Ruminant Dermatology, Pt. 2 Flashcards

1
Q

Where are screwworm infestations most common?

A

SOUTH - Texas, Florida, Mississippi

  • seen in horses, dogs, and deer
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2
Q

What is miasis?

A

fly strike - infestation with maggots that eat dead flesh (flies) or live flesh (screwworms)

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3
Q

How do animals with miasis present? How is it treated?

A

sick, depressed, maggots and odor observed

  • clean and debride wound
  • antibiotics
  • systemic dewormer + fly spray
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4
Q

What conditions predispose to miasis?

A
  • docked tails - decreased wound around tail + wound
  • moist and unhygienic environments
  • wooly breed sheep (Marinos) - can hide flies under wool and maintain a moist environment
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5
Q

What extreme measure has been taken decrease sheep death to miasis?

A

mulesing - removal of strips of wool-bearing skin from around the rear

  • wool around here typically retains feces and urine, which attract flies
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6
Q

What ectoparasite is seen in this sheep’s coat?

A

sheep ked - wingless fly susceptible to Ivermectins

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7
Q

What is Stephanofilariasis?

A

infection caused by parasite carried by the horn fly presenting with ventral midline and udder dermatosis in adult cows

  • common, not very serious
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8
Q

What are the 2 species of warbles (Hypodermiasis) that affect cows? Where are each found?

A
  1. H. bovis - northern US, migrates to spinal cord —> killing with OP can cause downers or bloat DO NOT TREAT IN WINTER
  2. H. lineatum - northern and southern US, migrates to esophagus

bee-like, hairy adults live for 1 week

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9
Q

What is the lifecycle of warbles like?

A
  • eggs laid on extremities in spring and summer
  • eggs hatch and larvae penetrate the skin and migrate to the spinal cord or esophagus to over-winter for 2-4 months —-> DO NOT TREAT
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10
Q

What is the cut-off to treating warble infections?

A

DO NOT TREAT November 15th to March 15th (Ivermectin)

  • killing larvae in the spinal cord and esophagus can cause paralysis or bloat
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11
Q

Cattle warbles:

A

hypodermiasis - “blow holes” where larvae penetrate the skin and eventually return Jan-Feb and after March

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12
Q

How do cattle present with copper deficiencies?

A

lighter colored coats

  • treat with copper supplementation
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13
Q

When are fleas most commonly a problem? Where do infestations most commonly originate? What signs are associated with infestation?

A

summer

barn cats —> fleas are not species-specific (Ctenocephalides felis)

rubbing, scratching, chewing, anemia, emaciated calves

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14
Q

What must treatment of fleas focus on?

A

treatment of affected and unaffected animals AND the environment

  • insecticides and insect-growth regulators
  • removal of staw bedding
  • treat any barn cats
  • remove feral cats from premises
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15
Q

What is the most common cause of ringworm in cattle? How is it transmitted?

A

Trichophyton verrucosum

ZOONOTIC - direct contact with infected animal or fomite (incubates for 1-6 weeks)

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16
Q

How do ringworm lesions progress? Where are lesions most commonly found?

A
  • thickened and scaled skin = circular and crusty
  • hair loss as hairs are invaded and break off
  • expand for 4-8 weeks and resolve with immune response

head, neck - alopecia, scaling

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17
Q

Ringworm:

A
  • halter is a common fomite!
  • begins with crusting
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18
Q

What groups of cattle are commonly infected with Ringworm?

A

show cattle —> in contact with multiple other cows and bulls regularly

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19
Q

What are 5 predisposing factors to ringworm?

A
  1. age - young animals more susceptible
  2. immunity - prior exposure improves host resistance
  3. environment - more common in the winter, crowding
  4. remains in the environment for years
  5. nutrition - poorly conditioned animals are more susceptible
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20
Q

How is ringworm diagnosed? Treated?

A

general appearance +/- scraping of margins

  • lime sulfur
  • Captan
  • Iodine
  • topical and systemic Thiabendazole
  • systemic Griseofulvin
  • Ketoconazole
21
Q

How can ringworm be controlled?

A
  • vaccine available in Europe - Ringvac Bovis LTF-130
  • keep possible fomites clean between use in different cows/bulls
22
Q

What fungus commonly infects show lambs? Why is it more common in show lambs?

A

Trichophyton —> contagious to humans!

frequent washing removes lanolin in the wool that usually protects lamb

23
Q

How are show lamb fungal infections controlled?

A
  • anti-fungals
  • prevent contact with infected (quarantine!)
  • clean possible fomites, don’t share equipment
  • anti-fungal sprays on environment, equipment, shelter, and fences (can remain for years!)
24
Q

What are common treatments used for ringworm in sheep? How long?

A
  • chlorhexidine
  • 1:10 dilution of hypochlorite (Chlorox)
  • 1:300 dilution of Captan (not approved for use in farm animals, irritating to skin)
  • iodine + baby oil
  • povidone iodine

daily for 5 days, then weekly for 3 treatments

25
Q

What species of ringworm most commonly affects swine? What age is most susceptible? What predisposes?

