shock Flashcards
what does cardiogenic shock result from?
decreased myocardial contractility
symptoms of cardiogenic shock?
cool/pale skin
altered mental status
thready pulse
crackles/dyspnea
dec. output!
heart rate in cardiogenic shock
high
BP/MAP in cardiogenic shock
low
CVP in in cardiogenic shock
high
wedge in cardiogenic shock
high
SVR in cardiogenic shock
high
CO in cardiogenic shock
low
reason for hemodynamic change in cardiogenic shock
sympathetic/RAAS response
medical management in cardiogenic shock
+ inotropes (dobutamine, dopamine, digoxin, milrinone)
diuretics
nitrates
opioids
vasopressors
nursing interventions in cardiogenic shock
afterload reducer
preload reducer
HOB elevated
intraaortic balloon pump
surgical intervention for cardiogenic shock
intraaortic balloon pump
complications in cardiogenic shock
dysrhythmias
med dependence (dobutamine)
acute tubular necrosis (kidney failure)
what is important to remmebr about intraaortic balloon pump for cardiogenic shock
goes through fem
bleeding/pulse concern
increases O2 supply and decreases demand
in terms of shock, which kinds of shock are “pool” “pump” and “pipe” problems
cardiogenic = pump problem
hypovolemic = pool
early septic = pipe
what does hypovolemic shock result from?
-dec. blood/intravascular volume
-internal & external fluid shifts
symptoms in hypovolemic shock
cool/pale! & diaphoretic!
dec. LOC!
tachycardia
hypoTN
postural vs
tachypnea
oliguria
heart rate in hypovolemic shock
high
BP/MAP in hypovolemic shock
low (no volume to pump)
CVP in hypovolemic shock
low
wedge in hypovolemic shock
low
SVR in hypovolemic shock
high (compensation)
CO in hypovolemic shock
low (no volume)
why are there hempdynamic changes in hypovolemic shock
vasoconstriction and compensation from sympathetic/RAAS
med treatment in hypovolemic shock
IV FLUID OR BLOOD TO REPLACE VOLUME
vasopressors??
nursing interventions in hypovolemic shock
modified trendelenburg
complications in hypovolemic shock
tubular necrosis
shock liver from dec. perfusion
what is early septic shock from?
release of toxins from bacteria -> damage to vascular endothelium -> water leak
-altered vascular permeability
s/sx of early septic shock
warm/dry/flushed skin!
fever
increased WBCs
HR in early septic shock
high
BP/MAP in early septic shock
slightly low
CVP in early septic shock
low
wedge in early septic shock
low
SVR in early septic shock
low
(bacteria causes vasodilation)
CO in early septic shock
normal
(or high)
reasons for hemodynamic changes in early septic shock
altered vascular permeability, bacteria causes vasodilation
(protein leakage -> fluid follows)
medical management of early septic shock
**fluids/antimicrobials
**vasopressors and low dose steroids after fluids
nursing interventions for early septic shock
**cooling blanket/antipyretic
ventilation
which forms of shock look similar symptomatically?
cardiogenic and late septic
what does late septic shock come from?
progression of early shock, irreversible vascular issues
**contractility worsens
s/sx of late spetic shock
**cold/clamy
diaphoretic
dec. LOC
dec. urine
signs of increased preload
HR in late spetic shock
high
BP/MAP in late spetic shock
severely low
CVP in late spetic shock
high
wedge in late spetic shock
high
SVR in late spetic shock
high (from vasopressors and compensation)
CO in late spetic shock
very low
med management of late spetic shock
+ inotropes (dobutamine dopamine)
vasopressors
corticosteroids
antibiotics
nursing interventions for late spetic shock
supportive care like DNR/CC
complications of late septic shock
MODS
death
which lab value is high in sepsis
lactate
what are the 3 kinds of distributive shock?
septic, anaphylactic, neurogenic
indicatord of shock
MAP < 60
high lactate
high bicarb (ABGs)
what are the 3 stages of shock? (progression order)
compensatory, progressive, refractory
what is a rare form of shock from massive dilation of blood vessels as a result of loss of sympathetic tone
neurogenic shock
which form of shock has signs of hypotension, bradycardia, and hypothermia?
neurogenic
treatment of neurogenic shock
treat cause
volume replacement
vasopressors
pathways used to recognize sepsis
SIRS and qSOFA
what are the 3 aspects of qSOFA
low bp
altered mentation
high resp rate
what 4 aspects make up the SIRS criteria
temp >38
HR >90
RR >20 OR PaCO2 <32
WBC >12000
when are blood products administered?
when hgb is 7.0-9.0 or lower