neuro pt. 2 (SAH) Flashcards

1
Q

what are the 3 hematoma/hemorrhage types

A

subarachnoid
epidural
subdural

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2
Q

bleeding into the subarachnoid space

A

subarachnoid hemorrhage (SAH)

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3
Q

what is a subarachnoid hemorrhage (SAH) usually caused by?

A

rupture of cerebral aneurysm

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4
Q

where are cerebral aneurysms usually found?

A

circle of willis

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5
Q

where does a cerebral aneurysm send arterial blood?

A

into subarachnoid space to mix with CSF

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6
Q

clinical manifestations of SAH

A

-after rupture = “worst headache of life” or WHOL
-n/v
-dec. LOC / irritability
-signs of meningeal irritation

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7
Q

signs of meningeal irritation in SAH

A

-stiff/painful neck
-photophobia, blurred vision
-fever
-+ Kernig/Brudzinski sign

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8
Q

describe positive kernig and brudzinski sign for meningela irritation / SAH

A

KErnig = Knee Extension painful
brudziNsKi = Neck flexion leads to Knee flexion

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9
Q

diagnostics of SAH

A

based on clinical presentation
CT
-if CT -, lumbar puncture

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10
Q

what will CSF look like after SAH

A

bloody

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11
Q

what does cloudy CSF indicate

A

infection like bacterial meningitis
NOT SAH

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12
Q

what is used to identify exact location of SAH in preparation for surgery

A

cerebral angiography

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13
Q

SAH clinical management before surgery

A

prevent aneurysm from rupture!
-minimal stimulation
-pharm management (antiHTN, stool softener, antipyretic)

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14
Q

surgical management of SAH

A

SURGICAL CLIPPING
coiling

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15
Q

describe surgical clipping and coiling to treat SAH

A

clipping = clamp off aneurysm, then rupture
coiling = detachable coild fill aneurysm to prevent rupture

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16
Q

complication post SAH

17
Q

s/sx of vasospasm post SAH

A

change LOC
blurred vision
HA
langauge impaired
hemiparesis
seizures

18
Q

what do vasospasms cause

A

decreased cerebral blood flow -> dec. oxygen to brain tissue
and
accumulation of lactic acid

19
Q

what is most important to ccomplish with vasospasm management?

A

keep cerebral artery full and open

20
Q

what is the calcium channel blocker shown to improve outcomes with vasospasm?

A

nimodipine

21
Q

when is nimodipine contraindicated for vasospasm

A

hypotension

22
Q

describe “triple H” therapy for vasospasms

A

hypervolemic expansion
hemodilution
hypertension (induced)

23
Q

how is hypervolemia accomplished for triple h therapy

A

IV crystalloids and colloids

24
Q

what should be avoided when accomplishing hypervolemia with vasospasm and why

A

hypotonic solutions
shift fluid to brain -> cerebral edema

25
what should be monitored with hemodilution of triple h therapy
hemoglobin
26
what is the goal for induced HTN of triple h therapy
BP > 20 mm Hg over baseline (150-160 is ideal, has to stay under 200)
27
what are the 3 main complications resulting from SAH?
hydrocephalus seizures rebleeding
28
what causes hydrocephalus in SAH
blood clots in subarachnoid space obstruct arachnoid villi to reabsorb CSF
29
what may hydrocephalus after SAH require?
ventriculoperitoneal shunt (VP shunt)
30
what complication of SAH happens if the aneurysm is not repaired?
rebleeding
31
what is a device to monitor/control ICP
ventriculostomy
32
which order will prevent vasospasm? 1) nimodipine 60mg q4h 2) 0.45% NaCl @ 100 3) nitroprusside to keep systolic < 120 mmhg 4) lisinopril 10mg daily
a) nimodipine