Repro Flashcards

1
Q

When do we start doing CTGs ?

A

27 weeks - before then it is common to look abnormal

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2
Q

How do we interpret ctgs?

A

DR C BRAVADO

DR - determine risk eg small baby
c - contractions - present? Spontaneous/augmented (oxytocin)/regular/how many in 10 mins - labour (3/4 in 10 mins)
BR - Baseline fetal heart rate - 120 normal
A - accelerations
V - variability
D - decelerations
O - overall impression

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3
Q

Reassuring values - Hr and variability on ctg

A

HR - 110-160
Variability- 5-25bpm

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4
Q

Acceleration and deceleration amount on a ctg

A

15bpm change - last for at least 15 seconds

5bpm = 1 little box
15 sec = half a box

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5
Q

What is shouldering on a ctg and is it reassuring?

A

When there is a quick increase in heart rate before and after a deceleration

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6
Q

What is one of the most important part of ctg to look at?

A

Variability

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7
Q

Sinusoidal ecg means what

A

NEEDS delivery ASAP

sign if bleeding

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8
Q

What does terbutaline do?

A

Slows down contractions (tar sp= slow)

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9
Q

What is the greene climeric scale and when do we use it?

A

Menopause symptoms and severity

Used to monitor and decide what hrt therapy

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10
Q

Still having periods, but menopause symptoms do we begin treatment?

A

Yes

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11
Q

Following gnhr analogues will periods resume?

A

Yes

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12
Q

Vaginal estrogen do we still give progesterone?

A

No as such a low dose

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13
Q

Hrt risks

A

VTE
cardiovascular
Breast cancer

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14
Q

Why do we get urinary in continuance in menopause

A

Due to atrophy (same embryological tissues as vag so same atrophy)

Can give topical oestrogen

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15
Q

anaemia
pain mnestrual cycle

A
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16
Q

3 criteria for hypernemesis gravidarum

A

5% weight loss
Dehydration (ketones)
Electrolyte imbalance (hypokalaemia)

17
Q

What do we need to exclude before we can diagnose hypernemesis gravidarum ?

A

Gi - peptic ulcers, cholecystitis, gastroenteritis, hepatitis, pancreatitis, uti/pyelonephritis, metabolic conditions, neurological condition/drug induced

18
Q

Green vs yellow vommitting

A

Green is obstruction (lower down so has bile)
Yellow is some problem higher up

19
Q

Electrolyte abnormalities in hypernemesis gravidarum

A

Hyponatraemia
Hypokalaemia
Thiamine low

20
Q

Low thiamine can lead to what and what wou,d. e the symptoms

A

Wernikes emcephalopathy
Opthalmiokagia
Ataxia
Confusion

21
Q

What scoring system can we use for hypernemesis?

A

PUQE score to determine severity

22
Q

Hypernemesis gravidarum treatment

A

No complications:
PUQE 3-12: antiemetics, lifestyle and dietary changes
PUQE 13: thiamine, antiemetics, normal saline and potassium. Ambulatory day care until no ketouria. No ketones in uria = discharge.

Complications = inpatient. No antiemetics = steroids. MDT. VTE prophylaxis

23
Q

Why don’t we give dextrose in hypernemesis gravidarum?

A

It can ,sad to wernikes encephalopathy

24
Q

Can iron exacerbate nausea and vommitting?

A

Yes

25
Q

First line antiemetics + hypermisis

A
  1. Cyclizine 50mg PO/IM/IV 8hrly
  2. Prochlorperazine 5-10mg 6-8 hourly PO. Am also be given PR
  3. Promethazine 12.5-25mg 4-8hrly
  4. chlorpromazine 10-25g 5+6 hourly po. Also can be given PR
26
Q

When do we want to avoid giving ondansatron?

A

First trimester - can lead to congenital anomalies