A

Microsporum nanum —> ubiquitous in the environment + ZOONOTIC

adults

poor sanitation and high humidity

26
Q

How is ringworm treated in swine?

A

most commonly spontaneously heals

  • topical fungicides
  • disinfect premises with bleach
27
Q

What cattle are most commonly affected by warts? How do lesions appear?

A

young, unexposed cattle (not for teat warts!)

papillomatosis - proliferative, pedunculated, most common over neck and head —> commonly regress spontaneously in 6 months, longer if immunocompromised

28
Q

What is the most common cause of ear warts in cattle? How are they treated?

A

contaminated ear tag equipment

vaccination +/- cryotherapy and debulking

29
Q

Which of the following is false in regard to bovine skin warts?

a. bovine skin warts may be spread by ear tattooers
b. bovine skin warts are mostly a problem in young cattle
c. wart vaccines have been proven to be quite effective in preventing warts

A

C

large amounts of strains

30
Q

How are penile warts managed?

A
  • debulk
  • cryotherapy

be careful of the urethra, may spread of bulls mount each other

31
Q

What are the 3 types of photosensitization?

A
  1. PRIMARY = photodynamic agent ingested (St. John’s wort)
  2. SECONDARY = liver damage (Rape, Lantana)
  3. CONGENITAL = aberrant pigment synthesis (congenital porphyria)
32
Q

What clinical sign is associated with photosensitization? How is it treated?

A

sloughing of light-colored hair areas

remove cause (photodynamic/hepatotoxic plants) and sunlight

33
Q

Photosensitization:

A

light colored areas affected

34
Q

What causes Dermatophilosis? What are some predisposing factors?

A

Dermatophilus congolensis (G+) - rain rot, carriers likely

rain and skin damage

35
Q

What are the most common signs of Dermatophilosis? What areas are specifically affected in llamas, cattle, sheep, and goats?

A

roughened coats with paint-brush lesions when plucked - painful and NOT pruritic

  • LLAMAS = not pruritic
  • CATTLE = back, sides, neck
  • SHEEP = coronary band
  • GOATS = tail and ears of kids, muzzle, dorsum, scrotum, and legs of adults
36
Q

How is Dermatophilosis diagnosed?

A
  • typical presentation with no pruritis
  • smear of scabs or skin biopsy show train tracks of microorganisms
37
Q

What are 3 common options for treating Dermatophilosis?

A
  1. TOPICALS - iodophors, lime sulfur (body washes, sprays, dips)
  2. ANTIBIOTICS - PPG, OTC, Ceftiofus
  3. typically self-limiting, can do nothing
38
Q

How is Dermatophilosis controlled?

A
  • keep cattle dry
  • dispose of crusts and lesions
  • good nutrition
  • control anything that can weaken the skin

not very practical

39
Q

In what animals is SCC common?

A

white pigmented/haired animals —> UV damage

  • mucocutaneous junctions common
40
Q

How does caseous lymphadenitis present?

A
  • unilateral LN swelling filled with caseous (very contagious) material
  • internal LN swelling causes systemic signs
41
Q

What causes contagious echthyma? What ruminants are most commonly affected? What can cause outbreaks?

A

sore mouth, orf = Parapox virus —> ZOONOTIC

sheep, goats, llamas

persists for years in scabs/soil —> lambing/kidding

42
Q

What clinical signs are associated with orf?

A
  • lesions most common on lips, muzzle, oral cavity, udder, and coronary bands (MUCOCUTANEOUS JUNCTIONS)
  • painful —> dams won’t let offspring nurse —> starvation in offspring
  • affected offspring can infect udders
43
Q

How is orf diagnosed?

A
  • presumptive diagnosis based on signs
  • skin biopsy and histopath
44
Q

How is orf treated?

A
  • usually self-limiting within 3 weeks
  • TLC for affected young
45
Q

How can orf be controlled?

A

vaccine available - fairly effective, but only recommended in farms with infections (live vaccine infects negative individuals)

46
Q

Which of the following are burrowing mites?

a. Chorioptes bovis
b. Psoroptes natalensis
c. Sarcoptes scabiei

A

C

47
Q

Which of the following is false regarding hypoderma?

a. adults are hairy, bee-like flies that only live for a short time
b. eggs are laid on the lower leg or flank of cattle
c. eggs hatch, larva penetrate the skin, and migrate to either the spinal cord or abdominal aorta depending on species

A

C

48
Q

What is the depicted insect that can be found in the wool of sheep?

a. flytick
b. ked
c. wool spider

A

B

49
Q

Ringworm will spontaneously regress with time, but some owners want animals treated. Regardless of the topical compound used for treatment, where should the product be put?

a. central aspect of lesion
b. periphery of lesion
c. outside the lesion in the haired skin to stop spread

A

B