

21
Q

S __, __, Epi: The Right Cocktail? It showed improved NEUROLOGICAL survival by 13.9%

A

epineprhine, vasopressin and steroids.

22
Q

EtC02< 10-20 mm Hg after ___ mins ACLS strongly associated with failure of resuscitation

A

after 20 minutes. also cardiac standstill on echo is a positive predictor of poor outcome

23
Q

• Initial objectives of post resuscitation care are to:

A

– Optimize cardiopulmonary function and systemic
perfusion, especially perfusion to the brain

– Try to identify the precipitating causes of the arrest

– Institute measures that may improve long-term,
neurologically intact survival

24
Q

temperature relationship with resuscitation

A

fever is a large negative in post cardiac resuscitaiton care. in all we’re still doing hypothermic practices. (33-36 degrees)

25
Q

pathophysiological processes of post cardiac arrest syndrome

A
  • brain injury )hypoxic0ischemic encephalopathy)
  • myocardial dysfunction
  • systemic ischemia/reperusion injury
26
Q

Predictors of poor outcome from coma/cardiac arrest:

• Absence of ___ or
corneal responses 72
hours

• Absent ___ response
other than extensor 72
hours

• Myoclonic status
___

• Elevated ___ (biomarker)

A

• Absence of PUPILLARY or
corneal responses 72
hours

• Absent MOTOR response
other than extensor 72
hours

• Myoclonic status
EPILEPTICUS

• Elevated NSE

27
Q

NOTE

A
28
Q

outline aspects of the CASPRI score

A
29
Q

Ho w to Neuroprognosticate?

A

• Physical exam remains the gold standard

• No sign or test in isolation should be the basis
for decisions •

Constellation of findings is important • In the TTM era allow time and more time…

30
Q
A