Shock Flashcards

1
Q

Define shock

A

-An acute circulatory crisis marked by hypotension and inadequate blood flow

-this can be severe and potentially fatal symptoms develop as the tissues become starved of oxygen and nutrients

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2
Q

What are the 3 basic stages of shock?

A
  1. compensatory stage- when arterial pressure and tissue perfusion are reduced, compensatory mechanisms are activated to maintain perfusion to the heart and brain- warm stage
  2. progressive stage- begins as compensatory mechanism fail to maintain cardiac output and tissues become hypoxic because of poor perfusion- cold stage
  3. irreversible or refractory stage- as shock syndrome progresses, permanent organ damage occurs as compensatory mechanisms can no longer maintain cardiac output
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3
Q

How is shock treated and cared for?

A

-re-establish haemodynamic stability (fluid bolus up to 100mls/ kilo can be given to replace circulating volume, but can dilute haemoglobin)

-establish adequate ventilation and oxygenation (oxygen given with a non rebreathe mask)

-medications such as inotropic (change force of heart contractions), antibiotics, noradrenaline

-increase cardiac function by reducing the work and improving efficiency of the heart

-provide adequate nutrition and metabolic state

-prevent any complications that result

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4
Q

What are the possible complications of shock?

A

-acute respiratory distress syndrome (ARDS)
-acute kidney injury (AKI)
-multi-organ dysfunction (MODS)
-disseminated intravascular coagulation (DIC)

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5
Q

What is preload?

A

the stretching of the heart muscle before contraction and the volume of blood in the heart’s chambers at the end of diastole

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6
Q

What is contractility?

A

force exterted by myocardium to push blood out into aorta or pulmonary artery

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7
Q

What is afterload?

A

the amount of resistance the heart must overcome to open the aortic valve and push the blood volume out into the systemic circulation

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8
Q

Explain the pathophysiology of shock?

A

-Blood circulating volume affects cardiac output, which creates arterial blood pressure

-if BCV drops, cardiac output drops, which causes hypoxia in tissues from inadequate perfusion

-this causes acidosis and vasoconstriction, which increases BP, but acidosis can also result in multi-organ failure

-arterial BP decreases renal blood flow, possibly causing renal failure

-reduced renal blood flow causes the RAAS response in the distal convoluted tubule, which increases water retention and therefore BCV increases

-infection or increased acidosis causes more tissue mediators to be released, which can result in arteriolar and capillary dilation, which can increase permeability, making them leaky, reducing BCV

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9
Q

Explain the symptoms of shock and what causes them

A

-increased respiratory rate= helps alleviate systemic acidosis by removing excess CO2
-gradual drop of oxygen saturation= less blood flow around body= hypoxia

-increased HR (tachycardia)= heart pumping faster and harder to keep blood circulating
-falling BP= able to compensate, a pre terminal sign, of the last signs, indicating cardiac arrest may occur
-increased capillary refill time and cold hands and feet= body shutting down peripherally

-decrease in conscious level and pupil change= reduced blood flow to brain and dropping BP

-reduced urine output= indicates low BCV

-temperature changes

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10
Q

What are the 3 causes of shock?

A
  1. Hypovolaemia= loss of fluid (BCV) occurs as a result of dehydration, D&V, injury, trauma
  2. Cardiogenic= inadequate pump, issues with contractibility, heart failure may occur as a result of congenital anomalies, infections, myopathy
  3. distributive= can be anaphylactic shock (allergen), neurogenic (spinal injury), or septic (infection). Hypotension may occur in this type of shock
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