Section 1: The Heart part 1 Flashcards

describe the location of the heart in the chest, the layers of the pericardium and the heart name and label the chambers of the heart and the great vessels entering and leaving the heart

1
Q

where is the heart located?

A

in between the 2nd and 5th intercostal space which is the same as in between the 3rd costal cartilage and 6th costal cartilage, near the midclavicular line

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2
Q

how many layers does the heart have

A

3
pericardium
myocardium
endocardium

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3
Q

describe the fibrous pericardium

A

outer sac
made of fibrous tissue
fibrous, inelastic, protects and prevents over-distension of the heart

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4
Q

describe the serous pericardium

A

inner layer
continuous double layer of serous membrane
single layer of endothelial cells, folded in on itself
forming a double membrane around the heart with an enclosed space between the layers

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5
Q

describe the layers of the serous pericardium

A

parietal pericardium, outer layer, lines the fibrous pericardium
visceral pericardium (epicardium), inner layer, firmly attached to the myocardium

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6
Q

describe the myocardium

A

specialised cardiac muscle, striated but not under voluntary control
each fibre/cell has a nucleus and one or more branches and is rich in mitochondria
sheet arrangement enables contraction of heart to be coordinated

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7
Q

describe the endocardium

A

thin membrane lines chambers and valves
single layer of flattened epithelial cells
is continuous
endothelium lines the blood vessels
smooth to minimise friction

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8
Q

name the right side of the heart

A

superior vena cava, branches of right pulmonary artery, right pulmonary veins, pulmonary valve, right atrium, right atrioventricular valve (tricuspid valve), right ventricle, inferior vena cava, thoracic aorta

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9
Q

name the left side of the heart

A

arch of aorta, branches of left pulmonary artery, branches of left pulmonary veins, left atrium, aortic valve, left atrioventricular valve (bicuspid/mitral), left ventricle, chordae tendineae, papillary muscle, interventricular septum

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10
Q

describe the chordae tendineae

A

prevents backflow by preventing the valves opening upwards

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11
Q

describe the papillary muscles

A

anchor the chordae tendineae to the ventricular wall

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12
Q

Describe the role of the sinoatrial node

A

Depolarises, initiating a new heartbeat
Impulse spreads through atrial muscle triggering atrial contraction then reaching the AVN

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13
Q

Describe the role of the AVN

A

Transmits the electrical signals from the atria into the ventricles
There is a delay to allow atria to finish contracting before ventricles start

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14
Q

Describe the role of the av bundle or bundle of his

A

Within the ventricular myocardium the branches break up into Purkinje fibres.
With those it transmits electrical impulses from the AV node throughout the ventricular myocardium triggering ventricular contractions

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15
Q

How is heart rate/force decreased

A

Vagus nerve (parasympathetic) supplies the SA and AV nodes and atrial muscle. Stimulation of vagus nerve reduces the rate of SA nod firing, decreasing the rate and force of heartbeat

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16
Q

Describe how heart rate/force is increased

A

Sympathetic nerves (accelerator) supply the SA and AV nodes and the myocardium, stimulation increases the rate and force of the heartbeat

17
Q

Where is the rate and force of contraction controlled

A

Cardiovascular centre in medulla oblongata

18
Q

Describe atrial systole

A

SA node triggers wave of contraction that spreads over atria myocardium, emptying atria and filling ventricles. Delay of AV transmission as impulse reaches AV node so atria can finish emptying before ventricles contract

19
Q

Describe ventricular systole

A

Av node triggers own electrical impulse spreads to ventricular muscle via av bundle, bundle branches and purkinje fibres,ware of contraction moves to apex and ventricular walls pumping blood into the pulmonary artery and aorta

20
Q

How does pressure change in atrial systole

A

Pressure rises in atria, theatriovertricular valves open, blood flows into ventricles and gravity helps if thorax is upright

21
Q

How does pressure change in ventricular systole

A

High pressure generated during ventricular contraction forces the atrioventricularvalves to close, preventing backflow of blood into the atria as it exceeds atrial pressure

22
Q

Describe complete cardiac diastole

A

Rest period between beats the myocardium in atria and ventricles is completely relaxed.

23
Q

how is blood pressure controlled (short-term)

A

baroreceptor reflex and chemoreceptors

24
Q

describe the baroreceptor reflex in high blood pressure

A

aortic and carotid baroreceptors stimulated, so increased input to CVC. decreased cardiac output occurs by parasympathetic activity to heart and sympathetic activity inhibited to the heart. sympathetic activity inhibited to blood vessels causes vasodilation of blood vessels. blood pressure falls

25
Q

describe the baroreceptor reflex with low blood pressure

A

aortic and carotid baroreceptors inhibited so decreased input to CVC. increased cardiac output occurs by parasympathetic activity to heart and sympathetic activity increased to heart. sympathetic activity increased to blood vessels causing vasoconstriction of blood vessels

26
Q

describe how chemoreceptors control blood pressure when pO2 fall and pCO2 and pH rise

A

carotid and aortic chemoreceptors detect change so send signals to CVC to increase sympathetic activity to heart and blood vessels so stroke volume and heart rate increases and vasoconstriction of blood vessels so blood pressure rises

27
Q

describe how chemoreceptors control blood pressure when pH and pCO2 falls and pO2 rises

A

carotid and aortic chemoreceptors detect change and send signal to CVC to decreases sympathetic activity to heart and blood vessels causing stroke volume and heart rate to decrease and vasodilation of blood vessels so blood pressure decreases

28
Q

describe he renin-angiotensin mechanism (RAAS) and how it controls blood pressure

A

kidneys respond to a reduction in BP and kidney blood flow by reducing excretion
excrete renin and renin activates angiotensinogen into angiotensin 1 which is then converted to angiotensin 2 by ACE
it is a powerful vasoconstrictor and triggers the release of aldesterone cause sodium retention and increased excretion of K so water reabsorption is increased therefore the body’s fluid load and circulating blood volume so BP is increased

29
Q

describe how atrial natriuretic peptide (ANP) controls blood pressure

A

a hormone released by the heart causing sodium and water loss from the kidney and reduces BP